adaptations to aerobic exercise Flashcards
acute responses to aerobic exercise
- Ventilation: Increases d/t neural input and feedback from central and peripheral receptors
- Heart Rate: Increases d/t reduced PNS influence and increased sympathetic drive and hormonal effects
- Stroke Volume: Increases d/t increased SNS drive (increased contractility) and increased EDV
- Blood Pressure
o SBP increases d/t increase in cardiac output
o DBP remains unchanged or may decrease
o slightly d/t vasodilation - Redistribution of blood flow
- Oxygen consumption
O2 deficit responses to aerobic exercise
O2 deficit is the difference between the oxygen requirement and the oxygen utilistation at the commencement of exercise.
- EPOC. Excess Post Exercise Oxygen Consumption.
o Increased oxygen consumption due to increased metabolic demand post exercise (Increased HR and ventilation, Replenishing Mb stores, Increased body temperature, Anabolic processes)
- How does training affect O2 deficit?
- How does EPOC change with exercise intensity?
aerobic training
- Improves the ability to sustain a particular level of physical effort
- Occurs via improvements in functional capacities related to oxygen transport and utilisation
- Fick equation VO2 = Q x a-vO2 difference
- Central adaptations: Increase Q
- Peripheral adaptations: Increase a-vO2 difference
central adaptations (HR and SV)
- How do we increase Q? Heart Rate o Reduced at rest and during o submaximal exercise o Training increases vagal tone o Lower cardiac O2 cost o Permits greater filling time
Stroke volume
- Increases with training d/t increased left ventricular volume and mass
- Increased SV
o Load placed on the heart during contraction (EDV (Preload), Afterload = pressure in the wall of the left ventricle during ejection, i.e. higher blood pressure)
central adaptations (plasma volume and blood flow)
Plasma volume
- Increase of ~10-20% following 3-6 aerobic training sessions
- Increased EDV
- Increased SV
- Temperature regulation
- Subsequent increase in Hb
Blood flow
- Distribution of blood flow to more oxidative muscle fibres
- Reduced splanchnic and renal blood flow
- Increase in skeletal muscle blood flow
o d/t increase in cardiac output and increase in cross-sectional area of capillaries
peripheral adaptations
Mitochondria - Increase size and number - Increased oxidative enzymes in the mitochondria o e.g. citrate synthase o d/t tissue hypoxia
Capillaries
- Increased number of capillaries
o d/t increased shear stress (pressure) on capillaries
- Results in increased O2 delivery
o Increased transit time
o How does this increase muscle oxygenation?
Fibre type
Type IIx –> Type IIa –> Type I
Myoglobin
- Possibly small increase in intramuscular myoglobin content
adaptations to aerobic training (response determined by)
- Magnitude of response is determined by
o Genetics
o Previous training status
o Training load (Training load = Training intensity x volume)
aerobic performance (Determined by)
Determined by
- VO2 max, Critical Power, Economy
VO2max
o The highest rate that oxygen can be taken up and used by the body during exercise
o A result of both central and peripheral adaptations to training
Anaerobic threshold
o Power Speed/pace/power at “threshold” is perhaps the most important determinant of endurance performance
o Closely linked terms: Critical power, Ventilatory Threshold, Lactate Threshold, MLSS (Maximum Lactate Steady State)
when does anaerobic threshold occur
o ~40-60% VO2max in untrained individuals
o ~70-75% VO2max in trained individuals
o Up to 80-90% VO2max in elite marathon runners
o Up to 85-90% VO2max in elite distance swimmers
o ~60-85% VO2max in sprint athletes
critical power
- Defn: Maximum Intensity at which work can be sustained indefinitely
- But practically ~60mins
- Critical Power (CP)
- Note: W’ is NOT AWC as shown in the diagram
ventilatory threshold
Intensity of exercise where a non-linear increase in VE is observed
o inc CO2
o inc H+
CO2 + H2O H2CO3 HCO3- + H+
lactate threshold
- Intensity of exercise where lactate begins to accumulate in the blood
o Lactate production outstrips utilisation
o Associated with inc H+
training causes threshold to occur at a higher work rate
implications for performance
- Why is CP/ AT a more important determinant of performance than VO2max?
- Consider 2 athletes with the same VO2max (4.0 L.min-1)
- AT of athlete A = 70% of VO2max and athlete B = 85% of VO2max
- Athlete B will be able to maintain a higher work rate before H+ and CO2 begin to accumulate in the blood
training to improve aerobic threshold
Largely peripheral adaptations
- Increased capillary density
- Increased mitochondria
- Changes in LDH and PDH enzymes (Decreased lactate production)
- Increased myoglobin content
- Increased fat utilisation
Train at ~AT for prolonged periods of time
- Often referred to as “tempo” or “threshold” training
- Be careful with volume