NEURO PPQ Flashcards

1
Q

Question 1

Describe three exercises necessary to retrain oro-facial function following progressive bulbar palsy as a result of motor neuron disease. (6)

A

· Jaw closure

· Lip closure

· Elevate posterior third of tongue

· Facial exercises

o Check for puffing, facilitate with cold fingers inside mouth

o Male patient-shaving grooming facilitate with mirror; don’t ice for too long

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2
Q

Mrs Strange was stabbed in the neck four months ago and was diagnosed with C4 complete quadriplegia. She presents with increased spasticity and has been referred to you as an out-patient for management.

2.1 Discuss any four factors you should include when prescribing caregiver education to her family. (8)

A
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3
Q

Explain three techniques that you could use to manage her spasticity. (6)

A

● Movements either passive or assisted - the faster you move the pt the higher the tone becomes, always start with slow movements from proximal to distal. By performing slow movements on the proximal part of the limbs you will find that there is already a change in the distal part of the limb.
● Relaxing techniques - teach the pt to take deep breaths and relax, while performing this the muscles will also relax and let go.
● Correct positioning (in wheelchair and in bed) to prevent shortening of muscles, muscles should always be in a lengthened position.
● Heat therapy - Wrap limbs/entire body in warm towels to allow physiological processes to relax muscles.
● Hydrotherapy can also be used.
● Weight bearing - standing frames or tilt tables depending on the patients’ neurological level and what would be the safest for them. You could also perform weight bearing exercises for upper limbs in puppy prone or in four-point. When targeting the upper limbs you could use slow rhythmical rocking to influence the spasticity.
● Slow stroking/ deep pressure - apply a squeeze/deep pressure over the tightened muscle to allow for voluntary relaxation of that muscle.
● PNF- use rhythmic initiation and very slow reversals to get the antagonistic muscle to work.
● Antagonistic muscle stimulation with functional electrical stimulation to overcome spasticity.
● Advocate for your pt to be placed on medications like Baclofen, Diazepam or botulinum toxin especially if they experience pain with spasticity.

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4
Q

Explain three techniques that you could use to manage her spasticity. (6)

A

· Range of motion excs

· Prolonged passivee stretching

· Active assisted excs

· Serial splinting

· Hydrotherapy and ultrasound

· Phenol or alcohol or botulinum toxin

· Surgery is indicated after 6/52 of intensive physiotherapy without improvement

· Joint approximation

· Weight bearing with weight shifting

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5
Q

Compare and contrast Parkinson‘s disease and Parkinson‘s Syndrome. (10)

A
  1. Parkinson’s Disease (PD)
    ·Progressive neurodegenerative disorder of the extrapyramidal system.
    Lack of dopamine

·Aetiology: Unknown cause that leads to dopamine deficiency

·Signs and Symptoms
-Tremor
-Rigidity
-Bradykinesia
-Impaired balance
-Gait problems
-Affects one side of the body more than the other

·Treatment
Medical mx:
-Replacement therapy →
-Levodopa
-Pallidotomy
-Cell transplantation
-Progressive not curable

Parkinson’s Syndrome
·General term that refers to a group of neurological disorders that cause movement problems similar to those seen in Parkinson’s disease.
Lack of dopamine

·Aetiology: Known cause including drugs, vasculature and some palsy’s that lead to dopamine deficiency
·Signs and Symptoms:
-No tremor
-Rigidity
-Bradykinesia
-Impaired balance.
-Affects both sides of the body equally

·Treatment:
Medical mx:
-Replacement therapy → Levodopa
-Stopping medication causing symptoms

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6
Q

List four forms of multiple sclerosis. (2)

A

· Progressive-relapsing multiple sclerosis-steady decline since onset with superimposed attacks

· Secondary progressive multilpe sclerosis-initial relapsing-remitting multiple sclerosis that suddenly begins to have decline without periods of remission

· Primary progressive multiple sclerosis-steady increase in disability without attacks

· Relapsing-remitting multiple sclerosis-unpredictable attanks which may or may not leave permanent deficits followed by periods of remission

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7
Q

Discuss four physiotherapy management strategies used during the terminal stage of motor neuron disease. (8)

A

· Position changing

· Respiratory care

· Pressure care

· Back care for carers

· Advice on feeding positions and diet

· Counseling to patient and carer-refer to social worker

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8
Q

Tabulate any four comparisons and contrasts between spasticity and rigidity. (½x8=4)

A

Spasticity
·Pattern of muscle Movement: UL Flexors
·Nature of tone: Velocity dependent increase in tone
·Tendon reflexes: Increased
·Clinical signs: UMN (Pyramidal sign)
·Tested through: Quick movements

Rigidity
·Pattern of muscle Movement: Flexors and extensors Equally
·Nature of tone: Constant throughout ROM
·Tendon reflexes: Normal
·Clinical signs: Extrapyramidal sign
·Tested through: Slow movements

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9
Q

Ms Jones is a C4 complete tetraplegic and has developed a Stage III pressure ulcer on her sacrum and is placed on strict bed rest in prone-lying. Discuss the role of the physiotherapist in the management of her pressure ulcer. (8)

A

Role of physiotherapy

· Education the patient on what pressure ulcers are, how to prevent them and possible complications associated with them.

· Prevention-through effective pressure relief, frequent positioning every 10-15 minutes and optimum seating patterns

· Curative- this can be done using conservative management such as electrolyte substitution and position change.

· Promotive- family education on positioning, transfers, wound care and cleaning (hygiene), as well as providing information about PUs and associated complications

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10
Q

Explain how you would manage a patient with Parkinson’s disease who struggles with initiation of walking. (8)

A

· Gait initiation by starting with high long steps.

· Trunk mobilisation with emphasis on arm swing.

· Movements should be broken up into small components

· Conscious effort (attention) on the task.

· Where possible, avoid simultaneous task performance

· A mirror can also be used to correct posture.

· Repetition important

· Walking aid use = restriction of normal walking patterns

· Motor-planning problems may complicate aid use.

· One can provide a walking aid so as to reduce falling episodes.

· Proper assessment for selection and use of walking aid necessary

· Freezing =When the patient is approaching the “freezing” time e.g. approaching a door way or about to turn they can:

o count their steps aloud

o stamp their feet

o lift their feet as if stepping over something

o or they can look at anything that has a pattern on it e.g. a rock and again repetition is the best way to learn.

· Lines on the floor and on steps.

· Break tasks down into components, teach each component and then task as a cognitive activity.

· Don’t walk/eat and talk at same time

· Chan et al (1993) reported significant changes in stride length, velocity and cadence in single-subject design following facilitation of pelvic-trunk counter-rotation.

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11
Q

Discuss the expected sensory problems associated with multiple sclerosis. (6)

A

· Paraesthesia: pins and needles sensation

· Pain: usually muscular in origin

· Loss of proprioception: makes coordinated movements difficult

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12
Q

Muscle strengthening is an important focus of physiotherapy management for patients in the plateau phase of Guillian-Barre syndrome.
Describe the focus of an exercise programme in this phase and give four examples of exercises you would include. (5)

A

Focus: Muscle strength central stability
Exercises:
● Kneeling
● Rhythmic stabilisation
● Crawling
● Big physioball exercises

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13
Q

A patient with Guillain Barre syndrome may experience periods of postural hypotension which needs to be taken into consideration by the physiotherapist. Discuss this statement. (10)

A

Postural hypotension refers to a drop in blood pressure upon assuming an upright position. This can have implications for physiotherapists in managing and providing care for these patients due to the following reasons:

· Autonomic Dysfunction: GBS is characterized by immune-mediated damage to peripheral nerves, including the autonomic nervous system. The autonomic nervous system regulates various involuntary functions in the body, including blood pressure control. In GBS, dysfunction of the autonomic nerves can lead to impaired regulation of blood pressure, resulting in postural hypotension.

· Risk of Falls and Safety Concerns: Postural hypotension can increase the risk of falls and related injuries, especially when patients with GBS attempt to stand up or change positions. Physiotherapists need to be aware of this potential issue and take appropriate precautions to ensure patient safety during mobility exercises and transfers. They may need to implement strategies such as gradual positioning changes, providing support during transitions, and closely monitoring blood pressure responses.

· Exercise and Orthostatic Intolerance: Physical activity and exercise are important components of rehabilitation for patients with GBS. However, postural hypotension can lead to orthostatic intolerance, where symptoms worsen upon assuming an upright posture. Physiotherapists should carefully monitor the patient’s response to exercise and modify the intensity, duration, and frequency of therapy sessions to prevent excessive drops in blood pressure and minimize orthostatic symptoms.

· To effectively manage postural hypotension in patients with GBS, physiotherapists may collaborate with other healthcare professionals, such as physicians or occupational therapists, to develop a comprehensive care plan. This plan may include strategies to improve blood pressure regulation, such as appropriate fluid intake, wearing compression stockings, and optimizing medication management under the guidance of a medical professional.

· Regular monitoring of blood pressure before, during, and after therapy sessions can help guide treatment decisions and ensure patient safety.

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14
Q

Explain spasms under the following headings:

a) Type of impairment (2)

b) Outcome measure used to assess spasms (1)

A

a) Manifests as a muscular problem due to an upper motor neuron lesion and decreased inhibition of the reflexes. These are often a result of exaggerated reflexes in response to sensory stimuli.

b) Penn Spasm Frequency Scale

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15
Q

Describe two supra-spinal level inhibitory influences whose absence could contribute to the occurrence of spasms. (4)

A

Basal Ganglia - which helps with refinement of and inhibition of certain movements. Absence results in akinesia, rigidity, dyskinesia on the contralateral side

Vestibulocerebellum - which helps with maintenance of balance. It’s absence will lead to bilateral/ipsilateral ataxia with intention tremors

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16
Q

Explain autonomic dysreflexia under the following headings:

a) Causation (3)
b) Prevention (3)
c) Management (3)

A

a) Causation (3)

· Anything that can cause discomfort to a neurologically intact person can trigger autonomic dysreflexia in a pt with SCI

· Distended bladder or rectum are common stimuli

· Stimulation of the skin from pressure, pain, heat or cold

· Bowel- over distended or irritation

· Skin-related disorders

· Sexual activity

· Other

o Heterotopic ossification

o Acute abdominal conditions

o Skeletal fractures

b) Prevention (3)

· Call doctor to administer an antihypertensive agent with rapid onset and short duration

· Educate pt on

o positioning to enhance comfortability, avoid laying flat supine. Opt for semifowlers

o relaxation positions,

o ACBT with focus on BC

o Monitor vitals

c) Management (3)

General management of Autonomic/Sympathetic dysreflexia (the goal of Rx is to identify and remove the cause of the dysreflexia and thus lower the BP)

· Check the pt’s BP

· If BP is elevated sit them up immediately with feet down to promote orthostatic reduction of BP. If pt unable to sit, elevate head of bed to 90 degrees

· Loosen any clothing or constrictive devices

· Monitor the urinary catheter for any blockage or twisting during movement

· Call doctor to administer an antihypertensive agent with rapid onset and short duration

· Document the episode

· Deterrence/prevention

· Good bladder and bowel care

· Patient education

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17
Q

Explain the physiotherapy management of a patient with Guillain Barre syndrome during the acute phase of the disease. (10)

A

Physiotherapy Mx: Acute stage (usually up to day 10)

· Respiratory care (ACBT, PD, Manual tech, Sxn..)

· Musculoskeletal: Joint protection (e.g. AFO), Maintenance of joint movement (e.g. AP’s/PA’s – should know when to choose between the two) and soft tissue length

· Skin: positioning & circulatory exercises

· Circulation: Prevent DVT & Postural hypotension

· Psychological support: family and patient

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18
Q

Mr Napo presents with a history of slow onset asymmetrical muscle weakness that has a patchy distribution, increased tone and reflexes, and has poor vision. Write short notes about the pathology of his condition. (12x½ =6)

A

Mr Napo has multiple sclerosis which is an inflammatory immune mediated demyelinating disease affecting the brain, spinal cord, and optic nerves. The myelin sheath (fragments and disintegrates) is mainly affected, and nerve axons are affected secondarily.
There are patchy areas of demyelination, and this is followed by gliosis.
Loss of myelin sheath and oedema results in decreased nerve impulse conduction. Remyelination occurs, however the re-myelinated axons and myelin sheath are thinner and shorter. Further clinical decline occurs when remyelination fails thereby leaving the axons in a vulnerable state. This failure could be due to a deficiency in the precursor cells, a failure of precursor cell recruitment, or a failure of precursor cell differentiation and maturation. Uhthoff’s phenomenon/syndrome could occur when an increase in body temperature aggravates the decrease in conduction of nerve impulses. Heat increases the hydrolysis of acetylcholine therefore decreased motor unit recruitment leading to muscle weakness and fatigue.

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19
Q

Explain the term “freezing” with regards to Parkinson’s disease. (4)

A

Freezing is a part of the gait problems faced by patients with Parkinson’s disease and it occurs when the patient has to transition from one movement to another such as turning a corner, standing from seated position. This also occurs because the patient struggles to initiate, transition and stop automatic movements. This often occurs when pt’s with Parkison’s disease are due for their Dopaminergic medication. Freezing episodes can be triggered by transitional movements or an attempt at performing multiple tasks at once.

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20
Q

Mr Ncube has been diagnosed with a Brown Sequard lesion resulting in ipsilateral and
contralateral signs and symptoms following a stab wound to the lower back.

4.1. Name the tract(s) that are involved in the ipsilateral effects and the associated signs
and symptoms. (6)

A

Brown Sequard Lesion is the hemi-section of the spinal cord, and it has the best prognosis for ambulation. Recovery takes approximately 6 months, and it commences in the ipsilateral proximal extensors then the distal flexors.
4.1. Posterior column – loss of vibration, form perception, two point discrimination, proprioception, and motor function below the level of the injury.

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21
Q

4.2. Name the tract(s) that are involved in the contralateral effects and the associated signs and symptoms. (2)

A
  • Spinothalamic tracts (hemi-anaesthesia/hemi-analgesia)- Loss of pain, temperature sensation and light touch below the level of the injury
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22
Q

Describe the tenodesis grip and its importance when managing a patient with a C6 complete
spinal cord injury. (3)

A

● Tenodesis grip – passive finger flexion with thumb opposition when performing wrist extension.
● Grip strength can be achieved with a flexor-hinge orthosis/brace. This is important when managing a patient with a complete C6 SCI because it can be utilised to facilitate grasping in tetraplegic patients with intact wrist extension against gravity. This gives the patient more independence with functional tasks such as grooming, dressing, feeding, and personal hygiene activities without the assistance of the caregiver.

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23
Q

Name and discuss two oro-facial problems following motor neuron disease with regards to the possible causes and physiotherapy management. (9)

A

● Swallowing problems (dysphagia) – this is a result of progressive bulbar palsy which results in LMN signs and symptoms and restricted function of the tongue. The tongue is weak and wasted and sits in the mouth with fasciculations. The patient presents with a hypoactive gag reflex which increases the risk of aspiration, sand decreased lip and jaw closure. The patient will also have a collection of saliva in the mouth leading to drooling. The PT can teach the patient postural techniques where he/she adjusts the head and body position when feeding to optimize swallowing and reduce the risk of aspiration. Referral to a dietician to alter the diet (texture and consistency of foods) as liquids may be easier to swallow compared to solid foods.

● Dysarthria – this is a result of progressive bulbar palsy which results in LMN signs and symptoms and restricted function of the tongue. The patient will present with slurred speech, poor articulation, reduced speech volume and an altered pitch. This is due to muscle weakness of the muscles controlling the tongue, lips, palate, and vocal cords. The PT can assist through strengthening and coordination of the affected muscle groups by using deep breathing exercises as they strengthen the respiratory muscles to help maintain control over speech volume and quality. Referral to a speech therapist will be necessary to ensure the patient gets exercises for articulation.

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24
Q

Explain how technology can affect the building blocks of a health care system. (Explain only
three building blocks). Service delivery, Health workforce, Information, Medical products, vaccines+technology, financing, and leadership and governance (6)

A

● Acquisition of expensive medical equipment (MRI/CT scans, TENS) can impose a financial burden on the healthcare system; and not all healthcare facilities will be afforded such equipment. Lack of such equipment in certain districts will limit diagnostic capacity of the healthcare workforce there and thus result in poor service delivery. This will have an overall burden on the healthcare workforce in the districts with this technology as they will have to work harder to cover more patients; which also compromises quality service delivery.
● Advances in technology will make it easier to collect and store information which will help the healthcare workforce to make better decisions about patient management thereby improving overall patient outcomes. This will also enable the patients to be active participants in their healing process as they will be able to easily access information regarding their health via the internet or by having easy communication methods with their healthcare provider.
● Improvements in technology within a healthcare system will enable improved and efficient service delivery to the target population of the hospital. For example, transitioning from a paper-based record keeping system to a computer-based system; this will make it easier to keep patient records and integrate patient information across hospitals in different provinces.

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25
Q

Explain the relationship between the rehabilitation process and occupational health. (10)

A

● The relationship between the rehabilitation process and occupational health is closely intertwined, as both concepts are focused on maintaining and enhancing the well-being of individuals within the context of their work environments.
● Occupational health is a multidisciplinary legislative requirement that aims to protect and promote the physical, mental and social well-being in the workplace. The goals of OH are to prevent risks and hazards in the workplace, thereby promoting physical and psychological health. OH encompasses early risk diagnosis (to limit disability), and adaptation of the work environment according to the capabilities of the workers. Through risk assessment in the workplace, OH workers can be able to mitigate any further consequences of occupational risks with a rehabilitation process.
● The rehabilitation process aims to restore physical, psychological and mental well-being following injury or disability thereby improving quality of life and reintegration into the work environment.
● OH practitioners play a crucial role in identifying workplace hazards and implementing prevention strategies to minimise the occurrence/consequence of occupational hazards. Practitioners are also responsible for creating return-to-work programs for workers that have sustained injury through collaboration with other specialists to ensure gradual and safe reintegration that aligns with the physical and psychological capabilities of the worker. Adaptation of the work environment is often necessary following injury or disability and must be facilitated by the practitioners. An integral part of the rehabilitation process is worker education on appropriate ergonomics in their respective work environment to reduce likelihood of injury or hazard exposure. Evaluation of the steps taken to prevent hazard exposure or to adapt the environment to fit worker capabilities must be done to ensure that changes are beneficial to the worker and that he/she is coping.

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26
Q

Outline the rationale behind the district health information system.

A

● To support improved district health management and planning
● To provide comprehensive, timely, reliable and good quality routine evidence for tracking and improving health care delivery
● Facility-based information does not reach the requirements of the DHIS
● To improve community health status by having objective information to base decisions on.
● Monitoring and evaluating district services

27
Q

Define three principles that guide the formulation and implementation of public health laws.(6)

A

● PHL requires public understanding and support: the public must understand the laws and consequences for not abiding.
● Mutual accommodation: laws that are passed should consider cultural and ethnic diversity
● PHL requires intersectoral initiative for development and implementation
● Appropriate enforcement mechanisms: ethical enforcement of new laws
● Community resources should be solicited for enforcement
● Implementation and enforcement requires financial commitment

28
Q

Name and provide the characteristics of the two levels of care in a district health system. (8)

A

● Primary care
-first point of contact with HCS
-homes clinics, level 1 health centres (200-250 beds)
-variable operating hours (8-24 hours)
-comprehensive range of services that are free (by-pass fee)

● Secondary Care
-district hospital
-250-500 beds
-first level of referral/ services (24hrs)
-specialists- rehab ,operating theaters and chronic care is available

● Tertiary care
-academic hospitals (500-1000+ beds)
-all specialists are available
-associated with medical schools

● Quaternary Care
-highly specialised hospitals (specific conditions)
-TB hospitals, Organ transplants.

29
Q

Define the five functions of a district health system.

A

● Managing, planning, organising
● Finance and resource allocation
● intersectoral action
● HR development
● Community participation

30
Q

Discuss four physiotherapy management strategies used during the terminal stage of motor neuron disease. (8)

A

●Position changing
●Respiratory care
●Pressure care
●Back care for carers
●Advice on feeding positions and diet
●Counseling to patient and carer-refer to social worker

31
Q

Briefly discuss the management of rigidity in Parkinson’s disease. (10)

A

● We CANNOT use techniques that apply to spasticity to reduce tone in PD
● Slow rhythmical trunk rotations
● Maintain the muscle length through muscle stretching.The stretching can be passive or active
● Relaxation techniques
● Patients should be taught self-stretching exercises as soon as is possible

32
Q

Contractures are a common complication of upper motor neuron lesions. Write short notes on the aetiology, prevention and management of contractures. (8)

A

33
Q

Pressure sores are a common complication after spinal cord injury. Discuss pressure sores under the following headings:

4.1 Precipitating factors (4)

A

● pressure, skin irritants, or temperature extremes

● Lack of bed mobility/immobility in bed

34
Q

List the requirements for normal swallowing. (5)

A

● Jaw and lip closure

● Elevation of the posterior third of the tongue

● Elevation of the borders of the tongue

● The stimulus of something to swallow

● The sitting position

● Control of breathing in relation of swallowing

● Normal gag reflex-prevents choking

35
Q

Tabulate four differences in the clinical presentation between a patient with Guillain Barré syndrome and a patient with stroke. (8)

A

36
Q

Describe three exercises that can be used to counteract the simian posture typical to Parkinson’s disease. (6)

A

● Do repetitive exercises that facilitate a total extensor response.

● Prone position and then shoulder horizontal abduction at a cadence of 50-58.

● The patient can also hang from an overhead bar for short periods.

● The patient can also be advised to lie in prone at night.

● A mirror can also be used to correct posture.

● The emphasis here is on extension

37
Q

An increase in body temperature can aggravate the decrease in conduction of nerve impulses in patients with multiple sclerosis. Discuss how this increase in temperature impacts on physiotherapy treatment. (6)

A

An increase in body temperature can aggravate the decrease in conduction of nerve impulses

● heat can be generated by muscle contraction/external sources

● Heat increases hydrolysis of Acetylcholine = ↓ motor unit recruitment = weakness/fatigue

● This is known as Uhthoff’s phenomenon/syndrome
The increased body temperature leads to less motor unit recruitment and thus increased fatigue.

●The inflammation caused by the heat may also lead to more pain during movement.
The factors impact physiotherapy treatment in that you cannot work a patient to the point of fatigue, or overheat them.
●Patients need to take frequent breaks and drink water to cool down.
●Another impact on physiotherapy treatment is that you need to choose your treatment modalities carefully- hydrotherapy is usually done in a luke warm pool as patients find relief from the heat, but these patients may need to be in a cooler pool. Rigorous exercise is also not appropriate for them as they may overheat.
●Heat therapy to relieve sore muscles is also not indicated.

38
Q

Describe the physiotherapy management for preventing chest complications in a patient with a T8 complete spinal cord injury. (5)

A

Chest physiotherapy techniques such as 3 cycles of ACBT, percussions and vibrations

postural drainage

Assited cough technique,

incentive spirometry

PEP devices like blowing bubbles or inflating balloons

Thoracic excs such as trunk flex,ext,rotation to mobilse secretions

39
Q

How should you differentiate clinically between central cord syndrome and posterior cord syndrome for a lesion at C5/6? (6)

A
40
Q

Outline the general treatment principles of a patient with motor neuron disease (excluding orofacial management). (9)

A

General management (depends on the disease stage)

Management should be in line with patient’s expectations/wishes

Prophylactic physio for respiratory function

Positioning to maximise ventilation/perfusion. some advice on positioning includes

Don’t lay flat

Lye on back

Educate on bed mobility to prevent pressure sores

Frequent repositioning

Wait till digestion is complete

Educate on wheelchair mobs

bed mobs

Standing frame

Educate caregivers on lifting and transfers

Care-giver strain index

Clear secretions

Maintain ROM and muscle length

Minimise joint damage

Medical: Riluzole-to decrease glutamate activity

41
Q

Discuss the role of physiotherapy in muscle strength training in patients with Parkinson’s disease. (8)

A

● Physiotherapy plays a crucial role in the management of Parkinson’s disease (PD), especially when it comes to addressing muscle strength training. Parkinson’s disease is a neurodegenerative disorder characterized by motor symptoms such as tremors, rigidity, bradykinesia (slowness of movement), and postural instability. These symptoms can lead to muscle weakness, decreased range of motion, and overall functional decline. Physiotherapy, including muscle strength training, can help mitigate these issues and improve the quality of life for individuals with Parkinson’s disease.

● Here’s how physiotherapy and muscle strength training are important for patients with Parkinson’s disease:

● Improvement of Muscle Strength and Endurance: Muscle weakness is a common symptom in Parkinson’s disease, which can lead to difficulties in performing daily activities. Physiotherapy employs strength training exercises to target specific muscle groups, helping to improve muscle strength and endurance. This, in turn, can enhance the patient’s ability to perform tasks like walking, getting up from a chair, and maintaining balance.

● Neuroplasticity and Motor Learning: In Parkinson’s disease, there are disruptions in the brain’s normal patterns of movement control due to the loss of dopamine-producing neurons. Physiotherapy interventions focus on promoting neuroplasticity, which is the brain’s ability to adapt and rewire itself. Through targeted exercises and repetitive movements, physiotherapy can facilitate the creation of new neural pathways, helping the brain to compensate for the damaged areas and improve motor function.

● Enhancement of Balance and Posture: Impaired balance and posture are common problems in Parkinson’s disease, leading to an increased risk of falls. Physiotherapists employ exercises that challenge the patient’s balance and stability, such as weight shifting, standing on one leg, or performing specific yoga poses. These exercises not only strengthen the muscles but also improve proprioception (the sense of body position) and coordination, ultimately reducing the risk of falls.

● Customized Treatment Plans: Physiotherapists tailor their interventions to the individual needs and progress of each patient. They assess the patient’s specific motor deficits, muscle imbalances, and functional limitations. Based on this assessment, they design personalized exercise regimens that focus on areas requiring improvement, ensuring that the treatment is tailored to the unique challenges posed by Parkinson’s disease.

● Gait Training: Walking difficulties are a hallmark of Parkinson’s disease. Physiotherapy can address gait abnormalities by incorporating exercises to improve stride length, step initiation, and overall walking pattern. Gait training can significantly enhance the patient’s ability to move more smoothly and confidently.

● Motivation and Adherence: Parkinson’s disease can lead to depression, anxiety, and decreased motivation for physical activity. Physiotherapists not only provide guidance on exercises but also offer emotional support and motivation to help patients stay engaged with their exercise routines. This support can be essential in maintaining adherence to the prescribed exercise program.

● Collaborative Care: Physiotherapists often work closely with other healthcare professionals involved in the management of Parkinson’s disease, such as neurologists, occupational therapists, and speech therapists. This collaborative approach ensures that the patient’s overall care plan is well-coordinated and addresses various aspects of the disease

42
Q

Why could remyelination fail to occur in patients with multiple sclerosis? (4x½=2)

A

This failure could be due to a deficiency in the precursor cells, a failure of precursor cell recruitment, or a failure of precursor cell differentiation and maturation.

43
Q

Caregiver education and self-responsibility programmes are essential components of rehabilitation. Discuss this statement. (8)

A

Caregiver Education:

● Knowledge and Understanding: Caregivers, including family members and professional caregivers, need to be well-informed about the condition, treatment plan, and specific needs of the individual undergoing rehabilitation. Understanding the nature of the condition, potential challenges, and the rehabilitation process empowers caregivers to provide effective support.

● Safe and Effective Assistance: Rehabilitation often involves exercises, therapies, and treatments that require proper execution to ensure safety and effectiveness. Caregivers who are educated about proper techniques can provide assistance with activities of daily living, mobility exercises, and medication management, minimizing the risk of injury or complications.

● Adherence to Treatment Plans: Caregivers play a pivotal role in ensuring that the individual adheres to the recommended treatment plan. This includes assisting with medication schedules, attending therapy sessions, and encouraging the completion of exercises or activities prescribed by healthcare professionals.

● Emotional Support: Chronic conditions and disabilities can take a toll on the emotional well-being of both the individual undergoing rehabilitation and their caregivers. Educated caregivers are better equipped to provide emotional support, offer coping strategies, and identify signs of distress or mental health issues.

● Preventing Caregiver Burnout: Providing care to someone undergoing rehabilitation can be physically and emotionally demanding. Caregiver education helps caregivers recognize signs of burnout and stress and provides strategies to manage their own well-being while caring for the patient.

Self-Responsibility Programs:

● Empowerment: Self-responsibility programs empower individuals undergoing rehabilitation to actively participate in their own recovery. When patients take an active role in their rehabilitation journey, they often experience improved motivation, engagement, and a sense of control over their health.

● Adherence and Compliance: A significant challenge in rehabilitation is ensuring that patients consistently follow prescribed exercises, therapies, and lifestyle changes. Self-responsibility programs encourage patients to take ownership of their health and adhere to treatment plans, which can lead to better outcomes.

● Lifestyle Modification: Many rehabilitation plans require patients to make lifestyle changes, such as dietary modifications, exercise routines, and stress management techniques. Self-responsibility programs educate patients about the importance of these changes and guide them in making sustainable choices.

● Long-Term Management: Rehabilitation is not just about short-term recovery; it also involves equipping patients with skills to manage their condition in the long run. Self-responsibility programs provide patients with tools to monitor their health, recognize early signs of relapse, and take appropriate actions to prevent setbacks.

● Sense of Achievement: Successfully participating in a self-responsibility program can boost an individual’s self-esteem and confidence. Achieving milestones and recognizing progress can enhance motivation and foster a positive attitude toward rehabilitation.

44
Q

What is the difference between chronic inflammatory demyelinating polyneuropathy and Miller Fisher syndrome? (2)

A

Chronic inflammatory demyelinating polyneuropathy (remits and relapses) and

Miller Fisher syndrome (with ataxic gait and paralysis starting in the eyes) also presents like GBS.

CIPD:
- Presents with symmetrical weakness in the body, a form of GBS that has periods of remission and relapsing.
- Affects the peripheral nerves (demyelination)
MFS:
- Presents with a ataxic gait and starts with paralysis of the eyes.
- Primarily involves cranial nerves.

MFS- is a variant of GBS it can also be classified as Acute inflammatory demyelinating polyneuropathy, symptoms include weakness of the respiratory system, ataxia, areflexia. MFS is associated with the involvement of the lower cranial and facial nerve and there is usually poor muscle coordination. A key difference between MF and GBS is that MF presents with eye muscle paralysis.

CIDP- symptoms include weakness of the respiratory system, decreased tone and reflexes, motor fibres are more affected than sensory fibres and decreased proprioception.
CIDP does not have an identifiable preceding illness. Duration of onset should be 8-14 days to be classified as CIDP
The main difference is that CIDP is a chronic condition while GBS is acute. GBS is once off and CIDP remits and relapses.
MFS- presents with ataxic gait and starts with involvement of the eyes (paralysis).

45
Q

Describe four spinal shock motor signs. (8)

A

Muscle paralysis

Disuse atrophy-is late onset and very light

Increased reflexes

Clonus

Clasp-knife spasticity

Absent abdominal and cremasteric reflexes

Positive Babinski

Spastic bowel and bladder

Autonomic disturbances

Loss of sweating below level of lesion

Vasodilation with hypotension (the hypotension is orthostatic in nature)

46
Q

Patients with early-stage Parkinson’s disease have problems with carrying out simultaneous activities and sequencing of movement patterns.

Discuss this statement with reference to the physiotherapy intervention strategies that should be used. (12x½ =6)

A

Bradykinesia, Akinesia, Hypokinesia

Use rhythmic initiation and slow reversals

Use of external stimulation

Watch the legs of somebody next

The use of momentum e.g. to roll over, stand and walk can be helpful.

Gait initiation by starting with high long steps.

Trunk mobilisation with emphasis on arm swing.

Movements should be broken up into small components/ Break tasks down into components, teach each component and then task as a cognitive activity.

Conscious effort (attention) on the task.

Where possible, avoid simultaneous task performance

47
Q

A home programme is critical in the physiotherapy management of a patient with multiple sclerosis.

Discuss this statement. (4)

A

Physiotherapy management Can be subdivided into the following:

Marked neurological signs but walking without assistance

Occasional wheelchair user

Full time wheelchair user

Management of other accompanying symptoms (e.g. fatigue, spasticity, muscle weakness, cerebellar dysfunction, etc)

Primary aims of Physio Mx

To maintain /increase ROM

Encourage postural stability

Prevent contractures

Encourage weight bearing

Prevent pressure sores, chest infections (and all other complications of immobility)

Increase independence (e.g. mobility aids, strengthening, fatigue Mx)

48
Q

Mrs Mthombeni (50 years old) and Mr Mohamed (61 years old) sustained spinal cord injuries four weeks ago and are admitted in your rehabilitation units. Mrs Mthombeni was diagnosed with Brown Sequard Syndrome and Mr Mohamed with Central Cord Syndrome.

3.1 Compare and contrast the presentation of Brown Sequard Syndrome to that of Central Cord Syndrome. (10)

A
49
Q

Discuss how the differences in presentation would influence your approach as the physiotherapist for the management of these two patients. (6)

A

Swallowing problems (dysphagia) – this is a result of progressive bulbar palsy which results in LMN signs and symptoms and restricted function of the tongue. The tongue is weak and wasted and sits in the mouth with fasciculations. The patient presents with a hypoactive gag reflex which increases the risk of aspiration, sand decreased lip and jaw closure. The patient will also have a collection of saliva in the mouth leading to drooling. The PT can teach the patient postural techniques where he/she adjusts the head and body position when feeding to optimize swallowing and reduce the risk of aspiration. Referral to a dietician to alter the diet (texture and consistency of foods) as liquids may be easier to swallow compared to solid foods.

Dysarthria – this is a result of progressive bulbar palsy which results in LMN signs and symptoms and restricted function of the tongue. The patient will present with slurred speech, poor articulation, reduced speech volume and an altered pitch. This is due to muscle weakness of the muscles controlling the tongue, lips, palate, and vocal cords. The PT can assist through strengthening and coordination of the affected muscle groups by using deep breathing exercises as they strengthen the respiratory muscles to help maintain control over speech volume and quality. Referral to a speech therapist will be necessary to ensure the patient gets exercises for articulation.

50
Q

Mrs Mbeki was diagnosed with Parkinson’s disease three years ago.
List five impairments and the subsequent functional limitations Mrs Mbeki may present with. (10)

A
  1. Tremors: Functional Limitation: Mrs. Mbeki might have difficulty performing tasks that require fine motor skills, such as writing, using utensils, or buttoning clothes.
  2. Bradykinesia (Slowness of Movement): Functional Limitation: She could struggle with tasks that involve quick movements, like getting up from a chair, walking, or turning around quickly.
  3. Muscle Rigidity: Functional Limitation: Mrs. Mbeki might find it challenging to perform tasks that involve bending, twisting, or stretching her muscles, making activities like getting dressed, reaching for objects, or bending down more difficult.
  4. Postural Instability/Lack of postural reflexes: Functional Limitation: She may experience balance problems, making it harder to stand without support, walk confidently, or perform activities that require maintaining balance, such as climbing stairs.
  5. Freezing: Functional Limitation: Mrs. Mbeki might have difficulty initiating/stopping walking, making turns or experience a sudden “freezing” of her feet while walking, which can increase the risk of falls and make it harder to move around safely.
51
Q

What are the signs and symptoms of postural hypotension in a patient with GBS? (3)

A

● Dizziness
● Blurred vision or double vision
● Fainting

52
Q

Why does postural hypotension occur more frequently in GBS? (2)

A

Autonomic disturbances are frequently seen in GBS due to the peripheral vasoconstriction, sensation loss and the fact that the autonomic system regulates blood pressure.

GBS disrupts the autonomic nervous system’s ability to regulate blood pressure effectively upon standing, leading to postural hypotension. By understanding this mechanism, healthcare professionals can take steps to manage postural hypotension and ensure the safety of patients with GBS during rehabilitation and daily activities.

53
Q

Discuss the precautions that a physiotherapist should take when mobilising a patient with GBS and postural hypotension. (5)

A

● Dont directly move to standing. Move from supine - fowlers - sitting - leaning forward - standing
● Ask pt if they are feeling dizzy or lightheaded or nauseous
● Have a chair ready if you do mobilise
● Stand close to the patient in case they faint or fall

54
Q

A patient with GBS may present with residual paralysis. Which four nerves are likely to be affected and why? (5)

A

All these nerves pass through an entrapment

Median nerve – Carpal tunnel

Ulnar nerve – Ulnar sulcus

Peroneal nerve – Fibula head

Distal branches of tibial nerve – Tarsal tunnel

55
Q

Mrs Nkomo was a passenger in a taxi when it was involved in a head-on collision with

another taxi three months ago. She sustained a fracture dislocation of T12/L1 and

presents with a complete ASIA A spinal cord injury. She has extensor spasms in the lower

limbs and she still has poor static sitting balance. She can roll and come from lying to

sitting.

Briefly discuss how you would improve Mrs Nkomo‘s poor static sitting balance. (10)

A

When starting to work on static sitting balance you would have to work on both long sitting and high sitting balance as the pt will need this to perform certain ALDs. In a sitting position a patient will need their hands free to perform ADLs.

Firstly assess how the pt is maintaining balance (do they place their hands behind them or next to them to maintain balance while sitting) also how long is the pt able to sit independently without falling over or losing balance. This will give them a reference point to measure improvement. In terms of their hand placement to maintain balance the first aim of treatment will be to move their hands to the edge of the plinth. When bringing the hands forward the pt’s centre of gravity is within their base of support and will aid in balance. This will build confidence and aid to overcome the fear of falling. Start with hands behind back and move them to the front of the plinth. The patient alternates between these two positions with the PT behind them to prevent them from falling. When hands are moved to the front to the plinth the exercise can be progressed to one hand in the air while maintaining sitting balance and then both.

When in high sitting prop the pt feet into a supported 90 degree angle, place a mirror in front of them, while you as PT hold up their upper body. Give less and less support until pt finds their balance. Start off with their hands holding on to the plinth in front of them, then hands on lap then arms crossed over the chest. Create a goal with the pt (2 min) they must try to reach 2 min of independent sitting. Then progress the exercise to change in hand position and then let the pt sit on less compliant surfaces like a mattress or a balance pad or a pillow.

Weight shift exercises will help with proprioception and finding midpoint. Have the pt weight shift from left to middle to right. Start these exercises with facilitating the movement and the pt using their arms as contact points on the plinth, then progress to the pt performing this exercise independently while still using their hands. The pt should finally be able to perform this exercise by weight shifting from side to side without any help from their upper limbs.

Equilibrium reaction exercises. The pt sits (either in long sitting or high sitting) with a mirror in front of them. The PT applies forces to the pt from different angles to disturb their equilibrium and the pt should gain their balance back with balance strategies. Start with small forces and then progress to big forces.
Stretching exercises stretching of the hamstring muscles via active or passive will allow the pt to have more range of movement and will also assist in balance in the long sitting position.

Lean out of BOS and return. This exercise leads more to improve dynamic sitting balance however it is useful for the pt to have when performing ALDs in sitting. Have the pt reach for objects out of their base of support and place the objects in different levels. This allows pt to reach out of BOS and allows them to find their balance again when returning. This also helps to overcome fear of falling.

Rhythmic stabilisation: This will help in engaging core and trunk muscles for central stability.

56
Q

Discuss the physiotherapy management of two orofacial problems that a patient with motor neuron disease(progressive paralysis due to loss of motor neurones) and pseudobulbar palsy is likely to present with. (8)

A

Motor neuron disease/Amyotrophic lateral sclerosis/Lou Gehrig’s disease is linked to glutamate related excitotoxicity causing progressive loss/degeberation of anterior horn cells (LMN) accompanied by astrocytic gliosis (astrocytes explode), and pseudobulbar palsy is caused by progressive loss/degeneration of corticobulbar tracts (UMN). UMN-brisk reflexes, spasticity; LMN-reduced reflexes, flaccidity.

Difficulty Swallowing can result in:

Drooling which may present problems with oral hygiene

Aspiration

Difficulty ingesting food- poor nutrional state which may need to use a nasogastric tube

physiotherapy can refer to Speech therapy, OT and dietician

Facilitate with two fingers up and down movement with swallowing bolus. PT to use index thumb at Adam’s apple

Facial expression

Raising eyebrows

Closing eye

Retracting the lip-for swallowing

Ventilation-incomplete glottic closure

PT mx: Positioning in semifowlers or upright sitting

ACBT with focus on BC

Motor aspects of speech production

Refer to ST

Tearing due to lax lower eye lid-causing dry eye which results in eye problems

Droolling-mx includes

Cold microfibre cloth moving UP+DOWN+LEFT+RIGHT

Lemon water/juice, or something sour

Mouth exercises

56
Q

Discuss the role of a physiotherapist in the management of a patient at the end stages of Parkinson’s disease. (7)

A
  1. Hoehn and Yahr 1

Self-management support

Prevent inactivity

Prevent fear to move or fall

Improve physical capacity

Reduce pain

Delay onset activity lmitations (motor learning, up to HY3)

2.Hoehn and Yahr 2-4

Maintain or improve activities, especially:

Transfers

Balance

Manual activities

gait

  1. Hoehn and Yahr 5

Maintain vital functions

Prevent pressure sores

Prevent contractures

Support carers/nurses

57
Q

Mrs Martins was diagnosed with multiple sclerosis (MS) one year ago. She travels to work via public transport (walks 2km from home to the taxi rank) and works as a cook in a hospital kitchen. Her condition has been following an exacerbation and remission course. She has come to you for treatment and advice now just after she had another exacerbation. She has spasticity, weakness in her lower limbs and fatigue.

Explain possible causes of fatigue after MS and how can you assist her to manage her fatigue. (13)

A

Causes
● Working against spastic limbs which increases energy expenditure
● Muscle contraction increases body temperature which increases hydrolysis of ACh and there reduced motor unit recruitment= fatigue and weakness
● Decreased nerve conduction due to decreased internodal distance
● Weakness and poor endurance due to immobility and deconditioning
Management:
● Managing/reducing spasticity: relaxation techniques, stretching, weight bearing.
● Cardiovascular fitness and strength training program that is specific to patient- include breaks in between, deep breathing exercises. This is to prevent deconditioning.
● Education on triggers and energy conserving techniques such as walking for shorter distances.
● Staying and working in cooler environments, taking cold showers to regulate temperature.

58
Q

Define neural plasticity. (2)

A

The ability of cells to undergo alterations in their form and function depending on environmental influences.
The ability of the brain to change and adapt by forming new connections with neurons or reorganising current neural networks based on external environmental stimuli and learning.
Neural plasticity refers to the brain’s ability to adapt and change its structure and function throughout life.
● Can be induced by manipulating the periphery to invoke changes in target neurons’ environment

Mechanisms of neural plasticity.
● recruiting of silent/latent synapses
● synaptic hyper effectiveness/potentiation
● synaptic reclamation/recovery
● axonal or collateral sprouting
● terminal regeneration (slow)
● denervation supersensitivity

For example, after a stroke damages brain tissue, the brain can reroute nerve signals around the injured area. This allows for some recovery of lost functions, like movement or speech.

59
Q

Describe two possible determinants of increased muscle tone in the lower limbs post spinal cord injury. (4)

A

Degree of motor unit activation > spinal stretch reflex activity
Intrinsic elastic properties of muscle, tendon and connective tissues
Environmental and sensory input

60
Q

Explain the phasic stretch reflex. (3)

A

The phasic stretch reflex, also known as the myotatic reflex or deep tendon reflex, is a reflexive response of a muscle to a sudden stretching or lengthening stimulus. The phasic stretch reflex is a protective mechanism that operates at the spinal cord level to quickly and involuntarily contract a muscle in response to a sudden stretch, helping to maintain muscle tone, joint stability, and overall motor control.
1. Rapid Muscle Contraction: When a muscle undergoes a sudden stretch or lengthening, specialized sensory receptors called muscle spindles within the muscle are activated which detect changes in muscle length and initiate the phasic stretch reflex. The sensory information from the muscle spindles is transmitted to the spinal cord via sensory neurons.
2. Monosynaptic Reflex Arc: The phasic stretch reflex follows a monosynaptic reflex arc, meaning that it involves a direct communication between the sensory and motor neurons in the spinal cord without involvement of the brain. The sensory neurons carry the information from the muscle spindles to the spinal cord, where they synapse with motor neurons that innervate the same muscle. This direct connection allows for a quick and automatic response to the stretching stimulus.
3. Muscle Contraction and Reflexive Movement: Upon receiving the sensory input indicating muscle stretch, the motor neurons in the spinal cord are activated. The motor neurons then send signals back to the muscle, causing it to contract. The reflexive muscle contraction helps to resist the stretch and stabilize the joint involved. The phasic stretch reflex plays a role in maintaining posture, protecting against overstretching, and ensuring the stability and integrity of the musculoskeletal system.

61
Q

Mrs Smith was diagnosed with Parkinson’s disease two years ago. She is able to walk independently but for short periods of time and uses a wheelchair for part of the day. She is attending physiotherapy and complains of difficulties coming out of the wheelchair when at home.

3.1 Explain to Mrs Smith why she is struggling with getting out of the wheelchair. (3)

A

Mrs Smith might be struggling to get out of the w/c for various reasons.
She might be struggling with the sequencing of the movement to go from sit to stand out of the wheelchair.
She might have muscle weakness from disuse in her lower limbs that causes her to have fatigue after walking and then when she wants to get out of the w/c again she is too tired to.

62
Q

Discuss the physiotherapy management of Mrs Smith at this stage. (7)

A

Strength:
● Strengthening using PNF techniques, weights, mat exercises, hydrotherapy, etc.
Bradykinesia, akinesia, hypokinesia:
● Rhythmic initiation and slow reversals
● Use of external stimuli such as watching the legs of someone else
● Use of momentum to complete movements such as sit to stand
● Gait initiation using long high steps
● Trunk mobilisation
● Breaking tasks into small components
● Avoid simultaneous tasks
● Conscious movements
Flexed posture:
● Prone positioning
● Shoulder horizontal abduction in prone
● Encourage pt to sleep in prone
● Hanging from an overhead bar for short periods
● Mirror to correct posture
Balance:
● Weight transfers progressed to balance reaction training
● Repetition
● Reaching for objects out of BOS
● Dressing training
Freezing:
● Conscious actions:
○ Count steps out loud
○ High steps
○ Stamp their feet
● Look at something with a pattern on it
Rigidity:
● Slow rhythmic trunk rotations
● Stretching to maintain muscle length
○ Passive or active
○ Teach self stretching
● Relaxation techniques