Oral medicine and OMS: Kang, DMD Flashcards

1
Q

What is gingivitis?

A
  • inflammation of tissues surrounding the teeth (gums)
  • can be necrotizing ulcerative gingivitis, or medication-influenced gingivitis
  • tx: oral hygiene, scaling/root planing, periodontal surgery
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2
Q

What is periodontal disease?

A
  • inflammation surrounding teeth causing loss of bone and PDL
  • progressive loss of attachment leads to significant loss of alveolar bone and attachment
  • periodontitis associated with systemic disease
  • tx: oral hygiene, scaling/root planing, periodontal surgery
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3
Q

What is alveolar osteitis?

A

this is inflammation of the alveolar bone, aka “Dry socket”:
After tooth extraction, a blood clot forms and will eventually be replaced by granulation tissue and bone. When the blood clot fails to form or is lost, it’s called “dry socket” - occurs following 1-3% of all extractions, 25% of wisdom teeth extractions
Sx: extreme pain and foul odor
Tx: pain meds, medicated dressings, remove debris, flush socket and oral hygiene, abx not necessary

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4
Q

Fungal infections of the mouth; Pseudomembrane and Erythematous candidiasis

A

Candidiasis
- opportunistic infection of candida in the mouth

Pseudomembrane candidiasis (thrush):

  • affected area becomes tender with red and white areas; white areas are debris mixed with candida - can scrape away
  • tx: remove underlying factor, nystatin oral, clotrimazole lozenge, Fluc/Keto/Itraconazole

Erythematous candidiasis (more common than thrush but often overlooked)

  • no white flecks, rather varying degrees of erythema/petechiae
  • commonly from dental trauma - debate re: true infection or host tissue response
  • subtypes: acute atrophic candidiasis follows course of Abx, denture stomatitis from wearing dentures
  • tx: oral hygiene, keep dentures out at night
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5
Q

What dental procedures require prophylaxis for bacterial endocarditis, and which do not?

A

Do: for someone with prosthetic cardiac valves, previous endocarditis, congenital HD, or heart transplant with valvulopathy undergoing:

  • Extractions
  • Periodontal procedures
  • Dental implant placement

Don’t:

  • Fillings
  • Local anesthetic injection
  • Removal of primary teeth
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6
Q

What is torus mandibularis and torus palatinus?

A

Torus mandibularis: common exostosis that occurs on the posterior aspect of lingual aspect of mandible; cause maybe environmental (masticatory stress) vs. genetics. Bilateral 90%; present in 7-10% of the population

Torus palatinus: common exostosis that occurs in the palate; not present until late teens/adulthood; no malignant potential. Present in 9-40% of population.

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7
Q

What is hairy tongue and what is the treatment?

A

The accumulation of keratin on filiform papillae on the dorsal tongue (spares ant. and lat. borders).
Cause is likely from poor oral hygiene and smoking; appears black from bacteria and staining from smoking/debris.
Found in .5% of adults and is benign.

Treatment:
Eliminate predisposing factors such as smoking; improve oral hygiene; periodic scraping with a tongue scraper or toothbrush

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8
Q

What is hairy leukoplakia?

A
  • an opportunistic infection most commonly in HIV patients that presents with a “hairy” or corrugated surface on the lateral border of the tongue; short white strands that project from the surface epithelium
  • associated with EBV
    Tx: nothing specific; can give anti-retrovirals
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9
Q

What is oral erythema migrans “geographic tongue”?

A
  • this subtype of erythema migrans involves only the dorsal and lateral aspects of the tongue mucosa
  • presents as areas of erythema surrounded by elevated yellow scalloped borders
  • a very common entity of unknown etiology and pathogenesis
  • affects 1-3% of the population
  • Tx: none; reassurance
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10
Q

What is an aphthous ulcer?

A
  • destruction of the oral mucosa; appears to be a T cell mediated immunologic reaction
  • common, present in 20% of population
  • multiple causes which may include allergies, hormones, trauma, stress, smoking
  • Tx: usually not necessary, can use OTC anesthetics or topical steroids
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11
Q

What is leukoplakia?

A
  • Leuko=white / plakia=patch
  • defined by the WHO as a “white patch or plaque that cannot be characterized clinically or pathologically as any other disease” – a clinical term and diagnosis of exclusion
  • tx: biopsy for definitive diagnosis; excision for any dysplasia
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12
Q

Ondontogenic cysts

A
  • these are odontogenic-derived epithelial cysts; subclassified into developmental and inflammatory
  • Inflammatory cysts: Periapical cyst; Residual cyst
  • Developmental cysts: (DECLOG) Dentigerous cysts; Eruption cyst; Calcifying odontogenic cyst; Lateral periodontal cyst; Odontogenic keratocyst; Glandular odontogenic cyst
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13
Q

What is a dentigerous cyst?

A
  • a cyst that originates by the separation of the follicle from around the crown of an unerupted tooth
  • not painful unless secondarily infected
  • 2nd most common cyst; usually in the posterior mandible or maxilla; associated with wisdom teeth; can grow to a large size and cause bony expansion/facial fullness
  • tx: enucleation
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14
Q

Odontogenic keratocyst (OKC)

A
  • rare, benign but locally aggressive and recurrent odontogenic cyst; resemble both cyst and tumor
  • histo: lined by stratified squamous epithelium with a parakeratinized and often corrugated surface; basal cells are hyperchromatic, cuboid-columnar, and have palisaded nuclei; usually no inflammation
  • peaks in teens/20s but can occur at any age; in kids associated with basal cell nevus syndrome
  • 2x in mandible > maxilla; small/unilocular to large/multilocular
  • can cause bony expansion and tooth mobility but most are asymptomatic
  • tx: marsupialization, enucleation and curettage, or resection
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15
Q

Ameloblastoma

A
  • tumor of odontogenic epithelium
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16
Q

Bisphosphonate related necrosis of the jaw (BRNOJ)

A
  • bisphosphonates cause decreased bone turnover (inhibited resorption) and antiangiogenesis; basically can’t address impending infections
  • stages of infection/disease:
    1. exposed bone; no bone or soft tissue inflammation
    2. painful areas of exposed bone; with soft tissue or bone inflammation
    3. most advanced stage, +/- fracture; extensive exposed bone and soft tissue inflammation
  • Dx:
    1. Exposed bone in maxillofacial region, for >8 weeks
    2. Current/previous bisphosphonate therapy
    3. No hx of radiation therapy of the jaws
17
Q

What is verrucous carcinoma?

A
  • a low grade variant of oral squamous cell carcinoma; slow growth but locally aggressive (mets are rare)
  • Lesion appears diffuse, well demarcated, painless, thick plaque with papillary projections
  • associated with HPV 6, 11, 16, 18, and chewing tobacco or snuff (where tobacco is habitually placed), generally men >60yo
  • Tx: surgical excision with adequate margins
18
Q

Squamous cell carcinoma of the mouth

A
  • tongue and floor of mouth are the most common sites of origin for primary SCC in the oral cavity
  • clinical features are variable–can be superficial or deep, ulcerative, exophytic or endophytic
  • ulcerative lesions = irregular borders, induration, and can bleed
  • biopsy with a punch or wedge excision is used to confirm the diagnosis
  • if the lesion is suspicious and the biopsy does not show malignancy, then the biopsy should be repeated
  • tx: staging, imaging, excision, radiation/chemo, FU
19
Q

SCC of the lip

A
  • SCC is more common on lower lip than upper (upper lip cancer is usually BCC); accounts for 25-30% of oral SCC
  • 2 General Presentations:
    1. slow growing exophytic mass (most common) - behaves like cutaneous SCC associated with UV light
    2. eroding destructive invasive ulceration (less common); more related to pipe/cigarette smoking
20
Q

What is Ludwig’s angina?

A
  • an aggressive and rapidly spreading cellulitis that involves the bilateral sublingual, submandibular, and submental spaces
  • presents as fever, swelling of the mouth/face/neck, unable to tolerate secretions
  • associated with recent dental work or caries