OOP midterm Flashcards

1
Q

homeostasis

A

Tendency of the body to seek and
maintain a condition of balance or
equilibrium within its internal
environment, even when faced with
external changes

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2
Q

Allostasis

A

body’s attempt to compensate for a stressful
situation in a protective manner

When threatened, the neuroendocrine-immune network-
capable of altering normal homeostatic rhythms
– As threat diminishes, feedback control systems should suppress
levels of allostatic compounds, returning body to normal function
– Disease processes will activate allostatic response

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3
Q
  • Frequent activation of stress response
A

damages body chronically through activation
of the hypothalamic-pituitary adrenal (HPA) axis
– Effects add up progressively (cumulative)

Allostatic load: price paid for chronic exposure
to stress-mediated neuroendocrine adaptations
– Long-term exposure to allostatic chemical
environment (catecholamines, cortisol, cytokines)
* Long-term activation of allostatic mechanism—
gradual destruction of organ systems
– Gradual loss of effectiveness of feedback pathways to
reestablish normal homeostasis

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4
Q

Allostatic load:

A

Allostatic load: price paid for chronic exposure
to stress-mediated neuroendocrine adaptations
– Long-term exposure to allostatic chemical
environment (catecholamines, cortisol, cytokines)
* Long-term activation of allostatic mechanism—
gradual destruction of organ systems
– Gradual loss of effectiveness of feedback pathways to
reestablish normal homeostasis

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5
Q

Biomechanical Model

A

patient has T4/T3 restriction, so they would have trouble walking

Uses structural/mechanical perspective to assess patient
* Structural impediment caused by a dysfunction of muscles, joints,
&/connective tissue

Can lead to disturbances in various body functions

  • Assess patient for a structural impediment (somatic dysfunction)
    – By using OMT to correct somatic dysfunction, patient regains associated structural,
    vascular, neurologic, metabolic, & behavioral functions
  • Objective: Optimize patient’s adaptive potential through restoring
    structural integrity & function
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6
Q

Respiratory-Circulatory Model

A

Evaluation & treatment:
– Maximize capacity & efficiency of respiratory-circulatory functions

maintenance of extra- & intra-cellular environments
– Delivery of oxygen & nutrients
– Removal of cellular waste products

OMT addresses dysfunction in:
– Respiratory mechanics
– Circulation
– Flow of body fluids

restoring fluid circulation

ex: compressed nerved—-> light headed

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7
Q

neurological model

A
  • Sensory & protective
  • Focuses on impairments of neural function caused by or cause pathophysiologic responses in
    the other 4 domains

relationship between somatic and autonomic NS

  • Considers influence on neuroendocrine immune network by:
    – Spinal facilitation
    – Proprioceptive function
    – Autonomic nervous system
    – Activity of nociceptors (pain fibers)

compression of vangus nerve, anxiety

IMPAIRMENT OF A NEURAL FUNCTION

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8
Q

neurological model

OMT focus and goal of treatment

A
  • OMT focus
    – Reduction of mechanical stresses
    – Balance of neural inputs
    – Elimination of nociceptive drive
  • Goal of treatment:
    – Re-establish normal/optimal neural function
    – Attain autonomic balance and flexibility
    – Address neural reflex activity
    – Remove facilitated segments
    – Decrease afferent nerve signals
    – Achieve pain relief

compression of vangus nerve —> anxiety

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9
Q

Metabolic-Energy (Nutritional) Model

A

Recognizes that the body seeks to maintain a balance between
– Energy production, Distribution, Expenditure
* The body’s ability to restore & maintain health requires energy-efficient response to
infectious agents & repair of injuries
* Proper nutrition enables normal biochemical processes, cellular functions, &
neuromusculoskeletal activity

injury to MSK burdens the energy

diabetic—> weight gain —-> strain on body

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10
Q

Behavioral (Biopsychosocial) Model

A
  • Recognizes that patient’s health includes:
    – Mental, emotional, & spiritual state of being
    – Personal lifestyle choices
  • Health is often affected by:
    – Environmental, socioeconomic, cultural, & hereditary factors
    – Various emotional reactions & psychological stresses
  • Environmental toxicities, inactivity, lack of exercise, use of addictive substances, poor
    dietary choices can diminish a patient’s adaptive capacity, make him/her vulnerable to
    infections and/or organ/system failure
  • Provide patient EDUCATION on:
    – Health
    – Disease & lifestyle choices
    – Mental outlook
    – Preventative care

diabetic—> weight gain—>strain on body

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11
Q

Somatic Dysfunction:

A

“Impaired or altered function of related components of the
somatic (body framework) system: skeletal, arthrodial, and myofascial structures, and
related vascular, lymphatic, and neural elements.”
– Impaired joint mechanics
– Altered mechanics of the associated soft connective tissue (e.g., abnormal
resting length of muscle, alteration of the tension of ligament, aberrant tension
across fascia, strain in the dura, and congestion in the extracellular matrix)
– Dysfunctions of arterial supply, venous and lymphatic drainage, and nerve
conduction
– Changes in the tissues resulting from the foregoing to include tissue texture
change (ropiness,

SAM’S RV LANE

Skeletal,
Arthrodial
Myofascial Structures
Related Vascular
Lymphatic and Neural Elements

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12
Q

Primary Somatic Dysfunction

A

– The somatic dysfunction that maintains a total pattern of dysfunction including
other secondary (“key lesion”)
– Initial or first somatic dysfunction to appear temporally
– Sudden trauma (usually an external force)
– Postural imbalances, micro trauma and repetitive trauma

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13
Q

Secondary Somatic Dysfunction

A

– Compensation for a primary problem
– Compensation for musculoskeletal defects
– Reflex response to visceral disease
– Reflex response to emotional stress
– Compensation for a hereditary imbalance

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14
Q

what are the 4 tenets of osteopathic medicine

A
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15
Q

structure and function relationship example

A
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16
Q

1892

A
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17
Q

1874

A
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18
Q

1918

A

R.K. Smith, M.D., D.O. presented information collected by the AOA at the Annual
Convention of the American Association of Clinical Research in New York City on
October 18, 1919:
* 2,445 DOs reported on their patients
– Influenza
* 110,120 cases treated
* Only 257 deaths (approximately 0.25% mortality rate)
* Reported MD patient mortality rate was 5%
– Influenza-associated Pneumonia
* 6,258 cases treated
* Only 635 deaths (approximately 10% mortality rate)
* Reported MD patient mortality rate was >30% (3x higher than DO rate)

DO had less death rates overall!!!

INC patient volume for DOs

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19
Q

1961

A
  • 1961 - California Osteopathic Association merged with California Medical Association
  • 1961 - The College of Osteopathic Physicians and Surgeons in Los Angeles changed
    to an allopathic institution, becoming the California College of Medicine (now the
    University of California Irvine School of Medicine)
  • 1962 - Qualified and consenting D.O.s were conferred M.D. degrees
    – About 2,000 D.O.s converted their degree to M.D.
    – No additional training/education was required, only an administrative fee of $65
    – New degrees only recognized within California & specialists unable to practice as such
    – D.O.s would no longer be licensed in the state
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20
Q

HOMEOSTASIS VS ALLOSTASIS

A

Homeostasis- process how body keeps itself in threshold where body can survive

Allostasis—how the body adapts to stressors ….. When the body is under a lot of stress, the baseline will shift. “body’s attempt to compensate for a stressful situation in a protective manner”

The big goal of Osteopathy is to restore homeostasis

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21
Q

palpation

A

Application of variable manual pressure upon the surface of the body for the
purpose of determine the shape, size, consistency, position, inherent motility and
health of the tissues beneath.

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22
Q

Observation of Static Landmark

A
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23
Q

Observation of Static Landmark- in the Mid-Gravity
Line/Plumb Line

  • Mid-heel point
  • Pubic symphysis
  • Umbilicus
  • Xiphoid process
  • Mid-sternum
  • Episternal notch
  • Symphysis menti
  • Glabella
A
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24
Q

Anterior Static Landmarks in the Mid-Gravity
Line/Plumb Line

A
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25
describe the 5 models
26
Somatic Dysfunction:
Somatic Dysfunction: “Impaired or altered function of related components of the somatic (body framework) system: skeletal, arthrodial, and myofascial structures, and related vascular, lymphatic and neural elements. SAM'S RV LANE” * Somatic dysfunction is a functional disorder * If pure somatic dysfunction is diagnosed and removed, normal function is restored
27
Acute Somatic Dysfunction
28
Chronic Somatic Dysfunction
29
Acute vs. Chronic Tissue Texture Changes
Acute Moist Boggy Edema Red opposite to amber is chronic
30
TART
31
__________barrier * The limit of motion imposed by anatomic structure; the limit of passive range of motion * The point past which tissue disruption occurs (sprain/strain) and disruption of the joint will occur
Anatomic Barrier
32
__________ Barrier * The limit of active motion * As far as the patient can go without assistance
Physiologic Barrier
33
________ Barrier * Somewhere between the physiologic and anatomic, barrier of motion
elastic barrier "feathers edge"
34
_________Barrier * A functional limit [within the active range of motion] that abnormally diminishes the normal physiologic range * Cannot achieve full range of motion.
Restrictive
35
_____ barrier a restriction of joint motion associated with pathologic change of tissues (example: osteophytes)
Pathologic barrier * a restriction of joint motion associated with pathologic change of tissues (example: osteophytes)
36
Erythema Friction Rub
37
landmarks to find spinal process
38
NAME the Planes and Axes of Motion
39
Superior Facet Orientation
40
rules of 3
41
Fryettes principles
42
Type I vs Type II Somatic Dysfunctions
43
categories of somatic dysfunction
fascial and ligamentous restrictions articular restriction muscular restriction edematous FAME
44
direct vs indirect treatments
45
46
What is the classification of OMT that applies a vector of force in towards the restrictive barrier
Direct technique
46
Name 3 direct MT and 3 Indirect OMT
Direct Soft tissue Myofascial release MUSCLE ENERGY TECHNIQUE INDIRECT myofascial release
47
ACTIVE vs PASSIVE
Active- patient does something MET Passive- patient does not do anything Soft tissue MFR
48
What is the absolute contraindication for all OMT
Lack of pt consent Lack of somatic dysfunction
49
What tissues form layer and sheets that envelops and separate tissues..
FASCIA
50
7 stages of Spencer technique (Week 5) and what does it treat ?
EFC CAARD treats shoulder
51
How do you perform direct myofascial release
“Direct MFR:The dysfunctional myofascial tissues are loaded and restrictive barrier is engaged with a constant force.” 1. Paplate soft tissue. 2.Find area of restriction in all three planes ( up/down, left/right, and clockwise/counterclockwise) 3. Stack all three planes and move in the direction of restriction Hold and wait for it to soften ]. Follow the creep if the tissue softens and allows you to travel further into the restriction area. BRING FASCIA TO DIRECT BARRER AND HOLD FOR 60 SECONDS
52
what are the 4 different type of MET?
53
What is an absolute contraindications for muscle energy besides lack of consent or somatic dysfunction
Fracture in area Patient cannot follow directions– CANT DO ON PERSON IN COMA.
54
28. How many seconds does a patient contract for during a cycle of muscle energy?
3 to 5 seconds
55
29. How much force is typical applied by the patent vehicle contracting
3 to 5 oz
56
30. How many times repeat muscle energy
3 to 5 times
57
31. How long is the post-isometric relaxation?
1 to 2 seconds
58
Isometric vs isotonic(concentric, eccentric) vs isolytic
Isometric– contract, muscle length stays same Isotonic- tone in muscle stays same, muscle shorten; mostly concentric -Concentric- While a contracting muscle, under a constant load, experiences a decrease in the distance between that muscle’s origin & insertion points (approximation) -Eccentric- muscle lengthens Isolytic - muscle lengthening while it is being contracted, A form of eccentric contraction designed to break adhesions using an operator-induced force to lengthen the muscle and in which, the counterforce is greater than the patient force.
59
things to keep in mind during soft tissue
60
Unilateral Thoracic and Lumbar Prone Pressure-soft tissue
61
suboccipital release
62
barriers
63
extrinsic vs intrinsic force vs inherent forces
inherent ROM? spontaneous motion of every cell, organ system, and their component units within the body.
64
direct vs indirect technique and what forces do they use
65
techniques that we have learned so far use active, passive, direct or indirect approach
66
soft tissue technique
67
muscle energy technique (MET)
68
________________ * A group of direct techniques that usually involve lateral stretching, linear stretching, deep pressure, traction &/or separation of muscle origin and insertion while monitoring tissue response and motion changes by palpation. * Historically considered a form of myofascial treatment.
soft tissue technique
69
________________ A direct treatment method which the patient’s muscles are employed upon request, from a precisely controlled position, in a specific direction, and against a distinctly executed physician counterforce. * Contraction of an antagonistic muscle would help relax the agonistic muscle.
muscle energy technique
70
-------- * An osteopathic method in which the restrictive barrier is engaged in one or more planes of motion and then a rapid, therapeutic force of brief duration traveling a short distance is applied within the anatomic range of motion of a joint. * Aka: Thrust treatment method
RAPID SHORT
71
________ A direct treatment method employing a low velocity/moderate to high amplitude force applied to a dysfunctional joint through either: – A repetitive springing motion or – A single movement of the joint through the restrictive barrier.
LOW velocity , Springy motion
72
__________ – A system of diagnosis and treatment, first described by A.T. Still MD, DO, & his early students, which engages continual palpatory feedback to achieve release of myofascial tissues.
MYOFASCIAL RELEASE (MFR
73
MYOFASCIAL RELEASE (MFR) Indications and Physiologic Effects
Indications and Physiologic Effects
74
_________a generalized term for the sheets and layers of connective tissue that envelop specific structures and segregate one structure, organ, or area from another.
75
principles of myofascial release
76
Fascial System
77
what layer of facia do we work with?
78
functions of fascia
ppppp
79
myofascial release
80
MFR Contraindications
81
_______palpable resistance to motion of an articulation or tissue ______relative palpable freedom of motion of an articulation or tissue _______the capacity of fascia and other tissue to lengthen when subjected to a constant tension load
* Bind: palpable resistance to motion of an articulation or tissue * Ease: relative palpable freedom of motion of an articulation or tissue * Creep: the capacity of fascia and other tissue to lengthen when subjected to a constant tension load * MFR allows for fascial creep which are associated with release of energy (heat, electromagnetic, etc.
82
how should the pressure be in myofascial release?
83
MFR General Considerations/Rules? (how do you perform MFR)
84
steps for Prone Thoracic/Thoracolumbar Junction MFR(Direct)
85
steps for Thoracic Inlet/Outlet MFR Seated, “Steering Wheel” (Direct)
86
do you need to diagnose the patient before doing Soft tissue?
87
Soft Tissue Techniques – Indications
88
how do we perform a soft tissue technique?
The technique tension or force applied should match the tension in the patient’s tissue * Modify the force applied as the patient’s tissue responds in real time
89
Unilateral Thoracic and Lumbar Prone Pressure
uses kneading
90
Bilateral Thoracic Prone Pressure with Counterpressure
91
Lumbar Prone Pressure with Counterleverage
92
AGR principles
93
Suboccipital Release, things to note
94
normal end feel motions after range of motion
BAS C
95
Abnormal End-Feel DURING RANGE OF MOTION
96
MUSCLE ENERGY INDICATIONS
97
Post-Isometric Relaxation Muscle Energy Technique (MET) STEPS
98
MET mechanism
99
post isometric relaxation vs reciprocal inhibition
the problematic (agonist muscle) is used as the contracting muscle during MET. The Golgi organ will send signals to the muscle to relax during the post-isometric relation and then you can take it to the next feather's edge RECIPROCAL INHIBITION For example, the bicep is the problem during reciprocal inhibition, but we will use the tricep to perform a post-isometric relaxation. This will cause the bicep to relax. Patient pushes toward barrier (where it doesn't want to go)
100
what muscle contraction is most commonly used during MET
101
Concentric Isotonic Muscle Contraction
102
Eccentric Isotonic Muscle Contraction
103
Isolytic Muscle Contraction
104
4 different types of MET
105
Post isometric relaxation goal method physiology force of contraction
106
reciprocal inhibition goal method physiology force of contraction
107
Respiratory Assistance goal method physiology force of contraction
108
oculocephalogyric reflex goal method physiology force of contraction
109
muscle energy mechanism of action
110
Deep Fascia of The Upper Extremity
111
effect of muscle energy
112
Evaluation for Radial Head Somatic Dysfunction
113
steps for MET Elbow: Posterior Radial Head Somatic Dysfunction (Radioulnar Pronation Dysfunction)
114
Three common gears that get jammed in the body are:
*Joints- most common *Muscles *Fascia.
115
what happens when fascia slide past eachother?
Fascial sheets must continually slide against other sheets. Frequently two sheets get stuck together (forming an adhesion) and can no longer glide due to that adhesion. These commonly form after injuries, surgeries or sustained periods of immobility.
116
______ is used to define Osteopathic Manipulative Techniques
The two most common forms of muscle energy are “joint mobilization” and “post-isometric relaxation
117
Post Isometric Relaxation and Joint Mobilization I
118
Post Isometric Relaxation and Joint Mobilization II
119
post isometric relaxation steps
120
understand this!!
121
Post Isometric Vertebral ME
122
123
124