Nose Flashcards

1
Q

nasal trauma

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

peri orbital cellulitis definition

A

an infection of the periorbital soft tissue characterised by erythema and oedema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

peri orbital cellulitis epidemiology

A

0-15 yrs old
peak <10 yrs
males more common
bi-modal seasonal variation - peak in late winter/early spring
occurs in 35% of children with sinus related infections

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

types of peri orbital cellulitis

A

pre septal and post septal, separated by orbital septum
occurs as a result of contigious spread from surrounding periorbital structures such as paranasal sinuses…ethmoidal sinusitis most common cause

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

why children peri orbital cellulitis

A

the thinner and dehiscent bone surface of their lamina papyracea and increased diploic venous supply in comparison with adulthood, in addition to relatively incomplete immunologic development in this age group

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

causes peri orbital cellulitis

A

sinus infection
dacrocystitis, dental infection, endophthalmitis, trauma, foreign bodies, insect bites, skin infections (impetigo), eyelid lesions (chalazia, hordeola), and iatrogenic causes such as eyelid and oral procedures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

organisms peri orbital cellulitis

A

Streptococcus pneumoniae, Haemophilus influenzae, Moraxella catarrhalis, Staphylococcus aureus, group A streptococcus, and upper respiratory tract anaerobes. Staphylococcus aureus is usually the most common pathogen.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

clinical features peri orbital cellulitis

A

eyelid oedema
erythema of upper eyelid
re-septal cellulitis will present with normal vision, absence of proptosis, and full ocular motility without pain on movement.
worsening oedema
purulent discharge from ipsilateral osteomeatal complex and sphenoethmoidal recess

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

signs orbital cellulitis

A

Proptosis
Ophthalmoplegia
Decreased visual acuity
Loss of red colour vision – first sign of optic neuropathy
Chemosis
Painful diplopia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

peri orbital cellulitis classification

A

chandler classification
pre
post
subperiosteal abscess
intra orbital abscess
cavernous sinus thrombosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

examination peri orbital cellulitis

A

Assessment of routine vital signs
Dentition
Anterior rhinoscopy
Appearance of the nasal mucosa in general and middle meatus area
Discharge should be swabbed and sent for culture
comprehensive opthalmic examination
sepsis screen
CT scan

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

ddx peri orbital cellulitis

A

Vesicles of herpes zoster ophthalmicus
Erythematous irritation of contact dermatitis
Raised, dry plaques of atopic dermatitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

tx peri orbital cellulitis

A

Mild pre-septal cellulitis in adults and children older than 1 year of age, treatment is typically rendered on an outpatient basis with empiric broad spectrum oral antibiotic
IV fluids, analgesia
tx underlying cause - sinusitis
early drainage or orbital abscess

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

complications periorbital cellulitis

A

visual related - vision loss, papilloedema, neuritis and atrophy, impaired ocular motility, eyelid inflammation
life threatening - meningitis, encephalomeningitis, cavernous sinus thrombosis, sepsis IC abscess, vomiting, seizures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

most common cause of epistaxis in children

A

nose picking
narrower nasal airways - mor prone to bleeding and inflammation in general
Mucosal irritation – e.g. dry air, URTIs, steroid use
Clotting abnormalities – either hereditary or acquired. Epistaxis is usually the major presenting feature of common clotting abnormalities such as Von-Willebrand’s disease or genetic conditions such as hereditary haemorrhagic telangiectasia (HHT)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

<2 children epistaxis

A

rare
referred to ENT for investigation

17
Q

pathophysiology epistaxis

A

most comes from little’s area - confluence of vessels originating from ICA and external carotid arteries

18
Q

recurrent epistaxis

A

underlying coagulopathy and a full clinical and family history should be explored. Recurrent epistaxis can rarely be caused by an underlying JNA (juvenile nasal angiofibroma) which are most common in males aged 12-20

19
Q

risk factors epistaxis

A

Activities involving altitude e.g. skiing
Strenuous physical activities with risk of nasal trauma or straining/raising ICP e.g. rugby, gymnastics
Coagulopathies
Hayfever or regular URTIs
Medication use (rare)

20
Q

examination epistaxis

A

see source on anterior septum of affected side
see evidence of septal haematoma

21
Q

septal haematoma tx

A

urgent review by ENT and drainage to avoid permanent disfigurement

22
Q

ddx epistaxis

A

foreign body - ruled out in unilateral offensive nasal discharge

23
Q

investigations epistaxis

A

rarely needs investigating
if large vols - blood tests - underlying coagulopathy
recurrent epistaxis - me nostril or in males aged 12-20 should be referred to ENT for flexible nasal endoscopy (FNE) to exclude a JNA or other unilateral pathology e.g. polyp

24
Q

initial management epistaxis

A

lean child first
spit blood out
pinch the soft part of nose and hold for 15 mins at least
check for cessation of bleeding
apply ice pack

25
Q

primary acre/A and E management epistaxis

A

look for bleeding points inside nose using bright light
local anaesthetic/decongestant spray - co-phenylcaine
nasal cautery with silver nitrate - can stain skin
circular motion with silver nitrate sitkc from outside to centre of bleeding point
if continues - anterior or posterior nasal packing
reduction of any nasal fractures
FBC, clotting profile

26
Q

definitive management epistaxis

A

pts should be discharged with naseptin ointment BD for 2 weeks
- must check for peanut allergy so must be checked

27
Q

reoccurrence risks

A

-Strenuous physical activity

-Bending forwards e.g. to tie shoelaces

-Hot drinks/food/showers (steam can irritate the inside of the nose)

-Blowing their nose

-Picking their nose

-Spicy food

28
Q

red flags epistaxis

A

-Persistent unilateral bleeding

-Weight loss

-Visual disturbance

-Facial pain

29
Q
A