Mental Disorders Flashcards

1
Q

Intellectual Developmental Disorder

A

ETIOLOGY:
1) hereditary, developmental, congenital, or traumatic factors -> cerebral hypoxia and malnutrition -> neural death

S/S:

1) Social deficits (empathy, making friends, emotional intelligence, interpersonal skills)
2) Conceptual deficits (reasoning, memory, language)
3) Practical deficits (executive function - organizing, hygiene, planning)

TREATMENT:

1) treat or manage underlying causes
2) exact therapy may vary with deficits

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2
Q

Learning Disorders

A

ETIOLOGY:
1) impaired visual, language, or memory processing -> impaired cognitive processes related to learning

S/S:

1) falling below school benchmark (based on mental age and standards)
2) low self-esteem, dropping out of school, interpersonal deficits

TREATMENT:

1) tutoring
2) stimulants if related to ADHD

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3
Q

Stuttering

A

ETIOLOGY:
1) stress and genetic factors -> stuttering (onset 2-7 years)

S/S:
1) repeated sounds, prolonged sounds, filled or unfilled spaces, broken words

TREATMENT:
1) speech therapy

BONUS FACTS:
1) spontaneous recover common into adulthood

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4
Q

Autism Spectrum Disorder (ASD)

A

ETIOLOGY:

1) RISK FACTORS include being male
2) unclear, but genetic factors involved

S/S:
1) communication impairment (absent nonverbal communication, delayed or absent verbal communication), repetitive motions, resistance to changes

TREATMENT:

1) behavioral therapy
2) risperidone (antipsychotic)

BONUS FACTS:

1) most ASD individuals manage adaptive behaviors into adulthood
2) Risperidone side effects include mammary development and secretion

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5
Q

Attention-Deficit Hyperactivity Disorder (ADHD)

A

ETIOLOGY:
1) genetic factors -> suspected abnormality in the prefrontal cortex -> impaired attention and impulse control

S/S:

1) difficulty maintaining attention, poor organization
2) squirming and fidgeting, and excessive talking

TREATMENT:

1) methylphenidate (stimulants)
2) vitamin supplements

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6
Q

Oppositional Defiant Disorder

A

ETIOLOGY:
1) poor child temperament, poor child management, and stressful life events

S/S:
1) arguing with adults, annoying others, disobeying, blaming others, irritability

TREATMENT:

1) family therapy and psychotherapy
2) possible risperidone (antipsychotic)

BONUS FACTS:
1) poor prognosis if diagnosed instead as conduct disorder

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7
Q

Tourette Disorder

A

ETIOLOGY:
1) Risk factors include family history; streptococcal infection

S/S:
1) nonrhythmic vocal (grunts, repetitive sounds, clicks) and motor (neck-jerking, blinking, grimaces) tics that changed over time

TREATMENT:
1) haloperidol (antipsychotic)

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8
Q

Alzheimer Disease

A

ETIOLOGY:

1) RISK FACTORS include chromosome 21 defects, autoimmune disorders, and neurochemical deficits
2) characterized by amyloid plaque and neurofibrillary tangles

S/S:
1) memory issues, communication issues, intellectual and physical impairment, sleep disturbances, irritability

TREATMENT:

1) donepezil (anticholinesterase)
2) haloperidol (antipsychotic)
3) paroxetine (SSRI)

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9
Q

Vascular Dementia

A

ETIOLOGY:
1) fibrous atheromatous plaque, embolism, minute aneurysms -> cerebral hypoxia and decreased nutrition -> neural death

S/S:
1) impairments in memory and judgments, changes in personality, poor executive function, apathy, depression, irritability

TREATMENT:

1) aspirin (antiplatelet)
2) carotid endarterectomy (surgical excision of tunica intima)

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10
Q

Dementia from Head Trauma

A

ETIOLOGY: any traumatic head injury that leads to cerebral edema and damaged blood vessels can cause hypoxia and decreased nutrition to brain cells

S/S: impaired intellectual and cognitive functioning

TREATMENT: treat injury to reduce further hypoxia, rehabilitative programs

PROGNOSIS: varies with extent of cerebral insult

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11
Q

Alcohol Abuse

A

ETIOLOGY:
1) PREDISPOSING FACTORS: family history, psychosocial stressors

S/S: 

1) frequent alcohol intoxication (behavioral issues, anxiety, impaired judgments, poor motor function, poor impulse control)
2) alcohol tolerance and withdrawal
3) peripheral neuropathy, cirrhosis, pancreatitis

TREATMENT:

1) rehabilitation program, group therapy, cognitive-behavioral therapy
2) disulfiram (antabuse targeting ethanol)

BONUS FACTS:
1) greatly increases risk of pancreatitis, cirrhosis, and peripheral neuropathy

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12
Q

Schizophrenia

A

ETIOLOGY: (unclear, but genetics, being male, stress, hypoxia, and infection may play a role) pathology theorized to be linked to dopamine, glutamate, and/or serotonin

S/S: positive effects (inappropriate affect, disorganized thinking, delusions, hallucinations) and/or negative effects (flat affect, poor executive function, social and occupational dysfunction, catatonia)

TREATMENT: atypical antipsychotics (risperidone), then typical antipsychotics if unresponsive (haloperidol)

PROGNOSIS: (prodromal to active to residual phase) best outcomes seen with acute rather than chronic schizophrenia, but best with compliance to treatment. Risk of extrapyramidal side effects with treatment

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13
Q

Bipolar Disorder

A

ETIOLOGY:
1) family history, stress, electrolyte imbalances, endocrine dysfunction (predisposing f.) -> suspected dopamine and/or serotonin imbalance

S/S:

1) manic episodes (grandiose or bizarre behavior, high arousal, lack of sleep, disorganized thoughts, euphoria, impaired judgment)
2) depressive episodes (flat effect, avolition, sleep disturbances)
* cycles

TREATMENT:

1) lithium carbonate (mood-stabilizer)
2) gabapentin (anticonvulsant for mood stabilizing)
3) antidepressants (during depressive episodes)

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14
Q

Major Depressive Disorder

A

ETIOLOGY:

1) (Predisposing Factors) psychosocial pressures, chronic illness, and alcohol dependency
2) Strongly linked to genetic and organic factors

S/S:
1) low-self esteem, avolition, anhedonia, inability to concentrate, social withdrawal, anxiousness

TREATMENT:

1) psychotherapy
2) fluoxetine (SSRI)
3) deep-brain stimulation (electro convulsive therapy)
4) atypical antipsychotics

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15
Q

Generalized Anxiety Disorder

A

ETIOLOGY:
1) genetic predispositions, stress, endocrine dysfunction, neurochemical imbalances

S/S:

1) free-floating anxiety, rumination, sleep disturbances
2) diarrhea, muscle tension, high blood pressure

TREATMENT:

1) psychotherapy, relaxation techniques (i.e. breathing exercises) 2) lifestyle changes
3) anxiolytic drugs

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16
Q

Panic Disorder

A

ETIOLOGY: (unclear) stress, endocrine dysfunction, neurochemical imbalances, and familial/genetic factors may play a role

S/S: episodes of unfocused anxiety (derealization or depersonalization, palpitations, high blood pressure, muscle tension, nausea)

TREATMENT: various forms of psychotherapy, relaxation techniques (breathing exercises) lifestyle changes, anxiolytic drugs

PROGNOSIS: best with compliance to treatment and strong support network

17
Q

Phobic Disorders

A

ETIOLOGY: (unclear) severe stress, genetic predispositions, and prior conditioning may play a role

S/S: anxiety in response to phobic stimulus (situation or object usually not harmful in nature)

TREATMENT: cogntive-behavioral therapy, other forms of psychotherapy, anxiolytic drugs

PROGNOSIS: best with compliance to treatment and strong support system

18
Q

Obsessive-Compulsive Disorder

A

ETIOLOGY: (unclear, but can be related to psychosocial stresses, endocrine dysfunction, and/or genetic predispositions) suspected abnormality in prefrontal cortex

S/S: obsessions (invasive and unwanted thoughts) and/or compulsions (behavioral urges, usual extremes of natural executive behaviors)

TREATMENT: psychotherapy, lifestyle changes, relaxation techniques, anxiolytic drugs

PROGNOSIS: best with compliance to treatment and strong support system

19
Q

Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptoccocal infections (PANDAS)

A

ETIOLOGY:
1) streptococcal infection -> autoimmune activity

S/S:

1) mood swings, separation anxiety, detachment from caregivers, sleep disturbances
2) exacerbation of pre-existing mental disorders (tics, hyperactivity)

TREATMENT:

1) antibiotics, possible steroids streptococcal infection
2) cognitive-behavioral therapy
3) SSRIs (OCD)

BONUS FACTS:
1) likely to recur with streptococcal infection

20
Q

Post-Traumatic Stress Disorder (PTSD)

A

ETIOLOGY:
1) traumatic events (human-mediated or natural disaster) -> PTSD develops weeks, months, or years later

S/S:
1) strong physiological response to triggering stimuli, insomnia, nightmares

TREATMENT:

1) cognitive-behavioral therapy, pet therapy
2) benzodiazepines (for sleep)
3) anxiolytic drugs or SSRIs

21
Q

Somatization Disorder

A

ETIOLOGY: (unclear) may be linked to family history and extreme periods of stress

S/S: pain in at least four body areas, GI symptoms (nausea, vomiting), sexual dysfunction (irregular menses or erectile/ejaculatry dysfunction)

TREATMENT: rule out any other medical condition, psychotherapy

PROGNOSIS: generally very poor, complete remission unlikely

22
Q

Conversion Disorder

A

ETIOLOGY: in response to extreme stress, anxiety converted into physical symptoms as defense mechanism

S/S: disrupted voluntary motor function (tics, paralysis, tremors) impaired sensory function (paresthesia, anesthesia, analgesia, hyperesthesia), impaired speech, visceral symptoms (headaches, GI issues, difficulty swallowing)

TREATMENT: symptom-based, possible psychotherapy and hypnosis

PROGNOSIS: varies based on level of stress, but may resolve within few weeks. Recurrence is likely

23
Q

Pain Disorder

A

ETIOLOGY: related to clinical-pathologic factors (musculoskeletal diseases, neurologic diseases, malignancies) and/or psychological factors

S/S: severe pain that may interfere with daily functioning

TREATMENT: treat underlying pathologies, possible narcotic analgesics, possible psychotherapy

PROGNOSIS: varies based on pathology

24
Q

Hypochondriasis

A

ETIOLOGY: unclear, but different from malingering

S/S: (from a clinical perspective) preoccupation with illness, vague symptoms, generalized history

TREATMENT: treat any underlying pathologies, psychotherapy

PROGNOSIS: varies based on willingness to attend therapy

25
Q

Factitous Disorder

A

ETIOLOGY: (unclear, behavior may be incentivized by attention)

S/S: individual may exaggerate their or someone else’s symptoms, self-imposed illnesses (i.e injecting foreign material), tampering with medical equipment

TREATMENT: believed to be untreatable (patients often find another caregiver when confronted), remove child from caregiver if caregiver-imposed

PROGNOSIS: varies based on duration of condition (worse if chronic)

26
Q

Gender Dysphonia

A

ETIOLOGY: (uncertain)

S/S: gender identity differs from sex, discomfort with assigned gender role, adopting of behavioral habits of opposite sex, possible low-self esteem from ostracism

TREATMENT: possible hormone therapy, possible surgical intervention, psychotherapy for low self-esteem

PROGNOSIS: varies on extent of patient’s feeling and social network

27
Q

Insomnia

A

ETIOLOGY: contributing factors include pain, stimulants (caffeine, amphetamines), anxiety, jet lag, noisy/hot/cold/bright/uncomfortable room

S/S: difficulty falling or staying asleep, nonrestorative sleep, irritability

TREATMENT: remove contributing factors if possible, psychotherapy if anxiety-induced, possible melatonin or benzodiazepine-class hypnotics

PROGNOSIS: varies on exact etiology, better if patient able to modify contributing factors

28
Q

Parasomnias (Sleep Arousal Disorders)

A

ETIOLOGY: psychological factors, brain tumors, high fevers, certain drugs, and genetic factors may precipate disorders

S/S: parasomnias include sleepwalking (occurs during NREM S3, no recall), night terrors (NREM 3, no recall), and nightmares (REM)

TREATMENT: preventing injury, eliminate psychological factors, eliminate precipitating drugs

PROGNOSIS: varies on exact etiology, but tends to recede into adulthood

29
Q

Narcolepsy

A

ETIOLOGY: strong genetic factors with REM stage occuring earlier in sleep

S/S: recurring compulsions to sleep (usually during monotonous activtiy)

TREATMENT: sleep-pattern changes, possible stimulants (methylphenidate or dextroamphetamine

PROGNOSIS: varies on response to therapy

30
Q

Sleep Apnea

A

ETIOLOGY: obesity, genetic defects, or bronchitis leads to respiratory tract obstruction

S/S: breathing may stop during sleep, nonrestorative sleep

TREATMENT: encourage weight loss, continuous positive air pressure (CPAP) machines, possible uvulopalatopharyngoplasty (UPPP)

PROGNOSIS: varies on etiology

31
Q

General treatment for personality disorders?

A

1) Psychotherapy (usually dialectal behavioral therapy, or DBT): identifying and altering thoughts and behaviors for interpersonal and emotional benefit
2) Possible drug therapy for comorbid disorders (antidepressants, anxiolytics)

32
Q

Cluster A Personality Disorders

A

Paranoid PD: suspicious and distrustful of others

Schizoid PD: prefer isolation, detached from others

Schizotypal PD: difficulty making or maintaining relationships, cognitive distortions related to everyday relationships

33
Q

Cluster B Personality Disorders

A

Antisocial PD: disregards social norms, can be manipulative or aggressive

Borderline PD: abrupt shifts in mood, unstable relationships, fear of rejection

Histrionic PD: desires attention, grandiose or promiscuous behavior

Narcissistic PD: extreme self-esteem, may react aggressively to criticism

34
Q

Cluster C Personality Disorders

A

Avoidant PD: fear of being judged by others, poor self-esteem, may feel socially inept

Dependent PD: relies on others to make decisions, may be clingy, fears being alone

Obsessive-Compulsive Personality Disorder: desires control, obsessed with perfection