Mastitis - Clinical Presentation Flashcards
How is the milk different in clinical and subclinical mastitis?
Clinical - can see clots
Subclinical - only know when perform CMT/ assess cell counts
When does most infection which results in mastitis occur?
During the dry period
What may be responsible for mastitis which is seen in the first 50 days of milking?
Infection in dry period
Either only noticed when cows brought inside, or less able to fight infection due to metabolic demands of milking
What are the two broad categories of sources of bacteria which can cause mastitis?
Contagious
Environmental
How can poor cubicles lead to increased rate of mastitis?
Cows will lie down in passageways
Udders come into contact with more faeces
Which bacterial causes of mastitis are more ‘opportunistic’ ?
E. coli
Strep uberis
Strep dysgalactiae
Which bacterial causes of mastitis are more ‘host adapted’ ?
Strep agalactiae
Staph aureus
Strep dysgalactiae
Where might klebsiella be found?
Likes wet conditions - leaking water troughs
What factors affect the presentation of mastitis?
Host
Environment
Agent
Why should you be cautious of low reported levels of mastitis on farm?
suggests poor farm detection
How can lower cell counts benefit milk?
Makes it last longer
When is there a new case of mastitis in the same cow which has previously had mastitis?
in DIFFERENT quarter
How can mastitis affect the quality and quantity of milk?
Quantity - loss of actual and potential
Quality - residues from antibiotics/analgesics
- High SCC - financial penalty
How can the milking process increase the risk of mastitis?
How can you minimise this risk?
If vacuum too extreme, or milking too long
Teat canal can ‘pop out’ resulting in keratosis
More susceptible to infection
PRE-STRIP and prep before milking
Describe the basic progression of events which results in clinical mastitis.
Exposure to pathogen
Entry in the teat and mammary gland
Establishment of infection
What is ALWAYS seen in clinical mastitis?
Changes in the milk