nurs 360 exam 3 Flashcards

1
Q

What are the complications for someone following a radical neck dissection with flap reconstruction?

A

➱ Drop/dysfunction and poor cosmesis (visible neck depression), high mortality

complications with flap –
➡Hemorrhage, hematoma formation, and rupture of the intrajugular vein or carotid artery
➡Chyle Leak (lymphatic leak in the thoracic duct)
➡Neurologic complications: stroke or nerve injury
➡Nutritional status (they can’t eat- must get their nutrition other ways such as NG tube)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What are the assessment findings for someone following a radical neck dissection with flap reconstruction?

A

➡ Postoperatively: patient is monitored for hemorrhage (raised site), wound infection (leakage [lymphatic fluid, saliva, gastric contents]), and altered respiratory status (airway, breathing, circulation)
★Swelling
★Low O2 sats
★ABC’s; watch for cold and blue flap
➡As they heal: Assess range of motion and determine if there has been a decrease due to nerve or muscle damage. Assess nutritional support.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What are the nursing interventions and nursing considerations for providing nasogastric nutrition?

A

➡ Management of tube feeding
Insert tube through the nose to the stomach and verify using x-ray and pH testing
✷pH 0-4 gastric
✷pH greater than 4 = in the intestine
✷pH greater than or equal to 5.5 =lungs
➡ Observe the color-gastric aspirate usually cloudy and green but may also be off white, tan, bloody, or brown
➡Flush tube with water or normal saline after feeding and before and after medication are administered through the tube

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What would you educate on the client prior to insertion of a nasogastric tube?

A

Education of tube feeding

➡Signs and symptoms of dumping syndrome: fullness, nausea, osmotic diarrhea, tachycardia,
➡Prevention: give slow the feeding and room temperature feeding, decrease the amount of water you are distilling
➡NG education: patient will gag until the tube is passed the gag reflex

➡Every 4 hours on your tube feeds, 30 mL flush

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what assessment findings should you be alert for in patients receiving tube feedings?

A

➡ tube placement, pt’s position (head elevated 30 degrees
➡ signs of dehydration (dry mucous membranes, thirst, decreases urine output)
➡ signs of infection (to avoid infection, replace any formula given by an open system every 4 to 8 hours with fresh formula; change tube feeding container every 24 hrs)
➡signs of complications ( if suspected, check gastric residual vL before each feeding or in the case of cont. feedings every 4 hours)
➡intake and output
➡weekly weights

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

what is continuous feeding

A

slow infusion over long periods, slow feeds usually better tolerated for critically ill, patients who are aspiration risks, patients at risk for for intolerance and small bowel feedings.

When small-bore feeding tubes for continuous infusion are irrigated after administration of medications, a 20-mL or larger syringe is used because the pressure generated by smaller syringes could rupture the tube.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what is intermittent infusion?

A

➡ Intermittent - Drip
✷Bolus
✷Gravity

Give by bolus; use 20 ml or larger syringe to flush small bore tubes (less pressure, less likely to rupture tubing; The feeding is paused, and the tube is flushed with at least 15 mL of water before and at least 15 mL of water after medication administration (30 mL total). Each medication should be prepared and administered separately, with a 15-mL flush provided between medications.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

difference between open and closed feeding system

A
☆Open
   ➱needs mixing
   ➱Change tubing Q24h
   ➱Can be used for bolus, intermittent or continuous 
   ➱feedings, push, gravity or pump
   ➱Hang time is usually 4-8 hours
☆Closed
   ➱pre-filled, premixed
   ➱needs a pump
   ➱can hang for 24 hours
   ➱less risk of infection
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is dumping syndrome? this is a complication of…

A

this is a complication of enteral nutrition

Formula infused into the small intestine too quickly or formula bypasses the stomach too readily into the small intestine; expansion of the intestinal wall; this leads to bloating, cramping, diarrhea, dizziness, diaphoresis, and weakness

Dumping syndrome- feelings of fullness, nausea, cramping, Disease, diaphoresis, diarrhea
Leads to dehydration, hypotension and tachycardia
Small intestine may adapt better is osmolality is increased slowly.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

G tube vs. J tube

A

➱J tubes good for those who can’t have anything in the stomach but still get food into the gut
✰Typically does not last as long
✰Endoscopically placed gastrojejunal tube in the jejunum for the purpose of administration of nutrition, fluids and medications.

➱G tubes someone can have a G tube for the rest of their life
✰Opening created in stomach, preferred over nasogastric for long term maintenance, (use greater than 4-6 weeks)
✰Preferred in comatose patients because the gastroesophageal sphincter remains closed
Percutaneous endoscopic gastrostomy (PEG) tube

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are the potential problems associated with g tubes and j tubes?

A

Potential problems

➱Wound infection, cellulitis, and leakage
➱GI bleeding
➱Premature dislodgment of the tube
➱Tube obstruction/clogging

☆Monitoring and managing potential complications by checking for drainage, comitus, and stool Aspiration is a potential risk with tube dislodgement, especially with nasally inserted tubes.
☆Skin care. It is normal to see scant serous drainage a few days post insertion. After approx 1 wk, the site can be cleaned 2x a week with soap and water and left open to air. Candida may appear in warm moist areas of the body.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Complications of enteral feedings

A

✰ Mechanical tube displacement➱ replace rube
✰ Aspiration➱elevate head of bead, check residual before feeding for intermittent check residual every 4 hour for continuous.
✰ GI (v/d/n)➱ decrease feeding rate, change formula, administer at room temperature
✰ Metabolic hyperglycemia ➱ monitor glucose, osmolality , reduce infusion rate, give insulin if needed
✰ dehydration➱ flush with water or normal saline
✰ formula drug interactions➱ check compatibility, flush tubing prior to and after medication

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Nasogastric tube management?

A

every four hours on your tube feeds, 30mL flush

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What is gastritis? What are the symptoms?

A

➱inflammation of the gastric or stomach mucosa

➱Symptoms:
   ✰Acute--> possible signs of shock 
-anorexia
-epigastric (rapid onset of symptoms)
-hematemesis 
-hiccups 
-melena or hematochezia 
-N/V

✰Chronic–> anemia & fatigue

  • belching
  • early satiety
  • intolerance of spicy or fatty foods
  • N/V
  • pyrosis
  • sour taste in mouth
  • vague epigastric
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What are the nursing interventions of gastritis?

A

➱ANITBITOICS, H2, PROTON PUMP ,

Acute gastritis is usually self-limiting, client may have decreased appetite, instruct to avoid EtOH, and food, may need IV fluids, NG tube, intubation, antacids, H2 blockers, PPI’s,
➱Surgery may be needed in necrosis or gastric outlet obstruction
➱Chronic gastritis is managed by diet modification (decrease caffeine), rest, reduce stress, avoid EtOH, NSAIDS, antibiotic for H. Pylori, quit smoking

Nursing interventions:
★No food or fluids by mouth. With IV therapy, monitor I&Os and serum electrolyte values. Introduce solid food as soon as possible, but report any symptoms that indicate repeat episodes of gastritis. Discourage intake of caffeinated beverages, alcohol, and smoking. Administer any pharmacotherapy prescribed.
★Promote fluid balance- monitor daily I&Os, assess electrolyte values every 24 hours. Watch for signs of hemorrhagic gastritis (especially vomiting/defecation of blood)
★Relieve pain- avoid foods or beverages that irritate gastric mucosa. Regularly assess levels of pain/discomfort.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

H2 blocker medications examples, MOA, and AE

A

reduce the amount of acid produced by the cells in the lining of the stomach

cimetidine, famotidine, nizatidine and ranitidine

side effects are cholinergic effects: constipation, dry mouth, dry skin, ringing int he ears, runny nose

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

How would you manage a GI bleed associated with gastritis and assess if it is getting better or worse?

A

Erosive gastritis my cause bleeding which may manifest as blood in vomit or as melena (black, tarry stools) or hematochezia (bright red, bloody stools

The nurse must always be alert to any indicators of hemorrhagic gastritis, which include hematemesis (vomiting of blood), tachycardia, and hypotension. All stools should be examined for the presence of frank or occult bleeding. If these occur, the primary provider should be notified, and the patient’s vital signs are monitored as the patient’s condition warrants. Guidelines for managing upper GI tract bleeding are discussed later in this chapter.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Why do we worry about pernicious anemia in clients with gastrointestinal disorders? What are the signs and symptoms of pernicious anemia? How is it treated?

A

Pernicious anemia is a decrease in red blood cells that occurs when the intestines cannot properly absorb vitamin B12. Vitamin B12 deficiency can occur in patients with disorders such as inflammatory bowel disease, or in patients who have had GI surgery such as ileal resection, bariatric surgery, or gastrectomy

signs and symptoms: smooth, sore, red tongue and mild diarrhea, extremely pale, particularly in the mucous membranes, confused; more often, paresthesias in the extremities (particularly numbness and tingling in the feet and lower legs). They may have difficulty maintaining their balance because of damage to the spinal cord, and they also lose position sense (proprioception)

treatment: b12 shots once a month

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What are the symptoms of peptic ulcer disease? What are the complications? What is perforation and what are the signs and symptoms?

A

Peptic ulcer disease: excavation formed in the mucosal wall, caused by erosion

Symptoms: 
dull, gnawing pain or burning sensation in the mid epigastrium or back
Pain after eating
Pyrosis
Vomiting
Constipation or diarrhea
Bleeding 
Symptoms often accompanied by sour eructation (burping) 
Complications: 
Hemorrhage
Perforation
Penetration
Gastric outlet obstruction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

What are the symptoms of peptic ulcer disease? What are the complications?

A

Peptic ulcer disease: excavation formed in the mucosal wall, caused by erosion

Symptoms: 
dull, gnawing pain or burning sensation in the mid epigastrium or back
Pain after eating
Pyrosis
Vomiting
Constipation or diarrhea
Bleeding 
Symptoms often accompanied by sour eructation (burping) 
Complications: 
Hemorrhage
Perforation
Penetration
Gastric outlet obstruction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

What is perforation and what are the signs and symptoms?

A

–Perforation
➱Signs and symptoms include:
-Sudden, severe upper abdominal pain (persistent and increasing in intensity); pain may be referred to the shoulders, especially the right shoulder, because of irritation of the phrenic nerve in the diaphragm
-Vomiting
-Collapse (fainting)
-Extremely tender and rigid (boardlike) abdomen
-Hypotension and tachycardia, indicating shock

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

How is peptic ulcer disease with h. pylori treated?

A

Management goal - eradicate H pylori, manage gastric acidity - antibiotics , proton pump inhibitor (“zoles”), and H2 blockers, completing treatment is crucial.
used for 10-14 days.

Maintenance H2 Blockers usually for 1 year post healing.

Combination of antibiotics, proton pump inhibitors, and sometimes bismuth salts that suppress or eradicate H. pylori
Recommended combination drug therapy is typically prescribed for 10-14 days and may include triple therapy with two antibiotics, plus a proton pump inhibitor or quadruple therapy with two antibiotics plus a proton pump inhibitor and bismuth salts
Research currently being conducted for vaccine against H. pylori
Avoid use of NSAIDS
Maintenance dosages of H2 blockers are usually recommended for 1 year
Look at most common antibiotics; know side effects of flagyl

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

What are preventative measures for “dumping syndrome”?

A
Dumping syndrome is the rapid passage of food to stomach, causing diaphoresis, diarrhea, hypotension; usually occurs 10-15 minutes after eating 
   ➱ restrict fluids w/ meals 
   ➱ avoid stress after eating 
   ➱eat smaller, frequent meals
   ➱lie down after eating
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Metronidazole aka flagyl

A

Dizziness, headache, stomach upset, nausea, vomiting, loss of appetite, diarrhea, constipation, or metallic taste in your mouth may occur

education: do not drink alcoholl

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

What is a “stress ulcer”? Why do they occur? How are they prevented?

A

Stress ulcer occurs after physiological stress. May occur due to burn injuries, multiple organ disorders,

Medications: prophylactic H2 Blockers or PPIs (end in “zoles”)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Proton-pump inhibitor

A

class of medications that cause a profound and prolonged reduction of stomach acid production

examples: 
omeprazole (Prilosec, Prilosec OTC, Zegerid)
lansoprazole (Prevacid)
pantoprazole (Protonix)
rabeprazole (Aciphex)
esomeprazole (Nexium)
dexlansoprazole 

ae:
In general, PPIs are believed to have few adverse effects, as they are generally well tolerated. Patients have experienced few minor side effects of short-term PPI use, such as headache, rash, dizziness, and gastrointestinal symptoms including nausea, abdominal pain, flatulence, constipation, and diarrhea

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

Describe the care for a client who is status post esophagojejunostomy for gastric cancer. What do you monitor for?

A

The patient undergoing gastric surgery may experience complications, including hemorrhage, dumping syndrome, bile reflux, and gastric outlet obstruction. Postoperative bleeding from the surgical site is a common complication of gastric surgery. Bleeding may be severe (hemorrhage) and manifest as vomiting large amounts of bright red blood, which may result in hemorrhagic shock

Monitor for dumping syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Anti-ulcer agents/ proton pump inhibitors (PPI)

A

Omeprazole, Pantoprazole

nursing considerations: Monitor CBC, electrolytes.
Avoid alcohol, NSAIDS, aspirin, and foods that may cause irritation

side effects: Abdominal pain, vitamin b 12 deficiency, systemic lupus erythematosus, fatigue, headache
Clostridioides difficile-associated diarrhea (CDAD), Hypersensitivity reactions, vitamin B12 deficiency

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

glucagon

A

used before endoscopy procedures as an aid during radiologic exams to temporarily inhibit GI movement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Metronidazole

A

antibiotic

used for peptic ulcer disease caused by H. pylori. Stops the growth of the bacteria

nursing considerations: No alc, cannot be pregnant

AE: Seizures, dizziness, headache, aseptic meningitis, encephalopathy, psychosis, stevens-johnson syndrome, abdominal pain, anorexia, nausea,

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

Clarithromycin

A

anti-infectives agents

used for ulcers; often administered with metronidazole and a PPI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

Sucralfate

A

anti-ulcer agent
used in the prevention of duodenal ulcers

Take on empty stomach, separate 30 mins of antacids. Causes a decrease in abdominal pain. Keep taking it even if you are feeling better

AE: constipation

33
Q

Loperamide

A

anti-diarrheal

diarrheal treatment

avoid using other CNA depressant while on this med. DONT DRINK. watch liver function

AE: cardia arrest, torsades de point

34
Q

Psyllium

A

laxative

AE: Bronchospasm, cramps, intestinal or esophageal obstruction, N/V

35
Q

docusate

A

laxative-stool softener
used for constpiation

short term use: DO NOT USE WITH MINERAL OIL

36
Q

octretide

A

first line treatment for esophageal varices
-given before endoscopy in patients with variceal bleeding

MOA: -Suppresses gastrin levels (PUD)
-Causes selective splanchnic vasoconstriction by inhibiting glucagon release and used mainly in the management of active hemorrhage (esophageal varices)

DO NOT USE IF PREG

AE: -cholelithiasis

  • ileus
  • pancreatitis
  • hypoglycemia
  • abdominal cramping
37
Q

Cimetidine

A

Histamine 2 agonist

GI ulcers and GERD
Anti-ulcer agent, Histamine 2 agonist
Should be used 6-8 weeks for complete peptic ulcer healing, patients who are at higher risk require a maintenance dose for 1 year.

confusion, dizziness, drowsiness, hallucinations, headache, ARRHYTHMIAS, constipation, diarrhea, nausea, gynecomastia, AGRANULOCYTOSIS, APLASTIC ANEMIA

38
Q

Describe the teaching for clients following gastrectomy. How would you educate them on optimizing their nutrition?

A

injection of vitamin B12 will be required for life, because intrinsic factor, secreted by parietal cells in the stomach, binds to vitamin B12 so that it may be absorbed in the ileum. This deficiency in vitamin B12 metabolism can result in decreased production of red blood cells, or pernicious anemia;

nurse assesses for signs of dehydration (thirst, dry mucous membranes, poor skin turgor, tachycardia, decreased urine output) and reviews the results of daily laboratory studies to note any metabolic abnormalities (sodium, potassium, glucose, BUN). Antiemetic agents are given as prescribed.

39
Q

Constipation

A

related to inadequate intake of fluid and bulk

nursing interventions
➱ record intake and output, color and consistent of stool and frequency of bowel movements to form the basis of an effective treatment plan
➱promote ample fluid intake
➱encourage the pt to walk and exercise to stimulate intestinal activity
➱discourage routine use of laxatives

40
Q

diarrhea

A

related to malabsorption, inflammation, or irritation of the bowel

nursing interventions
➱assess level of dehydration and electrolyte status
➱monitor the pt’s weight daily to detect fluid loss or retention
➱test stool for occult blood
➱assess fecal impaction. liquid stool may seep around an impaction

41
Q

bowel incontinence

A

related to neuromuscular involvement

➱establish a bowel schedule for the pt-1/2 hour after a meal is desirable for active peristalsis

42
Q

ileostomy vs. colostomy

A

➱ Ileostomy: widespread colonic obstruction may require near-total removal of the colon and rectum. SMALL INTESTINE to the surface of the abdomen, specifically the ileum (which is the last part of the small intestine before it opens into the large intestine). ALWAYS LIQUID STOOL. Huge risk for skin breakdown and increased risk of dehydration and electrolyte imbalance.

➱ Colostomy: an opening created to bring the LARGE INTESTINE to the surface of the abdomen (the large intestines are also called the colon). the causes for the surgery are due to rectal cancer.

Ascending: liquid stool
Transverse: lose to partly formed stool
Descending/Sigmoid: similar to normal consistency

stomas are pink and beefy red

43
Q

What diseases are related to malabsorption syndrome? How are they treated?

A

Sphincter defects:
Zollinger-Ellison syndrome, AIDS, parasites, Clostridiumdifficile

CELIAC: avoid gluten
lACTOSE INTOLERANCE: lack of lactase, avoid lactose

44
Q

differentiate between Ulcerative colitis and Crohn’s Disease

A

ulcerative colitis→ affects the mucosa and submucosa of the colon. Begins at the rectum and sigmoid colon and extends upward into the sigmoid colon.
★The stool typically contains pus and mucus and recurrent bloody diarrhea. Assess the pt for spastic rectum and anus, abdominal pain, anorexia, fever, toxic megacolon

Crohn’s disease→ can affect any part of the GI tract. Lymph nodes enlarge and lymph flow in the submucosa is blocked. The lymphatic obstruction causes edema, mucosal ulceration, fissures, abscesses.. Oval, elevated lymph follicles are called Peyer’s patches-develop on the lining of the small intestine. AS the disease progresses, the fibrosis thickens the bowel walls.
★right lower abdominal pain, cramping tenderness, n/d → chronic cases: diarrhea 4-6x a day
★ increased wbc count, increased ESR, hypokalemia, hypocalcemia, and decreased hemoglobin

45
Q

Know acute abdomen (ex: appendicitis)

A

Appendicitis occurs when the appendix becomes inflamed. Mucosal ulceration triggers inflammation, which temporarily obstructs the appendix. The obstruction blocks mucus outflow. Pressure in the now distended appendix increases. Bacteria multiply and inflammation and pressure continue to increase, restricting blood flow to the organ= abdominal pain.

☆ an appendectomy is the only effective treatment for appendicitis. If peritonitis develops, treatment involves GI intubation, parenteral replacement of fluids and electrolytes and administration of antibiotics.

46
Q

Parenteral nutrition, when to start, when it’s indicated, what to monitor

A

Complications: infection and hyperglycemia
Indicated when enteral nutrition is not an option, pt is malnourished, cannot tolerate orally

Nursing: Daily weights, I/Os, glucose, CBC,

monitor: glucose

47
Q

crohns disease

A

inflammatory conditions of any of the large or small intestine

assess
* severe abdominal pain, cramp in the lower right quadrant, chronic diarrhea,
* mucus, pus, FAT in stools
* increased temp
* decreased weight
diagnostic test-upper GI-barium enema, colonoscopy

nursing management- diet-high protein, high calorie, low fat, and low fiber

48
Q

Ulcerative colitis

A

inflammation condition of the colon characterized by eroded areas od the mucous membrane nad tissues beneath it

assess

  • diarrhea 10-20 stools per day
  • blood, pus, mucus in stool; n pain
  • fecal incontinence
    • dehydration
  • weightloss
  • weakness, cachexia,
  • metabolic acidosis
49
Q

what causes acute abdomen

A

appendicitis

50
Q

GERD

A

gastroesophageal reflux disease aka ACID REFLUX
USE cimetidine

This is a chronic disease that occurs when stomach acid or bile flows into the food pipe and irritates the lining. Acid reflux and heartburn more than twice a week may indicate GERD.

Complications of GERD include dental erosion, ulceration in the pharynx and esophagus, laryngeal damage, esophageal strictures, adenocarcinoma and pulmonary complications
–pH testing, endoscopy or barium swallow

51
Q

which electrolyte is out of whack in diarrhea?

A

potassium

52
Q

what psyllium is (metamucil)

A

it is fiber

For constipation💩

53
Q

What causes gastric outlet syndrome?

A

Food stops going through stomach

Associated with ulcers, gastritis, stricture

54
Q

Percutaneous endoscopic gastrostomy (PEG) tube

nursing considerations

A

enteral nutrition
nursing management- x ray verified, pH aspirate

flush with water or normal saline

55
Q

Geriatric considerations with gallstones and how they may present differently

A

Surgical removal is more common

Appear as: septic shock, oliguria, hypotension, tachycardia, tachypnea, altered mental status

56
Q

how does someone with gallstones present?

A
Burping after meals
Indigestion
Severe upper right quadrant pain
N/V
May have no symptoms
abdominal pain, distension
57
Q

What causes gall stone? (Cholelithiasis)

A

Causes:An increased concentration of bile substances

  • Obesity
  • Pregnancy
  • Diabetes
  • Hypothyroidism
  • Liver cirrhosis
58
Q

gallbladder inflammation is called…

A

Cholecystitis

Inflammation of the gallbladder
Causes: bacteria
Nursing: NPO, antibiotics

59
Q

pancreatitis

A

Inflammation of the pancreas

Possible causes: tumors, cysts, ulcers, etc
clinical manifestations: N/V, fluid/electrolyte imbalances, fever, jaundice, hyperglycemia, weight loss, ascites
Nursing: NPO, PPIs, Antiacids, Treating fluid and electrolytes, monitor for infection, TPN, avoid alcohol

60
Q

pt has jaundice but no liver disease…

A

Jaundice without liver disease: hemolytic jaundice

Increased destruction of red blood cells
Fecal and urine uro-bilirubinuria is increased, no bilirubin in the urine
No symptoms, but predisposes to CNS effects and gallstones

61
Q

what causes jaundice?

A

bilirubin excess

62
Q

Priorities for pt’s with confusion

A

O2 and toxicity

63
Q

signs and symptoms of alcoholic cirrhosis

A
Cirrhosis
Complications: 
* Portal hypertension
 * Esophageal Varices
       --Throwing up blood, bloody stool, discomfort
Nursing 
->diet : High Carb High protein
->Reduce ascites: sodium/fluid restrictions, 
->Monitor fluid and electrolyte
->Daily weights
->Abdominal girth

Alcoholic Cirrhosis

  • Caused by alcoholism and poor nutrition
  • Confusion, fatigue, jaundice, bleeding, hypertension, varices, ascites
64
Q

Hepatic encephalopathy

A

*Life threatening disease caused by liver disease, hepatic failure

  • Ammonia is secreted into the brain and causes CNS depression
    • ->Can increase in GI bleeds
  • Clinical signs: mental changes, motor changes, asterixis,
  • Metronidazole

ENCEPHALOPATHY IS CAUSED BY AMMONIA

65
Q

care of patient undergoing pancreatitis

A

Review care of patient undergoing paracentesi: procedure that removes fluid (peritoneal fluid) from the abdomen through a slender needle.

  • Position: supine
  • monitor: monitors the respiratory status because large volumes of ascites can compress the thoracic cavity and inhibit adequate lung expansion. The nurse monitors serum ammonia, creatinine, and electrolyte levels to assess electrolyte balance, response to therapy, and indications of hepatic encephalopathy.
  • Post paracentesis circulatory function
    * Albumin infusions help to correct decreases in effective arterial blood volume that lead to sodium retention. The use of this colloid reduces the incidence of post paracentesis circulatory dysfunction with renal dysfunction, hyponatremia, and rapid reaccumulation of ascites associated with decreased effective arterial volume
66
Q

rebound ascites is treated with…

A

albumin

67
Q

What treats hepatic encephalopathy?

A

Administer lactulose, brings down ammonia by making the patient defecate
* Lactulose: Works by drawing water from your body into your colon, which softens stools and causes you to have more bowel movements.

68
Q

Nursing education pre and post for endoscopy

A

PRE_OP: NPO status, start an IV, no ibuprofen, consent, educate on throat numbness due to anesthesia

POST_OP: maintain NPO status until gag reflex and bowel sounds return
observe for vomiting, aspiration, respiratory distress

69
Q

what is done for TPN patients

A

total parenteral nutirtion

remember- aseptic routine and swallow studies for pt’s after a long period of time

70
Q

Know IBS-C and IBS-D: know expected drugs and treatments

A

Irritable bowel syndrome: marked by chronic symptoms of abdominal pain ,alternating constipation and diarrhea, excess flatulence,

constipation (classified as IBS-C)
Lubiprostone, a chloride channel regulator in the gut, can be prescribed for patients with IBS-C

diarrhea (classified as IBS-D)
antidiarrheal agents (e.g., loperamide
71
Q

Magenisum Hydroxide

A

laxative

used in constipation

72
Q

Cholecystitis

A

inflammation of the gallbladder

73
Q

Cholelithiasis

A

Calculi, or gallstones, usually form in the gallbladder from the solid constituents of bile; they vary greatly in size, shape, and composition

Abdominal x-ray, ultrasound, radionucleotide imaging, oral cholecystography, endoscopic retrograde cholangiopancreatography ERCP

Management: banding, TIPS, shunting, Meds: Octreotide,
vasopressin (do not use in patients with HX coronary artery disease), beta blocker (“-lol”), sclerotherapy,

74
Q

care for PEG tubes

A

Elective change of the balloon every 3-6 months to extend expected lifetime of tube
Tube normally lasts 1-2 years with proper care
Clean skin around site 1-3 times a day
Rotate tube completely each day
Flush tube before and after feeding
If clogged, unclog as soon as possible

75
Q

how someone with gallstones presents aka choleithiasis

A
Burping after meals
Indigestion
Severe upper right quadrant pain
N/V
May have no symptoms
abdominal pain, distension

Geriatric considerations with gallstones and how they may present differently
Surgical removal is more common
Appear as: septic shock, oliguria, hypotension, tachycardia, tachypnea, altered mental status

76
Q

what causes cholelithiasis

A
Causes:An increased concentration of bile substances
Obesity
Pregnancy
Diabetes
Hypothyroidism
Liver cirrhosis
77
Q

laparoscopic vs open chelecytsctomy

A

Most done laparoscopically

Gall bladder removed through small incision
Open cholecystectomy is done surgically with a larger incision and has a higher mortality rate, chance of infection, and impairs gas exchange due to the higher and larger incision.
Why might be one indicated over another
Laparoscopic cholecystectomy reduces surgical risk, length of recovery, and decreases abdominal pain post op.
Know what complications of each are
Laparoscopic
Bile duct injury
Bile peritonitis
Open
Drainage, NG tube, and suctioning may be needed immediately post op
Know what causes gallstones (Cholelithiasis)

78
Q

position after appendectomy

A

high fowler