Ostertagia Flashcards

1
Q

Give the genus and species of the bovine equivalent of Teladorsagia

A

Ostertagia ostertagi

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2
Q

Ostertagia ostertagi are also known as?

A

The brown stomach worm

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3
Q

Where are Ostertagia ostertagi found in the body?

A

Abomasum

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4
Q

Which condition is caused by Ostertagia ostertagi?

A

Parasitic gastritis

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5
Q

Give some morphological features of Ostertagia ostertagi

A
  • 1cm in length
  • Slender
  • Pinky brown colour
  • Fine cervical papillae
  • Males have a bursa and spicules
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6
Q

How can Ostertagia ostertagi be differentiated from other GI worms?

A

o Site in the abomasum
o Size – medium, not as big as Haemonchus and not as small as Trichostrongylus
o Fine cervical papillae on the anterior end of the worm

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7
Q

Describe the life cycle of Ostertagia ostertagi up to L3

A
  • Direct, no immediate host
  • Spread directly from one cow to another via free living stages on pasture
  • L1-L3 is in faecal pat
  • L3 is the infective stage
  • L3 is ensheathed
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8
Q

How is L3 protected?

A

The L3 retains the cuticle of the L2 larvae so they are surrounded by a protective cuticle

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9
Q

Describe the eggs of Ostertagia ostertagi

A

90 x 45μm, barrel shaped, undifferentiated

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10
Q

How do the L3 of Ostertagia ostertagi develop and become infective?

A
  • Cattle eat L3 as they graze
  • L3 are swallowed and reach abomasum
  • Burrow into the gastric glands and develop into L4 and L5
  • L5 emerge into the lumen of the abomasum
  • L5 mature to adults and lay eggs
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11
Q

What is the pre patent period?

A

The time taken from when the cow first ingests a larvae through detection of eggs in the faeces of that animal

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12
Q

How long is the pre-patent period for Ostertagi ostertagi?

A

3 weeks

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13
Q

In a PM, how would an infection of Ostertagia be identified?

A

White/cream nodules created by the developing larvae within the gastric glands

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14
Q

Describe the pathogenesis of Ostertagia damaging the gastric glands

A
  • L4 and L5 develop in the gastric glands causing damage as the grow and feed
  • The damaged cells are replaced by undifferentiated epithelial cells
  • Loss of acid production causes an increase in abomasal pH
  • Loss of bacteriostatic effect
  • No conversion of pepsinogen to pepsin
  • Increased permeability of the mucosa
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15
Q

What are the functions of the gastric glands?

A
  • Maintains acid pH in abomasum
  • Bacteriostatic
  • Converts pepsinogen to pepsin
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16
Q

Which cells produce acid in the gastric glands?

A

Parietal cells

17
Q

What are the clinical signs of a Ostertagia infection?

A
  • profuse watery diarrhoea
  • weight loss
  • loss of appetite
18
Q

Describe type 1 bovine ostertagiosis

A
  • occurs in dairy replacement calves
  • disease occurs in the summer (august)
  • calves ingest large numbers of L3 in July
  • green watery diarrhoea
  • majority of calves in a group are affected
19
Q

Describe type 2 bovine ostertagiosis

A
  • yearling calves
  • less common
  • disease in late winter/early spring (january-march)
  • acute disease
20
Q

What are the clinical signs of type 2 bovine ostertagiosis

A

Intermittent diarrhoea, anaemia, thirst, weight loss

21
Q

Do higher levels of mortality occur in type 1 or 2 bovine ostertagiosis?

A

Type 2

22
Q

Define hypobiosis

A

The arrested development of L4 larvae within the host, in response to a trigger received by the free living L3

23
Q

What is the trigger for hypobiosis?

A

Drop of ambient temperature in autumn

24
Q

Do L3 hypobiose before or after they have been ingested?

A

After

25
Q

Name 3 GI nematodes found in the abomasum of cattle

A
  • Haemonchus contortus
  • Ostertagia ostertagi
  • Trichostrongylus axei
26
Q

Name 3 GI nematodes found in the small intestine of cattle

A
  • Nematodirus spp
  • Trichostrongylus spp
  • Cooperia spp
27
Q

Which nematode has a cotton wool like appearance with a cephalic vesicle?

A

Nematodirus spp

28
Q

Name 3 GI nematodes found in the large intestine of cattle

A
  • Chabertia spp
  • Oesophagostomum spp
  • Trichuris spp
29
Q

What biological process leads to Type II ostertagiosis?

A

Hypobiosis