LO1/2 Adverse Environments Flashcards
Thermoregulation
Heat production and excretion
Hypothalamus
thermostat of the brain
afferent
efferent
Skin to nervous system afferent pathways
Hypothalamus to body efferent pathways
Lowest temp a human can survive
is 13.7
hypothermia vs hyperthermia
Hyperthermia
Vasodilation (increased HR)
Hairs flat
Sweat – dermal layer of skin
Hypothermia
Vasoconstriction
Piloerection
Shivering
Thermolysis
release of stored heat
Mediated by parasympathetic nervous system
An increase in core temperature causes
the hypothalamus to send signals via Efarrant pathways in the parasympathetic nervous system causes vasodilation and sweating
Body’s reaction to daily production of heat energy and to hot environment
Vasodilation:
The person may have a complete loss of vasomotor control blood pools in the periphery and the patient could experience distributive shock
Radiation
body heat is lost to nearby objects without physically touching them (heat waves- sun)
Conduction
body heat is loss to nearby objects through direct physical touch (touch)
Convection
body heat is lost to surrounding air which becomes warmer, rises abd is replaced with cooler air (air moving over- an object-fan)
Evaporation
body heat causes precipitation which is lost from the body surface when changed from liquid to vapor (sweating)
Thermogenesis
Production of heat and energy for the body
Mediated by the sympathetic nervous system
Main method of dealing with cold stressors
Skin is the body’s thermostat.
Increases muscle tone and initiate shivering in the short term
Increases thyroid levels in the long term
Hypothalamus shunts blood to the core.
Sweating decreases.
Heat Illness
Increase in core body temperature
- -Due to inadequate thermolysis
- -Inability to get rid of the heat buildup in the body
Heat Cramps
Acute involuntary muscle pains
Usually in the lower extremities, the abdomen, or both
Occur because of profuse sweating and subsequent sodium loss
3 factors contribute: salt depletion, dehydration and muscle fatigue
Heat Cramps Treatment
Stop activity
Move the patient to a cool environment.
If the patient is too nauseated to take liquids by mouth, insert an IV catheter and infuse normal saline rapidly.
Do not massage the cramping muscles.
Heat Syncope
Typically occurs in nonacclimated people
Can occur with prolonged standing or when standing suddenly from a sitting or lying position
Peripheral vasodilation is thought to be the cause.
Treatment involves placing the patient in a supine position and replacing fluid deficits.
Water depleted
This form primarily effects geriatric patients.
Can effect active younger workers and athletes who do not adequately replace fluids in a hot environment
Sodium Depleted
May take hours or days to develop
Results from huge sodium losses from sweating but replacing only free water
Heat Cramps s/s
Temp < 40
No alt LOC
sweating
Headache, fatigue, dizziness, nausea, vomiting, and, sometimes, abdominal cramping
Skin is pale and clammy.
Fast and shallow respirations
Tachypnea
Rhabdomyolysis
muscle breaks down releases potassium which can kill you (brown urine)
“Summer flu”
Misdiagnosed
If untreated may progress to heat stroke
Heat Exhaustion – TREATMENT
Stop activity
Remove to cool area
Remove clothing
Replace oral fluids - water or electrolyte fluid, no stimulants
If decrease LOC withhold fluid
Monitor ABC and LOC, if changes treat as load and go
Cardiac monitoring, ETCO2