Neurology disorder Flashcards

1
Q

Bell’s palsy

A

Idiopathic facial nerve
palsy.
Acute, spontaneous, unilateral facial paralysis
cranial nerve VII palsy

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2
Q

Bell’s palsy presentation

A

Disappearance of the nasolabial fold

decrease tearing on the affected side

inability to wrinkle the forehead

loss of taste sensation
inability to pucker lips

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3
Q

treatment of Bell’s palsy

A

prednisone

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4
Q

concussion

A

mild traumatic brain injury
GCS =13 or > Glasgow coma scale

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5
Q

concussion treatment

A

physical rest 24-48h with a gradual return to activity

cognitive rest minimizes cognitive activities that exacerbate symptoms

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6
Q

recurrent concussion

A

may indicate overexertion during recovery

if a migraine history, a concussion may trigger migraine HA

promote sleep it help recovery

cognitive recovery may be longer than physical recovery

refer for symptoms lasting >21 days or escalating symptoms after injury or multiple concussions

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7
Q

After a concussion when back to school or work?

A

when able to focus for 30-45 mins at a time

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8
Q

back to sports?

A

return-to-school success

symptoms free, no meds

normal neuro exam

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9
Q

Back to sports ?

A

light aerobic exercise (increase HR)

sport-specific exercises (add movement

noncontact participation

full contact practice (assess status)

normal game/physical activity

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10
Q

Headaches?

A

primary headaches: migraine, tension, cluster

Secondary headaches: underlying pathology “ red flag” headache

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11
Q

NB

A

New onset headache in a middle/older adult (>50) is usually secondary

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12
Q

papilledema

A

swelling of the optic disc due to increase ICP
Almost always bilateral

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13
Q

CT vs MRI

A

CT : radiation,look at cavities, intracranial hemorrage, tumor, hard thing, bones
quick 5min

MRI: no radiation. look at soft tissue, brain , spinal cord, ligament, tendon, injuries
nonemergent

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14
Q

Dx of migraine without aura

A

-headaches last 4-72 hours
has 2 of these characteristics unilateral, pulsating quality, mod to severe intensity, aggravated by routine activity
-during headache: N and /or V , photophobia or phonophobia (at least 1)
-5 or more attacks have occurred with these characteristics
-no other reason for the headaches occurrence

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15
Q

Dx migraine with aura

A

25% of pt have aura
-2 attacks of migraine with aura
-visual. sensory, motor, and brainstem. retinal. or speech change fully reversible
-develops over 5-60mins , headache develop within 60minutes

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16
Q

treatment

A

first line -Acetaminophen
NSAIDs
triptans
calcitonin gene-related peptide CGRP
prophylactic agents : beta blockers propranolol, timolol
antidepressant amitriptyline, venlafaxine

anticonvulsants: valproate, topiramate

17
Q

Vertigo

A

either peripheral or central etiology

peripheral etiology involves the vestibular system (BPV)

central involves the brainstem or cerebellum (stroke)

18
Q

is vertigo peripheral or central

A

peripheral (vestibular)
-visual fixation stops nystagmus
-walking preserved
-hearing may be impaired
-normal neuro check
Central
-visual fixation does not stop nystagmus
-often falls with walking
-hearing usually intact
-abnormal neuro exam

19
Q

clue to etiology of vertigo

A

-Less than 1 minute, single episode - BPV
-Few minutes to hours, single, episode-migraine, TLA of labyrinth or brain stem
-Several mins or hours, recurrent- Meniere;s , vestibulopathy
-Days, prolonged and severe - vestibular neuritis, MS, brain stem infarct

20
Q

The Dix-Hallpike

A

The Dix-Hallpike maneuver is the gold standard for diagnosing benign positional paroxysmal vertigo caused by a posterior canal otolith. The patient is positioned recumbent with the head back and toward the affected ear, causing the otolith to progress superiorly along the natural course of the canal.

The Dix-Hallpike test diagnoses the condition and the Epley maneuver treats it by encouraging the tiny calcium carbonate crystals to move back where they belong.

21
Q

Treatment

A

-Meclizine-Antivert
-Dimenhydrinate-Dramamine
-Diphenhydramine-Benadryl

22
Q

Diabetic Neuropathy

A

-Distal symmetric polyneuropathy
DSPN= diabetic neuropathy
-15-20% of DM neuropathy patients have pain
-symptomatic neuropathy is not usually reversible

23
Q

Differential diagnosis of

A

=Metabolic-diabetes, B12, deficiency, hypothyroidism
=Toxic- Alcohol
= Diffuse motor without sensory- Guillain-barre syndrome, myasthenia gravis
=inflammatory infectious - sarcoidosis, lupus, Lyme disease

24
Q

Preventive care

A

-glycemic control
-modify risk factors (BMI, Bp, A1c, minimal EtOH consumption , avoidance of smoking ect)
-foot care

25
Q

Pharm treatment

A

SNRI: duloxetine (Cymbalta, Venlafaxine (Effexor)
TCA: amitriptyline, desipramine, nortriptyline
Gabapentinoid: pregabalin (Lyrica) , gabapentin (Neurontin)

26
Q

patient evaluation

A

-Screening in asymptomatic patients is NOT recommended
- history of cognitive/behavioral changes (from family or significant other)
-Med history asks about analgesics anticholinergics, psychotropics, sedative-hypnotics

27
Q

patient evaluation

A

-MMSE
-Montreal cognitive assessment (MoCA)
-if there is a discrepancy between cognitive testing and physical exam, further testing is need

28
Q

Depression

A

Depression can worsen cognitive impairment
Always screen for depression (consider PHQ2)

29
Q

Treatment of AD

A

Cholinesterase inhibitors: Donepezil, rivastigmine, galantamine.

NMDA receptor antagonist, memantine.
Advanced AD:
use in combo with cholinesterase inhibitors (donepezil/memantine)

30
Q

pharm for severe , persistent symptoms

A

Olanzapine (consider first)
Quetiapine (consider as an alternative)
May increase mortality. side effects

31
Q

cranial nerves

A

Oh Oh Oh To Touch And Feel Very Good Velvet AH
O-Olfactory
O-Optic
O-Oculomotor
T-Trochlear
T-Trigeminal
A-Abducens
F-Facial
V-Vestibulocochlear
G-Glossopharyngeal
V-Vagus
A-Accessory
H-Hypoglossal

32
Q
A