Module 3 UMN and LMN Flashcards
What is the locations and structures involved in UMN lesion?
central nervous system cortex, brain stem, corticospinal tracts, spinal cord
UMN diagnosis/pathology
stroke, traumatic brain injury, spinal cord injury
UMN tone
increased: hypertonia velocity dependent
UMN reflexes
increased: hyperreflexia, clonus
Exaggerated cutaneous and autonomic reflexes, +Babinski
UMN involuntary movements
muscles spasms: flexor or extensor
UMN strength
Weakness or paralysis ipsilateral stroke) or bilateral (SCI)
Corticospinal: contralateral if above decussation in medulla; ipsilateral if below distribution: nerve focal
UMN muscle bulk
Disuse atrophy: variable widespread distribution. especially of antigravity muscles
UMN voluntary movements
impaired or absent: dyssynergic patterns, obligatory mass synergies
LMN location and involved structures
cranial nerve nuclei/nerves
Spinal cord: anterior horn cell, spinal roots
peripheral nerve
LMN diagnosis/pathology
Polio, Guillain-Barre
Peripheral nerve injury
Peripheral neuropath
Radiculopathy
LMN tone
decreased or absent: hypotonia, flaccidity
not velocity dependent
LMN Reflexes
decreased or absent: hyporeflexia
cutaneous reflexes decreased or absent
LMN involuntary movement
with denervation: fasciculations
LMN strength
Ipsilateral weakness or paralysis
Limited distribution: segmental or focal pattern, root-innervated pattern
LMN muscle bulk
neurogenic atrophy: rapid, focal distribution, severe wasting
LMN Voluntary Movements
Weak or absent if nerve interrupted
Cerebral Cortex Corticospinal tracts diagnosis/pathology
stroke
Cerebral cortex corticospinal tract sensation
impaired or absent: depends on lesion location; contralateral sensory loss
cerebral cortex corticospinal tracts tone
hypertonia/spasticity velocity-dependent; clasp-knife
initial flaccidity: cerebral shock
Cerebral cortex corticospinal tracts reflexes
hyperreflexia
Cerebral cortex corticospinal tracts strength
Contralateral weakness or paralysis: hemiplegia or hemiparesis
Disuse weakness in chronic state
Cerebral cortex corticospinal tracts muscle bulk
normal during acute stage; disuse atrophy in chronic stage