Muscle Tone Flashcards

1
Q

What causes spasticity?

A

Hyper excitability of stretch reflex

  • UMN lesion; descending motor pathways from the cortex (pyramidal tracts) or BS (M and L vestibulospinal tracts, dorsal reticulospinal tract)
  • corticospinal tracts- moreso lateral
  • lesion produces disinhibition of spinal reflexes with hyperactive tonic stretch reflexes or a failure of reciprocal inhibition
  • Hyperexcitability of alpha motor neuron pool
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2
Q

What Dx’s do you most often see spasticity?

A
  1. CP
  2. MS
  3. SCI (even at same level)
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3
Q

What is chronic spasticity associated with?

A
  1. contracture
  2. abnormal posturing
  3. deformity
  4. functional limitations
  5. disability
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4
Q

during rapid movement, initial high resistance (spastic catch) followed by a sudden inhibition or letting go of the limb (relaxation) in response to a stretch stimulus

A

Clasp-knife response

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5
Q

cyclical, spasmodic alternation of muscular contraction and relaxation in response to sustained stretch of a spastic muscle

A

clonus

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6
Q

how do you elicit ankle clonus? how do you stop it?

A

quick stretch of PF (put into ankle DF)

  • leg in slight flexion
  • document if any beats are present, if you can count them, how many
  • stop = sustained pressure on the foot
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7
Q

Resistance to passive movements involving both agonist and antagonist muscles; both directions whether or not the muscle is on stretch

A

rigidity

- observed in PD

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8
Q

Indicates Corticospinal lesion in the BS btwn the Superior Colliculus and the Vestibular Nucleus

A

Decerebrate

- abnormal extensor posturing

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9
Q

Indicates Corticospinal Tract lesion at the level of the diencephalon (above the superior colliculus)

A

Decorticate

- abnormal flexion posturing

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10
Q

Impaired tone; Hyperkinetic movement disorder characterized by tone and involuntary repetitive twisting movements in large portions of body

A

Dystonia

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11
Q

What are the causes of dystonia?

A
  1. CNS lesion - Usually BG
  2. Genetic (Primary Idiopathic Dystonia)
  3. Neurodegenerative Disorders - PD; Wilson’s Disease = if on excessive l-DOPA
  4. Metabolic Disorders - Amino Acid or Lipid disorders
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12
Q

What are they types of dystonia?

A
  1. Focal = spastic torticollis, isolated writer’s cramp

2. Segmental = toritcollis, dystonic posturing of the arm

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13
Q

Diminished resistance to passive movement; Decreased or absent stretch reflexes; Hyperextension of joints common

A

hypotonia

- LMN lesion and spinal shock

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14
Q

What are the causes of hypotonia?

A
  1. lesions of ANT horn cells
  2. PN Lesions - Peripheral neuropathy, Cauda Equina Lesion. andRadiculopathy
  3. Cb Lesions = can produce mild decreases in tone and weakness
  4. UMN Lesions = cause temporary hypotonia = Spinal Shock or Cerebral Shock, depending on lesion location
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15
Q

When there is a problem with tone, what area of the brain is often involved?

A

BG

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16
Q

What is tone affected by?

A
  1. Medication*
  2. Environmental temperature*
  3. State of CNS (arousal and alertness)* – esp anxiety – if pt has this then can definitely increase spasticity
  4. Volitional movement
  5. Stress and anxiety
  6. Position
  7. Health
17
Q

What is the procedure for assessing tone?

A
  1. Palpation of the muscle belly
  2. Passive motion and speed up as you go
  3. All limbs/joints
  4. Compare right and left
  5. Compare UE’s and LE’s
  6. Cue your pt to stay relaxed
18
Q

What are the levels on the modified ashworth scale?

A
0 = No increase in muscle tone 
1 = Slight increase in muscle tone, manifested by a catch and release or by minimal resistance at the end of the range of motion when the affected part(s) is moved in flexion or extension 
1+ = Slight increase in muscle tone, manifested by a catch, followed by minimal resistance throughout the remainder (less than half) of the ROM 
2 = More marked increase in muscle tone through most of the ROM, but affected part(s) easily moved 
3 = Considerable increase in muscle tone, passive movement difficult 
4 = Affected part(s) rigid in flexion or extension
19
Q

What are the grades for DTRs?

A
0 = absent
1+ = present but depressed
2+ = normal
3+ = brisk
4+ = very brisk, hyperactive, w/clonus
20
Q

Abnormal obligatory movements that emerge w/spasticity [w/someone w/CNS disorder]; Inability to perform isolated movements at each joint, and will move in a certain pattern

A

Synergistic pattern

  • Ex: Stroke – reach out to grab something, & elbow flexes towards trunk – can’t stop this
  • PT records when the patterns occur, under what circumstances, and what variations are observed; Under moments of increased stress, increased fatigue, etc
  • decrease in synergy = condition improving
21
Q

How can you test strength in neuro patients who cannot isolate movements?

A

observe active movements

  1. Squats
  2. Sit/stands
  3. Heel raises
  4. Toe raises
    - can they do it? how long? how far down can pt go? what does the movement look like?