Nutrition in the LifeCycle 2 Flashcards

1
Q

Toddler

A
  • children between the ages of 1 and 3
  • stage of development characterized by rapid increase in gross and fine motor skills with subsequent increase in independence, exploration of the environment, and language skills
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2
Q

Preschool

A
  • children between the ages of 3 and 5
  • stage of development characterized by increasing autonomy, experiencing broader social circumstances, such as attending preschool, or staying with friends and relatives; increasing language skills, and expanding ability to control behavior
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3
Q

What is normal weight gain in toddlers and preschoolers respectively,

A

toddlers: 3-5 lbs a year, mostly in gut

preschoolers 4 lbs a year

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4
Q

How is growth monitored in toddlers? preschoolers?

A
Use calibrated scales & height board
Toddlers under age 2 years 
-Weighed without clothes or diaper 
-Determine recumbent length 
Children over age 2 years
-Weighed with light clothing 
-Measure stature with no shoes 
Recumbent length
-The length of toddlers < 24 months are measured in the recumbent position
Common problems:
-Error in measuring may result in errors in health status assessment  
-Use of calibrated equipment and plotting accuracy are vital
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5
Q

Physiological and Cognitive Developmental Milestones in Toddlers

A
walking progress by month
-15mo: crawl upstairs
-18mo: run stiffly
-24mo: walk up stairs one foot at a time
-30mo: alternate feet going up stairs
-36mo: ride a tricycle
cognitive
-toddlers "orbit" around parents
-transitions from self-centered to more interactive
-vocabulary expands
*10-15 words at 18mo
*100 words at 2 years
*3 word sentences by 3 years
-temper tantrums common
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6
Q

Cognitive Development of Preschool age children

A
  • egocentric: cannot accept another’s point of view
  • learning to set limits for himself
  • cooperative & organized group play
  • vocabulary expands to >200 words
  • begin using complete sentences
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7
Q

Development of feeding skills in Toddlers

A

gross and fine motor development improved
-9-10 months weaning bottle begins
-12-14 months completely weaned
-12 months refined pincer
-18-24 months able to use tongue to clean lips and has developed rotary chewing
Adults supervision vital to prevent choking

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8
Q

Feeding Behaviors in Toddlers

A
  • rituals in feeding are common
  • may have strong preferences & dislikes
  • food jags common (child eats the same thing for a long period of time and then won’t eat it at all)
  • serve new foods with familiar foods & when child is hungry
  • toddlers imitate parents & older siblings
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9
Q

What is a toddler’s appetite? What are appropriate portion sizes for toddler’s?

A
  • Slowing growth results in decreased appetite
  • Nutrient-dense snacks needed but avoid grazing on sugary foods that limit appetite for basic foods at meals
  • Toddler-sized portions average 1 tablespoon per year of age
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10
Q

Feeding Behaviors and Appeitite in Preschoolers

A
  • can use fork, spoon, & cup
  • spills occur less frequently
  • foods should be cut into bite-size pieces
  • adult supervision still required
  • appetite related to growth
  • appetite increases prior to the “spurts” of growth
  • include child in meal selection & preparation
  • portions?
  • Control amount eaten between meals to ensure appetite for basic foods
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11
Q

Temperament Theory

A

Temperament is defined as the behavioral style of the child, or the “how” of the behavior
Three temperamental clusters
-“easy”-adapts to regular schedules & accepts new foods
-“intermediate”/”slow-to-warm-up”-slow adaptability, negative to new foods, but can learn to accept new foods
-“difficult”-slow to warm up and may be negative to new foods; may have to give more exposures, have more patience
-“intermediate low” and “intermediate high”-a mixture of behaviors

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12
Q

Appetite and Satiety

A
  • consumption of foods high in sugar and/or fat before meals decreases intake of basic foods
  • offering large portions increases food intake and may promote obesity
  • restriction of palatable food increases preferences for the foods
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13
Q

Parent Child Feeding relationship

A

Parent or caretaker responsibilities:
-“What” children are offered to eat
-The environment in which food is served including -“when” & “where” foods are offered
Child’s responsibilities:
-“How much” they eat
-“Whether” they eat a particular meal or snack

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14
Q

Common micronutrient deficiencies in toddler’s and preschoolers

A
iron, calcium, zinc
RDA 1-3yrs
iron: 7mg
calcium: 500mg
zinc: 3mg
RDA 4-8yrs
iron: 10mg
calcium: 800mg
zinc: 5mg
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15
Q

Common Nutritional Problems in toddlers and preschoolers

A
Iron-deficiency anemia
Dental caries
Constipation 
Lead poisoning
Food security 
Food safety
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16
Q

Prevention and Intervention of Iron-Deficiency Anemia in Toddlers

A

Preventing Iron Deficiency:
-Nutrition-Limit milk consumption to 24 oz/d since milk is a poor source of iron
-Infants at risk should be tested at 9 to 12 months, -6 months later, and annually from ages 2 to 5
Intervention for Iron Deficiency
-Iron supplements
-Counseling with parents
-Repeat screening

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17
Q

Dental Caries in Toddlers

A
Prevalence: 
-1 in 5 children ages 2 to 4
Causes:
-Bedtime bottle with juice or milk
-Streptococcus mutans
-Sticky carbohydrate foods
Prevention:
-Fluoride—supplemental amounts vary by age & fluoride content of water supply
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18
Q

Constipation in Toddlers

A

Definition: Hard, dry stools associated with painful bowel movements
Causes: “Stool holding” and diet
Prevention: Adequate fiber
AI for fiber ages 1-3: 19g/d; 4-8: 25g/d

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19
Q

Lead Poisoning in Toddlers

A
  • Seen in ~2.2% of children ages 1-5
  • Low levels of lead exposure linked to lower IQ & behavioral problems
  • High blood lead levels may decrease growth
  • Reduce lead poisoning by eliminating sources of lead
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20
Q

Nutrition and Prevention of cardiovascular disease
in toddlers and preschoolers

A

Limit dietary saturated fats, trans fat & cholesterol
Acceptable total fat intake ranges:
-2 to 3 years—30 to 35% of calories
-4 to 18 years—25 to 35% of calories
For children at high risk of CVD limit saturated fat to < 7% of calories & cholesterol to < 200 mg

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21
Q

Vitamin and Mineral Supplements for prevention of nutritional deficiencies

A

A varied diet provides all vitamins & minerals needed
AAP recommends supplements for certain groups of children:
-From deprived families
-With anorexia, poor appetites or poor diets, or a -dietary program for wt mgmt
-Who consume only a few types of foods
-Vegetarians without dairy products
Vitamin D deficiency
-consequences: rickets

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22
Q

Dietary and PA guidelines for Toddlers and Preschoolers

A

Dietary guidelines

  • Offer a variety of foods, limiting foods high in fat & sugar
  • No specific # minutes of PA for kids 2-5, active play several times each day, developmentally appropriate and fun with variety
  • 60 minutes of vigorous physical activity each day (6-17 year olds)
  • MyPlate developed by the USDA for young children
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23
Q

Recommended v. Actual Intake for Toddlers and Preschoolers

A

Recommended
-Iron- meats, fortified cereal, dried beans and peas
-Fiber- fruits, vegetables, whole grains
-Fat- follow food guide pyramid
-Calcium- dairy, canned fish with soft bones, green -leafy vegetables, calcium-fortified beverages
-Fluids- beverages, foods and sips of water
Dietary intake of children ages 2-5
-Energy exceeded by 31%
-Consistently low “mean” intakes of zinc, folic acid, Vit D and Vit E
-Total fat is 31% of calories
-Sodium intake is 2150-2400 mg (rec 2300 mg)

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24
Q

Nutrition Intervention for Risk Reduction for T/P

A
Model program
Bright Futures in Practice: Nutrition
-Public food and nutrition programs
WIC
-WIC’s Farmers’ Market Nutrition Program
-Head Start and Early Head Start
*Breakfast, lunch and snack
*Monthly nutrition information
*Funding from USDA and meals in compliance with federal requirements
-Supplemental Nutrition Assistance Program (formerly Food Stamps)
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25
Q

Definition of Middle Childhood

A

between the age of 5-10

also part of school-age

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26
Q

Definition of pre-adolescence

A

for girls: ages 9-11
for boys: ages 10-12
also part of school aged

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27
Q

How is tanner staging determined

A

girls: breast buds and pubic hair
interval between breast budding and menarche is usually 2.25 years but can be as long as 6 years
boys: testes size and pubic hair

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28
Q

Normal growth development in childhood

A

Measurement techniques

  • Growth velocity will slow down during the school-age years
  • Should continue to monitor growth periodically
  • Weight and height should be plotted on the appropriate growth chart (charts for boys and girls)
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29
Q

Physiological development of school-aged children

A
  • Muscular strength, motor coordination, & stamina increase
  • In early childhood, body fat reaches a minimum then increases in preparation for adolescent growth spurt
  • Boys have more lean tissue than girls
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30
Q

Psychosocial issues in school-aged children

A
  • caregiver/peer interactions
  • body image
  • media influence
  • dieting behaviors (parents/peers)
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31
Q

Body image and excessive dieting in school-aged children

A
  • mother’s concern of her own weight may increase her influence over her daughter’s food intake
  • young girls are preoccupied with weight & body size at an early age
  • the normal increase in adiposity at this age may be interpreted as the beginning of obesity
  • imposing control & restrictions of “forbidden foods” may increase the desire and intake of those foods
32
Q

Common Nutritional Problems in school-aged children

A
  1. iron deficiency
    - less common in children then in toddlers (although rates are low they are still above the 2010 national health objectives)
    - prevention: encourage iron rich foods (meat, poultry, and fortified cereals & vitamin C rich foods to help absorption)
  2. Dental Caries
    - seen in half of children aged 6-8
    - prevention: limit sugar snacks, provide fluoride, choose fruits, veg, and grains, regular meal times and snacks, brush teeth after meals
33
Q

Characteristics of Overweight Children

A

compared to normal weight peers, overweight children

  • are taller
  • have advanced bones ages
  • experience earlier sexual maturity
  • look older
  • are at higher risk for obesity-related chronic diseases
34
Q

Nutrition and Prevention of CVD School-Aged Children

A
  • acceptable range for fat is 25%-35% of energy for ages 4-18
  • include sources of linoleic (omega-6) and alpha linolenic (omega-3) fatty acids
  • limit saturated fats, cholesterol, and trans fat
  • increase soluble fibers, maintain weight, include ample PA
  • diet should emphasize: F&V, low-fat dairy, whole grains, seeds, nuts, fish, & lean meats
35
Q

Dietary Recommendations for school aged children

A

iron: eat iron rich food
fiber: increase fresh F&V, whole grains and cereals
fat: decrease sat fat and trans fats
calcium & vit D: bone formation occurs during puberty, include dairy products and calcium fortified foods, vit D exposure to sunlight; if lactose intolerant: do not completely eliminate dairy products-decrease only to the point of tolerance

36
Q

Recommended versus Actual Intake in school-aged children

A

sat fat: intake is 12.6% of calories (recommended <7%)
total fat: intake excessive in AA b&g and Mexican American girls
caffeine: increasing because of soft drink consumption
fast food: 30.3% of children consume fast food each day

37
Q

Puberty

A

the time frame during which the body matures from that of a child to a young adult

38
Q

thelarche

A

breast bud development

39
Q

adrenarche

A

in males and females

the onset of androgen dependent signs of puberty (pubic hair, axillary hair, pimples)

40
Q

menarche

A

onset of menses

by menarche growth velocity almost stops

41
Q

What is the peak linear growth velocity for girls and boys

A

girls: growth spurt peaks at 12
boys: growth spurt peaks at 14

42
Q

What hormones are involved in puberty

A

gnrh
LH, FSH
Androgen

43
Q

What is bone age?

A

bone age is based on nutrients available more fat stores tells bone to calcify faster, bone age predicts how tall and matches puberty
Almost half of adult peak bone mass is accrued during adolescence
chronological age and bone age do not necessarily match

44
Q

What are modifiable and non modifiable determinants of puberty

A
nonmodifiable:
-genetics
-race/ethnicity
-SES
modifiable
-nutritional status (nutritional quality/energy adjusted intake)
-PA level
-energy balance
-general health
-geographic location
-adiposity (adipose tissue aromatize your homes and can use higher ammounts of estrogen and testosterone- aromotize=acts like an endorcrine organ)
-leptin
45
Q

Specific micro/macro nutrient concerns in pre/adolescence

A
iron
Calcium and Vit D: especially the role of soda and risk of osteopenia and osteoporosis (high phosphorous)
sugar
salt 
fat
fiber
46
Q

Anorexia Nervosa (AN)

A
  1. refusal to maintain body weight at or above a minimally normal weight for age and height (weight loss that exceed maintenance at a body weight 85% of that expected)
  2. Intense fear of gaining weight or becoming fat, even though they are underweight
  3. Disturbance in the way in which one’s body weight or shape is experienced, undue influence of body weight or shape on self-evaluation or denial of the seriousness of current body weight
47
Q

Types of AN

A

restricting type: no regular bingeing or purging (self-induced vomiting or use of laxatives or diuretics, enemas)
binge eating/purging type: regular bingeing/ or purging behavior

48
Q

Associated Diagnoses with AN

A

comorbidities include depression, anxiety or OCD

49
Q

Bulimia Nervosa

A
  1. recurrent episodes of binge-eating characterized by a. eating in discrete period of time, an amount of food that is definitely larger than most people would eat in a similar period of time and under a similar circumstance and b. a sense of lack of control over eating during the episode
  2. recurrent inappropriate compensatory behaviors to prevent weight gain such as self induced vomiting, misuse of laxatives, diuretics, enemas, or other medications, fasting or excessive exercise
  3. The binge-eating and inappropriate compensatory behaviors occure as least 1x/week for 3 months
  4. The disturbance does not occur exclusively during an episode of AN
50
Q

Associated Diagnoses with BN

A
impulsive behaviors:
-substance abuse
-self-mutilation/self-hard
-sexual promiscuity
-stealing
Depression
History of Sexual Trauma
51
Q

Binge-Eating Disorder

A
  1. Recurrent episodes of binging
  2. associated with 3 of the following:
    - eating faster than normal
    - eating until uncomfortably full
    - eating large amounts when not hungry
    - eating alone because of guilt or embarrassment
    - feeling disgusted/depressed/guilty
    - experiencing marked distress
    - on average at least 2x week for 6 months
  3. No inappropriate compensatory behaviors
52
Q

Treatment for Eating disorders

A
  1. Collaborative Outpatient Care
    - multidisciplinary team
    - medical stabilization and nutrition rehabilitation are the most important determinants of short term outcomes
    - mental health therapy
  2. Day Treatment Programs
    - intermediate level of care for patients who require more than outpatient but less than 24hr
    - used to prevent hospitalization or as a step down
    - 8-10 hours of care (meals, therapy, groups)
  3. Hospital-Based treatment
    - less common
    - more likely for AN than BN
53
Q

What are nutritional complications in Eating Disorders

A
1. Osteopenia
contributing factors:
-inadequate nutrition (Ca and Vit D)
-low body weight
-amenorrhea (low estrogen)
-elevated stress response (cortisol)
-fracture risk is 3x greater
2. hypercarotenmia (high vit A)
-yellowish/orange skin
3. Deficiencies of thiamin, riboflavin, and vit B6, contributes to cognitive dysfunction
54
Q

Etiology of eating disorders

A
  1. Psychodevelopmental factors:
    - bodily changes
    - life transitions
    - sexuality issues
  2. Sociocultural Factors:
    - peers
    - media
    - lifestyle
    - family values
    - abuse
  3. Neurochemical or genetic factors
    - brain chemistries
    - DNA
    - Personality structure
55
Q

Predisposing Precipitating and Perpetuating factors of eating disorders

A
  1. Predisposing factors
    - dieting
    - genetics (family history of Eating Disorders, substance abuse, depression)
    - individual (age, sex, personality, poor self-esteem, occupation)
    - familial (family dynamics, conflict skills, boundaries)
    - societal (pursuit of thinness, role confusion, dieting as a solution, weight reduction as salvation)
  2. Precipitating factors: triggers
    - major life changes (adolescence, marraige)
    - physical illness (cystic fibrosis, type I diabetes)
    - trauma (loss, grief, bullying)
  3. Perpetuating factos (keeps behaviors going)
    - reinforcing properties of weight loss
    - binge purge cycle
    - eating disorder as identity
    - starvation syndrome
56
Q

How were the BMI percentiles developed?

What do the percentiles indicate?

A

NHES II/III 1960s
NHANES I/II 1970s
NHANES III 1988-94
(Excluding ages >6)

BMI (sex & age specific)
Terminology
Current: 
 Overweight 
	(>85% , z-score >1.44)
 Obese 
	(>95%, z-score >1.96)
57
Q

Adiposity Rebound

A

the normal increase in body mass index that occurs after BMI declines to its lowest point at 6 years of age.

58
Q

Possible Factors affecting Adiposity rebound

A
possible mechanisms
-normal /low BMI before rebound
-rapid infant growth
-high protein low-fat intake?
* leads to early adiposity rebound 
which has the following affects
-increase is body fat (weight gain after rebound is 2x more fat)
-central adiposity
-increasing bone age
-early menarche
NOT affected by
-birth weight & length
-placental weight
-gestational age
59
Q

Social ecological model and influence of obesity

A
Individual: 
-food and beverage intake
-PA 
-energy intake and energy expenditure on not in balance
-genetics
-psychosocial
Interpersonal
-home & family
-school
-childcare
-physicians 
Community
-built environment
-restaurants
-supermarkets
-park
Society
-government
-agriculture
-media/advertising
-culture/social norms
60
Q

Intragenerational propagation of obesity

A

Adult Obesity–> leads to Obese Pregnancy (abnormal metabolic environment) (how much weight you gain during pregnancy)–> fetal/neonatal obesity –> childhood obesity (decrease insulin sensitivity) which increases the risk for adult obesity

61
Q

Individual risk factors for childhood obesity

A

Sleep
-for every additional hour of sleep for a 9 year old, 40% less likely to be an obese 12 year old
-possible mechanisms: (increase food intake when up later, hormonal changes leptin & ghrelin, decreased energy for PA)
Screen Time
-increase risk for obesity
-stimulates somatic arousal and attenuation of melatonin
-possible mechanisms (displacing PA, mindless eating in front of TV, highly palatable food advertising)

62
Q

Interpersonal risk factors for child obesity

A
  1. Family (parenting styles)
  2. School PE
    - only 18% of HS students had PE
    - recess replaced with class time
    - during PE students spent a small amount of time in vigorous activity
  3. School Food
    - high prevalence of competitive foods
63
Q

Parenting Styles and Obesity Risk

A

Authoritative:
-establish rules and guidelines that their children are expected to follow. However, this parenting style is much more democratic. Authoritative parents are responsive to their children and willing to listen to questions. When children fail to meet the expectations, these parents are more nurturing and forgiving rather than punishing. Baumrind suggests that these parents “monitor and impart clear standards for their children’s conduct. They are assertive, but not intrusive and restrictive. Their disciplinary methods are supportive, rather than punitive. They want their children to be assertive as well as socially responsible, and self-regulated as well as cooperative”
-characterized by high responsiveness and high demandingness
-lowest risk for obesity
Authoritatian:
- children are expected to follow the strict rules established by the parents. Failure to follow such rules usually results in punishment. Authoritarian parents fail to explain the reasoning behind these rules. If asked to explain, the parent might simply reply, “Because I said so.” These parents have high demands, but are not responsive to their children. According to Baumrind, these parents “are obedience- and status-oriented, and expect their orders to be obeyed without explanation
-characterized by high demanding and low responsiveness
-5-fold increase in obesity risk
permissive:
characterized by high responsiveness and low demanding
-2-fold increase in risk

64
Q

Health Consequences of Obesity

A
Gastrointestinal
-non alcoholic fatty liver disease
Endocrine/metabolic
-hyperandrogenism
-PCOS
-Type II diabetes
-impaired glucose tolerance
-insulin resistance
-metabolic syndrome and its components
Pulmonary
-obstructive sleep apnea
-obesity hypoventilation syndrome
-asthma
Adult Obesity
65
Q

How do nutrient recommendations change with increasing age? why?

A
Increased need:
calcium
vitamin D
vitamin B12
vitamin B6
protein
decreased need:
calories
vitamin A (older adults are more prone to vit A toxicity liver stores of vit A increase with age)
66
Q

Sarcopenia of Aging

A
  • a paucity of muscle
  • defined as the decrease in lean body mass
  • often associated with noncomitant increase in fat mass
  • total body weight may not change
  • much thicker layer of subcutaneous fat and a smaller muscle compartment, the fat infiltrates into muscle decrease of functioning of muscles in lower extremities
67
Q

Consequences of Sarcopenia

A
  • decreased resting energy expenditure
  • decreased insulin sensitivity
  • diminished muscle strength
  • increased risk of physical disability
  • increased risk of mortality
68
Q

Treatment of Sarcopenia

A

-Hormonal therapy (testosterone, DHEA, estrogen, growth hormone)
-exercise interventions
strength training & supplement trial with 90 yr olds
*exercise group improved in muscle strength by 100%, so did ex & supp but not statistically different from ex only, supplement only and control group did not improve at all
-nutritional supplements

69
Q

Prevalence of malnutrition in the elderly population

A

Community-dwellings: 3-11%
Nursing home residents: 17-65%
Hospital inpatients: 15-40%

70
Q

Prevalence of obesity and comorbidity & mortality

A

obesity increases with age

4/5 leading causes of death are associated with obesity (heart disease, cancer, stroke, diabetes)

71
Q

Nutrition Screening Initiative

A

A tool to detect under/over nutrition in a population of elderly
Can be administered in 5 minutes
Been shown to be very predictive of identifying individuals of under/overnutrition

72
Q

Atrophic gastritis -B12 deficiency

A
  • lining of stomach gets thinner as we get older and the decrease of the secretions becomes fewer
  • results in decreased secretion of hydrochloric acid, pepsin and to a modest degree intrinsic factor
  • in order for b12 to be absorbed you need acid and pepsin in the stomach B12 is bonded to peptides and the peptide bond needs to be broken in the stomach
  • people with atrophic gastritis cannot absorb b12
  • elderly are also more sensitive to b12 deficiency at lower-normal levels of b12
73
Q

Causes and Treatment of vitamin D and calcium deficiency in the elderly

A

Causes:
-Increased risk because of lack of sunlight
-habitually low dietary intake
-impaired synthesis in senile skin
Treatment:
-weight-bearing and muscle-strengthening exercise >3X/wk for all adults
-pro-active strategies to prevent falls for at-risk individuals
-avoidance of tobacco use and >2 alcoholic drinks/d

74
Q

what are the consequences of not treating a b12 deficiency?

A

It takes years to develop a B12 deficiency, but once developed the neurological symptoms are irreversible. Symptoms include deterioration of mental function change in personality and loss of physical coordination

75
Q

Tests and Treatment for B12 deficiency

A

Tests: MMA (methylamlonic acid)/homecysteine levels)
Treatment
Plasma B12 remains the ‘first line’ test
a level of >350 pg/mL (258 pmol/L) excludes deficiency
a level of <150 pg/mL (110 pmol/L) should be considered diagnostic of deficiency
levels between 150 and 350 should prompt a MMA level. If MMA is substantially elevated*, the the patient should be considered to have B12 deficiency
Those individuals whose B12 is 150-250 and whose MMA is normal should be monitored occasionally for slow transition to a frank deficiency