Module 5 Flashcards
Confusion
not a disease process or disease state but rather a symptom. Confusion is an inability to think quickly or coherently
usually demonstrated by inappropriate reactions to environmental stimuli, may arise suddenly or gradually, and may be either temporary or irreversible. Stressful events, lack of sleep or food, or sensory deprivation may precipitate confusion. Age no reliable factor
Dementia
is a decline in mental functioning, affecting memory, cognition, language, and personality
persistent or more severe confusion, with or without psychomotor hyperactivity characterized by a significant time span between symptom appearance and death
Cognitive impairment
refers to a decline in at least one of the following cognitive domains: language, executive function, attention, perceptual-motor function, social cognition, learning, and memory. The disturbance must interfere with independence in everyday activities and not be better accounted for by another neurocognitive disorder
Treatment of dementia- pharm
NMDA receptor agonist (memantine) and cholinesterase inhbitors (donepezil, rivastigmine)
most useful for moderate to severe AD, combo is more effective
non pharm tx of dementia
cognitive rehab
exercise
occupational therapy
recommended psychotic for dementia
risperidone, only used for 3 months
delirium
acute, abrupt onset
confusion fluctuates throughout day
inattention
once the cause is corrected, should return to previous state of cognitive functioning
metabolic disturbance/ confusion
fluid, electrolyte and acid base imbalance may b result of metabolic problems, which can produce confusion
dehydration
infectious process/ confusion
confusion may be result of infectious process that can cause extensive tissue and organ impairment, ischemia produces cell injury
Dizziness
is the sensation of unsteadiness or feeling off balance, faintness, light-headedness, and a feeling of movement within the head. Loss of consciousness rarely occurs, but the feeling of faintness encourages the patient to lie down, which may cause the feelings to disappear
brief episodes, result of inadequate blood flow and oxygen supply to brain
Vertigo
is the false sensation of rotation or movement of the patient or the patient’s surroundings. Vertigo may result from an inner ear disease or a disturbance of the vestibular center or pathway in the central nervous system (CNS).
Diagnoses of dizziness- 4 categories
peripheral vestibular disease
systemic disorders
CNS disorders
anxiety states
Questions to ask regarding dizziness
duration
severity
nature of episodes
associated symptoms
physical exam dizziness
exam ear (r/o cerumen)
whisper test
rinne and weber
thorough neuro assessment
Hallpike maneuver
The Hallpike maneuver
is performed by rotating the patient’s head to one side and then lowering it slowly to 30 degrees below the bodyline. The patient should be observed for nystagmus during head rotation and vertical positioning.
In patients with benign vertigo, there may be rotational nystagmus and possible severe vertigo, which usually occurs in one direction. This resolves quickly and cannot be reproduced after two to three repetitions.
The clinician should suspect a central lesion when the vertical nystagmus is of a longer duration and continues with each repetition
peripheral vestibular disease dizziness
Most often the problem is located in the labyrinth of the middle ear
dizziness, nausea and vomiting, diaphoresis, difficulty with balance, vertigo, tinnitus, fluctuating hearing loss, feelings of pressure in the ear, and diplopia
tx vertigo
meclizine or promethazine most common- suppress vestibular end organ receptors, inhibit vagal response
take med x1 week, then taper down
diamox
is used to decrease edema in the labyrinth
vertigo exercise
instructed to reproduce the feelings of vertigo by placing the affected ear down, then assume a supine position and hold that position until the vertigo disappears. The vertigo may return when the patient sits up. The patient should repeat these maneuvers at least five times a day or until the vertigo no longer returns. Patients with persistent symptoms should be referred for assessment of nerve function.
systemic disorder dizziness
aggravated by postural changes or exertion. Pallor, dyspnea, tachycardia, bounding pulse, weakness, hypotension, blurred vision, decreased breath sounds, headache, diaphoresis, and agitation suggest systemic problems
percentage of HA without cause
90%
types of primary headaches
tension-type headaches, migraines, and cluster headaches
Tension-type headache
, also referred to as a muscle contraction headache, presents as a mild to moderate bilateral, nonpulsating, tightening pain that is not aggravated by routine physical activity. It is usually not accompanied by nausea and vomiting or photophobia.
Migraine headache
may last for 4 to 72 hours and may or may not be precipitated by an aura. It is usually unilateral, of moderate to severe intensity with a pulsating quality, aggravated by routine physical activity, and accompanied by nausea, vomiting, and photophobia.
Cluster headache
usually occurs at night and may last from 15 to 180 minutes. There is usually severe unilateral orbital, supraorbital, and/or temporal pain that is accompanied on the same side of the face with sweating, lacrimation, nasal congestion, ptosis, rhinorrhea, eyelid edema, and/or conjunctival injection.
subtypes of migraine
with and without aura
aura
fully reversible neurologic symptoms: visual, motor, sensory, speech, brainstem, or retinal. The symptoms of an aura typically develop gradually and are usually followed by a headache. Auras are fully reversible and last 5 to 60 minutes
definition of migraine without aura (2 characteristics required)
unilateral location, pulsating quality, moderate to severe intensity, and exacerbation by physical activity. In addition, at least one of the following must be present: nausea or vomiting, photophobia, or phonophobia
chronic migraine definition
15 migraine days each month, for at least 3 months
women are _____x more likely to be affected by migraines
2-3
most common type of headache
tension, age peaks between 30-38
second mots common type of headache
migraine
African americans, non whites 2x likely to have migraines
foods that are known migraine triggers
sodium nitrate
alcohol
tyramine
phenylethylamine
caffeine
most painful type of headaches
cluster
temporal arteritis or GCA
Associated symptoms include scalp allodynia, jaw claudication, and concurrent polymyalgia rheumatica. Other symptoms are local swelling; tenderness and pulselessness of the temporal artery; and systemic symptoms of fever, anorexia, weight loss, and chills. Systemic markers of inflammation also are present. Prompt diagnosis is important since temporal arteritis can cause permanent vision loss if left untreated.
thunderclap headache
abrupt and severe, sudden onset headache that reaches maximal intensity in under 1 minute. This type of headache requires emergent evaluation since it is often caused by a subarachnoid hemorrhage
Arterial dissection,
characterized by cephalic pain or headache of sudden onset, often preceding transient ischemic attack (TIA) or stroke symptoms. Carotid or vertebral artery dissection causes acute unilateral neck pain that is sudden and often radiates to the ipsilateral face or eye. The headache is related to cervical manipulation, sustained exertion, or trauma. Recognition and proper treatment are important since arterial dissections can lead to stroke
Increased intracranial pressure
causes headaches that worsen with lying flat, bending over, or with the Valsalva maneuver. There may be an associated cranial nerve VI palsy, pulsatile tinnitus, or transient visual obscuration, including a graying out of vision, typically with position changes. Funduscopic examination reveals papilledema.
Encephalitis
is associated with a new-onset generalized headache, accompanied by confusion, altered level of consciousness, focal neurologic signs, or seizures. Signs of infection and meningismus may be present. Changes associated with these conditions can be detected with lumbar puncture (LP) and brain imaging
skull structures sensitive to pain
meninges, arteries, skull
Pain signals are transmitted from most structures in the head by branches of the
trigeminal nerve
It is thought that an abnormal reduction in serotonergic activity in the ________is a part of the hypersensitization in primary headache syndromes
thalamus
events of migraine
prodrome- hours to days before headache (drowsiness, depression, euphoria)
Unilateral headache- increased sensitivity
cluster headache duration
2-3 months, 1-2x/year
family hx-migraine
associated with 40% patients
typical migraine without aura
often unilateral
2-72 hours
pulsating
inhibiting daily activity
n/v, photo and phonophobia
typical migraine with aura
aura develops over minutes, lasts less than hour
visual symptoms most common
migraines and menses
migraines often occur after ovulation and before/during menstruation
for women who experience aura
use of any combined hormonal contraception is contraindicated= d/t 3x increase in risk of stroke
physical findings with subarachnoid hemorrhage
visual blurring
diploplia
fever
alteration of consciousness
nuchal rigidity
A symptom specific to temporal arteritis is
“claudication” of the muscles of mastication
A computed tomography (CT) scan or an magnetic resonance imaging (MRI) study is recommended if the patient’s headache
pattern is atypical, has changed in pattern or character, or is accompanied by seizures, personality changes, or an abnormal neurologic finding
labs for headache pt
CBC
CMP
UA
GCA labs
ESR strongly elevated
CRP elevated
biopsy of temporal artery is gold standard
HA red flags
- Progressive or fundamental change in headache that worsens over time
- Patient states, “This is the worst headache of my life!”
- New-onset headaches before the age of 5 or after age 50
- Persistent headache precipitated by a Valsalva maneuver, exertion, or sex
- Fever, acute glaucoma, hypertension, myalgias, weight loss, or scalp tenderness
- Neurologic signs and symptoms: confusion, altered level of consciousness, changes in memory, papilledema, sensory deficits, reflex asymmetry, or gait disturbances
- Headache with syncope or seizures
Tension type headache tx
NSAID, cool compresses, stress reduction
Not more than 2x/week
migraine tx- non pharm
ID and eliminate triggers
maintain strict sleep schedule
regular exercise
deep breathing, massage, hot/cold therapy
migraine tx- pharm
triptan , NSAID
cluster ha tx
SQ sumatriptan
intranasal zolmitriptan
O2
Paresthesia
is an abnormal sensation described as numbness or tingling, cramping, or pain without a known stimulus, felt along peripheral nerve pathways.
Paresis
is weakness
Tremors
are rhythmic involuntary muscle movements that result from alternate contraction and relaxation of opposing muscle groups. They are typically evident in patients with cerebellar or extrapyramidal disorders
resting tremor
occurs in a relaxed and supported extremity and ends with purposeful movement
intentional tremor
occurs when the patient attempts voluntary movement.
Musculoskeletal problems
most common cause of disability in workers
* Typically self limiting, however must r/o musculoskeletal emergencies
* Diagnosis dependent on patient’s self reporting symptoms
Categories of musculoskeletal complaints
o Acute or chronic
o Articular or nonarticular
o Inflammatory or non inflammatory
o Localized or systemic
articular complaints
involve synovium, synovial fluid, articular cartilage, intra articular ligaments, joint capsule
o Deep/diffuse pain, limited ROM on active and passive movement, swelling,crepitation, instability, locking, deformity
o Based on number of involved joints (monoarticular, periarticular, polyarticular)
non-articular complaints
involve extra articular ligaments, tendons, bursae, fascia, bone, nerve, skin
o Focal vs widespread
inflammatory muscle complaints
infectious or idiopathic
o Four signs of inflammation: erythema, warmth, pain swelling
non inflammatory musculoskeletal complaint
- Non inflammatory- r/t trauma, ineffective repair, neoplasm
when inspecting joint pain always compare
symmetry and sides
Pain over greater trochanter indicative of
hip trochanteric bursitis
hand numbness that awakens pt at night indicative of
carpal tunnel syndrome
Bony enlargement of distal interphalangeal joints indicative of
osteoarthritis
soft tissue swelling indicative of
rheumatoid arthritis
Two key portions of physical exam of musculoskeletal
ask pt to rise from chair without holding on
have pt walk
Lab tests for musculoskeletal complaints
CBC- r/o anemia, leukopenia
ESR, CRP
Rheumatoid factor
ANA
Lyme
Uric acid
Imaging for musculoskeletal complaints
Indicated when exam can’t localize structure causing symptoms, after significant trauma, loss of joint function, pain continues after conservative management, fracture or infection suspected, hx of malignancy
Acute Musculoskeletal Injury
- Acute pain of less than 6 weeks
- Damage to muscles, tendons, ligaments, nerves, bursae- most common are spasm, strain, sprain
Spasm
o - persistent, painful, reversible contracture of striated muscle
Strain
o - muscle injury caused by excessive tensile stress placed on muscle- results in stiffness, decreased function
“pulled muscle”, usually no redness/swelling, ROM may be limited
Sprain
o - stretching/tearing of ligaments when joint forced beyond normal anatomical range
often have hx of sudden fall/injury
o Redness/bruising over affected joint, active and passive ROM limited, pain when moving