NMJ

Question 1

NMJ: EPP

connects, where

axon - loses what = forms what

EPP - AP at (?) = influx = NT = influx = event = AP at (?) = influx = event = contraction

why no inhibitory in skeletal + alternative

myasthenia gravis & eaton lambert - patho/structure

Answer 1

NMJ
- connects neuron to muscle
- in presynaptic cell
- axon loses its myelin sheath = motor end plate (depression/NMJ)

EPP
- AP at alpha motor of agonist = Ca influx = exocytose ACh (NT) then go to nicotinic receptor = Na influx = EPP = AP at skeletal muscle (sarcoplasmic reticulum) = EPSP process (Ca > NT > Na > EPSP > AP) = muscle contraction
- inhibitory: none since ACh only NT in skeletal muscle. if inhibit = use golgi tendon organ (IPSP)

pathologies
- MG: ACh receptor is foreign
- eaton: attack Ca = no Ca/exocytosis of NT

Question 2

NMJ: Smooth & Cardiac Muscle

no, has what + E/I + cover, main nerve of ANS

EJP - contraction = vessel
IJP

wallerian - equilibrium = ACh = what

Answer 2

• no endplates
• varicosities/synapse en passant: no schwann; inhibitory
• ANS: use CN10

excitatory junctional potential
- peristaltic contraction = vasoconstriction

inhibitory junctional potential
- release NT that is inhibitory instead

wallerian
- equilibrium since shits move around & balance charges = ACh gets leaked out = many contraction (fasciculation)