otology Flashcards

1
Q

Middle ear is connected to the nasopharynx by what?

A

eustachian tube - provides ventilation and mucociliary clearance to the middle ear

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2
Q

where is the pars tensa, malleus, and pars flaccid

A

When looking at the ear drum, pars tensa is inferior to the umbo and malleus, malleus forms an oblique line, and pars flaccid is superior to the malleus

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3
Q

What does an audiogram measure

A

It measures hearing level (in dB) as a function of frequency (Hz). These pure tone thresholds measure the sensory (cochlear) aspect of hearing function

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4
Q

What is the minimum dB humans can detect

A

Humans can detect a one dB difference. On the audiogram, 0 dB is the normal hearing threshold, not zero sound intensity

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5
Q

What is a word recognition score

A

Represents the ability to discrimination words presentenced at normal conversational loudness levels (0-100%). This is a critical part of the hearing assessment and represents the “neural” aspect of hearing ability (inner hair cell, 8th nerve and central auditory pathways)

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6
Q

Four types of hearing loss and what structures are affected

A

conductive (external ear, tympanic membrane or middle ear), sensory (cochlear), neural (8th nerve, inner hair cell, central pathways), or sensorineural (combo of sensory and neural structures)

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7
Q

causes of conductive hearing loss

A

otitis media, TM perforation, Cholesteatoma, Otosclerosis (bones become fixed), Congenital aural atresia

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8
Q

signs and symptoms of acute otitis media

A

Rapid onset of signs (inflammation of TM, drainage, perforation) and symptoms (pain, fullness/pressure, hearing loss) of an ear infection in the presence of a middle ear effusion.

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9
Q

treatment of acute otitis media

A

1) Consider Watchful Waiting when uncertain in low-risk patients 2) Systemic Abx 3) Tympanocentesis for culture or acute pain control 4) Myringotomy and Mastoid Surgery in complicated cases

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10
Q

otitis media with effusion- signs/symptoms and treatment

A

Middle ear fluid in the absence of inflammation/infection. Pain/fullness/popping /hearing loss (can delayed speech and language development), recurrent episodes of AOM. Antihistamines, decongestants, antibiotics, steroids not effective. Treat with time (watchful waiting) or PE tubes

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11
Q

causes of sensory hearing loss

A

hair cell loss due to presbycusis, noise trauma, ototoxicity, genetic factors. Outer hair cells more susceptible than inner hair cells.

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12
Q

what is presbycusis

A

Gradual, progressive bilateral hearing loss caused by degenerative physiologic changes associated with aging. Decreased hearing threshold sensitivity. Decreased ability to understand supra-threshold speech. Central auditory process impairment. Associated with greater risk of alzheimers and other cognitive declines

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13
Q

What are the OSA regulations for chronic noise exposure

A

80 dB for 8 hrs or 90 dB for 4 hours

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14
Q

What is a noise notch

A

A form of sensory hearing loss where the audiogram shows normal hearing at low frequencies, then an abrupt decrease in hearing level in dB at a high frequency (ie. 4000Hz) but then hearin level in dB goes back up to near normal at higher frequencies (ie. 8000Hz). This occurs around 2000-4000Hz becuase the resonant frequency in the middle ear is around this frequency

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15
Q

What is ototoxicity

A

Ototoxicity is the tendency of certain therapeutic agents and other chemicals substances to cause functional impairment and cellular degeneration of the tissues of the inner ear, especially the inner ear hair cells and stria vascularis

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16
Q

common ototoxic agents

A

aminoglycosodes (gentamicin), cancer agents (cis-platinum), macrolides (erthromycin), ASA, quinine, loop diuretics, hydrocodone, viagra

17
Q

treatment of ototoxicity

A

stop agent, use corticosteroids, antioxidants (vitamin E), avoid noise exposure

18
Q

Two types of genetic causes of hearing loss

A

non-syndromic (70%) and syndromic (30%)

19
Q

most common causes of non syndromic hearing loss

A

Connexin 26 (autosomal recessive)

20
Q

most common causes of syndromic hearing loss

A

include Waardenburg’s (2%), Usher’s (4-5%), Pendred’s (5%), Brachio-oto-renal (7%), Alport’s (1%), and Jervell, Lange Nielson (0.25%).

21
Q

What is endolymphatic hydrops

A

A pathologic condition characterized by expansion of the endolymphatic compartment of the inner ear. Is associated with recurrent episodes of vertigo, sensorineural hearing loss, tinnitus and aural fullness that is refered to as Meneire’s disease

22
Q

stria vascularis function

A

maintains proper endolymph fluid homeostasis and the endonuclear potential by pumping K ions into the scala media

23
Q

How does loss of endonuclear potential affect hearing

A

it causes sensory hearing loss

24
Q

Causes of disordered inner ear fluid homestasis and treatment

A

Impaired blood flow due to: Vascular dysfunction/ vasculitis, systemic metabolic disorders (DM, hypothyroid, renal failure, arteriosclerosis), immune mediated (lupus, sarcoidosis, wegeners). Treated with corticosteroids

25
Q

hallmarks of neural hearing loss

A

asymmetry of hearing between the two ear and reduced speech perception scores.

26
Q

Causes of neural hearing loss

A

8th nerve tumors, auditory neuropathy, multiple sclerosis

27
Q

management of vestibular schwannoma

A

observation, resection, stereotactic radiosurgery

28
Q

treatment of sensorineural hearing loss

A

Air Conduction Hearing Aids, Implantable Hearing Aids, Cochlear Implants, Assistive Listening Devices, Speech and Language Training