Orthopedic Tx 400 -- Dislocations and atrophy Flashcards

1
Q

not able to hinge at hip?

A

trip another approach to same movement

–> E.g. flex at hip instead of bending forward

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2
Q

active support

A

E.g. support to the joint structures coming from E.g. muscle tissue

I.e.
Support from contractile tissue

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3
Q

atrophy is

A

loss/wasting of mm tissue

“A decrease in the size and number of your muscle fibers causes sarcopenia. With muscle atrophy, there’s a reduction in the size of the fibers, but the amount of fibers stays the same. Everyone experiences some amount of muscle loss as they age.”

—> NOTES SAY OPPOSITE (??):
“Atrophy is due to the reduction in the number of muscle fibers within the muscle as a whole, or in connection with a lesion (myogenic atrophy) or with nerve damage (neurogenic atrophy).”

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4
Q

myogenic atrophy – aka

A

disuse atrophy

Loss of mass and strength.

Caused by a lack of physical exercise/<ADL’s, often due to pain, sedentary lifestyle, medical conditions, or <activity levels, prolonged immobility (bed rest, cast).

Can usually be reversed with exercise, unless severe (damage).

During aging, there is a gradual reduction in MM function and mass. This is known as sarcopenia, and may be distinct from atrophy in its pathophysiology.

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5
Q

neurogenic atrophy

A

with nerve damage

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6
Q

NEUROGENIC ATROPHY (AKA true atophy)

A

This is the most severe type of atrophy.

Caused by the loss of motor innervation to a MM, from an injury or disease.

It occurs more suddenly than myogenic atrophy.

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7
Q

common causes of muscle atrophy

A

Alcohol-associated myopathy

Amyotrophic lateral sclerosis (ALS or Lou Gehrig’s disease)

Burns

Dermatomyositis and polymyositis

Guillain-Barre syndrome

Injury

Long-term corticosteroid therapy

Malnutrition

Motor neuropathy (such as diabetic neuropathy)

Muscular dystrophy

Not moving (immobilization)

Osteoarthritis

Polio

Rheumatoid arthritis

Spinal cord injury

Stroke

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8
Q

causes to specifically note

A

Alcohol-associated myopathy

Guillain-Barre syndrome

Long-term corticosteroid therapy

Osteoarthritis
Rheumatoid arthritis

Stroke

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9
Q

DISLOCATION

A

A dislocation is the complete dissociation of the articulating surfaces of a joint.

A subluxation is when the articulating surfaces of a joint remain in partial contact with each other.

A joint reduction is the medical term for the tractioning of the dislocated joint surfaces.

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10
Q

dislocatons can occur at

A

may occur at any joint, certain joints are more susceptible due to anatomical configuration.

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11
Q

most frequently dislocated joint

A

The Glenohumeral joint is the most frequently dislocated. Only a portion of the articulating surface of the humeral head comes into contact with the shallow glenoid fossa. It relies on ligaments and MM support.

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12
Q

other commonly dislocated jts

ACJ is also classified as separation/sprain (more typically classified as sprain)

A

Other commonly dislocated joints include the acromialclavicular joints, metacarpals, and interphalangeals.

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13
Q

the events during dislocation

A

In a dislocation injury, a portion of the joint capsule and surrounding ligaments are either completely torn or partially ruptured.

The nearby tendons, synovial sheaths, and articular cartilage may also be damaged.

Other soft tissue injuries, such as stains and contusions occur.

Complications may occur, such as NN & BV damage, and FX.

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14
Q

during subluxaiton

A

In a subluxation, the joint capsule is stretched. Sprains may be present.

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15
Q

cause of dislocaiton

A

The cause of a dislocation is a trauma-related sudden twist or wrench of the joint beyond its normal ROM. Can be direct or indirect.

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16
Q

what happens when a join is disocated once?

A

The dislocated joint is more susceptible to future dislocations or subluxations, leading to joint instability.

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17
Q

what about likelihood of pain after one dislocation?

A

This unstable structure may become painfully hypermobile.

18
Q

some risk factors / contributing factors for dislocaiton

A

Pathologies
(E.g. RA, hemiplegia),

Congenital Lig. Laxity,

Previous dislocations.

19
Q

GH jt dislocation

A

Commonly anteriorly dislocated (subcoracoid).

20
Q

GH dislocation common MOI

A

excessive ABD. and ER. Humeral head forced through the inf. Joint capsule (foramen of Weitbrecht) where it lodges inferior the the coracoid process.

21
Q

what can happen to anterior glenoid labrum during anterior dislcaiton of GH jt

A

There may be damage to the anterior glenoid labrum (Bankart lesion).

22
Q

anterior glenoid labrum LESION

A

Bankart lesion

23
Q

which nerve is comonly damaged during anteiror GH dislocaiton

A

Axillary NN can be injured.

24
Q

GH jt tx

A

Tx

reduction, sling

treat softtissue damage that occurred simultaneously

25
Q

posterior GH dislocaiton

A

Posterior dislocation is less frequent.

MOI- flexion, adduction, and internal rotation.

26
Q

patella dislocaiton

A

Patella

Dislocates Laterally

MOI- ER of the Tibia with knee flexion.

27
Q

lunate dislocaiton

A

FOOSH injury.

Radius displaces the lunate palmarly.

Open reduction may be required.

Median NN lesions possible

lunate necrosis possible.

28
Q

elbow dislocaiton (HU)

A

Usually w/ a fracture. Caused by a FOOSH. Radius and Ulna forced posteriorly.

Brachial artery and Median NV can be involved.

coronoid process commonly fractured

29
Q

hip dislocaoitn

A

Uncommon. Femur forced posterior. Acetabular fracture and sciatic NV may occur.

MVA (seated – knee hits dashboard)

30
Q

SSx acute

A

Snapping or popping noise is heard at the time of injury.

P is intense.

Joint appears deformed prior to reduction.

Local edema and heat.

Joint effusion may occur with capsular damage.

Hematoma is RED, BLACK, and BLUE.

<ROM from protective MM spasm, edema and P.

Cannot continue activity.

Post reduction: joint may be taped, splinted, casted, or in a sling.

31
Q

acute dislocaiton – complicaitons

A

Complications: strains, contusions, blood vessel and NV injury, fracture.

32
Q

early subacute – SSx – dislocaitons

A

Joint unstable. Hematoma is BLACK and BLUE.

P, edema and inflammation are reduced.

Adhesions are developing.

MM spasms decrease.

TrP’s in MM crossing the joint and compensatory.

Joint is taped, splinted, in a sling.

ROM is reduced (Dr. will advise for allowable range).

33
Q

SSx – late subacute (dislocaiton)

A

Hematoma is YELLOW, GREEN, and BROWN.

P, edema, and inflammation diminished.

Adhesions are maturing.

Spasms are replaced with > MM tone.

Joint is supported or immobilized.

<ROM.

MM crossing the joint are stabilizing the joint.

34
Q

dislocaiton (late stage – chronic) – SSx

A

Local P with joint stress. Adhesions matured.

HT and TrP’s present.

ROM is restricted.

A pocket of chronic edema may remain local to a ligament.

Tissue may be cool to touch.

Joint may be unstable in the direction of injury unless surgically repaired.

The joint may be immobilized up to 9 weeks post surgery.

There is loss of proprioception at the joint.

MM weakness or disuse atrophy can occur.

Supportive taping may be needed for activities that stress the joint.

35
Q

CIs – dislocaiton

A

During acute and subacute, testing other than P-free ROM is CI’d to prevent further injury.

Avoid removing MM splinting in acute and early sub-acute.

Distal circulatory work not appropriate in acute and early sub-acute.

Be aware of placing the joint in the MOI position.
No joint play.

No prolonged or severe H20 in acute/early sub-acute stages.

No remedial exercised during the acute stage.

If restoring ROM in the direction of MOI, ensure strength has fully returned.

Do not restore full ROM in a joint that has been surgically repaired.

36
Q

treatment goals – Acute

A

Reduce inflammation: Elevate, Ice.

Reduce edema: Lymph drainage, nodal pumping, unidirectional effleurage, stationary circles – all proximal.

Do not remove protective MM spasm.

On site work is contraindicated.

Maintain ROM: mid range PROM is used on Prox joints. (IF SAME MM NOT CROSSING PROXIMAL JOINTS)

37
Q

tx – early subacute (dislcaiton)

A

Contrast H2O

Continue to reduce edema.

Reduce (but do not remove) MM spasms.

Reduce TrP’s.

On site work is NOW INDICATED:
vibrations, gentle stroking, gentle finger tip kneading.

38
Q

NOTE EARLY SUBACUTE DISLOCAITON

A

ONSITE WORK IS INDICATED

39
Q

late subacute tx -dislocaiton

A

Contrast H2O

Reduce HT and TrP’s.

Prevent excess adhesions: onsite; small area is tx each time. Longitudinal MM separations; followed with cross-fiber stroking/gentle frictions. Ice post Tx.

Maintain ROM: Gentle joint play (not in direction of MOI), P-free active assisted ROM & mid range PROM

40
Q

chronic dislocaiton – tx

A

Deep moist heat.

Reduce adhesions: Cross fiber frictions to adhesions followed by passive stretch, then ice.

41
Q

homecare – dislocaiton

A

Acute: P-free AROM of prox. Joints. That’s all!

Early Sub-acute: P-free AROM prox. and dist. Submaximal P-free isometric exercises.

Late Sub-acute: Maximal P-free resisted isometric exercises. Gradually progress to isotonic; avoid full range in direction of MOI.

Chronic: Isotonic active resisted in all ranges. Gradually return to ADL’s.

42
Q
A