Muscle structure & training principles Flashcards

1
Q

muscle structure

A

contractile proteins
- myosin: thick filaments with globular heads
- actin: thin filaments
(actin, tropomyosin, troponin)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

mechanisms of muscle contraction

A
  • Myosin head binds to an active site on actin (‘cross bridge’)
  • Myosin head moves pulling actin filaments towards the centre of the sarcomere (‘power stroke’)
  • Sacromere shortens, muscle shortens, force is generated
  • Cross bridges detach
  • Dependent upon sufficient Ca2+ and ATP
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

sliding filament theory

A
  1. ATP is hydrolysed when myosin head is unattached
    - troponin-Ca2+ complex pulls tropomyosin away, exposing myosin binding sites
  2. ADP + Pi are bound to myosin as myosin head attaches to actin
  3. ADP + Pi release causes head to change position and acting filament to move
  4. binding of ATP causes myosin head to return to resting position
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

role of Ca2+ in muscle contraction

A
  • At rest, myosin and actin are unable to bind d/t tropomyosin and troponin
    o Tropomyosin covers the binding sites on actin
    o Troponin holds tropomyosin in place
  • Ca2+ binds to troponin moves tropomyosin away to expose myosin binding site on actin
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

role of ATP in muscle contraction

A
  • Myosin head contains a binding site for ATP
  • ATP –> ADP + P = cross bridge + power stroke
  • A new ATP binds to myosin to release it from actin
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

excitation-contraction coupling

A

Sequence of events that begin with a neural impulse and end with contraction

  • Excitation of a motor nerve
  • Propagation of an action potential
  • Events at the neuromuscular junction
  • Calcium release from sarcoplasmic reticulum
  • Sliding Filament Theory
  • Muscle contraction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

muscle fibre types

A
  • Type I fibres (slow twich)
    -Type II fibres (fast twitch)
    o Type IIa
    o Type IIx (IIb)
  • Differences in speed of contraction, maximum force production, oxidative capacity, fatigability…
  • Power production is the key determinant of recruitment
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Type I

A
  • Relatively small in diameter
  • Slow contractile speed
  • Low force production
- High oxidative (aerobic) capacity
o Many mitochondria
o Many capillaries
o Lots of myoglobin
o Great aerobic enzyme activity
  • Highly resistant to fatigue
  • Dominant muscle fibre during exercise below CP
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Type II

A
  • Relatively large in diameter
  • Fast contractile speed
  • High force production
  • High glycolytic capacity
    o Lots of glycolytic enzymes
    o Greater glycogen and PCr stores
  • Highly fatigable
  • Recruited during high-intensity exercise
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Type IIa

A
  • Moderately well-developed oxidative capacity
    o Many mitochondria
    o Moderate number of capillaries
  • Gradually recruited during exercise bouts >CP
    o i.e. High intensity aerobic exercise <40min – 1hr. Typical anaerobic exercise bouts
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Type IIx(b)

A
- Largely anaerobic
o High contractile speed, force, power
o Great glycolytic capacity
o High concentration of CP
o Slight oxidative capacity
o Highly fatigable
  • Recruited during short (<1min), very high-intensity anaerobic exercise, when requirement for force and/or speed is high
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

distribution of fibre types

A
  • Skeletal muscle contain both type I and type II fibres however the proportion of these fibres differ
  • Postural muscles – higher proportion of type I
  • Power muscles – higher proportion of type II
  • e.g. Gastrocnemius vs. Soleus
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

determinants of fibre type

A
  • Genetics ~50%
  • Environment ~50%: Training, physical inactivity
  • Significant differences between individuals
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

assessment of fibre types (muscle biopsy)

A
  • Site is anaesthetised
  • Small incision is made (1-3cm)
  • Needle is inserted into the muscle
  • 10-100mg sample is extracted (~grain of rice)
    o Not too big to reduce impact on performance
  • Samples from metabolically active sites
    o e.g. deltoids in swimmers
  • Sample is stained to identify fibre type
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

purpose of training

A
- Improvements in health and performance
o Cardiorespiratory fitness
o Muscular strength and endurance
o Body composition
o Flexibility
o Speed
o Agility 
o Power
- Adaptation: Key to training
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

principle of training (3)

A
  • Specificity
  • Overload
  • Reversibility
17
Q

specificity

A

Responses to training are specific to the type of exercise, the muscle groups involved and the energy systems used

SAID principles
- Specific Adaptation to Imposed Demands
o Adaptations are specific to the stimulus
o No adaptation = no improvement
o Lack of specificity results in a reduced training stimulus

18
Q

overload

A

Training load must be at a level beyond which the athlete is accustomed for improvements in performance

  • For continued improvements to occur the athlete must be progressively overloaded
    o As fitness increases the body becomes more resilient to stress, recovery is faster, and initial training loads are highly achievable
    o To ensure continued improvement, the stimulus must increase (or vary)
  • Progressive overload is the key to achieving improvements without overtraining
19
Q

adaptation

A
  • Improvement is only possible if this sequence is followed:
  • Increasing stimulus (load) –> adaptation –> performance improvement
  • If the load is always the same adaptation occurs in the initial stages followed by a plateau
  • Same stimulus –> plateau –> lack of improvement
  • If the stimulus is excessive or overly varied the athlete will be unable to adapt (maladaption)
  • Excessive stimulus–> maladaption –> decrease in performance
20
Q

overload and fatigue

A

A properly designed program will allow for adequate recovery while imposing sufficient stress

21
Q

reversibility

A
  • Training adaptation is not permanent

- Training adaptations will decay once the stimulus has been removed

22
Q

monitoring training loads

A
  • The FITT principle
  • Training loads can be monitored (and modified) by assessing:
    o Frequency – how often
    o Intensity – how hard
    o Time – how long
    o Type – training method(s)
23
Q

training intensity

A
  • Energy expenditure or work per unit time
    o The effort invested into a training session
  • Often regarded as the most significant component of the training stimulus for applying overload
  • Important to monitor
24
Q

determining training intensity

A
- Methods
o % of VO2max
o % of CP or LT etc
o RPE
o HR (Most common and accessible, Monitoring HR is an indirect estimate of O2 consumption in exercise, Used to indicate the overload placed on the body)
  • The maximal heart rate method
  • Training heart rate (THR) is calculated from maximal HR only
    o Maximum HR ~220 – age
    o THR = % of maximum HR
  • For trained athletes, THR for endurance training should be between 85-95% HR max
  • Lower for recreational athletes
25
Q

Karvonen’s HR reverse method (training intensity)

A

HRR = difference between resting and maximal HR
o HRR = HR max – HR rest
o Training HR (THR) can be determined as a % or HRR plus resting HR (Should be at least 60% HRR + HR rest)

  • e.g. HR max = 200 bpm HR rest = 60 bpm
  • HRR = 200-60= 140 bpm
  • 60% of 140 = 84
  • THR = 84+ HR rest (60) = 144bpm
    o For trained athletes: 80-85% HRR + HR rest