Musculoskeletal Flashcards
hormonal disturbances that alter bone function: calcitonin, estrogen, parathyroid, vitamin D
calcitonin- inhib osteoclasts (bone-crushing) (prevent bone breakdown) (inc calcitonin= stronger bones)
estrogen- stim osteoblast (bone building0
parathy- cause bones release Ca (can be weakened if overactive)
vitamin D- enhance Ca absorb
weight-bearing exercises
promote bone density (not incl swimming)
adl v iadl
adl- grooming, toileting, bathing, dressing and eating
iadl- instumental (phone, meds, shopping, transpor, meal prep and housework)
physical assessment
CMS posture, gait, joint mobility/abnormal pulse assessment sensation muscle tone/ strength curvature of spine
cms assessment
circulation- pulses, cap refil, temp and color
movement- paralysis?
sensation- inc pain, assess for paresthesia
lab- Ca
8.5-10.5 mg/dl
source- bones
indicates bone integrity or density
lab- phosphorus and vitamin d
2.5-5 mg/dl
d- 35-40 ng/ml
assess bone health, rely on renal system
kidney failure- dec Ca absorb (kidneys activate vitam D)
sources phos- nuts and meat (anything high in protein)
diary/ leafy greens
lab- estrogen
dec estrogen after menop inc risk fx
assessed through urine
xray
bone density, degen dis, joint irreg, spurs
CT
muscle and bone disorders (tumors and fx)
MRI
disk dis
osteomyelitis and ligamentous tears
soft tissue dis (musc, tendon, nerves, fat)
use of contrast dye
see vascularity of areas (ex tumor)
DEXA
bone mineral density study for id of osteopenia/ osteoporosis monitors trtmnt T score > -1 = normal < -2.5=OA
bone scan
find bone damage, dis or infection
radioactive IV
cold spots- cancer
hot- infection
role of aging on bones
dec bone mass and mineralization around age 30
dec ca avaliable
joint cartil dec (inc fx)
CT ligaments/tendons lose water content become more rigid
sarcopenia
dec muscle mass and strength
factors for fall risk
eyesight, dec coord, balance, polypharm, mentation, sensation, home environment
Osteoporosis- def
deterioration of bone tissue and density
Osteoporosis- risk factors
genetics- female, sm frame, white, asian
age- post meno females, low testosterone
low Ca, vitam D and phosp
low physical, non-weight bearing exercise
caffine, alcoh, smoking (nicotine impairs absorb zca, dec osteob fun and dec blood supply bones)
meds- corticosteroids, anti-seizure, heparin and thyroid
malabsorption (bariatric surgery), renal failure and hyperthyroidism
recommended Ca and vitamin D
Ca 1200mg
D- 800-1,000 (sunlight, supplements)
osteopor- patho
osteoclastic act> osteoblastic act
calcitonin (protective)dec
estrogen lvls dec (dec bone formation)
inc parathyroid (inc osteoclastic activity)
osteop- clincial manif
silent dis fx occur in spine (compression)= change height, posture progressive curvature of spine kyphosis- rounding in cervial lordosis- inward curving of lumbar spine
osteopor- prevention
prevention
FRAX- fracture risk assessment
labs- serum ca, phos, vit D, tesosterone (men)
if Ca abn then PTH
osteop- management
weight b exercise
vit d and ca supplements (post meno higher dose than pre)
meds slow bone absorption (bisphosphonates)
estrogen replacement
serms (selective estrogen receptor modulators)
calcitonin (dec bone resorption, not taken chronically)
bisphosphonates
inhib bone breakdown (stop osteoc)
serms
mimic estrogen effects
*better than direct estrogen (risk MI, stroke, breast ca, dvt)
selective estrogen recep modulators
low back pain- patho
lumbar region inc P from poor body mechanics most flexible portion of spine l4-5 and l5-s-1 common w/ age as discs degen
LBP- risk
occupations, obesity, age
radiculopathy
pain radiating down extremities form area of back affected
disc abnormal
degen- genetic, metab change assoc w/ aging, repeated stress or trauma (shrinks in size)
bulge- swelling
herniated- tears or cracks of disc (inc P on nerve root)
thinning- dec h2o and protein w/ aging