MSK Disease Definitions and Info Flashcards
Reduction?
Action to reposition a deformed limb into anatomical alignment.
ORIF?
Open Reduction Internal Fixation (reduction done after opening skin).
Sprain?
Tearing of ligament.
Strain?
Tearing of musculotendinous unit.
Valgus?
Deformation of limb away from the body midline.
Varus?
Deformation of the limb toward the body midline.
Flexum?
Lack or deficit of extension in the range of motion (ROM) of a joint.
Dull/ache?
Local pathology.
Burning/tingling?
Nerve-related.
Pain at rest that is relieved by movement and night pain?
Inflammatory.
Pain upon effort?
Mechanical pathology.
Diaphysis?
Long shaft of bone.
Metaphysis?
Between the epiphysis and diaphysis.
Epiphysis?
End of bone.
Epiphyseal plate?
Growth plate.
Articular cartilage?
Covers epiphysis.
Periosteum?
Bone covering (pain sensitive).
Medullary cavity?
Hollow chamber in bone.
Red marrow makes blood cells.
Yellow marrow is adipose.
Endosteum?
Thin layer lining the medullary cavity.
Open vs Closed?
Fracture is open when exposed to air (laceration or gross exposure).
Pathologic fracture?
Implies fracture through weakened bone.
Stress fracture?
Implies misuse or overuse.
Spinal stenosis?
Cause?
MC in who?
Narrowing of spinal canal with impingement of nerve roots. Caused by arthritis and spondylolysis.
Over 60 y/o.
Ankylosing spondylitis?
Risk factors?
Joint stiffness due to fusion; Chronic inflammatory arthropathy of axial skeletal.
Males 15-30 y/o AND HLA-B27 +.
Herniated disc MC where?
L5-S1 and L4-L5.
Compression fracture occur from what?
From jumping/falling from a great height but can occur in elderly or bc of ca.
What must you do in every pt who comes in with a compression fracture?
Rule out CA.
Spondylolysis?
MC?
MOA?
Pars interarticularis defect due to failure of fusion or stress fracture.
L5-S1.
Repetitive hyperextension trauma (football, gymnasts, weight lifters).
What is MC form of back pain in children and adolescents?
Spondylolysis.
Spondylothisthesis?
MC?
MOA?
Causes?
Forward slipping of the vertebra on another.
L4-L5.
Complication of spondylolysis.
Degen, trauma, malignancy, congenital anomalies.
Cauda equina?
MCC?
Spinal nerve compression in lumbosacral region.
Lumbar disc herniation.
Rheumatoid arthritis?
Onset age?
MC in men or women?
Etiology?
Chronic inflammatory autoimmune disease involving synovium of joints causing damage to cartilage and bone.
20-40 y/o.
Women.
Infection but genetic predisposition is necessary.
Reactive arthritis?
MC in who?
Reiter Syndrome?
Associated organism?
Asymmetric inflammatory oligoarthritis of LE preceded by infectious process.
HLA-B27 +.
No classic findings.
Salmonella, Shigella, Campylobacter, Chlamydia, Yersinia.
Polyarteritis nodosa?
What is involved?
Effects which?
Assx with?
Medium sized muscular arteries involved.
Renal, CNS, GI vessels (spares the pulmonary vessels).
Chronic Hep B & C, HIV and drug reactions.
Polymyalgia rheumatica?
Average onset?
MC in men or women?
Cause?
70 y/o.
Women.
Autoimmune.
Polymyositis?
Cause?
What else does it involve?
MC in men or women?
Muscle inflammation.
Though to be genetic plus environmental trigger leads to immune activation which results in chronic inflammation.
Skin.
Women.
Dermatomyositis is assx w/?
CA- remits once tumor is removed.
Fibromyalgia is MC in men or women?
Women.
Sjorgen Syndrome?
MC in women or men?
A multi organ autoimmune disease in which lymphocyte infiltrate and destroy exocrine glands.
Systemic sclerosis (scleroderma)?
What causes problems?
MC in men or women?
Onset?
Chronic connective tissue disorder that can lead to fibrosis. Cyotkines stimulate fibroblasts causing abnml collagen deposit.
High amount of collagen.
Women.
35-50 y/o.
Systemic Lupus Erythematous (SLE)?
Patho?
MC in who?
Autoimmune disorder leading to inflammation and tissue damage involving multiple organ systems.
Autoantibody production, deposition of immune complexes, complement activation and accompanying tissue destruction/vasculitis.
Most common in young females (20-40s), African American women.
Antiphospholipid syndrome?
What does it cause?
Idiopathic disorder characterized by venous or sartorial thromboses due to antibodies against negatively charged phospholipids.
Increased risk of arterial & venous thrombosis.
Juvenile (Idiopathic) RA?
Autoimmune mono or polyarthritis in children < 16 y/o for over 6 weeks.
Pelvic fractures?
Result of?
Complications?
High impact injuries.
DVT, sciatic nerve damage, and bleeding.
Hip dislocation? MMC? MC Type? What to check for anterior dislocation? Complications?
Head of femur “pops” out of acetabulum.
High impact injury like MVA.
Posterior.
Check for femoral artery and nerve compression.
Avascular necrosis, sciatic nerve injury, DVT, bleeding.
Hip fracture?
MC in?
Three types?
High risk of what?
Elderly especially women who are more prone to developing osteoporosis.
Femoral head fracture, intertrochanteric (b/w the greater and lesser trochanter), and subtrochanteric (below the trochanters).
DVT.
What happens if the femoral neck is fractured?
It may disrupt blood supply to the femoral head which may lead to avascular necrosis.
Slipped capital femoral epiphysis?
Risk factors?
Displacement of femoral head (epiphysis) from femoral neck through the growth plate.
Children 8-16, obese, African American, males during growth spurt, if before puberty suspect hormonal or system disorder (hypothyroidism, hypopituitarism).
Legg-Calve-Perthes Disease?
MC in?
Risk factor?
Idiopathic avascular osteonecrosis of femoral head in children due to ischemia of capital femoral epiphysis.
4-10 y/o males.
Coag disorders.
Femoral shaft fracture?
Occur after?
Also look for what fracture?
Lots of what assx w/ break?
Femur is strongest bone and surrounded by quadriceps and hamstrings.
High energy trauma.
Femoral neck fracture.
Bleeding.
MC long bone fracture?
Tibial-Often open fracture w/ soft tissue injury.
What physical assessment maneuver should you do for tibial/fibular fractures?
Check ROM in toes, dorsalis media and posterior tubular pulses (& compare) and for nerve injury.
Where does the blood supply to leg come from?
Tibial artery.
Genu valgum?
(knock-knee) deformity in which the tibia is bent or twisted laterally. May occur as a result of collapse of the lateral compartment of the knee and rupture of the MCL.