Memory and Amnesia Flashcards

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1
Q

The three steps of the memory process:

A
  1. Encoding = The processing of information into the memory system
  2. Storage = The retention of encoded material over time
  3. Retrieval = The process of getting the information out of memory storage
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2
Q

What is required for memory?

A

Attention.

Our memory is an organ of reconstruction - this is made more difficult if we do not pay attention. Retrieval requires effort but this can be easier if we are prompted by something we paid attention to.

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3
Q

What is the main brain structure for memory?

A

Hippocampus - it is one of the few parts of the brain where neurogenesis (plasticity/cell regeneration) occurs which may explain why memory can get lost in retrieval. It is also why it is important to keep re-learning things from the past

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4
Q

What is the capacity of human memory?

A

Potentially infinite - there is no ‘space’ to fill with our memory it is a constantly changing area that keeps being reconstructed.

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5
Q

What is forgetting?

A

Functional remembering (unless it becomes pathological)

It is not the opposite of remembering - It allows us to update/modify our memories. It would not be beneficial to remember verbatim information so we forget unused information.

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6
Q

Why is memory unreliable?

A

Memory is a subjective thing - witnesses of the same event will have different accounts of it depending on their focus/attention/past experiences

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7
Q

The information deficit model

A

‘If we rely on memories that aren’t precise, we will construct them based on our beliefs’ - this is a poor model as we all do this

For example:
the hidden tiger = the grass moves, could be the wind but humans assume it is a tiger and run (in order to survive). If our brains worked like computers, we would think ‘statistically it is the wind’ and wouldn’t run - this might be right, but the one time it isn’t, we would die

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8
Q

What is cognitive offloading? (the google effect)

A

This is the cognitive consequence of having free access to all information immediately as we want it - we remember less.

60 Ps were asked to type/learn 40 factual statements - half were told to save their document and half were told to erase it. The erase group remembered significantly more than the save group

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9
Q

Benefits of note taking (Coria and Higham, 2018)

A

found that recall of a lecture was best for those who took handwritten notes rather than those who passively listened or annotated a PowerPoint.

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10
Q

What is the testing effect?

A

memory of a topic increases when some of the learning period is spent retrieving the ‘to-be remembered’ information
Meaning, self-testing soon after learning slows forgetting and improves recall

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11
Q

Do expectations influence our memory?

A

Expectations influence perception and so therefore influence our memory.
for example - the man who thought he was abducted by aliens bought an alien proof hat and hasn’t encountered aliens since - this is because his beliefs changed, he believes it works and so no longer has the expectation of alien encounters.

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12
Q

How does collective representation influence memory? (clock)

A

Collective representation is a view or image of something held by members of society that elicit widespread individual memories.
For example:
A clock in a train station broke following a terrorist attack and became a symbolic as it stopped at the exact time of the attack.
The clock was then fixed but when they asked train station workers/commuters (who saw the clock almost every da) if it was working (it was) 92% said no
The 92% remembered the clock being broken as it was an iconic symbol

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13
Q

How does previous knowledge effect memory?

A

Bartlett (1932) War of the Ghosts
He designed this story to see if cultural background and unfamiliarity with a text would lead to distortion of memory when recalling the story.
His hypothesis was that memory is reconstructive and that people store and retrieve information according to expectations formed by cultural schemas

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14
Q

How does change blindness effect memory?

A

Attention is required to encode memories - but humans aren’t good at this.
A study was carried out where an actor asked a participant for directions then two people carrying a picture came between them (a distraction to change the actor) very few people noticed they were talking to a new person.
this creates issues with EWT and identification as we can’t know if a person’s memories are reliable

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15
Q

What are the two traditions of human neuropsychology?

A
  1. Classical neuropsychology approach = what functions are disrupted by damage to region x?
    - addresses questions of functional specialisation, tends to use groups study methods
  2. Cognitive neuropsychology approach = can a particular function be spared/impaired relative to other cognitive functions?
    - addresses question of what the building blocks of cognition are, tends to use single cases study method
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16
Q

Structure of neuropsychological assessment

A
  1. interview = exploring the symptoms
  2. screening = test battery to identify areas of problem
  3. neuropsychological examination = reach clinical labelling
  4. experimental tests = make precise diagnosis
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17
Q

How do we collect evidence for the structure of the mind?

A

We use individual cases - if we collect enough individual cases of the same phenomenon, we can produce an average.

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18
Q

How do we study individual cases in neuropsychology?

A

Deficits that arise as a result of brain damage are used to explore how the normal brain operates.

Conversely, theories derived from models of normal cognitive functions can help form questions asked in the examination/diagnosis/rehabilitation of people with cognitive deficits

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19
Q

Main questions of neuropsychology:

A
  1. clinical neuropsychology = what happened to cause the particular symptom?
    A = (possibly) deficits are because of damage to a particular area (localisation)
  2. Cognitive neuropsychology = what does the pattern of impaired and intact capabilities teach us about the way the normal brain works
    A = (possibly) deficits are because of damage to the psychological processes which mediate the impaired function
20
Q

What is the science of falsifiability

A

Developed by Karl Popper - ‘no matter how many instances of white swans we encounter, we only need to observe one black swan to justify the conclusion that not all swans are white’

This applies to neuropsychology as, if a person with brain damage performs normally in certain skills, we can conclude that function is not run by the damaged network.

21
Q

the uses of single cases is not limited to neuropsychology

A
  • Alexander Lura reported symptoms of neglect in individuals before we had a term for ‘neglect’ he simply reported his unusual observations
  • the ability to walk upright was thought to be due to brain size until they found ‘Lucy’ who had a small brain
22
Q

What is double dissociation and why do we need it?

A

Some tasks are more cognitively demanding than others - just because we can’t do a task doesn’t mean we have separate areas in the brain for difficult work and easy work.
When observing deficits, we need a double dissociation - this is when a patient shows function A is easier than function B while another patient shows function B is easier as this shows both tasks are of equal difficulty and it is their deficits causing the impairment.

23
Q

How do we study normality?

A

We create a reference group of ‘normal’ people with no brain legions and compare our patients with deficits to this reference group

24
Q

How does the brain recognise faces?

A

Facial processing is a top-down process whereby sensory input with even the slightest suggestion of a face can result in the interpretation of a face.

25
Q

What would a lesion in the fusiform area of the brain do?

A

Cause difficulty in representing/remembering faces (even familiar faces)

26
Q

Bruce and Young’s cognitive model of face processing

A

Face
|
Structural Encoding (uses the rest of the cognitive system for…) \
| \
Face recognition Direct visual processing
unit (familiar faces) (unknown faces)
|
Person identity nodes
|
Name retrieval

27
Q

What are the two paths of facial recognition?

A
  • Emotional recognition (e.g. blocked by capgras delusion)
  • Cognitive recognition
28
Q

What is amnesia?

A

An acquired impairment of explicit LTM - due to brain legions or psychological factors

(STM and implicit learning are preserved and other components may be spared)

There are many causes of amnesia e.g. head injury, stroke, trauma, stress, malnutrition, Alzheimer’s etc.

29
Q

What is Alzheimer’s?

A

Alzheimer’s is characterised by an increased loss of memory due to the shrinkage of the hippocampus (more shrinkage = more loss)

30
Q

Recognising damage in brain scans

A

Brain scans can show grey matter, white matter and brain fluid (dark parts). When comparing brain scans to a healthy brain, any patches that aren’t supposed to be there can represent damage to certain areas.

31
Q

What is STM?

A

short term memory is memory for information currently ‘in the mind’ - it is very limited in capacity

32
Q

What is LTM?

A

stored information that need not be presently accessed or even consciously accessible - potentially unlimited capacity.

33
Q

Types of LTM

A

Declarative (explicit) memory
- episodic memory (events)
- semantic memory (facts)
(tested by recall/recognition)

Non-declarative (implicit) memory
- procedural memory (skills)
- perceptual representation system
- classical conditioning
- non-associative learning
(tested by priming tasks)

34
Q

how do we study memory systems?

A

We look at patients with impairments to understand how the system should work

35
Q

the Case of HM

A

HM had his temporal lobes severed (no hippocampus) to stop epileptic seizures. His procedural memory remained in tact and he could even learn new skills. He suffered from severe anterograde amnesia whereby he could not remember his address, current age, last meal etc.

IMPAIRED EXPLICIT LTM - damage to medial temporal lobe

36
Q

what is the serial position curve

A

This is the idea that when free recalling a list of items, we remember the first things in the list and the last things in the list but forget items in the middle.
- primacy effect = first items can be committed to LTM
- recency effect = last items are still in STM

37
Q

the Case of KF (shallice and Warrington, 1970)

A

Provides double dissociation of separate systems of memory (opposite of HM)

  • normal LTM = primacy effect intact
    IMPAIRED STM / DIGIT SPAN (recency effect is impaired) - damage to left parietal lobe
38
Q

What is the issue with terms like memory or amnesia?

A

They are umbrella terms for more specific things - this is why we study them through individual cases as we can see specific issues

39
Q

the Case of KC

A

Suffered a severe head injury that damaged hippocampus, coronal and sagittal views. He provides evidence for selective retrograde amnesia

  • Tulving et al (1988) = can’t recall personal events from pre-trauma but can remember facts and general knowledge learned pre-trauma
40
Q

What would be impaired from a lesion in the hippocampus?

A

episodic memory

41
Q

What would be impaired from a lesion in the parahippocampal region?

A

semantic memory

since this is close to the hippocampus, episodic and semantic memory are usually impaired together e.g. HM

42
Q

Graham et al 2000

A

Suggest a double dissociation of:
- early Alzheimer’s patients (hippocampus atrophy)
- ‘semantic dementia’ patients (temporal lobe atrophy)

43
Q

What is the function of the prefrontal cortex?

A

Maintenance and manipulation of information. This includes working memory (STM) and working with memory (e.g. encoding into LTM)

Patients with prefrontal cortex lesions often have disorganised memory

44
Q

What is confabulation?

A

Patient will give false information they believe to be true due to prefrontal cortex damage = amnesia due to a lack of control of memory rather than due to a memory deficit

e.g. patient claimed to have 4 children after 4 months of marriage - he had actually been married 33 years but rationalised his 4 children through adoption, they were not adopted.

45
Q

Retrograde amnesia

A

Difficulty remembering events/info from before the time of brain injury

46
Q

Anterograde amnesia

A

Difficulty forming new memories after the time of brain injury

47
Q

How does conditioning test memory?

A

conditioning creates a dichotomy between implicit and explicit memories.

Milner et al (1968) showed HM could learn Gollin’s incomplete picture task (of a fish).

When the picture progression to a fish is removed - normal people answer fish when asked what they were just shown and amnesiacs can’t remember. BUT when show the first picture (which is barely an outline of a fish) they remember it is a fish = implicit learning