med surg lecture 2 Flashcards
WHAT ARE THE TWO DIVISIONS OF THE NERVOUS SYSTEM?
CENTRAL NERVOUS SYSTEM
PERIPHERAL NERVOUSE SYSTEM
WHAT IS THE MAIN CELL OF THE NEUROLOGICAL SYSTEM?
NEURON
THE CENTRAL NERVOUS SYSTEM CONSISTS OF:
BRAIN
SPINAL CORD
THE PERIPHERAL NERVOUS SYSTEM CONTAINS:
NERVES OUTSIDE THE BRAIN AND SPINAL CORD
INCLUDES THE AUTONONMIC SYSTEM
THREE TYPES OF NEURONS FOUND IN THE CENTRAL NERVOUS SYSTEM
AFFERENT
INTERNEURON
EFFERENT
THE AFFERENT NEURON IS RELATED TO:
SENSORY
THE INTERNEURON IS RELATED TO:
RELAY
THE EFFERENT NEURON IS RELATED TO:
MOTOR
AFFECT OR SENSE
AFFERENT NEURON
EFFECT OR ACTION
EFFERENT
NEURON THAT COLLECTS DATA
SENSORY
NEURON THAT INTERPRETS DATA
INTERNEURON
NEURON THAT RESPONDS TO DATA
MOTOR
HOW MANY SPINAL NERVES ARE THERE?
31 PAIRS
HOW MANY PAIRS OF CERVICAL NERVES ARE THERE?
8
HOW MANY THORACIC NERVES ARE THERE?
12
HOW MANY LUMBAR NERVES ARE THERE?
5
HOW MANY SACRAL NERVES ARE THERE?
5
HOW MANY PAIRS OF COCCYGEAL NERVES ARE THERE?
1
THE LARGEST PORTION OF THE BRAIN?
CEREBRUM
SURFACE OF THE BRAIN MARKED BY THICK RIDGES
GYRI
SHALLOW GROOVES OF THE BRAIN THAT DIVIDE THE GYRI
SULCI
DEEP SULCI ARE CALLED
FISSURES
SITS BETWEEN THE CEREBRUM AND THE MIDBRAIN
DIENCEPHALON
THE SECOND LARGEST REGION OF THE BRAIN
CEREBELLUM
PART OF THE BRAIN THAT CONSISTS OF THE MIDBRAIN, PONS, MEDULLA OBLONGATA
BRAINSTEM
WHAT CONNECTS THE TWO HEMISPHERES OF THE BRAIN?
CORPUS CALLOSUM
WHAT ARE THE FOUR LOBES OF THE BRAIN CALLED
FRONTAL
PARIETAL
OCCIPITAL
TEMPORAL
WHAT DOES THE FRONTAL LOBE OF THE BRAIN AFFECT?
PERSONALITY
WHAT DOES THE TEMPORAL LOBE OF THE BRAIN AFFECT?
HEARING AND SMELL
WHAT DOES THE PARIETAL LOBE OF THE BRAIN AFFECT?
TASTE
WHAT DOES THE OCCIPITAL LOBE OF THE BRAIN AFFECT?
VISION
WHAT RELAYS SENSORY INFORMATION BETWEEN BRAIN REGIONS?
DIENCEPHALON
WHAT IS INCLUDED IN THE DIENCEPHALON?
THALAMUS
HYPOTHALAMUS
PITUITARY GLAND
COORDINATES MOVEMENTS, CONTROLS POSUTRE, BALANCE AND FINE MOTOR MOVEMENTS, INVOLVED IN MOTOR LEARNING
CEREBELLUM
IF A PATIENT HAS AN INJURY TO THE CEREBELLUM WHAT ARE THEY AT AN INCREASED RISK FOR?
FALLS
REGULATES HEART RATE, BREATHING, BLOOD PRESSURE, SWALLOWING, REFLEXES
BRAIN STEM
3 LAYERS OF CONNECTIVE TISSUE THAT COVER THE BRAIN AND SPINAL CORD
MENINGES
LOCATION OF CEREBROSPINAL FLUID FORMATION, 2 LATERAL AND 2 MIDLINE
VENTRICLES
PERMITS EXCHANGE OF NUTRIENTS/WASTE BETWEEN BLOOD AND NEURONS AND IS A CUSHION/SHOCK ABSORBER
CEREBROSPINAL FLUID
HOW MANY CRANIAL NERVES ARE THERE?
12 PAIRS
WHAT ARE THE TWO DIVISIONS OF THE AUTONOMIC NERVOUS SYSTEM?
SYMPATHETIC
PARASYMPATHETIC
FIGHT OR FLIGHT
SYMPATHETIC
REST AND DIGEST
PARASYMPATHETIC
THE SYSTEM THAT ALLOWS THE BODY TO DO THINGS WITHOUT BEING TOLD WHAT TO DO
AUTONOMIC NERVOUS SYSTEM
CHEMICALS THAT INITIATE THE SYMPATHETIC NERVOUS SYSTEM
NEUROTRANSMITTERS
NEUROTRANSMITTERS IN THE SYMPATHETIC NERVOUS SYSTEM
EPINEPHRINE
NOREPINEPHRINE
PROMOTES NORMAL FUNCTIONING OF ORGAN SYSTEMS
PARASYMPATHETIC NERVOUS SYSTEM
NEUROTRANSMITTER OF THE PARASYMPATHETIC NERVOUS SYSTEM
ACETYLCHOLINE
WHEN THIS SYSTEM IS ACTIVATED THE HEART RATE WILL INCREASE
SYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE BLOOD PRESSURE INCREASES
SYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE HEART RATE SLOWS TO BASELINE
PARASYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE BRONCHIAL SMOOTH MUSCLE DILATES TO IMPROVE OXYGENATION
SYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE INCREASED GAS SECRETION
PARASYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE BLADDER EMPTIES
PARASYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE PUPILS DILATE TO ENHANCE VISUAL ACTIVITY
SYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE STORED ENERGY IS CONVERTED TO GLUCOSE FOR BRAIN AND MUSCLES
SYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE BOWELS EMPTY
PARASYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE BRONCHIAL SMOOTH MUSCLE CONSTRICTS TO BASELINE
PARASYMPATHETIC
WHEN THIS SYSTEM IS ACTIVATED THE PUPILS WILL CONSTRICT TO FOCUS THE EYE FOR NEAR VISION
PARASYMPATHETIC
WHAT IS INVOLVED IN A NEURO ASSESSMENT
LEVEL OF CONSCIOUSNESS/ORIENTATION
VITAL SIGNS
PUPILS
STRENGTH/MOVEMENT
SENSATION
DECORTICATE POSTURING
ABNORMAL FLEXION BOTH EXTREMETIES COME UP TO THE CORE, LEGS EXTEND INAPPROPRIATELY
DECEREBRATE POSTURING
ABNORMAL EXTENSION – EXTEND INAPPROPRIATELY AND TURN PALMS OUTWARDS AND TOES POINT OUTWARDS
GLASGOW COMA SCALE IS USED TO ASSESS WHAT
LEVEL OF CONSCIOUSNESS
WHAT IS ASSESSED WHEN USING GLASGOW COMA SCALE?
EYE OPENING RESPONSE
BEST VERBAL RESPONSE
BEST MOTOR RESPONSE
BEST SCORE ON GLASGOW COMA SCALE
15
LOWEST SCORE ON GLASGOW COMA SCLAE
3
WHEN A PATIENT HAS A SCORE OF 8 OR LESS WHAT DOES THIS MEAN?
PT CAN NO LONGER PROTECT THEIR AIRWAY AND YOU HAVE TO INTERVENE
LESS THAN 8 INTUBATE
IF PATIENT HAS AN IMPAIRMENT OF THE CEREBRAL FUNCTION WHAT TYPE OF POSTURING WILL THEY HAVE?
DECORTICATE
IF A PERSON HAS DAMAGE TO THE BRAINSTEM WHAT TYPE OF POSTURING WILL THEY HAVE?
DECEREBRATE
WHAT IS IT CALLED WHEN PUPILS ARE UNEQUAL IN SIZE?
ANISOCORIA
WHAT CAN CAUSE ANISOCORIA?
PRESSURE IN THE BRAIN
WHAT IS INVOLUNTARY EYE MOVEMENT CALLED?
NYSTAGMUS
abnormal sensation (burning, tingling, pins and needles) (NERVE INVOLVEMENT WOULD BE ABNORMAL FINDING)
PARESTHESIA
POOR BALANCE (STUMBLING, STAGGERING GATE)
ATAXIA
SLURRED SPEECH SEEN WITH STROKES IS CALLED
DYSARTHRIA
DIFFICULTY WITH SPEECH IS CALLED
DYSPHASIA
ABSENCE OF SPEECH IS CALLED
APHASIA
WHAT ARE THINGS YOU CAN DO TO COMMUNICATE WITH PATIENT IF THEY HAVE DIFFICULTY WITH COMMUNICATION?
YES AND NO QUESTIONS
PICTURE BOARDS
PEN AND PAPER
SQUEEZE HAND/BLINK FOR YES OR NO
BE PATIENT
WHEN ASSISTING IN ADLs WHAT CAN WE DO AS NURSES FOR PATIENTS?
ENCOURAGE INDEPENDENCE
MAINTAIN ROUTINE
DIFFICULTY SWALLOWING CAN CAUSE ASPIRATION
DYSPHAGIA
THERAPEUTIC MEASURES WITH NUTRITION WHEN TROUBLE EATING?
EVALUATE SWALLOWING
ELEVATE HEAD OF BED WITH MEALS
DONT LEAVE PT DURING MEAL TIMES
THICKEN LIQUIDS, HIGH CALORIC FOOD, SMALL AND FREQUENT SNACKS OR MEALS
ALWAYS PLACE A PATIENT IN WHAT KIND OF POSITION?
FUNCTIONAL POSITION
PERMANENT MUSCLE CONTRACTION FROM LACK OF USE
CONTRACTURES
WHAT SHOES SHOULD YOU PLACE ON A PATIENT WHO BED BOUND AND WHY?
HIGH TOP SHOES TO AVOID FOOT DROP
WHAT CAUSES A SEIZURE
ABNORMAL ELECTRICAL DISCHARGES IN THE BRAIN RELATED TO INSTABILITY OF NEURONAL CELL MEMBRANES
WHAT ARE THE TWO CLASSIFICATIONS OF SEIZURES
PARTIAL
GENERALIZED
SEIZURES THAT BEGIN ON ONE SIDE OF CEREBRAL CORTEX
PARTIAL
SEIZURE WHERE BOTH HEMISPHERES OF THE BRAIN ARE INVOLVED
GENERALIZED
WHAT DIAGNOSTIC TESTING IS USED TO DETECT A SEIZURE?
EEG - ELECTOENCEPHALOGRAPHY
WHEN SHOULD AN EEG BE DONE FOR SEIZURE DETECTION?
AT THE TIME IT IS HAPPENING
WHAT ARE ACQUIRED REASONS FOR EPILEPSY?
FEVER
BRAIN BLEED
BRAIN INJURY
BRAIN SWELLING
SYMPTOMS OF A SEIZURE WILL WHAT?
CORRELATE WITH THE AREA OF THE BRAIN WHERE THE SEIXURE BEGAN
SENSATION THAT A SEIZURE IS ABOUT TO OCCUR
AURA
TYPES OF AURAS
VISUAL DISTORTION
ODOR
SOUND
IF A PATIENTS SEIZURE BEGINS IN ONE CEREBRAL HEMISPHERE IS IS CLASSIFIED AS WHAT TYPE OF SEIZURE?
PARTIAL SEIZURE
IF SYMPTOMS OCCUR ON OPPSOITE SIDE OF BRAIN AFFECTED IT IS WHAT TYPE OF SEIZURE?
PARTIAL
REPETATIVE PURPOSELESS BEHAVIORS ARE CALLED
AUTOMATISMS
AUTOMATISMS, PARESTHESIA, VISUAL DISTURBANCES AND INVOLUNTARY MOVEMENTS ARE ALL SYMPTOMS OF WHAT KIND OF SEIZURE?
PARTIAL
MAINTAINS CONSCIOUSNESS DURING SEIZURE IS CALLED
SIMPLE PARTIAL SEIZURE
LOSS OF CONSCIOUSNESS DURING A SEIZURE IS CALLED WHAT
COMPLEX PARTIAL SEIZURE
A SEIZURE THAT INVOLVES BOTH CEREBRAL HEMISPHERES?
GENERALIZED SEIZURE
STARRING OR BREIF SUDDEN LAPSES IN ATTENTION ARE WHAT KIND OF SEIZURES?
PETIT MAL ALSO KNOWN AS ABSENCE SEIZURES
TONIC-CLONIC SEIZURES ARE ALSO CALLED WHAT
GRAND MAL
SIGNS AND SYMPTOMS OF GRAND MAL/TONIC-CLONIC SEIZURES
RIGIDITY FOLLOWED BY MUSCLE CONTRACTION AND RELAXATION
USUALLY LOSES CONSCIOUSNESS
PUPILS FIXED AND DILATED
CLENCHED JAW OR BITING
MAY TEMPORARILY STOP BREATHING
INCONTINENCE
WHAT SHOULD YOU DO WHEN A PATIENT IS HAVING A SEIZURE?
START TIMING
TURN PATIENT ON SIDE
PREVENT INJURY
PAD SIDE RAILS
MONITOR VITALS
WHAT SHOULD YOU NOT DO WHEN A PATIENT IS HAVING A SEIZURE
LEAVE PATIENT
RESTRAIN
ATTEMPT CPR
PUT SOMETHING IN THEIR MOUTH
GIVE THEM FOOD OR WATER
WHY DO YOU PUT SOMEONE ON THEIR SIDE DURING A SEIZURE?
PREVENT ASPIRATION
WHEN WILL YOU CALL 911 DURING A SEIZURE?
IF THE SEIZURE LASTS LONGER THAN 5 MINUTES
TROUBLE BREATHING AFTER THE SEIZURE
THE PERSON APPEARS INJURED
THEY ASK FOR MEDICAL HELP
WHAT IS THE PERIOD AFTER THE SEIZURE CALLED?
POSTICAL PERIOD
WHAT IS COMMON IN THE POSTICAL PERIOD seizures
DISORIENTATION
MAY REMEMBER WHAT HAPPENED, MAY NOT
HEADACHES
FATIGUE
WHAT SHOULD YOU DO FOR THE PATIENT DURING THE POSTICTAL PERIOD?
LET THE PATIENT REST
WHAT ARE SEIZURE PRECAUTIONS
PADDED SIDE RAILS
SUCTION
OXYGEN AND EMERGENCY AIRWAYS
CALL LIGHT
IF SURGICAL INTERVENTION IS USED TO PREVENT SEIZURES WHAT IS DONE?
RESECTION OF THE AREA OF THE BRAIN AFFECTED BY SEIZURES
WHAT ARE FOUR MEDICATIONS USED TO PREVENT SEIZURES?
CARBAMAZEPINE (TEGRETOL)
LEVETIRACETAM (KEPPRA)
PHENYTOIN (DILANTIN)
VALPROIC ACID (DEPAKOTE)
WHAT DO SEIZURE MEDICATIONS DO?
SUPPRESS ABNORMAL DISCHARGE OF NEURONS
WHAT ARE TWO COMMON SIDE AFFECTS OF SEIZURE MEDICATIONS
DROWSINESS
DEPRESSION
WHAT LABS WILL YOU MONITOR WHEN A PATIENT IS ON SEIZURE MEDICATION?
LIVER FUNCTION
KIDNEY FUNCTION
HOW LONG SHOULD YOU WAIT TO ADMINISTER EMERGENCY MEDICATIONS FOR SEIZURES?
5 MINUTES
WHY ARE EMERGENCY MEDICATIONS FOR SEIZURES GIVEN?
BECAUSE SEIZURES CAN CAUSE RESPIRATORY DEPRESSION AND WE NEED TO PROTECT BRAIN FUNCTION
WHAT ARE THE EMERGENCY MEDICATIONS THAT ARE GIVEN FOR SEIZURES?
LORAZEPAM (ATIVAN)
DIAZEPAM (VALIUM, DIASTAT)
WHAT ROUTES CAN EMERGENCY MEDICATIONS FOR SEIZURES BE GIVEN?
RECTALLY
IV
NASAL
30 MINUTES OF CONTINUOUS SEIZURE ACTIVITY WITHOUT RETURN OF CONSCIOUSNESS
STATUS EPILEPTICUS
WHAT IS SOMETHING CONCERNING ABOUT STATUS EPILEPTICUS
OXYGENATION
PROMPT INTERVENTION IS NEEDED TO PREVENT NEUROLOGICAL DAMAGE DURING WHAT?
STATUS EPILEPTICUS
THERAPEUTIC INTERVENTIONS DURING STATUS EPILEPTICUS
ENSURE AIRWAY AND OXYGENTATION
ADMINISTER DIAZEPAM OR LORAZEPAM
ICU ADMISSION
INDUCED COMA
IF YOU ARE MAXED OUT ON DIAZEPAM OR LORAZEPAM WHAT WOULD BE THE NEXT STEP?
INDUCED COMA WITH PENTOARBITAL
WHAT IS A SYMPTOM OF NEUROCOGNITIVE DISORDER/DISEASE OR MEDICATION THAT IS NOT A NORMAL PART OF AGING?
DEMENTIA
INTELLECTUAL FUNCTIONING THAT DEMENTIA CAUSES
INPAIRED NORMAL ACTIVITES AND RELATIONSHIPS
IMPAIRED PROBLEM SOLVING AND EMOTIONAL CONTROL
PERSONALITY CHANGES
BEHAVIORAL CHANGES
MEMORY PROBLEMS
disorders that cause cognitive decline
NEUROCOGNITIVE DISORDER
HOW IS MEMORY AFFECTED WITH DEMENTIA?
RECENT MEMORY AFFECTED FIRST THEN REMOTE MEMORY AFFECTED LATER
SIGNS AND SYMPTOMS OF DEMENTIA
MEMORY LOSS
WANDERING
APHASIA
BEHAVIORAL PROBLEMS
TOTAL DEPENDENCE
THERAPEUTIC MEASURES FOR DEMENTIA
MEDICATIONS TO DELAY PROGRESSION
DISCUSSION OF WISHES/END OF LIFE DECISIONS
PATIENT SAFETY
REORIENTATION
MAINTAIN ROUTINE
MONITOR NUTRITION
CEREBROVASCULAR ACCIDENT IS ALSO KNOWN AS
STROKE
HOW DOES A STROKE AFFECT THE BRAIN
INADEQUATE BLOOD FLOW TO BRAIN
DECREASED O2 AND GLUCOSE TO BRAIN CELLS
BRAIN CELL/TISSUE DEATH
WHAT ARE THE TWO TYPES OF STROKES?
ISCHEMIC
HEMORRHAGIC
BLOOD SUPPLY IS BLOCKED OR SLOWED
ISCHEMIC STROKE
RUPTURE OF CEREBRAL BLOOD VESSEL
HEMORRHAGIC STROKE
TWO TYPES OF ISCHEMIC STOKES
THROMBOTIC
EMBOLIC
WHAT ARE THE TWO TYPES OF HEMORRHAGIC STROKES?
SUBARACHNOID
INTRACEREBRAL
A BUILD UP IN AN ARTERY THAT LEADS TO OCCLUSION EX: CARDIOVASCULAR DISEASE LIKE FAT BUILD UP
THROMBOTIC STROKE
EMBOLUS CAUSES OCCLUSION IN VESSEL EX BLOOD CLOT LODGED OR FAT EMOLISM LODGED
EMBOLIC STROKE
bleeding on the surface of the brain CAUSES PRESSURE ON THE BRAIN
SUBARACHNOID HEMORRHAGE
BLEEDING IN THE DEEPER TISSUES OF THE BRAIN
INTRACEREBRAL HEMORRHAGE
STROKE CAUSED BY A TRAMATIC BRAIN INJURY
INTRACEREBRAL HEMORRHAGE
MODIFIABLE RISK FACTORS FOR STROKE
HTN
SMOKING
DM
CARDIOVASCULAR DISEASE
ATRIAL FIBRILATION
OBESITY
HIGH CHOLESTEROL
NONMODIFIABLE RISK FACTORS FOR STROKE
AGE
GENDER
HEREDITY
PRIOR STROKE OR HEART ATTACKS
WHY COULD ATRIAL FIBRILATION CAUSE A STROKE?
IRREGULAR HEART RHYTHM CAN CAUSE CLOT IN THE HEART THAT IS DISLODGED FROM HEART AND GOES TO BRAIN
STROKE WARNING SIGNS
SUDDEN:
NUMNBESS OR WEAKNESS
CONFUSION
CHANGE IN VISION
TROUBLE WALKING/DIZZINESS
SEVERE HEADACHES
ACCRONYM USED FOR A STROKE
F - FACE IS IT DROOPING
A - ARMS CAN YOU RAISE BOTH
S - SPEECH IS IT SLURRED OR JUMBLED
T - TIME TO CALL 911 RIGHT AWAY
SIGNS AND SYMPTOMS OF A STROKE DEPEND ON WHAT?
THE AREA OF THE BRAIN AFFECTED BY THE STROKE
SIGNS AND SYMPTOMS OF A STROKE
ONE-SIDED WEAKNESS/PARAKYSIS
ATAXIA (UNSTEADY GAIT)
DYSPHAGIA
SENSORY LOSS
MENTAL STATUS CHANGES
VISUAL DISTURBANCES
SPEECH DISTURBANCES
SEVERE HEADACHE
WHAT DIAGNOSTIC TEST SHOULD BE RAN IMMEDIATELY FOR A STROKE?
CT SCAN
WHAT DOES A CT SCAN SHOW WHEN DONE FOR A STROKE?
IF BLEEDING IS PRESENT OR NOT
DETERMINES TYPE OF STROKE
WHAT TYPE OF STROKE WILL NOT PRESENT ON CT SCAN RIGHT AWAY AND HOW LONG DOES IT TAKE TO SHOW UP
ISCHEMIC STROKE AND IT WILL SHOW UP DAYS LATER
WHAT ARE OTHER WAYS TO FIND OUT WHAT CAUSED A STROKE?
ELECTROCARDIOGRAM
LAB WORK
CAROTID DOPPLER OR ANGIOGRAM
TIME LOSS = WHAT?
BRAIN LOSS
THERAPUETIC MEASURES OF A STOKE
GET PT TO CT SCAN
MONITOR AND TREAT ABCS
NEURO ASSESSMENTS OFTEN
CONTROL HTN
THROMBOLYTIC THERAPY AND MEDICINE MANAGEMENT
EDUCATION
THE USE OF DRUGS TO BREAK UP OR DISSOLVE BLOOD CLOTS
THROMBOLYTIC THERAPY
WHAT MEDICATIONS ARE INVOLVED WITH THROMBOLYTIC THERAPY
TISSUE-TYPE PLASMINOGEN ACTIVATOR (tPA)
WHAT TIME FRAME SHOULD TISSUE TYPE PLASMINOGEN ACTIVATORS BE GIVEN
WITHIN 3 TO 4.5 HOURS AFTER SYMPTOMS BEGIN
HOW DO TISSUE-TYPE PLASMINOGEN ACTIVATORS WORK
THEY MAY REVERSE SYMPTOMS BY RETURNING BLOOD FLOW TO BRAIN
WHAT TYPE OF STROKE IS TISSUE-TYPE PLASMINOGEN ACTIVATORS GIVEN FOR?
ISCHEMIC STROKES
IF TISSUE TYPE PLASMINOGEN ACTIVATORS ARE GIVEN FOR A HEMORRHAGIC STROKE WHAT COULD HAPPEN?
SIGNIFICANT RISK FOR BLEEDING AND MAKING THINGS WORSE SINCE THERE IS ALREADY BLEEDING WITH THIS STROKE. WE WANT BLOOD TO CLOT WITH HEMORRHAGIC STROKE
WHAT ARE THE TYPES OF STROKE PREVENTION MEDICATIONS?
ANTIPLATELET AGENTS
ANTICOAGULANT AGENTS
CHOLESTEROL-LOWERING AGENTS
WHAT SHOULD THE NURSE MONITOR WHEN GIVING MEDICATIONS FOR STROKE PREVENTION?
SIGNS OF BLEEDING LIKE:
BRUISING
LEVEL OF CONSCIOUSNESS
BLACK STOOL/EMESIS
STROKE PREVENTION MEDICATIONS ARE USED FOR WHAT
AND PUT PATIENT AT RISK FOR WHAT
PROLONGS BLEEDING TIMES SO THEY HAVE A RISK OF BLEEDING
WHAT DO ANTIPLATELET AGENTS DO?
LOWER PLATELET AGGRESGATION (PLATELETS DONT STICK TOGETHER LIKE NORMAL)
TWO ANTIPLATELET AGENTS
ASPIRIN
CLOPIDOGREL (PLAVIX)
WHAT DO ANTOCOAGULANT AGENTS DO?
PROLONGING TIME FOR CLOTS TO FORM
TWO ANTICOAGULANT AGENTS?
WARFARIN (COUMADIN)
HEPARIN
WHAT DO CHOLESTEROL-LOWERING AGENTS DO?
Lower LDL cholesterol
WHAT ARE THREE CHOLESTEROL-LOWERING AGENTS?
ATORVASTATIN (LIPITOR)
LOVASTATIN (MEVACOR)
SIMVASTATIN (ZOCOR)
LONG-TERM EFFECTS FROM A STROKE
IMPAIRED MOBILITY
IMPAIRED SENSATION
IMPAIRED SPEECH/SWALLOW
IMPAIRED JUDGEMENT/EMOTIONS
AFTER-STROKE INTERVENTIONS
REDUCE RISK FACTORS
PREVENTATIVE MEDICATIONS
HELP WITH ADLS
ASSIST WITH DEFICITS