Medications (quick class/action) Flashcards

1
Q

Acetaminophen

A

Class:
-Analgesic/antipyretic

Action:

  • Inhibits the synthesis of prostaglandins which transmit pain signals and induce fever.
  • Reduces pain by blocking the signals produced by prostaglandins.
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2
Q

Adenosine

A

Class:
-Antiarrythmic

Action:
-Slows electrical conduction through AV node, and interrupts re-entry pathway, converting SVT to NSR.

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3
Q

Albuterol

A

Class:
-Bronchodilator : Sympathomimetic (beta-2 specific)

Action:

  • Relaxes bronchial smooth muscle by stimulating beta-2 adrenergic receptors.
  • Produces bronchodilation, relieves bronchospasm, and reduces airway resistance.
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4
Q

Amiodorone Hydrochloride

A

Class:
-Antiarrhythmic

Action:

  • Blocks sodium, potassium, and calcium channels in cardiac cells slowing conduction and prolongs repolarization.
  • Has alpha and beta adrenergic blocking properties causing negative inotropic effects and reduces peripheral vascular resistance (afterload).
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5
Q

Aspirin

A

Class:
-Platelet aggregation inhibitor

Action:

  • Inhibits normal tendency for platelets to accumulate inside injured or occluded coronary arteries, thereby improving blood flow through the vessels to better perfuse the heart.
  • Blocks formation of Thromboxin A2.
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6
Q

Atropine

A

Class:

  • Antiarrhythmic
  • Anticholinergic

Action:

  • Competes with acetylcholine for receptor sites blocking the PNS response at SA & AV nodes.
  • Increases heart rate by increasing electrical conduction through the heart.
  • Positive chronotropic properties with little or no inotropic effects.
  • Inhibits secretions by decreasing PNS effect on bronchial, salivary, sweat, and GI glands.
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7
Q

Atrovent

A

Class:
-Bronchodilator : anticholinergic

Action:

  • Antagonizes the action of acetylcholine, preventing the interaction of acetylcholine with muscarinic receptors in bronchial smooth muscle causing bronchodilation.
  • Dries respiratory tract secretions.
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8
Q

Calcium Chloride

A

Class:
-Electrolyte

Action:

  • Increases myocardial contractility.
  • Increases ventricular automaticity.
  • Reverses cardio-electric changes produced by hyperkalemia.
  • Shifts potassium back into the cell to prevent intravascular hyperkalemia.
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9
Q

Charcoal

A

Class:
-Absorbent

Action:

  • Binds and absorbs ingested toxins present in the GI tract.
  • Inhibits intestinal absorption, preventing systemic toxicity.
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10
Q

Dextrose (D50/D10)

A

Class:
-Carbohydrate

Action:
-Increases blood glucose by providing a quick release of free sugar into the circulation.

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11
Q

Diphenhydramine

A

Class:
-Antihistamine

Action:
-Binds to histamine receptor sites blocking H1 and H2 receptors
>H1 causes bronchoconstriction, airway swelling, and
vasodilation.
>H2 causes secretion of gastric acid.
-Does not prevent histamine release.

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12
Q

Dopamine Hydrochloride

A

Class:
-Vasopressor: Sympathomimetic (both alpha and beta properties)

Action:

  • At low and medium doses, selectively dilates blood vessels supplying the brain, kidneys, heart, and gastrointestinal tract.
  • At medium to high doses, increases cardiac output by improving contractility and stroke volumem thereby increasing blood pressure.
  • At high doses, causes vasoconstriction and increased heart rate.
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13
Q

Epinephrine

A

Class:
-Catecholamine: Sympathomimetic (both alpha and beta effects)

Action:
-On the bronci: bronchodilation (beta 2)
-On the peripheral vasculature : vasoconstriction (alpha)
-On the heart:
   >Increased heart rate (chronotropic)
   >Increased contractility (inotropic)
   >Increased AV conduction (dromotropic)
   >Increased automaticity (dromotropic)
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14
Q

Fentanyl Citrate

A

Class:
-Analgesic: Opioid Agonist

Action:
-Potent narcotic analgesic and sedative.

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15
Q

Glucagon

A

Class:
-Antihypoglycemis: Pancreatic hormone

Action:

  • Increases blood glucose by converting glycogen stored in the liver to glucose.
  • Inhibits synthesis of glycogen from glucose.
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16
Q

Ketamine

A

Class:
-Anesthetic/Analgesic

Action:

  • Low & slow dosing provides strong analgesic effects acting on the limbic system and cortex to block afferent transmission of impulses associated with pain perception.
  • High & rapid dosing provides fast acting dissociative anesthetia (blocks sensory perception) which can lead to distortion of sights, colors, sounds, self, and one’s environment.
17
Q

Lidocaine

A

Class:
-Antiarrhythmic/Anesthetic

Action:

  • Depresses depolarization and automaticity in the ventricles.
  • Increases the ventricular fibrillation threshold by increasing phase IV repolarization.
  • Amide-type local anesthetic.
18
Q

Midazolam

A

Class:
-Benzodiazepine: Sedative/Anticonvulsant

Action:

  • CNS depressant.
  • Produces anterograde amnesia, then sedation.
  • Stops and prevents seizures.
19
Q

Morphine Sulfate

A

Class:

  • Opioid
  • Narcotic analgesic

Action:

  • CNS depressant acting on opiate receptors in the brain.
  • Potent analgesic and sedative.
20
Q

Naloxone

A

Class:
-Opioid antagonist

Action:
-Reverses respiratory depression, sedation, and hypotensive effects of opioid overdose by occupying opiate receptor sites.

21
Q

Nitroglycerin

A

Class:
-Vasodilator

Action:
-Produces vasodilation by relaxing systemic venous and arterial vessels, thereby:
   >Decreasing preload & afterload
   >Decreasing myocardial workload and
   >Decreasing mycoardial O2 consumption
   >Dilates coronary arteries
22
Q

Normal Saline

A

Class:
-Electrolyte/isotonic crystalloid

Action:

  • Electrolyte solution, which is osmotically equivalent to blood.
  • Increases the circulating volume of the vascular system. (2/3 of infused volume leaves the vascular space within 1 hour)
23
Q

Ondansetron

A

Class:
-Antiemetic

Action:
-Blocks serotonin receptors in vagus nerve terminals and in small intestine that trigger the vomiting reflex.

24
Q

Sodium Bicarbonate

A

Class:

  • Electrolyte
  • Alkalinizing agent

Action:
-Reduces acidosis of causes alkalosis by direct release of bicarbonate ion into the circulation.