Neuro 3 Flashcards

1
Q

Which CNs are sensory only?

A

1,2,8
**therefore no LMN signs

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2
Q

Where do CNs originate from?

A

From cerebrum or brainstem
-ipsilateral signs (no crossing over)

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3
Q

Which CN is the only one that does not go through the thalamus?

A

CN I

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4
Q

CN II

A

Optic nerve
-issues if Blind, loss of menace or loss of PLR
-often circle towards side of lesion?

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5
Q

CN III

A

Occulomotor nerve

-deficits= dilated pupil, loss of PLR

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6
Q

CN V

A

Trigeminal Nerve
-deficits= decreased jaw tone, atrophy of mastication muscles, loss of facial sensation

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7
Q

CN VII

A

Facial nerve

Deficits:
-drooped face
-loss of blink
-loss of lip twitch, ear twitch
-decreased tear production

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8
Q

CN VIII

A

Vestibulocochlear nerve

deficits:
-loss of balance
-head tilt
-loss of hearing

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9
Q

CN IX and CN X

A

9= glossopharyngeal
10=vagus

deficits:
-dysphagia
-loss of gag reflex
-laryngeal paralysis

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10
Q

CN XII

A

Hypoglossal nerve

deficits:
-loss of tongue strength
-tongue atrophy
-tongue deviation to strong non lesion side (contralateral)

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11
Q

Horners syndrome

A

-loss of Sympathetic NS input to eye
- +/- miotic pupil
- +/- 3rd eyelid prolapse
-ptosis (small palpebral fissue)
-enophthalmous (sunken eye)

-peripheral vasodilation on side of the lesion (ear, nose, neck) = increased sweat
*in horses, lack on innervation= reduced sweating

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12
Q

3 parts that are affected in horners syndrome

A
  1. First order
    -rare. intracranial or cervical cord lesions
  2. Second order (preganglionic)
    -T1-T3, brachial plexus, nerve roots T1-T3, cranial mediastinal mass, cervical soft tissue trauma, neoplasia
  3. Third order (postganglionic)
    -otitis media/interna, neoplasia middle ear, retrobulbar injury or neoplasia
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13
Q

Whats the forebrain?

A

cerebral cortex and diencephalon (thalamus/hypothalamus)

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14
Q

Signs of a cerebral cortex/forebrain lesion

A

-seizures **almost always
-altered behaviour/mentation
-cortical blindness (opposite)
-diminished facial sensation (opposite)
-may circle/pace towards lesion
-inconsistent/mild loss of proprioception on side opposite of lesion
-gait may be normal
-hemi-neglect (hypoalgesia/blindness)

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15
Q

Signs of a brainstem lesion

A

-altered mental state (depression, stupor, coma)
-ipsilateral proprioceptive ataxia and UMN hemiparesis
-multiple cranial nerve deficits
-+/- vestibular signs

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16
Q

Lesions of vestibular system

A

-head tilt
-balance problems
-vestibular ataxia
-nystagmus
-proprioceptive ataxia if brainstem disease

17
Q

Vestibular disease

A

-central and peripheral
-head tilt towards lesion
-spontaneous nystagmus at rest (fast phase away from side of lesion) = horizontal or rotary
-nystagmus may get worse or only be apparent with changes in body position (positional nystagmus)
-vomiting
-motion sickness

18
Q

Peripheral vestibular disease signs

A

-nystagmus always horizontal or rotary and does not change direction with changed head position

-posture and proprioception is normal

-other CNs are not affected. EXCEPT can see concurrent horner’s and/or CN7 paralysis if middle/inner ear disease

19
Q

Central vestibular disease

A

-vertical nystagmus; changes direction as change in head position
-abnormal postural reactions/proprioception on same side as lesion
-other cranial nerves (except 7) affected

20
Q

Signs of cerebellar disease

A

-mentally normal
-strong, hypermetric
-exaggerated limb responses; goose stepping
-loss of fine tuning or motor control
-may see head tremor or intention tremor

21
Q

Paradoxical vestibular disease

A

Head tilt towards the normal side (opposite of the lesion)

-occurs because cerebellar vestibular nuclei should inhibit brainstem vestibular nuclei and if a cerebellar lesion is present then inhibition so normal side has less activity than abnormal side

22
Q

Paradoxical vestibular disease signs

A

-head tilt to one side

-postural reaction deficits and other CN deficits on opposite side (due to brainstem lesion)

-lesion on the side with postural reaction deficits and CN deficits (side opposite head tilt)