Midterm II Diseases and Conditions Review Flashcards

1
Q

Describe what Iron Deficient RBCs Look Like

A
  • microcytic

- hypochromic (small and pale)

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2
Q

Describe what Pernicious RBC Look Like

A
  • macrocytic
  • normochromic
    ( large, red RBCs)
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3
Q

Which type of Anemia has many Neurological Symptoms

A

Pernicious Anemia

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4
Q

What Can Folate Deficient Anemia in Pregnancy Cause

A
  • CV disease

- neural tube defects

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5
Q

What is the difference between Hodgkin’s and Non-Hodgekin’s Lymphoma

A
  • hodgekins has a local start at a younger age while non is more severe, affects multiple nodes, and spreads erratically in older people
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6
Q

Hypovolemic Shock

A
  • low fluid volume in blood
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7
Q

Cardiogenic Shock

A
  • impaired contractility of heart
  • not common
  • more severe (weak to no pulse and little to no urine output)
  • neuro symptoms
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8
Q

Distributive Shock (three types and general symptoms)

A
  • vasodilation
  • decreased BP
  • warm and pink
  • bounding pulse
    1. Anaphylactic- wheezing, edema, hives
    2. Septic- fever, increased WBC
    3. Neurogenic- spinal injury, increased resp rate
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9
Q

Variant Angina

A
  • vessel spasm caused by too much SNS and decreased vasodilators
  • cyclical pain at night
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10
Q

Stable Angina

A
  • atherosclerosis
  • narrowing of vessel lumen
  • triggered by increased cardiac oxygen demand
  • predictable pain
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11
Q

Unstable Angina

A
  • atherosclerosis

- prone to rupture

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12
Q

Bacterial Meningitis

A
  • rapid onset
  • dangerous
  • purpura
  • tight hams and neck
  • CSF glucose test
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13
Q

Left Heart Failure

A
  • inability to push blood into systemic circulation
  • pulmonary backflow
  • lung symptoms (frothy pink sputum)
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14
Q

Right Heart Failure

A
  • inability to push blood into pulmonary circulation
  • systemic backflow
  • hypoxemia
  • diuretics used to unload heart
  • edema
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15
Q

Acute Diabetes Complications

A
  1. Hypoglycemia
    • not enough food too much insulin or exercise
  2. Diabetic Ketoacidocis
    • lack of insulin
    • kussmaul resps
  3. HHNS
    • profound fluid loss
    • slow progression
    • hospitalization
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16
Q

Chronic Diabetes Complications

A
  1. Macrovascular
    • heart issues
    • MI/Stroke
    • foot ulcers
    • gangrenous feet
  2. Microvascular
    • ischemia of eyes and kidneys
  3. Neuropathies
    • nerve ischemia
    • nerve demyelination
  4. Infection
    • impaired WBC
    • increased glucose
17
Q

Hyperthyroidism

A
  • grave’s
  • autoantibodies act as TSH
  • protruding eyes (xophthalmas)
  • dehydrating
  • everything speeds up
18
Q

Hypothyroidism

A
  • Hashimoto’s
  • periorbital edema
  • everything slows down
19
Q

Hypercortisolism

A
  • Cushing’s syndrome
  • ectopic ACTH production
  • iatrogenic (longterm glucocorticoid therapy)
  • increased blood glucose, buffalo hump, moonface etc
20
Q

Hypocortisolism

A
  • Primary- addison’s

- Secondary- sudden withdrawal of glucocorticoid therapy

21
Q

Signs of a Stroke

A

F- face
A- arms
S- speech
T- time

22
Q

Ischemic Thrombotic Stroke

A
  • plaque in brain prone to rupture

- blood flow obstruction

23
Q

Ischemic Embolic Stroke

A
  • bits of clot elsewhere block blood flow to brain
24
Q

Hemorrhagic Stroke

A
  • spontaneous blood vessel rupture

- increased fatality

25
Q

Provoked Seizure

A
  • from another underlying disorder (fever)
26
Q

Unprovoked Seizure

A
  • recurrent
  • genetic
  • auras
  • epilepticus
27
Q

Primary Brain Neoplasm

A
  • from supporting brain cells
  • risks increase up to 70 yrs
  • Supra- adult
  • intra- kid
28
Q

Types of Focal TBI

A
  1. Skull Fracture
  2. Contusions- brain bruise
  3. Hematomas- clotted blood
29
Q

Types of Diffuse TBI

A
  1. Concussions
  2. CTE- post concussion, progressive, leads to Alzheimer’s
  3. DAI- increased morbidity, shaken baby, damages axons, unpredictable outcome
30
Q

Chronic Venous Insufficiency

A
  • venous return over long period

- causing venous status uclers

31
Q

Dyslipidemia

A

H better then L, L leads to atherosclerosis

32
Q

DVT

A
  • lower extremities
  • stasis of blood flow
  • hypocoagulability
  • increased embolism risk if untreated
33
Q

Steps of Atherosclerosis

A
  1. edothelium is injured allowing LDL enters epithelium
  2. LOL oxidized by RAO
  3. Macrophages engulf oxidized LDL
  4. LDL turn into foam cells
  5. Build up of foam cells lead to lipid pool and then to plaque
    - pain in legs when walking
34
Q

SIADH

A
  • increased ADH
  • decreased urine
  • decreased serum sodium
  • hypotonic
35
Q

Diabetes Insipidus

A
  • decreased ADH
  • hypertonic
  • cretism in kids
36
Q

Autonomic Dysreflexia

A
  • sympathetic response below injury

- disconnect between PNS and SNS