adaptations to aerobic exercise Flashcards

1
Q

acute responses to aerobic exercise

A
  • Ventilation: Increases d/t neural input and feedback from central and peripheral receptors
  • Heart Rate: Increases d/t reduced PNS influence and increased sympathetic drive and hormonal effects
  • Stroke Volume: Increases d/t increased SNS drive (increased contractility) and increased EDV
  • Blood Pressure
    o SBP increases d/t increase in cardiac output
    o DBP remains unchanged or may decrease
    o slightly d/t vasodilation
  • Redistribution of blood flow
  • Oxygen consumption
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2
Q

O2 deficit responses to aerobic exercise

A

O2 deficit is the difference between the oxygen requirement and the oxygen utilistation at the commencement of exercise.
- EPOC. Excess Post Exercise Oxygen Consumption.
o Increased oxygen consumption due to increased metabolic demand post exercise (Increased HR and ventilation, Replenishing Mb stores, Increased body temperature, Anabolic processes)

  • How does training affect O2 deficit?
  • How does EPOC change with exercise intensity?
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3
Q

aerobic training

A
  • Improves the ability to sustain a particular level of physical effort
  • Occurs via improvements in functional capacities related to oxygen transport and utilisation
  • Fick equation VO2 = Q x a-vO2 difference
  • Central adaptations: Increase Q
  • Peripheral adaptations: Increase a-vO2 difference
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4
Q

central adaptations (HR and SV)

A
- How do we increase Q?
Heart Rate
o Reduced at rest and during
o submaximal exercise
o Training increases vagal tone 
o Lower cardiac O2 cost
o Permits greater filling time

Stroke volume
- Increases with training d/t increased left ventricular volume and mass
- Increased SV
o Load placed on the heart during contraction (EDV (Preload), Afterload = pressure in the wall of the left ventricle during ejection, i.e. higher blood pressure)

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5
Q

central adaptations (plasma volume and blood flow)

A

Plasma volume

  • Increase of ~10-20% following 3-6 aerobic training sessions
  • Increased EDV
  • Increased SV
  • Temperature regulation
  • Subsequent increase in Hb

Blood flow
- Distribution of blood flow to more oxidative muscle fibres
- Reduced splanchnic and renal blood flow
- Increase in skeletal muscle blood flow
o d/t increase in cardiac output and increase in cross-sectional area of capillaries

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6
Q

peripheral adaptations

A
Mitochondria 
- Increase size and number
- Increased oxidative enzymes in the mitochondria
o e.g. citrate synthase
o d/t tissue hypoxia

Capillaries
- Increased number of capillaries
o d/t increased shear stress (pressure) on capillaries
- Results in increased O2 delivery
o Increased transit time
o How does this increase muscle oxygenation?

Fibre type
Type IIx –> Type IIa –> Type I

Myoglobin
- Possibly small increase in intramuscular myoglobin content

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7
Q

adaptations to aerobic training (response determined by)

A
  • Magnitude of response is determined by
    o Genetics
    o Previous training status
    o Training load (Training load = Training intensity x volume)
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8
Q

aerobic performance (Determined by)

A

Determined by
- VO2 max, Critical Power, Economy

VO2max
o The highest rate that oxygen can be taken up and used by the body during exercise
o A result of both central and peripheral adaptations to training

Anaerobic threshold
o Power Speed/pace/power at “threshold” is perhaps the most important determinant of endurance performance

o Closely linked terms: Critical power, Ventilatory Threshold, Lactate Threshold, MLSS (Maximum Lactate Steady State)

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9
Q

when does anaerobic threshold occur

A

o ~40-60% VO2max in untrained individuals
o ~70-75% VO2max in trained individuals
o Up to 80-90% VO2max in elite marathon runners
o Up to 85-90% VO2max in elite distance swimmers
o ~60-85% VO2max in sprint athletes

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10
Q

critical power

A
  • Defn: Maximum Intensity at which work can be sustained indefinitely
  • But practically ~60mins
  • Critical Power (CP)
  • Note: W’ is NOT AWC as shown in the diagram
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11
Q

ventilatory threshold

A

Intensity of exercise where a non-linear increase in VE is observed
o inc CO2
o inc H+

CO2 + H2O H2CO3 HCO3- + H+

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12
Q

lactate threshold

A
  • Intensity of exercise where lactate begins to accumulate in the blood
    o Lactate production outstrips utilisation
    o Associated with inc H+

training causes threshold to occur at a higher work rate

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13
Q

implications for performance

A
  • Why is CP/ AT a more important determinant of performance than VO2max?
  • Consider 2 athletes with the same VO2max (4.0 L.min-1)
  • AT of athlete A = 70% of VO2max and athlete B = 85% of VO2max
  • Athlete B will be able to maintain a higher work rate before H+ and CO2 begin to accumulate in the blood
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14
Q

training to improve aerobic threshold

A

Largely peripheral adaptations

  • Increased capillary density
  • Increased mitochondria
  • Changes in LDH and PDH enzymes (Decreased lactate production)
  • Increased myoglobin content
  • Increased fat utilisation

Train at ~AT for prolonged periods of time

  • Often referred to as “tempo” or “threshold” training
  • Be careful with volume
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