Other Aphasia Types Flashcards
Mixed Nonfluent Aphasia - AC?
Poor AC
Not good enough to be Broca’s
But better than Global
Mixed Non fluent Aphasia - RC?
Mild RC deficits
Mixed Nonfluent Aphasia expressive
Like Broca’s Agrammatic Mod repetition deficits Decreased speech initiation Awkward articulation Effortful articulation Motor planning, programming
Mixed Nonfluent Aphasia word finding deficits?
yes-substantives, pictures, action words
Subcortical aphasias
Acquired language disorders d/t brain damage in subcortical, white matter structures and/or subcortical grey matter
What aphasia is INDIRECT consequence of disruption in this transfer/editing/processing system
subcortical aphasias
SOL for subcortical (thalamic) aphasias
Thalamic nuclei
BG
Internal Capsule
White matter/periventricular areas…?
What is the thalamus involved in?
- is a sensory-relay station -receives input from peripheral sensory receptors
- sends sensory stimuli to cortical structures for perception and process
- involved in motor processes,
- relays motor input from cerebellum to motor cortex
What happens if a lesion occurs in the thalamus
can’t relay sensory feedback from periphery, or transport motor info to and from the cerebellum.
Thalamic (subcortical) aphasia receptive skills
Typically Good AC
MAY be mod impaired
Thalamic (subcortical) aphasia expressive
-Anomia is hallmark Cant access words Confrontation naming deficits Rare paraphasias -Excellent repetition -Good syntax & artic -Just can’t access lexicon
Subcortical aphasias associated w/ lesions in BG
Relatively few studies characterizing subcortical aphasias due to lesions in the BG
“worse” if cortical lesion AND a subcortical BG lesion?
Subcortical Aphasias associated w/lesions in BG
Has some type of role in automatized speech and language?
Motor? Slow, nonfluent, effortful
Yet also cases of impaired comprehension
“Mixed” ?
With some features of confused/mis-used grammar
“Fluent”?
BG what do they do?
Modulates, regulates, refines voluntary motor movements
Internal capsule (subcortical) aphasia is
2-way hi-way connecting cortex to brainstem (BS) spinal cord (SC)
Internal Capsule (Subcortical) Aphasia- aphasic characterisitcs
Anterior IC lesions = non-fluent
Posterior IC lesions = fluent
Bi/Multilingual Aphasia
You can be aphasic in one or both languages
Are some cases of being aphasic in L1 today, and aphasic in L2 tomorrow
There is a diversity of patient-presentation:
Levels of severity & patterns of recovery between each language
Bi/multi-lingual aphasia involves different SOLs:
EFs (PFC, FL, & extensions…)
Attention, inhibition, working memory
Self-monitoring components of EFs
Might be “aphasic” due to atypical SOLs (PFC, FL), rather than “expected” SOLs of language circuits
