midterm 2 Flashcards
consciousness
our awareness of our environment/selves
dual processing
brains process info at 2 levels
serial processing (consciousness can only process 1 step at a time
unconscious processes super fast
parallel processing
blindsight
consciously, we are blind
unconsciously, we still have some vision
subliminal stimulation
we’re exposed to a stimulus and the exposure is super fast
–> info gets to brain but never reaches consciousness
cannot see object consciously but can still influence behaviours
serial processing
consciousness can only process one step at a time
parallel processing
processes multiple pirces of info simultaneously
subconscious processing (priming)
we are exposed to a stimulus and are aware of it, HOWEVER we don’t know that the stimulus is unconsciously activating in our brain memories and the information linked to it
–> can affect behaviour
where does consciousness come from
the brain
attention
allow us to focus our awareness on a tiny point of stimulation around us
value (attention)
ability to pay attention to valuable & essential things for survival
(t/f) attention is an unlimited resourse
FALSE
is attention selective
YES. focusing attention on a certain stimuli causes us to disregard other stimuli in the environment
inhibition (attention)
paying attention to a specific stimuli causes the brain to inhibit us from processing other info
–> sharpens attention
cocktail party effect
at a party –> very loud and noisy but can focus attention on an interesting convo
in spite of this, if someone said our name we’re likely to pick it up
dichotic listening
used to study selective attention
2 diff msgs in each ear at the same time –> patients asked to pay attention to one ear only
we still pick up info from he other ear
Corteen and Wood Experiment (1972)
experiment to examine DEGREE we use dichotic listening
conditioned to associate city names with an electric shock
–> city names were played in unattended ear
–> used GSR (galvanic skin response) measurements ( measures arousal in sympathic nervous system)
–> 38% response rate for recognized city names, 10% for unrelated words
passive (attention attraction)
bottom-up process
stimuli in environment grabs our attention bc there is something that stands out about it
active (attention attraction)
top-down
we intentionally chose what we are going to pay attention to
inattentional blindness
we’re going to fail to detect a significant stimulus in our environment
change blindness
we’re engaged with a stimulus but don’t notice the change in the stimulus
intentional change detection
we’re told that the stimulus is going to change, but we still have a hard time detecting the change
divided attention (multitasking)
consciousness can’t multitask, we tend to slow down and make errors
visual neglect
attention disorder
part of the world becomes non existent for us
can happen in the right or left hemisphere depending on what’s dmged
ex. u only shave one side of ur face –> u can see the other side but ur not aware of it
fatal familial insonmia
rare hereditary disease that affects THALAMUS
can die from lack of sleep 12-18 months after symptoms start
electroencephalograms (EEGs)
measure activity across surface of brain
electrooculograms
used to measure movements of eyes in sleep
electromyograms
used to measure tension and muscles of jaw
2 observable patterns when awake
relaxed wakefulness, awake and alert
alpha waves
8-12 Hz
more regular and predictable than beta waves
relaxed wakefulness
alpha waves
when person is quietly resting
beta waves
13-30 Hz
desynchronized and erratic waves –> many neural circuits processing information
stages of non-REM sleep
STAGE 1, STAGE 2, slow wave sleep (SWS_
STAGE 1 (non-REM sleep)
theta waves
transition from relaxed state –> early sleep (very light)
theta waves
3.5-7.5Hz
awake and alert (stages of sleep)
beta waves
STAGE 2 (non-REM sleep)
sleep spindles, K complex
sleep spindles
(12-14Hz)
occur around 2-5 times/min
thought to play a role in memory consolidation
higher sleep spindles associated with higher IQ test scores
K complex
bursts of energy on EEGs
occur around once/min
can be triggered by unexpected noises, still wouldn’t have a sense of sleep when woken
prepares brain to enter SWS
slow wave sleep (SWS)
delta waves
15-20 min after STAGE 2 starts
deepest stage of sleep, only a strong stimulus will wake u
groggy, confused when woken
delta waves
less than 4Hz
regular, high-amplitude waves
REM sleep (rapid eye movement)
theta waves, beta waves
45 min after SWS starts
brain is very active with vivid dreams, but easier to wake than SWS
REM sleep antonia
generally become paralyzed during REM sleep
what happens to blood flow to brain during REM sleep
blood flow reduced, but visual association cortex and prefrontal cortex receive a large proportion of oxygenated blood
function of SWS
important for resting brain over body
important for explicit memories
regions with highest activity during waking hours is resting
function of REM
benefits brain’s ability to absorb and process information
rebound phenomenon (REM)
there is a need for a certain amount of REM sleep
–> if brain is deprived from REM for a couple days, brain tries to enter REM more quickly and for a longer period of time
increased % of time spent in REM in infants
insomnia
inability to fall asleep/inability to remain asleep
sleep hygeine
habits and behaviours conducive to sleeping well
conditioned insomnia
learned insomnia
–> going to bed becomes associated with inability to fall asleep
among the most commonly diagnosed forms of primary insomnia
idiopathic insomnia
child onset insomnia
neurophysiological abnormality in the CNS
begins in childhood, more resistant to treatment
hypersomnia
excessive sleepiness, caused by poor sleep during the night
sleep apnea
intake of oxygen is reduced as person sleeps
–> brain sends signal to body as blood oxygen decreases –> sleeper wakes
treatment for sleep apnea
CPAP: pressurized air mask htat pushes pressurized air through airway
narcolepsy
rare genetic neurodegenerative disorder that has several symptoms:
- sudden and extreme need to sleep
- cataplectic attacks
- paralysis with hallucinations
cataplexy/cataplectic attacks (narcolepsy)
paralysis experience during REM initiates and inappropriate times
initiated by emotionally engaging events
kinds of hallucinations (narcolepsy_
hypnagogic: upon onset of sleep
hypnopompic: just before waking
circadian rhythym
daily clocks, closer to 25 hours
zeitgebers
time givers/cues
–> our clock is reset every morning by cues associated with morning activity
reliable stimuli in the environment tht provide information about the time of day
what resets our daily clock
presence/absence of light
suprachiasmatic nucleus (SCN):
your body’s timekeeper
sends signals to several regions of the brain → ex. pineal gland
pineal gland secretes melatonin
psychoactive drugs
drugs that influence/affect function of brain/NS
where do drugs produce their effects
synapse
–> interfere with communication between neurons
dopamine
pleasure molecule
what do most street drugs agonize
dopamine
tolerance (drugs)
can develop a tolerance if we abuse a drug
–> need to consume more and more to get the desired effect
neuroadaptation
when we use drugs, our brains adapt to that by changing themselves (plasticity)
brain may stop production of certain NT or shut down receptors for certain NT
withdrawal
happens when you stop abusing a drug
–> can get uncomfortable and painful symptoms
drug dependence
physical: need drug to function normally
psychological: feel like we can’t live without it
2 major types of drugs
- depressants
- stimulants
- hallucinogens
depressants
reduce, slow down activity of the NS/brain
alcohol
depressent at all dosages
what area of the brain is affected by alcohol
hippocampus
–> part of brain that deals with memories
–> memories of a drunk night are hazy
barbiturates (depressants)
- more addictive
- more powerful
- known as downers
benzodiazepines (depressants)
- highly addictive
- known as tranquilizers
- eg. xanax - valium
stimulants (drugs)
psychoactive drugs that speed up/increase activity of NS/brain
(t/f) depressants have a lethal aditive effect
TRUE
nicotine
damages ur DNA
1 cigarette = lose 11/12 min of life
why cant u stop smoking
nicotine is highly addictive –> enhances the activity of multiple NTs:
- acetylcholine
- norepinephrine
- dopamine
nicotine’s dual effect
- sluggish –> smoke –> perk up
- anxious –> smoke –> calm down
effects of long-term use of nicotine on acetylcholine
reduces levels of acetylcholine
cocaine
stimulant that blocks reuptake of some NTS –> enhances:
- dopamine
- norpinephrine
- serotonin
can end up with chronic permanent depression that requires meds`
formication
feeling as if we have bugs/insects crawling in skin
stereotypic behaviours (cocaine)
non sensical behaviours that we repeat while under the influence of cocaine
amphetamines
stimulant
combats effects of hunger and fatigue
agonizes dopamin
–> inhibits reuptake of dopamine and stimulates release of it from terminal buttons
hallucinogens
directly influence sensory systems and interpretation of reality
psychedelics
have most profound effects on consciousness
LSD
man-made
areas of the brain that usually talk to each other communicate more/stop talking to each other
which part of the brain does LSD affect
thalamus
–> explains why LSD users experience fusing of senses
marijuana
attaches to cannabinoid receptors found all over brain
active ingredient: THC
increases dopamine release
what NTs are inhibited by marijuana
norepinephrine, acetylcholine, glutamate, GABA
whats more likely to lead to addiction: inhaled drug, digested drug
INHALED
classical conditioning
we learn to associate 2 events/stimuli
–> learn that one event signals arrival of another
stimulus triggers response
respondant behaviour
response that occurs in the presence of a stimulus
operant conditioning
learn to associate a behaviour with its consequences
operant behaviour
organism initiates the behaviour and it produces consequences
fundamental principle (OC)
behaviour is controlled by its consequences
–> desired consequence –> likely to repeat it
law of effect (OC)
when a behaviour produces an undesirable/desirable consequence, we are more likely to repeat it
thorndike
stipulated the law of effect and started research on operant conditioning
–> instrumental conditioning
skinner
linked and associated with operant conditioning
strict behaviourist
ABCs (OC)
A = antecedent
B = behaviour
C = consequence
differential vs non-differential consequences
Differential: receiving different consequences
–> leads to FASTER LEARNING
Non-differential: receiving the same consequence
reinforcer
consequence of a behaviour that makes the behaviour more likely to repeat in the future