Oral Pain and its mangament (no lecture) Flashcards

1
Q

What are the structures in the oral cavity and the maxillofacial region?

A

teeth, gingiva, mucosa, salivary glands, muscles, bones, ligaments, tendons, blood and lymphatic vessels, taste buds, nerves

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2
Q

What is the most prevalent pain in the facial region?

A

toothache
-odonalgia

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3
Q

what percentage of people report tooth ache in the past 6 months?

A

12-14%

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4
Q

Are all toothahces the same?

A

nah fam

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5
Q

What are odotogenic?

A

pulpal and periodontal

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6
Q

What are non-odontogenic?

A

sinus/nasal, myofascial, neurovascular, neuropathic, cardiogenic, systemic, idiopathic

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7
Q

What are other sources of orofacial pain?

A

mucosal pain, TMDs, orofacial neuropathic pain, neurovascular pain, sleep disorders

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8
Q

What does TMD involve?

A

joint sounds

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9
Q

What is pain?

A

unpleasant sensory and emotional experience assiociated with actual or potential tissue damage or described in terms of such damage

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10
Q

Why do we feel pain?

A

instills protectivebehavior but if unabated, pain can be harmfuil

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11
Q

What are the principles of pain?

A

-always subjective
-may or may not be tied to a stimulus
-always a consequence of an emotioanl experience and psychological state

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12
Q

How do we experience pain usually?

A

-environmentla stimulus
-receptor activaiton
-generation of action potential
-transmission through primary afferent to dorsal horn
-projection from dorsal horn to brain for perception and interpretation

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13
Q

What are the biological factors of pain experience?

A

-genetics
-physiology
-neurochemistry
-tissue heath

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14
Q

What are the psychological factors of pain experience?

A

perceived control, self-efficacy, catastrophic thinking, hypervigilance, depression, anxiety, anger

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15
Q

What are the social factors of pain experience?

A

socioeconomic status, social, skepticism, operant, social support, social learning

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16
Q

What is the descending spinal pathway

A

-endogenous opoid signaling
-non-opioid inhibitatory neurotransmitters

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17
Q

What is the descending supraspinal pathway?

A

-influenced by psychological
-neurons from the cortex and amygdala
-periaqueductal gray and rostroventral

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18
Q

What is involved with supraspinal modulation?

A

psychological, emotional, and placebo

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19
Q

What is involved with spinal modulation?

A

neurotransmitters, neuropeptides, interneurons, endogenous opioids, central sensitization

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20
Q

What is involved with peripheral modulation?

A

peripheral sensitization, inflammatory mediators, intense/repetitive/prolonged noxious stimulus

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21
Q

What is pain modulation?

A

dynamic process- can occur at multiple levels of the ascending and descending pathway

22
Q

What is neuronal sensitization?

A

arise when neurotransmitters are left to linger in the synapse
-due to failure in diffusion, enzymatic destruction, and reuptake

23
Q

What does neuronal sensitization do?

A

prolong the effect of the post-synaptic neurons, allow subthreshold input to recruit a response, spontaneous discharge, and increase the size of receptive field

24
Q

What is normal input of neuronal sensitization?

A

increased responsiveness of nociceptive neurons

25
What is the the subthreshold input of neuronal sensitization?
recruitment of a response
26
What is spontaneous discharge of neuronal sensitization?
increased size of receptive field
27
What is peripheral sensitization?
nociceptive neurons at periphery of receptive field -increased responsiveness of noiceptive neurons
28
What is reduced threshold to stimulation?
primary hyperalgesia
29
What is hyperalgesia?
an increased pain experience in response to a painful stimulus
30
What is central sensitization/
nociceptive neurons in the central nervous system
31
What is allodynia?
pain resulting from a stimulus that does not normally provoke pain
32
What can cause allodynia?
dysfunctional endogenous pain control
33
What is the revised gate control theory?
myelinate non-nociceptive affect fiver can activate inhibitory interneurons modulating nocicpective transmition
34
What does revised gate control theory do?
instinctively wave, hold, clench, your fingers when they burn
35
What is diffuse noxious inhibitor control?
thresold for nociception can be raised when another noxious stimulus is provoke in another pain area -pain inhibits painWh
36
What is the placebo effect?
psychological -leads to released of endogenous analgesic substance
37
What are the different ways to categorize different pain?
neurophysiology, structures involved, timing
38
What is nocieptive pain?
-pain resulting form damage or threatened damage to non-neural tissue -activation of nociceptor
39
What is neuropathic pain?
-pain resulting from the presence of a lesion or disease of the somatosensory nervous system
40
What is nociplastic pain?
-new concept -pain that arises from altered nociception -does not satisfy the defintions of nociceptive or neuropathic pain -possible for a patient to present with nociceptive and nociplastic pain at the same time
41
What is acute pain?
pain with close temporal relationship to a stimulus, injury, or disease -tends to respond to treatment in a linear dose-dependent fashion
42
What is chronic pain?
more than 3 months -does not typically respond to treatment in a linear dose-dependent fashion -presence of other/multiple ongoing pains is a predictor for transition from acute to chronic -more influence of psychosocial factor -more difficult to treat
43
Most common psychosocial disorders?
-anxiety -major depression -personality disorders -pain distress
44
What are coping mechanisms for psychological disorders?
-internal locus of control -perceived control -catastrophic thinking -hypervigilance -fear avoidance
45
What are the assessments for pain?
-pain intensity -pain distress -pain-related interference (functional limitation, disability) -oral habits
46
What is homotopic pain?
when the site of the pain is the source of the pain
47
What is heterotropic pain?
when the site of the pain is not the source
48
What is central pain?
source is central but percieved peripherally
49
What is projected pain?
pain follows same nerve distribution as primary source
50
What is referred pain?
pain in different nerve than primary source and is spontaneous -not aggravated by palpation -does not respond to anesthesia -does not cross midline
51
What is the most important part of pain management?
understanding why the patient is in pain
52
What are examples of opioids?
codeine, oxycodone, morphine, hydromorphone, and meperidine