nurs 360 exam 3 Flashcards

1
Q

What are the complications for someone following a radical neck dissection with flap reconstruction?

A

➱ Drop/dysfunction and poor cosmesis (visible neck depression), high mortality

complications with flap –
➡Hemorrhage, hematoma formation, and rupture of the intrajugular vein or carotid artery
➡Chyle Leak (lymphatic leak in the thoracic duct)
➡Neurologic complications: stroke or nerve injury
➡Nutritional status (they can’t eat- must get their nutrition other ways such as NG tube)

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2
Q

What are the assessment findings for someone following a radical neck dissection with flap reconstruction?

A

➡ Postoperatively: patient is monitored for hemorrhage (raised site), wound infection (leakage [lymphatic fluid, saliva, gastric contents]), and altered respiratory status (airway, breathing, circulation)
★Swelling
★Low O2 sats
★ABC’s; watch for cold and blue flap
➡As they heal: Assess range of motion and determine if there has been a decrease due to nerve or muscle damage. Assess nutritional support.

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3
Q

What are the nursing interventions and nursing considerations for providing nasogastric nutrition?

A

➡ Management of tube feeding
Insert tube through the nose to the stomach and verify using x-ray and pH testing
✷pH 0-4 gastric
✷pH greater than 4 = in the intestine
✷pH greater than or equal to 5.5 =lungs
➡ Observe the color-gastric aspirate usually cloudy and green but may also be off white, tan, bloody, or brown
➡Flush tube with water or normal saline after feeding and before and after medication are administered through the tube

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4
Q

What would you educate on the client prior to insertion of a nasogastric tube?

A

Education of tube feeding

➡Signs and symptoms of dumping syndrome: fullness, nausea, osmotic diarrhea, tachycardia,
➡Prevention: give slow the feeding and room temperature feeding, decrease the amount of water you are distilling
➡NG education: patient will gag until the tube is passed the gag reflex

➡Every 4 hours on your tube feeds, 30 mL flush

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5
Q

what assessment findings should you be alert for in patients receiving tube feedings?

A

➡ tube placement, pt’s position (head elevated 30 degrees
➡ signs of dehydration (dry mucous membranes, thirst, decreases urine output)
➡ signs of infection (to avoid infection, replace any formula given by an open system every 4 to 8 hours with fresh formula; change tube feeding container every 24 hrs)
➡signs of complications ( if suspected, check gastric residual vL before each feeding or in the case of cont. feedings every 4 hours)
➡intake and output
➡weekly weights

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6
Q

what is continuous feeding

A

slow infusion over long periods, slow feeds usually better tolerated for critically ill, patients who are aspiration risks, patients at risk for for intolerance and small bowel feedings.

When small-bore feeding tubes for continuous infusion are irrigated after administration of medications, a 20-mL or larger syringe is used because the pressure generated by smaller syringes could rupture the tube.

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7
Q

what is intermittent infusion?

A

➡ Intermittent - Drip
✷Bolus
✷Gravity

Give by bolus; use 20 ml or larger syringe to flush small bore tubes (less pressure, less likely to rupture tubing; The feeding is paused, and the tube is flushed with at least 15 mL of water before and at least 15 mL of water after medication administration (30 mL total). Each medication should be prepared and administered separately, with a 15-mL flush provided between medications.

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8
Q

difference between open and closed feeding system

A
☆Open
   ➱needs mixing
   ➱Change tubing Q24h
   ➱Can be used for bolus, intermittent or continuous 
   ➱feedings, push, gravity or pump
   ➱Hang time is usually 4-8 hours
☆Closed
   ➱pre-filled, premixed
   ➱needs a pump
   ➱can hang for 24 hours
   ➱less risk of infection
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9
Q

What is dumping syndrome? this is a complication of…

A

this is a complication of enteral nutrition

Formula infused into the small intestine too quickly or formula bypasses the stomach too readily into the small intestine; expansion of the intestinal wall; this leads to bloating, cramping, diarrhea, dizziness, diaphoresis, and weakness

Dumping syndrome- feelings of fullness, nausea, cramping, Disease, diaphoresis, diarrhea
Leads to dehydration, hypotension and tachycardia
Small intestine may adapt better is osmolality is increased slowly.

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10
Q

G tube vs. J tube

A

➱J tubes good for those who can’t have anything in the stomach but still get food into the gut
✰Typically does not last as long
✰Endoscopically placed gastrojejunal tube in the jejunum for the purpose of administration of nutrition, fluids and medications.

➱G tubes someone can have a G tube for the rest of their life
✰Opening created in stomach, preferred over nasogastric for long term maintenance, (use greater than 4-6 weeks)
✰Preferred in comatose patients because the gastroesophageal sphincter remains closed
Percutaneous endoscopic gastrostomy (PEG) tube

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11
Q

What are the potential problems associated with g tubes and j tubes?

A

Potential problems

➱Wound infection, cellulitis, and leakage
➱GI bleeding
➱Premature dislodgment of the tube
➱Tube obstruction/clogging

☆Monitoring and managing potential complications by checking for drainage, comitus, and stool Aspiration is a potential risk with tube dislodgement, especially with nasally inserted tubes.
☆Skin care. It is normal to see scant serous drainage a few days post insertion. After approx 1 wk, the site can be cleaned 2x a week with soap and water and left open to air. Candida may appear in warm moist areas of the body.

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12
Q

Complications of enteral feedings

A

✰ Mechanical tube displacement➱ replace rube
✰ Aspiration➱elevate head of bead, check residual before feeding for intermittent check residual every 4 hour for continuous.
✰ GI (v/d/n)➱ decrease feeding rate, change formula, administer at room temperature
✰ Metabolic hyperglycemia ➱ monitor glucose, osmolality , reduce infusion rate, give insulin if needed
✰ dehydration➱ flush with water or normal saline
✰ formula drug interactions➱ check compatibility, flush tubing prior to and after medication

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13
Q

Nasogastric tube management?

A

every four hours on your tube feeds, 30mL flush

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14
Q

What is gastritis? What are the symptoms?

A

➱inflammation of the gastric or stomach mucosa

➱Symptoms:
   ✰Acute--> possible signs of shock 
-anorexia
-epigastric (rapid onset of symptoms)
-hematemesis 
-hiccups 
-melena or hematochezia 
-N/V

✰Chronic–> anemia & fatigue

  • belching
  • early satiety
  • intolerance of spicy or fatty foods
  • N/V
  • pyrosis
  • sour taste in mouth
  • vague epigastric
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15
Q

What are the nursing interventions of gastritis?

A

➱ANITBITOICS, H2, PROTON PUMP ,

Acute gastritis is usually self-limiting, client may have decreased appetite, instruct to avoid EtOH, and food, may need IV fluids, NG tube, intubation, antacids, H2 blockers, PPI’s,
➱Surgery may be needed in necrosis or gastric outlet obstruction
➱Chronic gastritis is managed by diet modification (decrease caffeine), rest, reduce stress, avoid EtOH, NSAIDS, antibiotic for H. Pylori, quit smoking

Nursing interventions:
★No food or fluids by mouth. With IV therapy, monitor I&Os and serum electrolyte values. Introduce solid food as soon as possible, but report any symptoms that indicate repeat episodes of gastritis. Discourage intake of caffeinated beverages, alcohol, and smoking. Administer any pharmacotherapy prescribed.
★Promote fluid balance- monitor daily I&Os, assess electrolyte values every 24 hours. Watch for signs of hemorrhagic gastritis (especially vomiting/defecation of blood)
★Relieve pain- avoid foods or beverages that irritate gastric mucosa. Regularly assess levels of pain/discomfort.

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16
Q

H2 blocker medications examples, MOA, and AE

A

reduce the amount of acid produced by the cells in the lining of the stomach

cimetidine, famotidine, nizatidine and ranitidine

side effects are cholinergic effects: constipation, dry mouth, dry skin, ringing int he ears, runny nose

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17
Q

How would you manage a GI bleed associated with gastritis and assess if it is getting better or worse?

A

Erosive gastritis my cause bleeding which may manifest as blood in vomit or as melena (black, tarry stools) or hematochezia (bright red, bloody stools

The nurse must always be alert to any indicators of hemorrhagic gastritis, which include hematemesis (vomiting of blood), tachycardia, and hypotension. All stools should be examined for the presence of frank or occult bleeding. If these occur, the primary provider should be notified, and the patient’s vital signs are monitored as the patient’s condition warrants. Guidelines for managing upper GI tract bleeding are discussed later in this chapter.

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18
Q

Why do we worry about pernicious anemia in clients with gastrointestinal disorders? What are the signs and symptoms of pernicious anemia? How is it treated?

A

Pernicious anemia is a decrease in red blood cells that occurs when the intestines cannot properly absorb vitamin B12. Vitamin B12 deficiency can occur in patients with disorders such as inflammatory bowel disease, or in patients who have had GI surgery such as ileal resection, bariatric surgery, or gastrectomy

signs and symptoms: smooth, sore, red tongue and mild diarrhea, extremely pale, particularly in the mucous membranes, confused; more often, paresthesias in the extremities (particularly numbness and tingling in the feet and lower legs). They may have difficulty maintaining their balance because of damage to the spinal cord, and they also lose position sense (proprioception)

treatment: b12 shots once a month

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19
Q

What are the symptoms of peptic ulcer disease? What are the complications? What is perforation and what are the signs and symptoms?

A

Peptic ulcer disease: excavation formed in the mucosal wall, caused by erosion

Symptoms: 
dull, gnawing pain or burning sensation in the mid epigastrium or back
Pain after eating
Pyrosis
Vomiting
Constipation or diarrhea
Bleeding 
Symptoms often accompanied by sour eructation (burping) 
Complications: 
Hemorrhage
Perforation
Penetration
Gastric outlet obstruction
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20
Q

What are the symptoms of peptic ulcer disease? What are the complications?

A

Peptic ulcer disease: excavation formed in the mucosal wall, caused by erosion

Symptoms: 
dull, gnawing pain or burning sensation in the mid epigastrium or back
Pain after eating
Pyrosis
Vomiting
Constipation or diarrhea
Bleeding 
Symptoms often accompanied by sour eructation (burping) 
Complications: 
Hemorrhage
Perforation
Penetration
Gastric outlet obstruction
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21
Q

What is perforation and what are the signs and symptoms?

A

–Perforation
➱Signs and symptoms include:
-Sudden, severe upper abdominal pain (persistent and increasing in intensity); pain may be referred to the shoulders, especially the right shoulder, because of irritation of the phrenic nerve in the diaphragm
-Vomiting
-Collapse (fainting)
-Extremely tender and rigid (boardlike) abdomen
-Hypotension and tachycardia, indicating shock

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22
Q

How is peptic ulcer disease with h. pylori treated?

A

Management goal - eradicate H pylori, manage gastric acidity - antibiotics , proton pump inhibitor (“zoles”), and H2 blockers, completing treatment is crucial.
used for 10-14 days.

Maintenance H2 Blockers usually for 1 year post healing.

Combination of antibiotics, proton pump inhibitors, and sometimes bismuth salts that suppress or eradicate H. pylori
Recommended combination drug therapy is typically prescribed for 10-14 days and may include triple therapy with two antibiotics, plus a proton pump inhibitor or quadruple therapy with two antibiotics plus a proton pump inhibitor and bismuth salts
Research currently being conducted for vaccine against H. pylori
Avoid use of NSAIDS
Maintenance dosages of H2 blockers are usually recommended for 1 year
Look at most common antibiotics; know side effects of flagyl

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23
Q

What are preventative measures for “dumping syndrome”?

A
Dumping syndrome is the rapid passage of food to stomach, causing diaphoresis, diarrhea, hypotension; usually occurs 10-15 minutes after eating 
   ➱ restrict fluids w/ meals 
   ➱ avoid stress after eating 
   ➱eat smaller, frequent meals
   ➱lie down after eating
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24
Q

Metronidazole aka flagyl

A

Dizziness, headache, stomach upset, nausea, vomiting, loss of appetite, diarrhea, constipation, or metallic taste in your mouth may occur

education: do not drink alcoholl

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25
What is a “stress ulcer”? Why do they occur? How are they prevented?
Stress ulcer occurs after physiological stress. May occur due to burn injuries, multiple organ disorders, Medications: prophylactic H2 Blockers or PPIs (end in “zoles”)
26
Proton-pump inhibitor
class of medications that cause a profound and prolonged reduction of stomach acid production ``` examples: omeprazole (Prilosec, Prilosec OTC, Zegerid) lansoprazole (Prevacid) pantoprazole (Protonix) rabeprazole (Aciphex) esomeprazole (Nexium) dexlansoprazole ``` ae: In general, PPIs are believed to have few adverse effects, as they are generally well tolerated. Patients have experienced few minor side effects of short-term PPI use, such as headache, rash, dizziness, and gastrointestinal symptoms including nausea, abdominal pain, flatulence, constipation, and diarrhea
27
Describe the care for a client who is status post esophagojejunostomy for gastric cancer. What do you monitor for?
The patient undergoing gastric surgery may experience complications, including hemorrhage, dumping syndrome, bile reflux, and gastric outlet obstruction. Postoperative bleeding from the surgical site is a common complication of gastric surgery. Bleeding may be severe (hemorrhage) and manifest as vomiting large amounts of bright red blood, which may result in hemorrhagic shock Monitor for dumping syndrome
28
Anti-ulcer agents/ proton pump inhibitors (PPI)
Omeprazole, Pantoprazole nursing considerations: Monitor CBC, electrolytes. Avoid alcohol, NSAIDS, aspirin, and foods that may cause irritation side effects: Abdominal pain, vitamin b 12 deficiency, systemic lupus erythematosus, fatigue, headache Clostridioides difficile-associated diarrhea (CDAD), Hypersensitivity reactions, vitamin B12 deficiency
29
glucagon
used before endoscopy procedures as an aid during radiologic exams to temporarily inhibit GI movement
30
Metronidazole
antibiotic used for peptic ulcer disease caused by H. pylori. Stops the growth of the bacteria nursing considerations: No alc, cannot be pregnant AE: Seizures, dizziness, headache, aseptic meningitis, encephalopathy, psychosis, stevens-johnson syndrome, abdominal pain, anorexia, nausea,
31
Clarithromycin
anti-infectives agents | used for ulcers; often administered with metronidazole and a PPI
32
Sucralfate
anti-ulcer agent used in the prevention of duodenal ulcers Take on empty stomach, separate 30 mins of antacids. Causes a decrease in abdominal pain. Keep taking it even if you are feeling better AE: constipation
33
Loperamide
anti-diarrheal diarrheal treatment avoid using other CNA depressant while on this med. DONT DRINK. watch liver function AE: cardia arrest, torsades de point
34
Psyllium
laxative AE: Bronchospasm, cramps, intestinal or esophageal obstruction, N/V
35
docusate
laxative-stool softener used for constpiation short term use: DO NOT USE WITH MINERAL OIL
36
octretide
first line treatment for esophageal varices -given before endoscopy in patients with variceal bleeding MOA: -Suppresses gastrin levels (PUD) -Causes selective splanchnic vasoconstriction by inhibiting glucagon release and used mainly in the management of active hemorrhage (esophageal varices) DO NOT USE IF PREG AE: -cholelithiasis - ileus - pancreatitis - hypoglycemia - abdominal cramping
37
Cimetidine
Histamine 2 agonist GI ulcers and GERD Anti-ulcer agent, Histamine 2 agonist Should be used 6-8 weeks for complete peptic ulcer healing, patients who are at higher risk require a maintenance dose for 1 year. confusion, dizziness, drowsiness, hallucinations, headache, ARRHYTHMIAS, constipation, diarrhea, nausea, gynecomastia, AGRANULOCYTOSIS, APLASTIC ANEMIA
38
Describe the teaching for clients following gastrectomy. How would you educate them on optimizing their nutrition?
injection of vitamin B12 will be required for life, because intrinsic factor, secreted by parietal cells in the stomach, binds to vitamin B12 so that it may be absorbed in the ileum. This deficiency in vitamin B12 metabolism can result in decreased production of red blood cells, or pernicious anemia; nurse assesses for signs of dehydration (thirst, dry mucous membranes, poor skin turgor, tachycardia, decreased urine output) and reviews the results of daily laboratory studies to note any metabolic abnormalities (sodium, potassium, glucose, BUN). Antiemetic agents are given as prescribed.
39
Constipation
related to inadequate intake of fluid and bulk nursing interventions ➱ record intake and output, color and consistent of stool and frequency of bowel movements to form the basis of an effective treatment plan ➱promote ample fluid intake ➱encourage the pt to walk and exercise to stimulate intestinal activity ➱discourage routine use of laxatives
40
diarrhea
related to malabsorption, inflammation, or irritation of the bowel nursing interventions ➱assess level of dehydration and electrolyte status ➱monitor the pt's weight daily to detect fluid loss or retention ➱test stool for occult blood ➱assess fecal impaction. liquid stool may seep around an impaction
41
bowel incontinence
related to neuromuscular involvement ➱establish a bowel schedule for the pt-1/2 hour after a meal is desirable for active peristalsis
42
ileostomy vs. colostomy
➱ Ileostomy: widespread colonic obstruction may require near-total removal of the colon and rectum. SMALL INTESTINE to the surface of the abdomen, specifically the ileum (which is the last part of the small intestine before it opens into the large intestine). ALWAYS LIQUID STOOL. Huge risk for skin breakdown and increased risk of dehydration and electrolyte imbalance. ➱ Colostomy: an opening created to bring the LARGE INTESTINE to the surface of the abdomen (the large intestines are also called the colon). the causes for the surgery are due to rectal cancer. Ascending: liquid stool Transverse: lose to partly formed stool Descending/Sigmoid: similar to normal consistency stomas are pink and beefy red
43
What diseases are related to malabsorption syndrome? How are they treated?
Sphincter defects: Zollinger-Ellison syndrome, AIDS, parasites, Clostridiumdifficile CELIAC: avoid gluten lACTOSE INTOLERANCE: lack of lactase, avoid lactose
44
differentiate between Ulcerative colitis and Crohn's Disease
ulcerative colitis→ affects the mucosa and submucosa of the colon. Begins at the rectum and sigmoid colon and extends upward into the sigmoid colon. ★The stool typically contains pus and mucus and recurrent bloody diarrhea. Assess the pt for spastic rectum and anus, abdominal pain, anorexia, fever, toxic megacolon Crohn’s disease→ can affect any part of the GI tract. Lymph nodes enlarge and lymph flow in the submucosa is blocked. The lymphatic obstruction causes edema, mucosal ulceration, fissures, abscesses.. Oval, elevated lymph follicles are called Peyer’s patches-develop on the lining of the small intestine. AS the disease progresses, the fibrosis thickens the bowel walls. ★right lower abdominal pain, cramping tenderness, n/d → chronic cases: diarrhea 4-6x a day ★ increased wbc count, increased ESR, hypokalemia, hypocalcemia, and decreased hemoglobin
45
Know acute abdomen (ex: appendicitis)
Appendicitis occurs when the appendix becomes inflamed. Mucosal ulceration triggers inflammation, which temporarily obstructs the appendix. The obstruction blocks mucus outflow. Pressure in the now distended appendix increases. Bacteria multiply and inflammation and pressure continue to increase, restricting blood flow to the organ= abdominal pain. ☆ an appendectomy is the only effective treatment for appendicitis. If peritonitis develops, treatment involves GI intubation, parenteral replacement of fluids and electrolytes and administration of antibiotics.
46
Parenteral nutrition, when to start, when it’s indicated, what to monitor
Complications: infection and hyperglycemia Indicated when enteral nutrition is not an option, pt is malnourished, cannot tolerate orally Nursing: Daily weights, I/Os, glucose, CBC, monitor: glucose
47
crohns disease
inflammatory conditions of any of the large or small intestine assess * severe abdominal pain, cramp in the lower right quadrant, chronic diarrhea, * mucus, pus, FAT in stools * increased temp * decreased weight diagnostic test-upper GI-barium enema, colonoscopy nursing management- diet-high protein, high calorie, low fat, and low fiber
48
Ulcerative colitis
inflammation condition of the colon characterized by eroded areas od the mucous membrane nad tissues beneath it assess * diarrhea 10-20 stools per day * blood, pus, mucus in stool; n pain * fecal incontinence * dehydration * weightloss * weakness, cachexia, * metabolic acidosis
49
what causes acute abdomen
appendicitis
50
GERD
gastroesophageal reflux disease aka ACID REFLUX USE cimetidine This is a chronic disease that occurs when stomach acid or bile flows into the food pipe and irritates the lining. Acid reflux and heartburn more than twice a week may indicate GERD. Complications of GERD include dental erosion, ulceration in the pharynx and esophagus, laryngeal damage, esophageal strictures, adenocarcinoma and pulmonary complications --pH testing, endoscopy or barium swallow
51
which electrolyte is out of whack in diarrhea?
potassium
52
what psyllium is (metamucil)
it is fiber | For constipation💩
53
What causes gastric outlet syndrome?
Food stops going through stomach Associated with ulcers, gastritis, stricture
54
Percutaneous endoscopic gastrostomy (PEG) tube | nursing considerations
enteral nutrition nursing management- x ray verified, pH aspirate flush with water or normal saline
55
Geriatric considerations with gallstones and how they may present differently
Surgical removal is more common Appear as: septic shock, oliguria, hypotension, tachycardia, tachypnea, altered mental status
56
how does someone with gallstones present?
``` Burping after meals Indigestion Severe upper right quadrant pain N/V May have no symptoms abdominal pain, distension ```
57
What causes gall stone? (Cholelithiasis)
Causes:An increased concentration of bile substances - Obesity - Pregnancy - Diabetes - Hypothyroidism - Liver cirrhosis
58
gallbladder inflammation is called...
Cholecystitis Inflammation of the gallbladder Causes: bacteria Nursing: NPO, antibiotics
59
pancreatitis
Inflammation of the pancreas Possible causes: tumors, cysts, ulcers, etc clinical manifestations: N/V, fluid/electrolyte imbalances, fever, jaundice, hyperglycemia, weight loss, ascites Nursing: NPO, PPIs, Antiacids, Treating fluid and electrolytes, monitor for infection, TPN, avoid alcohol
60
pt has jaundice but no liver disease...
Jaundice without liver disease: hemolytic jaundice Increased destruction of red blood cells Fecal and urine uro-bilirubinuria is increased, no bilirubin in the urine No symptoms, but predisposes to CNS effects and gallstones
61
what causes jaundice?
bilirubin excess
62
Priorities for pt's with confusion
O2 and toxicity
63
signs and symptoms of alcoholic cirrhosis
``` Cirrhosis Complications: * Portal hypertension * Esophageal Varices --Throwing up blood, bloody stool, discomfort Nursing ->diet : High Carb High protein ->Reduce ascites: sodium/fluid restrictions, ->Monitor fluid and electrolyte ->Daily weights ->Abdominal girth ``` Alcoholic Cirrhosis * Caused by alcoholism and poor nutrition * Confusion, fatigue, jaundice, bleeding, hypertension, varices, ascites
64
Hepatic encephalopathy
*Life threatening disease caused by liver disease, hepatic failure * Ammonia is secreted into the brain and causes CNS depression - ->Can increase in GI bleeds * Clinical signs: mental changes, motor changes, asterixis, * Metronidazole ENCEPHALOPATHY IS CAUSED BY AMMONIA
65
care of patient undergoing pancreatitis
Review care of patient undergoing paracentesi: procedure that removes fluid (peritoneal fluid) from the abdomen through a slender needle. * Position: supine * monitor: monitors the respiratory status because large volumes of ascites can compress the thoracic cavity and inhibit adequate lung expansion. The nurse monitors serum ammonia, creatinine, and electrolyte levels to assess electrolyte balance, response to therapy, and indications of hepatic encephalopathy. * Post paracentesis circulatory function * Albumin infusions help to correct decreases in effective arterial blood volume that lead to sodium retention. The use of this colloid reduces the incidence of post paracentesis circulatory dysfunction with renal dysfunction, hyponatremia, and rapid reaccumulation of ascites associated with decreased effective arterial volume
66
rebound ascites is treated with...
albumin
67
What treats hepatic encephalopathy?
Administer lactulose, brings down ammonia by making the patient defecate * Lactulose: Works by drawing water from your body into your colon, which softens stools and causes you to have more bowel movements.
68
Nursing education pre and post for endoscopy
PRE_OP: NPO status, start an IV, no ibuprofen, consent, educate on throat numbness due to anesthesia POST_OP: maintain NPO status until gag reflex and bowel sounds return observe for vomiting, aspiration, respiratory distress
69
what is done for TPN patients
total parenteral nutirtion remember- aseptic routine and swallow studies for pt's after a long period of time
70
Know IBS-C and IBS-D: know expected drugs and treatments
Irritable bowel syndrome: marked by chronic symptoms of abdominal pain ,alternating constipation and diarrhea, excess flatulence, constipation (classified as IBS-C) Lubiprostone, a chloride channel regulator in the gut, can be prescribed for patients with IBS-C ``` diarrhea (classified as IBS-D) antidiarrheal agents (e.g., loperamide ```
71
Magenisum Hydroxide
laxative | used in constipation
72
Cholecystitis
inflammation of the gallbladder
73
Cholelithiasis
Calculi, or gallstones, usually form in the gallbladder from the solid constituents of bile; they vary greatly in size, shape, and composition Abdominal x-ray, ultrasound, radionucleotide imaging, oral cholecystography, endoscopic retrograde cholangiopancreatography ERCP Management: banding, TIPS, shunting, Meds: Octreotide, vasopressin (do not use in patients with HX coronary artery disease), beta blocker (“-lol”), sclerotherapy,
74
care for PEG tubes
Elective change of the balloon every 3-6 months to extend expected lifetime of tube Tube normally lasts 1-2 years with proper care Clean skin around site 1-3 times a day Rotate tube completely each day Flush tube before and after feeding If clogged, unclog as soon as possible
75
how someone with gallstones presents aka choleithiasis
``` Burping after meals Indigestion Severe upper right quadrant pain N/V May have no symptoms abdominal pain, distension ``` Geriatric considerations with gallstones and how they may present differently Surgical removal is more common Appear as: septic shock, oliguria, hypotension, tachycardia, tachypnea, altered mental status
76
what causes cholelithiasis
``` Causes:An increased concentration of bile substances Obesity Pregnancy Diabetes Hypothyroidism Liver cirrhosis ```
77
laparoscopic vs open chelecytsctomy
Most done laparoscopically Gall bladder removed through small incision Open cholecystectomy is done surgically with a larger incision and has a higher mortality rate, chance of infection, and impairs gas exchange due to the higher and larger incision. Why might be one indicated over another Laparoscopic cholecystectomy reduces surgical risk, length of recovery, and decreases abdominal pain post op. Know what complications of each are Laparoscopic Bile duct injury Bile peritonitis Open Drainage, NG tube, and suctioning may be needed immediately post op Know what causes gallstones (Cholelithiasis)
78
position after appendectomy
high fowler