Mat P3 WORKBOOK Flashcards

1
Q

what factors predispose women to DVTs?

A

inactive during labor and during early puerperium bc this inc risk of blood clot formation
prolonged time in birthing room w legs in stirrups
preexisting obesity and pre weight gain >recommended
preexiseting varicose veins
develop a postpartum infection
hx of previous thrombophlebitis
older than 35 or inc parity
high incidence in family
smoke cigarettes bc nicotine causes vasoconstriction

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2
Q

strategies to prevent DVT development?

A

ambulation, limiting time in stirrups or make sure well padded, support stockings for first 2 weeks after birth, instruct to remove them twice daily to look at skin for inflm, might be prescribed aspirin q4h

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3
Q

why is it dangerous to massage the area over a clotted area?

A

bc can loosen the clot causing a PE

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4
Q

measures to prevent development of mastitis?

A

-making certain baby is positioned correctly and grasps nipple properly
helping a baby release a grasp on the nipple before removing the baby from the breast
-washing hands between handling perineal pads and touching breasts
-expose nipples to air once a day
-vit K ointment daily to soften nipples
-encouraging women to begin breastfeeding on an unaffected nipple

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5
Q

what organism are associated with nosocomial mastitis?

A

infant usually acquired staphylococcus aureus, a MRSA infect or candidiasis

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6
Q

assessment findings associated with mastitis

A

usually unilateral, although epidemic mastitis bc originates from infant may be bilateral. Feels painful and appears swollen and red. Fever comes within first hours and breast milk becomes scant. Sonogram may be done

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7
Q

medical and nursing intervention to treat mastitis

A

abx effective against penicillin resistant staph such as diclozacillin or cephalosporin

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8
Q

should you continue breast feeding with mastitis?

A

yes if possible bc keeping breast empty prevents frowth of more bacteria. sometimes hurts from sucking so want to express milk until abs works (3 days or so)

cold or ice compress and supportive bra for pain

warm, wet compress dec inflm and edema

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9
Q

why are women prone to UTIs after delivery?

A

Because often woman are catheterized at the time of childbirth or during postpartal period and bacteria are introduced there.

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10
Q

Describe the assessment findings and therapeutic management of a UTI after delivery.

A

burning on urination, possibly hematuria, frequency, sharp pain on voiding, may have low grade fever and low andm pain,

urin spec but use a sterile cotton swab to tuck into vagina to avoid local discharge

mgmt: broadspectrum abs such as amoxicillin or ampicillin (make sure it is safe for breastfeeding)

lots of fluids

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11
Q

what does oxytocin do?

A

it is a hormone that stimulates smooth muscle contraction (uterus) similar to contractions. has vasopressor and antidiuretic effects

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12
Q

what does misoprostil do?

A

This medication may also be used in the hospital to assist with childbirth only at the time of delivery (e.g., cervical ripening, induction of labor) and for the treatment of severe bleeding after delivery.

inserted vaginally

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13
Q

what does ergometrine do?

A

type of medication called an ergot alkaloid and used to help prevent and control bleeding after childbirth.
acts on 3 different types of receptors walls of vessels and uterus. cause blood vessels to constrict and uterus to contract. contractions help deliver placenta. reduces blood flow to uterus which reduces blood loss

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14
Q

what does hemabate do?

A

IM injection to stop bleeding following pregnancy

also used to induce abortions.

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15
Q

what is ophthalmia neonatorum?

when does it occur?

how do you treat?

A

eye infection that occurs at birth during first month of life. generally bilateral conjuctivae become fiery red with thick pus and eyelids are edematous

occurs 1-4 days of life

treat with IV ceftriaxone and penicillin
sterile saline in eyes using dropper or bulb syringe

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16
Q

when do neonatal absintince symptoms appear and fade? (Drug-dependent mother)

A

24-48 hours and lasts about 2 weeks

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17
Q

if drug dependent mom was on methadone maintenance, will baby still show symp?

A

yes

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18
Q

how can you test if mom was using during birth? hint: test on infant

A

test in mec or urine sample of first hour of birth- narc metbaolites

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19
Q

what might present in preschool age if cocaine using mom?

A

maladaptive coping behaviours

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20
Q

cocaine infants have problem with?

A

sucking.
quiet dark room

woman advised not to BF

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21
Q

what are some common drugs used to counteract absintince symp?

A

morphine and phenobarbital. Others are methadone, chlorpromazine and diazepam,

22
Q

which babies are at risk for RDS?

A

preterm infants, infant of diabetic mothers, infants born by c/s or those who have dec blood perfusion of the lungs. surfactant

23
Q

when does surfactant usually form?

A

usually forms in the 34th week of gestation

24
Q

what are the S&S of RDS and when do they first appear?

A

(long) difficult resps at birth. After resuscitation they have hours or days of no symptoms bc initial release of surfactant but will have subtle symp such as: low boy temp, nasal flaring, sternal and subcostal retractions, tachypnea (more than 60RR), cyanotic mucous membranes. Within hours, expiratoy grunting occurs caused by the closure of the glottis,aas it tries to inc the p in alveoli on expiration in order to keep them from collapsing. Infants become cyanotic and their po2 and o2 sat level fall on RA

25
Q

as distress cont infant may exhibit?

A

Auscultation- fine rales and diminished breath sounds bc of poor air entry. As distress inc an infant may exhibit:

  • seesaw resps (inspiration- ant chest wall retracts and abdomen protrudes and expiration the sternum rises)
  • Heart failure, evidenced by dec output and edema of the extremities
  • Pale gray skin
  • Periods of apnea
  • Bradycardia
  • Pneumo
26
Q

Describe the action of surfactant and CPAP or PEEP oxygen administration.

A

CPAP or PEEP (positive end-expiratory pressure) exerts pressure on the alveoli at the end of expiration and help keep alveoli from collapsing and also supply o2.

27
Q

what is the risk of too much o2 on immature infant?

A

ROP (retinopathy of prematurity) or bronchopulmonary dysplasia

28
Q

what is transient tachypnea of the newborn? (TTN)

A

at birth, rapid reps d/t retained lung fluid normal (up to 80bpm)

29
Q

what is the cause of TTN?

A

slight dec in the production of of phosphatidylglcerol or mature surfactant, it is a direct result of retained lung fluid which limits the amount of alveolar surface that is available for oxygen exchange

30
Q

normal newborn RR and TTN rate?

A

normally rapid rate of 80 then goes to 30-60

TTN stays 80-120

31
Q

why is feeding difficult with TTN?

A

child cannot suck and breath rapidly at the same time

32
Q

which babies are more at risk for TTN?

A

born by C/s, infants whose mothers received lots of fluid during labor, preterm infants

33
Q

what nursing actions are required if an infant has TTN?

A

Close observation is priority. Watch carefully to be certain the inc effort is not tiring and other signs of resp distress such as nasal flaring or retractions are not occurring bc rapid resp rate is often first sign of resp obstruction. O2 may be necessary. Mild glucosteroid to reduce resp tract inflm

34
Q

when should TTN resolve?

A

eOnset is about 6 r and peaks in 36 hrs and then begins to fade around 72 hours as lung fluid is absorbed and resp activity is effective

35
Q

how soon does fetus have mec?

A

as early as 10 weeks gestation

36
Q

what causes mec aspiration syndrome?

A

if hypoxia occurs, stimulates the vagus nerve which relaxes the rectal sphincter and mec is released into the amniotic fluid

37
Q

why is MAS a concern?

A

Cau cause serious resp distress in 3 ways: inflm of bronchioles bc foreign substance, bloc small bronchioles b y mechanical plugging, and it can dec surfactant prod through lung trauma. Hypoxemia, co2 retention and inrapulmonar and extrapulmonary shunting can occur. Secondary infection of injured lung tissue can lead to pneumonia

38
Q

What signs and symptoms (assessment findings) will the nurse note with MAS?

A

difficulty establishing resps at birth. Apgar is low. Almonst immediately, tachypnea, retractions and cyanosis begin. After initiation of resps, an infants resp rate may remain rapid (tachypnea) and coarse bronchial sounds may be heard on auscultation. May continue to have retractions bc the inflm of the bronchi tends to trap air in the alveoli, limiting the entrance of oxygen. Air trapping may also cause englargement of anteroposteriod diameter of the chest. Poor gas exchange wth po2 and blood gases. CXR will show bilateral coarse infiltrates in the lungs with spaces of hyperaeration (peculiar honey comb effect) diaphragm will also be pushed downward by over-expanded lungs

39
Q

in MAS- why might the infants chest (anteroposteroid diameter) be enlarged?

A

air trapping

40
Q

what causes hemolytic disease of the newborn (hyperbilirubinemia)

A

Results from destruction of red blood cells by a normal physiologic process as the newborn breaks down excess red blood cells formed in utero. Can be from Rh incompatability or ABO incompatability

41
Q

What does the term hydrops fetalis mean?

A

severe form of Rh incept at birth. hydrops refers t the edema and fetalis to the lethal state.
total body swelling

42
Q

when does mom start to form Ab if baby is pos (rh)

A

within 72 hours after birth

43
Q

why are high bilirubin levels dangerous?

A

they are dangerous because the liver cannot convert all the indirect bilirubin produced too direct bilirubin so jaundice is extreme. High levels above 20mg/dl in a term infant causes brain damage from bilirubin-induced neurlogic dysfunction, a wide spectrum of disorders can occur from mild dysfunction to ABE (invasion of bilirubin into brain cells)

44
Q

explain the effect of early feeding (hyperbilirubenmia)

A

Initation fo early feeiding (urge mom to BF 8-10 times a day for first 2 days)- bilirubin is removed from the body by being incorporated into feces- sooner the elimination the sooner it is removed.

45
Q

effect of phototherapy for hyperbilirubinemia

A

Phototherapy- exposure to light triggers liver to assume function. Infant is exposed to liht such as quartz halogen, cool white daylight, or special blue fluorescent light. Placed 12-30 in above newborns bassinet or incubator. Generally scheduled for phototherapy when the total serum bilirub level rises to 10-12 mg/dl at 24 hours of age preterm is lower than this. Might admin iV immunoglobulin with phototherapy. Infants eyes are covered cause exposure to retina is damaging. Cover with dressing or cotton balls over a mask. Stools will be bright green and loose urine may be dark fro urobilinogen formation.

46
Q

what are the normal total serum bilirubin (TsB) levels? What TsB level becomes dangerous?

A

Normal I think? Is 0-3mg/100ml in cord blod? It is dangerous if it rises above 20mg/dl or as low as 12mg/dl in term infants

47
Q

Why does hemorrhagic disease of the newborn happen?

A

def of vit k- essential for formation of prothrombin by the liver. Lack of causes impaired blood coagulation. Formed by action of bacteria in the intestine but babys intestine is sterile at birth w minimal amonts of vit k until normal flora is established. Admin vit k to mom often protexts newborn.

48
Q

what med is given to babies with hemorrhagic disease of the newborn

A

1mg vit K IM

49
Q

what is necrotizing enterocolitis?

A

Intestinal dysfunction that develops in 5% of infants in ICY nurseries.
he bowel develops nectroic patches interfering with digestion and possibly leading paralytic ileus, perforation and peritonitis. Occurs bc of anoxia to the bowel and may result as a complication of exchange transfusion or an episode of dyspnea.

50
Q

what causes ROP (retinopathy of prematurity?

A

Caused by vasoconstriction of immature retinal blood vessels. Happens bc of high conc of oxygen exposure and vessels constrict, endothelial cells in the periphery of the retina proliferate causing retinal detachment and possible blindness