Non Surgical Therapy_Teresa 2021+Hoda 2022 Flashcards

1
Q

What is the rationale for non-Sx mechanical therapy?

A

1- Eliminate plaque biofilm accumulating surface characteristics (Rough surfaces –> 4x increase in plaque formation - Quirynen 1993)
2- Reduce inflammation
3- To control disease progression:
- Loe 1965 -biofilm is the primary etiology
- Harrell and Nunn 2001 - Px that don’t receiver Perio tx had increased PD at a rate of 0.3 mm/year

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is the tissue response to sterile & sterile calculus?

A

Allen and Kerr 1965:
Study in Guinea pigs
- Sterile calculus –> mild foreign body reaction
- Non-sterile calculus –> marked inflammatory response with granulation tissue formation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What are the limits of SubG scaling?

A

Stambaugh 1981:
- Curette efficiency: PD to which tooth surface the tooth can be instrumented to a plaque and calculus free surface = 3.73 mm (Average) & (Maximum = 4.07 mm)
More efficient on M/D than Bucc/Ling)
- Curette limit: Maximum PD to which evidence of instrumentation on the root could be detected = 6.21 mm (Average = 5.52 mm)
However, the curette efficiency maxed out at 4 mm due to resistance of pocket wall to be distended by the instrument shank for proper blade angulation in deeper pockets.

  • Average instrumentation time:
  • Max. posterior tooth –> 39 min
  • Mandibular posterior tooth –> 25 min
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

According to Heitz-Mayfield & Lang 2013, what is the critical PD for SRP and MWF(Access flap)? What

A

Critical PD is the PD below which a specific tx approach would result in CALoss and above which it will result in CALgain. Those numbers are based on a longitudinal RCT in 15 patients with advanced PDitis (Lindhe et al 1982)
SRP –> 2.9 mm
MWF (Access flap) –> 4.2 mm
5.4 mm –> is the PD starting from which access flap would result in sig. higher CALgain than SRP. This means that between PD 2.9-5.4 mm, non Sx therapy is preferred

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What classical study showed that plaque biofilm is the 1ry etiology of disease?

A

Loe et al. 1965
Aim:
OH was withdrawn and the changes in the microbial flora and gingiva was observed
* 12 study participants (9 dental students, 1 perio faculty, and 2 laboratory technicians)
* Patients were told not to perform any oral hygiene and inflammatory changes were observed (GI, PI)

Results:
- 3 subjects developed gingivitis in 10 days.
- 9 subjects took 15-21 days.
- different progression rates and models were present among individuals.
- Gingival inflammation was resolved within one week resuming oral hygiene.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is the effect of PD reduction on the clinical and microbiological changes?

A

Mombelli and Nyman et al.1995:
Design:
- 7 patients aged 30-60 years with generalized periodontitis.
- OHI + SupraG debridement
- Flaps + osseous of angular bony defects. While the control teeth were carefully debrided and thoroughly root planed, no root instrumentation was performed on the test teeth + APF
Results:
- Similar pattern of response in the test and control sites over a one year post therapy –> PD + CAL were sig. reduced 1 month after surgery and remained at a lower level.
- Sig. decrease in total anaerobic viable bacterial counts and Gram-ve anaerobic rods (Pg, Fusobacterium and C. Rectus) decreased significantly in both groups.
Conclusion:
These findings corroborate the concept that the reduction of selected SubG microorganisms is the key element for the success of periodontal therapy, rather than the removal of tooth substance and mineralized deposits by root instrumentation.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What is the architecture of biofilm on natural teeth?

A

Ziginge 2010:
3D architecture with Red complex bacteria in the middle and superficial layer

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Is attachment possible between calculus and Junctional epithelium?

A

Listgarten 1973:
Calculus sterilized with CHX allows the attachment of Junctional epith. via hemidesmosomes.

From the abstract:
“Methods: Following SRP of 2 rhesus monkeys, gingiva was maintained by 1x daily intra-oral swabs with 2 % CHX, supplemented by toothbrushing and interdental cleansing with toothpicks 3X/ week.
- 1 monkey maintained for 3 months on this regimen, the 2nd monkey was maintained for only 3 weeks followed by a period of 12 days without oral hygiene.

Results: The JE is able to form an attachment to a calculus mass which contained bacterial cells. Electron microscopy revealed that this attachment was mediated through a dental cuticle, a basement lamina and hemidesmosomes. The ultrastructure of the epithelial attachment to calculus was indistinguishable from that which exists between junctional epithelium and various tooth surfaces. These results may be due to the inhibiting effect of chlorhexidine gluconate on plaque formation and possibly a concomitant lowering of the toxicity of calculus.”

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Do you think that calculus detection by periodontists is accurate?

A

Sherman et. al 1990) evaluated the accuracy of clinical detection of calculus by periodontists, he examined 101 teeth (which were deemed for extraction due to perio/pros reasons)

476 surfaces (4 surfaces of non-molar teeth and 6 surfaces for molars) were evaluated before and 1 week after scaling and root planing (hand + US). After 3-month maintenance, extractions done, then microscopic evaluation of remaining calculus.

Microscopically: 57.7% of surfaces had residual calculus. However, clinically only 18.8% were recorded.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What is the difference in calculus removal with and without flap at different PD?

Caff likes to remove calc

A

Waerhaug evaluated calculus efficiency in relation to diff. PD:
- Shallow: 83%
- Moderate: 38%
- Deep: 11%

Caffessee et al. 1986: compared the calculus-free surfaces when only SRP or Flap/MWF + SRP was done.

SRP only:
- PD 1-3 mm –> 86%
- 4-6 mm –> 43%
- > 6 mm –> 32%
SRP + Flap:
- PD 1-3 mm –> 86%
- 4-6 mm –> 76%
- > 6 mm –> 50%

Ca

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What are the limitations of SRP in furcations?

A

Bower 1979: assessed the width of molar furcations:
- 81% of furcations were =< 1 mm
- 58% of furcation areas were < 0.75 mm.
On the other hand, the width of the commonly used curettes were smaller than the width of furcations, the narrowest curette width is 0.8 mm (Gracey curette)

Bower also measured root concavity in Max and Mand. molars:
- Max: 94% of MB roots had 0.3 mm concavity
- Mand: 100% Mesial root had 0.7 mm concavity, and 99% of distal root had 0.5 mm concavity

  • Regardless of the type of instrumentation, it is expected that there will be residual calculus, but when combining open flap debridement WITH US instrumentation it will be more effective especially in narrow furcations (Matia et. al. 1986).
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Does PD measurement affect the success of Plaque control?

A

(Waerhaug 1978b) In 83% of the sites, plaque control was successful (reformation of JE in histo and seen when pocket depths were < 3mm. , and 39% when PD were 3-5mm. However, when pockets were > 5mm the success rate was only 11%.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

How long does it take oral flora to recolonize pocket after SRP?

A

Greenwell and Bissada 1984 –>
* following SRP microflora from diseased sites return to baseline levels after 4-8 weeks (~ 56 days)
*There was an increase in the proportion of nonmotile bacteria in sites with probing depths of ≤4 mm
Sbordone 1990 –> rebound of subG microflora takes 60 days after SRP

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Is there a difference in clinical improvement that can be achieved using hand instrumentation vs. US or laser vs. US?

A

(Badersten 1981) Compared Ultrasonic vs. Hand :
Patients with moderate- advanced periodontitis, split mouth design.
Both methods resulted in improvements of clinical parameters, NSSD between them.

(Sculean et. al. 2004a) Compared Laser vs. Ultrasonic: 20 patients (PD >4mm), split mouth
non-surgical therapy done with either:
1-Er:YAG laser device.
2-Ultrasonic.
They both resulted in comparable results in terms of (PD, BOP, CAL gain, recession

(Sculean et. al. 2004b) Compairing Ultrasonic vs. Hand: 38 patients recruited with moderate – advanced periodontal disease, they were randomly distributed to non-surgical therapy with either:
1- Vector ultrasonic system (working tip movement is parallel to the root).
2- Hand instrumentation.
There were No differences in parameters measured (PD, BOP, GI, CAL gain &recession).
Less working time with the first group.

(Muller et. al. 2014) RCT, split mouth design, compairing Ultrasonic vs. air polishing: 50 patients (with PD> 4mm):
1-air-polishing device.
2-US instruments.
NSSD between the groups.
Significantly less pain reported by patients in sites treated with air-polishing compared to US.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is the efficacy of endotoxin removal, time and pocket closure with hand vs. US instrumentation?

A

Nishimine and O’leary showed that US instrumentation results in 8X more endotoxin left behind than root planning

Copulos showed that US is more time efficient in achieving similar clinical outcomes compared to hand instrumentation.
US –> 3.9 min/tooth
Hand –> 5.6 min/tooth

Wesnstrom and Tomasi 2005: assessed the % of pocket closure (PD of =< 4mm) with 4Quads SRP vs. FM US
- 4 Quads SRP –> 66% pocket closure with 8.8 min/closed pocket
- FM US –> 58% with 3.3 min/closed pocket

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What is the effect of US vs. hand instrumentation on root surface roughness following OFD?

A

Schlageter 1996: US (regular & diamond tips) roughened root surface more than hand instruments

17
Q

How easy it is to remove endotoxin from root surface? Is total cementum removal possible?

A

Moore and Wilson: 99% of endotoxin can be removed by gentle cleaning: 39% via washing + 60% via brushing for 1 minute
- O’leary and Kafrawy: showed 50 strokes failed to completely remove cementum

18
Q

What should expect clinically after non Sx therapy in terms of PD reduction and BOP?

A

Cobb 1996:
- Intial PD 1-3 mm: 0.03 PD reduction & -0.34 mm CALoss
- Intial PD 4-6 mm: 1.29 PD reduction & +0.55 mm CALgain
- Intial PD ≥ 7mm: 2.16 PD reduction & 1.19 mm CAgain

Cobb 2022: Reduction in Bleeding on probing: A review of BOP reduction after SRP. When PD were 4 – 6.5 mm there was a reduction of BOP by 45% from baseline measurements.

DeVore et. al. 1990: Reduction in plaque scores: 24 patients, assessment of plaque indices before and 3 months after active periodontal therapy, they found SS reduction in plaque score in T2 compared to T0.

19
Q

Is complete calculus removal sufficient to regain perio health?

A

Cadosh and Zimmerman: Complete calculus removal was achieved after 9.3 strokes and this resulted in endotoxin reduction to minimal levels

20
Q

Can tissue attach on rough root surface?

A

Waerhaug 1956: normal attachment was observed histo. on roughened root surfaces.

21
Q

Can long Junc. Epith resist plaque?
“LJE is Magnum size”

A

Magnusson: Monkey study –> Long JE can resist plaque as normal attachment.

22
Q

Is there an added benefit from single vs. multiple instrumentation?

A

Badersten 1984: NSSD between single vs. multiple instrumentation (3x at 3 months intervals.

Anderson 1996: NSSD betwen single vs. 3x instrumentation –>

23
Q

Does it matter if we kept maintaining our periodontal patients or not?

A

Treated Patients:
- Annual tooth loss:
** Without maintenance:
Becker et. al 1984a:
Including hopeless teeth, they found that there will be 0.29 tooth/year.
Excluding hopeless teeth, they found it to be 0.22 tooth/year.
Most common tooth lost is Max 2nd molar
** With Maintenance:
Becker et. al. 1984b:
Including hopeless teeth = 0.24 tooth/ year.
Excluding hopeless teeth = 0.11 tooth/year.
Most common tooth lost is Max 2nd Molar.

24
Q

Does it matter if we treat our periodontal patients or not?

A

UnTreated Patients:
- Annual tissue destruction and tooth loss
Without maintenance:
** Loe et. al. 1986: Sri Lankan Tea laborers
No progression: 11% 0.05-0.09 mm / year
Moderate progression: 81% 0.05-0.5 mm / year
Rapid progression: 8% 0.1-1.0 mm / year

**Becker et. al 1979:
Including hopeless teeth, 0.61 tooth/year.
Excluding hopeless teeth, 0.36 tooth/year.
Most common teeth lost are Mand. 1st and 2nd molars.

25
Q

What should we expect to see (histologically) after SRP? When do we re-evaluate after mechanical therapy?

A

Engler and Ramfjord 1996 did a study on monkeys and found that healing with long junctional epithelium will take 2 weeks, but we need 4-6 weeks for connective healing by repair. In addition, most dramatic clinical changes occur during the first 4 weeks after SRP, and PD and CAL changes are stable up to 3 months (Sherman et. al. 1990a)

Waehaug 1978: 21 patients. 29 teeth deemed for extraction. SRP was done. within 2 weeks, healing by long junctional epithelium was evident in areas that had plaque and calculus.

Magnusson: Long junctional epithelium has similar “plaque resistance” as normal epithelium