Nerve and Tendon Injuries Flashcards

1
Q

How are tendons arranged macroscopically?

A

Components

  • Muscle origin (from bone)
  • Muscle belly
  • Musculotendinous junction
  • Tendon ± Sesamoid bone (e.g. patella) ± Tendon sheath
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2
Q

What are the different macroscopic layers found in tendons?

A

Fascicles of long spiralling collagen bundles

  • Endotenon - cover COLLAGEN BUNDLES
  • Epitenon - cover TENDON
  • Paratenon - fills space between tendon and its sheath
  • Tendon sheath - Connected to tendon by vincula
    • Synovial lining + fluid - allows gliding lubrication and nutrition
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3
Q

Why is movement so important for tendon health?

A

Immobility reduces

  • Water content
  • Glycosaminoglycan concentration and strength

Weakens tendon!!!

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4
Q

Can give some examples of types of tendon injuries?

A
  • Degenerative - e.g Achilles tendon
  • Inflammatory - e.g. De Quervan’s Tenosynovitis
  • Enthesiopathy - e.g. Tennis elbow
  • Traction apophysitis - e.g. Osgood Schlatter’s disease
  • Avulsion +/- bone fragment - e.g. mallet finger
  • Tear - Intrasubstance (Achilles), musculotendinous junction (plantaris syndrome)
  • Laceration/Incision
  • Crush/Ischaemia
  • Nodules - e.g. trigger finger
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5
Q

What is an apophysis?

A

A layer of bone over a growth plate; an area of structural weakness in a growing child or adolescent

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6
Q

What is traction apophysitis?

A

Powerful Tendons attaching to apophyses can cause chronic traction injuries. Once growth plates fuse, the problem will disappear

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7
Q

How do tendons heal?

A
  • Initiated by fibroblasts (from epitenon) and macrophages
  • 3 phases - inflammatory, fibroblastic (collagen-producing), remodelling
  • Weakest - 7-10 days
  • Most of original strength - 3-4 weeks
  • Max Strenght - 6 months
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8
Q

What are the basic principles of tendon rehabilitation?

A
  • Early movement (stress) - increases healing & strength, & reduces adhesions
    • Active
    • Passive
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9
Q

What are the different layers within the macrostructure of nerves?

A
  • Endoneurium - covers AXONS
  • Perineurium - covers FASCICLES (nerve bundles)
  • Epineurium - covers NERVE
  • Myelin sheath - surrounds neuron (Schwann cell, oligodendrocyte)
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10
Q

What type of nerve injuries can occur?

A
  • Neurapraxia
  • Axonotmesis
  • Neurotmesis
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11
Q

What is neurapraxia?

A
  • Reversible conduction loss
  • “Nerve in Continuity” - Axon remains intact, but myelin has been damaged
  • Often caused by local ischaemia and demyelination - e.g. compression
  • Prognosis good - weeks or months
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12
Q

What is Axonotmesis?

A
  • “Tube in Continuity” - Epineurium intact but disruption of axon continuity
  • Caused by stretching, crush or direct blow
  • Wallerian degeneration - follows injury; part of the axon separated from the neuron’s cell body degenerates distal to the injury - occurs with nerve fibre crush or cut
  • Fair Prognosis - sensory recovery>motor recovery
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13
Q

What is neurotmesis?

A
  • Complete nerve division
  • Caused by Laceration or avulsion
  • Endoneurial tubes disrupted - no guidance for nerves to repair, unlike axonotmesis
  • Prognosis poor - no recovery unless surgically repaired
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14
Q

What is dysasthesia?

A

Disordered sensation

  • Anaesthetic (numb)
  • Hypo- & hyper-aesthetic
  • Paraesthetic (pins & needles)
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15
Q

What is paresis?

A

Muscular weakness

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16
Q

How do nerves heal?

A

Very Slow

  • Proximal axonal budding occurs - after about 1 month delay
  • Regeneration - 1 mm/day or 1 inch/month
  • Pain returns first
  • Prognosis depends whether nerve is
    • Pure - only sensory or only motor
    • Mixed - both sensory and motor within same nerve
    • Distal/Proximal - proximal worse
17
Q

What is wallerian degeneration?

A

A process that results when a nerve fiber is cut or crushed, in which the part of the axon separated from the neuron’s cell body degenerates distal to the injury.

18
Q

How does achilles tendon rupture occur?

A
  • Pushing off with weightbearing forefoot whilst extending knee joint
  • Unexpected dorsiflexion of ankle
  • Violent dorsiflexion of plantarflexed foot
19
Q

Who is more at risk of Achilles tendon rupture?

A
  • Males
  • 30-40yrs old
20
Q

How does achilles tendon rupture present?

A
  • Sudden Pain + Popping Sound - in achilles area - feel like they’ve been hit in the heel
  • Palpable tender gap - particularly in first 24 hrs
  • Unable to heel raise
  • Positive Simmonds Test
21
Q

How would you investigate suspected achilles tendon rupture?

A

Clinical Diagnosis - simmond’s test

Imaging

  • MRI
  • X-ray - check for fractures if suspected?
22
Q

How would you manage someone with a ruptured achilles tendon?

A

Acute referral to orthopaedics

Conservative

  • PoP cast in equinus position - 10 wks

Operative

  • Open repair + earlier mobilisation - Re-rupture rate lower
23
Q

What is a vinculum?

A

Band of connective tissue, similar to a ligament, that connect a flexor tendon to a phalanx bone. They contain tiny vessels which supply blood to the tendon

24
Q

Risk factors for achilles tendon rupture

A
  • quinolone use (e.g. ciprofloxacin) is associated with tendon disorders
  • hypercholesterolaemia (predisposes to tendon xanthomata)