Neurology Flashcards
Which spinal lesions would you get normal to hyper-reflexive
C1-C5
What spinal lesions would give normal to hypo-reflexive?
C6-T2
Predisposition for cervical intervertebral disc disease
Large dogs associated with Hansen type II protrusions at disc
Doberman associated with cervical spondylomyelopathy
Signs of cervical intervertebral disc
- radicular pain or nerve root signature often with impingement of C5 and C8
- most common sign = cervical spinal pain, low head, neck carriage, neck guarding, stilted and cautious gait and spasms of muscle
Non surgical treatment for cervical intervertebral disc disease
- conservative indicated with 1st episode of pain only or with milked paresis
- strict cage confinement (4-6 weeks), corticosteroids and muscle relaxants
- harness during limited walking exercises
Surgical treatment for intervertebral disc disease
- persistent pain or moderate to severe neurological deficits
- removal of the disc
- ventral slot decompression - most commonly performed
Predisposition of thoracolumbar intervertebral disc disease
Chondrodystrophic breeds —> hansen type I in thoracolumbar region - progressively decreases from T12-T12 causally
Most common site for Hansen type I in large non-chondrodystrophic breeds - between L1 and L2
Signs of thoracolumbar intervertebral disc
- percute (<1hr), acute (<24hr), gradual (>24hr)
- persecute/acute disc extrusion = “spinal shock” or shiff-Sherrington posture
- (vary) spinal hyperesthesiia/paraplegia with/without pain perception
- dogs with back pain —> reluctant to walk - show kyphosis
Diagnosis of thoracolumbar IVDD
Signalmen’s, history, and neurological examination
CT, MRI, surgery
Differentials for thoracolumbar IVDD
Trauma, fibrocartilaginious emoblism, degenerative myelopathy, discospondilitis, neoplasia etc
Treatment for thoracolumbar IVDD
Non surgical - same as other IVDD
Surgical
- decompression alone is inadequate to restore spinal cord function when an extramural mass is greater than 4mm in diameter (so decompression and remove)
- decompressive procedures = dorsal laminectomy, hemilaminectomy and pediculectomy
Another name for degenerative lumbosacral stenosis
Cauda equina syndrome
Predisposition for cauda equina syndrome
Middle age dogs of medium - large breeds
GSD most commonly affected
7 yrs, 2x more likely in male than female
Working dogs that are heavily trained are prone
Cause of cauda equina syndrome
Degenerative lumbosacral stenosis (DLSS)
Pathogenesis of cauda equina
Soft tissue and bony changes, possibly in conjunction with abnormal motion of the lumbosacral joint, impinge on the nerve roots/vasculature of the cauda equina
Signs of cauda equina
- lumbosacral (caudal) pain
- maintain characteristic posture, keeping their lumbosacral joints flexed (increases the canal —> decreasing nerve root compression)
- pelvic limb lameness, tail paresis or paralysis
- urinary or faecal incontinence
Compression of sciatic nerve (L6-S2)
Abnormal proprioceptive positioning, muscle atrophy, parapetáis, reduced flexor reflex