orofacial pain Flashcards

1
Q

ways to measure pain

A

visual analogue scale
hospital anxiety and depression scale
mcgill pain questionnaire
oral impacts on daily performance

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2
Q

classification of orofacial pain

A

acute
chronic continuous
chronic acute

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3
Q

possible etiologies of acute orofacial pain

A
tmj
facial trauma
dental and oral causes
maxillary sinusitis
salivary gland disorders
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4
Q

what is most common form of non dental facial pain

A

TMD

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5
Q

how to differentiate between the different forms of TMD

A

palpate the joint and muscle
if joint hurts –> arthritic
if muscle hurts –> myalgic

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6
Q

in masticatory tmd, the discomfort/pain is usually located at

A
muscles of mastication
temple
retromolar pad
ear
neck
directly over TMJ area
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7
Q

characteristics of tmd

A

sudden onset of pain
pain is continuous, gets worse through out the day/night, worsen with function
deep aching pain

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8
Q

tmd aggravating factors

A

prolonged chewing
jaw movement
prolonged mouth opening eg at dental appointmetn

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9
Q

tmd associated factors

A

parafunctional habits eg clenching, bruxing
headache
migraines
tmj disc problems

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10
Q

treatment of tmd

A

Pharmacological:
NSAIDs, paracetamol, opioids
Injecting steroids/glucocorticoids into the joint (very effective if TMD due to arthritis associated with systemic inflammation)
Myalgia — diazepam, botox, TCA

Non pharmacological:
TMJ luxation — treat with manual repositioning
Jaw exercises, stretching, Occlusal correction
Arthrocentesis, discectomy with reduction, arthroscopy without reduction

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11
Q

burning mouth syndrome demographics

A

only occur in women

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12
Q

location for burning mouth syndrome

A

tongue (most common)
lips
palate
buccal mucosa

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13
Q

nature of pain or burning mouth syndrome

A

stinging, burning
continuous
mild to severe
itchy, sore

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14
Q

associated factors with burning mouth syndrome

A

depression
menopause (hormonal changes)
dry mouth
abnormal taste

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15
Q

management of burning mouth syndrome

A

exclude any secondary causes eg diabetes, drugs, anemia (full blood count, hematinics eg ferritin, b12 deficiency that can cause secondary burning mouth)

alphalipoic acid with gabapentin with or without benzydamine mouthwash for symptomatic relief around meal times

cognitive behavioural therapy to help you feel better about the pain

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16
Q

management of persistent idiopathic facial pain

A

this is a diagnosis of exclusion

anti depressants, cognitive behavioural therapy, acknowledge patient’s pain

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17
Q

location of post herpetic neuralgia

A

at the same site as where you had herpes zoster

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18
Q

presentation of post herpetic neuralgia

A

burning, tingling, itchy pain, may be sharp

moderate to severe

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19
Q

aggravating factors of post herpetic neuralgia

A

(≠initiating factors, pain is there all the time
light touch
eating

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20
Q

associated factors with post herpetic neuralgia

A

herpes zoster

ramsay hunt syndrome

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21
Q

management of post herpetic neuralgia

A

can be reduced by early treAtment of herpes zoster with acyclovir

spontaneous resolution, about 15% remain at one year

gabapentin, carbamzepine (must take blood test to test for allergic reaction which might result in acute erythema multiforme)

22
Q

presentation of post traumatic trigeminal neuropathic pain

A
continuous burning pain
unilateral
can be sharp and very severe
up to 6 months after trauma (more severe trauma can last longer)
triggered by touch, hot , cold
23
Q

management of post traumatic trigeminal neuropathic pain

A

reassurance that pain will pass quickly
anti convulsants
gabapentin

24
Q

presentation of persistent dentoalveolar pain

A

diagnosis of exclusion
pain is localised to tooth or tooth bearing area
continuous ache, dull, throbbing
may be aggravated by touch

25
management of persistent dentoalveolar pain
support, gabapentin, anticonvulsant
26
what is giant cell arteritis
inflammation of lining of arteries
27
giant cell arthritis may cause pain that is bilateral, or multifocal. giant cell arthritis at __ artery causes what symptom lingual, occipital, maxillary, retinal
lingual artery -- ischemic tongue, vascular necrosis of tongue occipital artery -- headache at back of head maxillary artery -- masticatory claudication retinal artery -- visual impairment due to giant cell infiltration in retinal arteries
28
nature of pain brough on by giant cell arteritis
continuous, uni or bilateral sudden onset dull, throbbing claudication pain (may mimic tmj)
29
diagnosis of giant cell arteritis
suspect if esr and crp high if suspicious, do temporal artery biopsy and look for periluminal dark halo on colour duplex ultrasonography give high dose steroids on speculation
30
presentation of post stroke pain
``` ipsilateral side as weakness (contralateral side to cerebral hemisphere insult) whole side of face continuous burning/prickling moderate pain may be delayed onset ```
31
migraine headache disorder is most common in what demographic
young females
32
etiology of migraine
abnormal 5-HT activity with cerebral oedema | association with TMD
33
presentation of migraine
unilateral Initially poorly localised, then localised (to temporal, frontal, orbital regions) may have aura episodic (attacks decrease in frequency with age)
34
aggravating factors for migraine
hormonal (OCP can increase frequency in those predisposed) diet flashing lights stress sleep deprivation
35
migraines may be associated with
photophobia nausea vomitnig tmj pain/tmd
36
treatment for migraines
acute treatment: 5 HT antagonist eg ergotamine, sumatriptan prophylaxis: beta blockers, calcium channel blockers, antihistamines
37
dental things to note when treating patient who is on prophylactic mediaction for migraines
patient may be taking propranolol which is non selective, should not give LA containing adrenaline (risk of hypertensive reaction) patient may be taking calcium channel blockrs eg verapamil which can cause gingival hypertrophy
38
what are the two types of trigeminal autonomic cephalgia
short unilateral neuralgiform pain with cranial autonomic features eg crying, sweating short unilateral neuralgiform pain with conjunctival injection and tearing eg lacrimation, nasal suymptoms, ptosis
39
presentation of trigeminal autonomic cephalgia
``` unilateral cluster headaches usually around the eyes (v1, v2) rapid, short lived attacks (seconds-minutes) sharp stabbing pain non refractory, frequent elicited by touch ```
40
management of trigeminal autonomic cephalgia
anticonvulsants, iamotrigine
41
the two types of trigeminal neuralgia
type 1: classical type episodic paroxysmal attack, short lasting (2s to minutes) usually in older patients nerve root compression at posterior cranial fossa by branch of pca type 2: longer lasting central cns aetiology eg brain tumour, demyelination disease
42
location of trigeminal neuralgia
unilateral | usually lower divisions ie v2, v3
43
nature of trigeminal neuralgia pain
``` episodic unilateral has refractory periods ie cannot elicit pain twice in a short period of time periods of remission electric shock with burning afterpain moderate to severe ```
44
aggravating factors of trigeminal neuralgia
light touch cold wind brushing teeth
45
treatment for trigeminal neuralgia
type 1 usually responds to medication. if not, neurosurgery type 2 mri to check for demyelination ``` cognitive behavioural therapy for fear steroids for demyelination antidepressants (depression due to pain) carbamazepine gabapentin with ropivacaine pregabalin ```
46
what neurosurgery do you do for trigeminal neuralgia
microvascular decompression, rhizotomy, peripheral neurolysis
47
what is the complication of carbamazepine
may cause acute erythema multiforme, need to take blood test to test HLA type before taking
48
presentation of glossopharyngeal neuralgia
triggered by swallowing, coughing, touching ear unilateral in ear, tongue base, tonsils radiating pain paroxysmal, 2s to minutes electric shock patient may get syncope due to severity of pain
49
management of glossopharyngeal neuralgia
carbamazepine gabapentin pregabalin lamotrigine
50
systemic diseases with characteristic facial pain/headaches
``` pagets hyperthyroidism metastatic disease multiple myeloma vitamin b defiicency SLE chemotherapy folic acid and iron deficiency anemia hyperparathyroidsm ```
51
red flags for orofacial pain
TUMOUR: spontaneous focal pain or altered sensation may indicate tumour invasion new onset headache with increasing severity, nausea, abnromal neurologic signs eg decreased sensation across face --> intra cranial tumour pregnancy + ear ache+ trismus + altered sensation in mandibular distribution --> acoustic nerve tumour CARDIAC: pain at angle of mandible esp left, brought on by exertion and relieved by rest --> cardiac ischemia SYSTEMIC: systemic signs of anorexia, fever, weight loss, malaise, sweating trigeminal neuralgia in patient under 50 years old may suggest multiple sclerosis (a type of demyelination disease) TEMPORAL ARTERITIS: suspect temporal arteritis when: - over 50 y/o, localised progressive headache, superficial temporal artery swelling and tenderness - jaw claudication, palpably tender superficial temporal arteries