Lower Extremity Injuries Flashcards
How should a suspected PROXIMAL FEMUR FRACTURE be managed ACUTELY?
- MEDICAL EMERGENCY
- Call EMS
- Treat for shock
- Splint before moving them
- test neurovascular structures periodically
- most require surgery
If an acetabular fracture is missed - what can result?
- Avascular necrosis of the femoral head
What would prompt URGENT HOSPITALIZATION with a Traumatic PELVIC FRACTURE?
- Disruption of the pelvic ring
- Abdominal organ injury or Hemorrhage
*no disruption of ring = treat based on symptoms
What is a test used to diagnose Femoral shaft Stress Fractures?
- FULCRUM TEST (assessor’s arm under thigh; pushes superior to inferior at distal thigh; +ve = pain and apprehension)
What types of femoral stress fractures are HIGH RISK? How are they treated?
- LATERAL FEMORAL NECK
- FEMORAL HEAD
- Surgical Stabilization OR STRICT non-weightbearing (until callous forms)
- typically occur at neck and are due to COXA VARA
What is the typical TREATMENT for LE STRESS FRACTURE?
- PROPER diagnosis is important
- refer to MD
- Weight bearing restrictions (Non or limited)
What is the typical CAUSE of Lower Extremity APOPHYSITIS? What are expected RTP timeframes?
- Chronic traction forces
- RTP =2-6 months (~3.1 month average)
How should an uncomplicated ACUTE AVULSION fracture in the lower extremity be treated?
- TTWB for 1-2 months
- gradual progression of strengthening and stretching of impacted muscle as tolerated
- if displaced - may need surgery
What anatomical factors increase the prevalence for hip LABRAL pathology?
- Coxa Vara
- increased center-edge angle (measures anterior coverage of femoral head by acetabulum)
- Retroverted femur
- Retroverted acetabulum
How should LABRAL PATHOLOGY of the hip be managed ACUTELY?
- REST
- Protected weightbearing
- avoiding TRANSVERSE plane movement
What are some DIFFERENTIAL DIAGNOSES for FAI?
- Osteitis pubis
- Athletic Pubalgia
- Lumbosacral Pathology
Does FAI typically present with palpatory tenderness?Weak hip flexors and abductors?
- No
- Yes
What are some STRUCTURAL DEFORMITIES that can contribute to hip instability?
- Shallow acetabulum (contributes to labral tears)
- Excessive acetabular retroversion or anteversion
- Inferior acetabular insufficiency
- Neck-shaft angle >140 deg (coxa valga)
What is FOCAL ROTARY INSTABILITY of the hip?
- Laxity at ligamentous (often iliofemoral ligament) or capsular structure DUE TO repeated forceful rotation at hip (i.e.: golf, ballet, martial arts, baseball)
What is the most common direction of HIP DISLOCATION? What is the MOI?
- ~85% POSTERIOR
- MOI = ant to posterior force on flexed and adducted hip
How should a HIP DISLOCATION be managed ACUTELY?
- EMERGENT reduction (avoid Osteonecrosis)
* evaluate neurovascular status of leg (cutaneous nerve function, distal pulses - popliteal and dorsalis pedis)
What is the HIP APPREHENSION sign? What is it used for?
- Passively EXTEND, ABDUCT, EXTERNALLY ROTATE = may report sensation of instability
- to test for atraumatic hip instability (hip is typically unstable in the posterior direction which is the most common direction)
What activities or movements should the athlete with hip instability avoid?
- FORCEFUL HIP EXTENSION
- FORCEFUL ROTATIONAL LOADING
*these movements stress passive restraints
What are some SPECIAL TESTS to determine atraumatic HIP INSTABILITY?
- FABER
- FADIR
- HIP IR >30 deg at 90 deg flex
- hip APPREHENSION sign
What are some PRIMARY treatment focuses with REHAB for patients with atraumatic hip instability ?
- FOCUS ON NEURO RE-ED
- STRENGTHEN HIP ABDUCTORS AND ROTATORS (to assist in supporting limited passive restraints)
Severe hip flexor injuries can lead to profound swelling which can cause what?
FEMORAL NERVE PALSY
What can be predictive for adductor strains?
Relative strength deficits between hip abductors and adductors
What is the most commonly injured hip adductor muscle?
- ADDUCTOR LONGUS
* has excellent blood supply - can rehab aggressively
What muscles are indicated in ATHLETIC PUBALGIA (sports hernia)?
PUBIC ATTACHMENTS OF:
- RECTUS ABDOMINUS
- HIP ADDUCTORS
*often attributed to imbalance - strong adductors and weak abdominals
Some ATHLETIC PUBALGIA signs and symptoms include…?
- intense, deep groin/lower abdominal pain
- worse with: SPRINTING, CUTTING, RESISTED SIT UP TEST
- pain with RESISTED ADDUCTION and PALPATION TO PUBIC RAMIS
What are SURGICAL OPTIONS for ATHLETIC PUBALGIA?
- ABDOMINAL WALL REPAIR
- ADDUCTOR TENOTOMY
- rehab starts ~4 weeks post op
- avoid trunk extension and rotation to protect surgical site
What is the difference between INTERNAL and EXTERNAL SNAPPING HIP SYNDROME?
- INTERNAL = iliopsoas tendon snaps over iliopectineal eminence OR femoral head
- EXTERNAL (most common) = IT band or GLUTE MAX snaps over greater trochanter (sagittal and transverse movements)
For patients with EXTERNAL SNAPPING HIP, what muscle tends to be overactive AND what muscle tends to be underactive?
- Overactive = TFL
- Under active = Glute max
*Have been shown to glute eccentric weakness
What chance do partial ACL ruptures >50% have of complete rupturing?
> 50% chance of progression to full rupture
At what point post ACL RECONSTRUCTION can you initiate jogging, hopping, jumping? What criteria must be met?
- 12 weeks
- STRENGTH AND ROM NORMALIZED
- NO LAXITY PRESENT - graft is secure
What is the MOI for the entire MCL (deep and superficial fibers)? What is the MOI for the SUPERFICIAL MCL?
- Entire MCL = knee extended
- Deep fibers = knee flexed >20 deg
Injury to the deep fibers of the MCL are associated with what other structures?
ACL, medial meniscus, bone bruise, or osteochondral injury
*Deep fibers attach to medial joint capsule and medial meniscus
When valgus stress testing the MCL, what is the difference in laxity between grades 2-1+, 2-2+ and 3?
2-1+ = 0-5 mm laxity, end feel present 2-2+ = 5-10 mm laxity, end feel difficult to determine 3= no end feel (complete rupture)
How are MCL tears treated? What is the timeline for RTP?
- Almost exclusively treated CONSERVATIVELY
- get terminal KNEE EXTENSION ROM ASAP
- RTP = grade 1 = 7-10 days
Grade 2 = up to 3 weeks - can brace (PT, MD dependent)
What is the POSTEROLATERAL COMPLEX region of the knee made up of?
- POSTERIOR JOINT CAPSULE
- LCL
- POPLITEUS TENDON
- POPLITEOFIBULAR LIGAMENT
What are some tests to aide in DIAGNOSIS of POSTEROLATERAL COMPLEX injuries?
- Knee Varys Test (laxity)
- Dial Test (laxity)- prone, flex knees, rotate tibias
- ER Recurvatum Test of Hughston (laxity)
What is the MOI for PCL injury?
- HYPERFLEXION
- BLOW TO FRONT OF KNEE (TIBIA) WHILE KNEE FLEXED (dashboard)
What do POSTEROLATERAL COMPLEX injuries usually occur in conjunction with?
- LCL sprains
- cruciate sprains
What are 3 tests to determine injury to the PCL?
- posterior sag sign
- posterior drawer test
- Clancy Step off test (thumbs palpate femoral condyle, IP joints palpate tibial plateau - which should be 1 cm in front of condyles; any deviation = positive)
What is the BIGGEST concern following PCL injury?
- RESIDUAL TIBIAL LAG (after reconstruction and surgery)
* Avoid hamstring exercises the first 6-8 weeks
How is a DISLOCATED patella (usually laterally) RELOCATED?
- MOVING FLEXED KNEE INTO FULL EXTENSION (ACTIVELY OR PASSIVELY)
How should a DISLOCATED PATELLA be managed ACUTELY?
- IMMOBILIZE
- ICE
- TRANSFER for imaging (due to risk for fracture or osteochondral injury)
What are BRACING recommendations post LATERAL PATELLA DISLOCATION?
- Immobilize 3-4 weeks
- post immobilization - progress to less restrictive brace with LATERAL BUTTRESS (limits lateral translation)
What is SURGICAL treatment of choice for LATERAL PATELLAR DISLOCATION if conservative treatment fails?
- MPFL repair or reconstruction
Why is a TIBIOFEMORAL DISLOCATION considered a medical emergency?
- Close proximity of NEUROVASCULAR structures in POPLITEAL FOSSA
- Can result in: vascular issue (loss of limb), or inability to walk (neurological compromise)
- IMMOBILIZE AND TRANSPORT
- sometimes it may REDUCE spontaneously = makes it difficult to diagnose
- if 3 or more ligaments have been injured = knee should be considered DISLOCATED (regardless of alignment)
What are signs and symptoms of TIBIOFEMORAL DISLOCATION acutely?
- Extreme pain
- Significant swelling
- Shock - WATCH FOR IT
How should TIBIOFEMORAL DISLOCATION be managed ACUTELY?
ACUTELY:
- Call EMS; Monitor neurovascular status (popliteal pulse, peroneal and tibial nerves)
- immobilize in EXTENSION for at least 4 weeks
How does knee MENISCAL injury type present differ as it’s related to AGE of the patient?
- YOUNGER = meniscus (with ACL) = peripheral injury (more likely to heal intrinsically)
- OLDER = tears in less vascular area = less amenable to repair
What are the RTP recommendations post partial meniscectomy ?
- ~6-8 weeks
* usually WBAT as tolerated immediately (no brace required)
What are the post-surgical requirements regarding weight-bearing status and brace use post MENISCUS REPAIR? What are ROM and strengthening restrictions? What is RTP timeline?
- ~4-6 weeks brace (locked into extension - depending on type and repair); WBAT or PWB (depending on repair, surgeon)
- flexion ROM <90 deg (first 6 weeks)
- strengthening progress as tolerated at 6 weeks
- RTP = 16-20 weeks
Differentiate between the 4 grades of OSTEOCHONDRAL INJURIES.
- Grade 1 = softening, swelling of cartilage
- Grade 2 = fissuring, fragmentation <0.5 inch
- Grade 3 = fissuring, fragmentation >0.5 inch
- Grade 4 = erosion down to subchondral bone
What are the signs and symptoms of OSTEOCHONDRAL INJURIES?
- PAIN and SWELLING with activity that relieves with rest
- no SPECIAL TESTS (best DX via radiographs, scope)
- symptoms may be MINOR until defect comes loose
What are the SURGICAL OPTIONS for OSTEOCHONDRAL INJURIES?
- Microfracture
- Osteochondral Autologous/Allograft Transplantation (OATs)
- Autologous Chondrocyte Implantation
- Debridement
- Lavage
What are the REHAB PRECAUTIONS post surgical intervention for OSTEOCHONDRAL injuries?
- Limited WB 4-6 weeks post (except with posterior patella, or in trochlear groove - can WBAT with brace in full extension)
- LOAD GRADUALLY
What is the path of the patella during knee movement from extension to flexion?
- TRANSLATES slightly medial as it enters the femoral condyles @20-30 deg flexion
- follows femoral condyle and TRANSLATES slightly laterally within the groove as it moves into greater flexion
What are some common activities that may EXACERBATE SYMPTOMS for those with PFPS?
- SQUATTING
- STAIRS
- KNEELING