Mass Transport In Animals Flashcards
Low O2 environment
Needs higher affinity
Dissociation curve shifts to left
“Load more oxygen into body”
High O2 environment
Graph shifted to right
Organism can afford to have lower affinity
Dissociates more in tissues
“Unload more oxygen into respiring tissues”
Smaller animals
Lose heat faster
Faster metabolisms
Need to unload more oxygen into cells
Graph shifted to right
Ventricles relax, atria relax (stage 3)
Atria fill again due to higher pressure in vena cava and pulmonary vein
Causes pressure to increase in atria, ventricles continue to relax until pressure in ventricles lower
Blood passively flows into ventricle as AV valves open
Atria contract to repeat process
Systole
Cardiac contraction
Diastole
Cardiac relaxation
Cords
Attach AV valves to ventricles to stop them being forced up into the atria when the ventricles contract.
Arteries
Muscular and elastic tissue
Stretch and recoil as heart beats
Maintains high pressure
Endothelium folded— stretching
Veins
Contain valves to prevent back flow (low pressure)
Blood flow helped by surrounding muscle contraction.
Why water re enters at venule end of capillary
Lower hydrostatic pressure due to fluid loss and increased concentration in plasma proteins
Development of Atheroma
Damage to endothelium
White blood cells and lipids in blood clump together under the lining (endothelium) to form fatty streaks
Builds up, along with connective tissue, hardens to form fibrous plaque
Partial blockage of artery- causes higher blood pressure and restricted flow
Aneurysm
Blood travels through weakened artery at high pressure
Pushes inner layers of artery through outer elastic layer to form a balloon like swelling
Bursts to form haemorrhage
Thrombosis
Atheroma can rupture endothelium, damaging it to leave a rough surface
Platelets and fibrin accumulate to form a blood clot at site of damage (thrombus)
Blocks artery
Can dislodge and block vessel elsewhere
Debris from rupture can cause another blood clot further down artery
Factors increasing risk of CVD
High blood cholesterol as cholesterol is a main constituent to the fatty deposits.
Diet high in saturated fat
Smoking (nicotine increases risk of high blood pressure, CO binds to haemoglobin, decreases amounts of antioxidants in the blood (antioxidants protect cells from damage—— Atheromas)
Increased blood pressure: overweight, lack of exercise, excessive alcohol consumption
High concentration of carbon dioxide (due to cell respiration)
Increases rate of oxygen unloading/ dissociation from haemoglobin
Curve shifts to the right
(Bohr effect)