osteoarthritis LP 2 Flashcards

1
Q

what is a chornic degenerative, progressive deterioration of a joint characterized by a loss of articular cartilage?

A

osteoarthritis

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2
Q

what two structures are damaged with OA?

A

subchondral bone and articular cartilage

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3
Q

what two things form from osteoarthritis?

A

acute low grade inflammation and osteophytes

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4
Q

what happens to the joint when the space between decreases?

A

rubbing of joints which leads to lay down or ossification and forms osteophytes

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5
Q

what is the first sign of OA?

A

presences of a capsular pattern forming

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6
Q

what posture can cause OA?

A

lordotic curve of the lumbar spine (anterior pelvic tilt)

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7
Q

would ligaments have a closed packed position with OA?

A

yes

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8
Q

what joints are most commonly effected by OA?

A

weight bearing
hips, knees, Si joint, L5 and S1
small joint in the hands and feet
lumbar and cervical joints

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9
Q

what is primary OA?

A

idiopathic due to number of factors

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10
Q

what factors cause primary OA?

A

aging
wear and tear
genetic factors

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11
Q

what is secondary OA?

A

identifiable cause such as trauma or pathology leading to the degeneration

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12
Q

what things can cause secondary OA?

A

mechanical incongruity
inflammation
bone disease
bleeding disorders
neurological disorder
excessive intra-articular steroid injections
endocrine disease

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13
Q

place the follow in the correct order of progression of OA:
articular cartilage
capsule
synovial membrane
muscles
subchondral bone
ligaments

A

articular cartilage
subchondral bone
synovial membrane
capsule
ligaments
muscles

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14
Q

what things will happen when articular cartilage becomes impacted from OA?

A

fibrillation (breaking down layers begging)
fissuring: (breaking down of deep layers)
erosion: (thinning of cartilage)
lipping: (extra cartilage laid down and forms rim or lip around edges)

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15
Q

what things will happen when subchondral bone becomes impacted from OA?

A

sclerosis: (bone becomes spongy and replaced by cortical bone)
cyst formation: (bone can’t hold weight and breaks down)
eburnation: (bone rubs together)
osteophytes formation: (osteophytes or bone spurs form)

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16
Q

when would you see low grade inflammation in OA?

A

when capsule becomes effected

17
Q

what are some early signs of OA?

A

few visible changes
swelling is low grade
decrease ROM
normal springy end feel
stiffness in the morning last about 1/2 hour and relieved by moving
muscle guarding

18
Q

what are some late signs of OA?

A

visible palpable changes in joint size and shape
swelling is still low grade
end feel is bony and premature
morning stiffens is longer then 1/2 hour
progressive loss of ROM (especially in PR)

19
Q

how would a flare up present?

A

inflammation signs (PRISH)
low grade edema

20
Q

what are the normal / typical signs of OA in a person?

A

normally in age 40+
onset pain and with weight bearing mostly
pain at night
muscle spasm
stiffness
loss of Range
muscle wasting and weakness
joint deformation

21
Q

what order would OA appear in men?

A

hips
knee
spine
ankle
shoulder
fingers

22
Q

what order would OA appear in women?

A

knee
fingers
spine
hip
ankle
shoulder

23
Q

what is degenerative disc disease?

A

degeneration of the annulus fibrosis of a disc. results on pain, tearing, scarring and clefts

24
Q

what areas of the spine are most likely to see DDD?

A

lumbar and cervical

25
Q

is ddd more common in men or women?

A

men

26
Q

what is a disc herniation?

A

bulging of the nucleus pulpous against the outer layers of the annulus fibrosis into the intervertebral space

27
Q

what is the tearing of both the inner and outer layers of the annual fibrosis causing the nucleus pulpous to bulge directly into the outside space?

A

disc prolapse

28
Q

what can cause disc trauma?

A

postural or occupational leading to excessive rotation
fixation of the spine
direct trauma

29
Q

what are some signs and symptoms of disc damage?

A

joint dysfunction
muscle spasms
osteophytes
radiating pain
nerve root damage or compression

30
Q

what’s the most common position of pain?

A

forward flexion

31
Q

what kind of questions could you ask someone with DDD or OA?

A

did you have any injections in any joints (commonly leads to OA)
what occupations do you have (desk work can cause postural changes over time leading to disc pain)
what position causes pain (flexion is a sign of DDD)

32
Q

what position causing facet joint pain?

A

extension and ipsilateral rotation

33
Q

what would you encourage clients to do in the early stages of OA?

A

move through pain

34
Q

what activities are beneficial for OA or any disc damage?

A

swimming
walking

35
Q

what’s a big part of treating OA or DDD?

A

correcting predisposing factors (posture and biomechanics)

36
Q

What are the CIS for OA?

A

No mobilizing bony blocks or hyper flexible joints

No hydro during flare up

No frictions around affected joints