Liver CTC Flashcards

1
Q

what is brighter on T1, liver or spleen?

A

liver

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2
Q

what is brighter on T2, liver or spleen?

A

spleen (spleen is a big bag of water that collects blood)

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3
Q

most likely case being shown if given a view of the gallbladder fossa

A

some kind of fatty lesion problem - like focal fatty infiltration or sparing

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4
Q

fat related artifacts on MR

A

Type 1 “chemical shift” Type 2 “india ink” (ip and oop)

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5
Q

chemical shift type 1 artifact occurs in what direction?

A

frequency encoding

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6
Q

how do you decrease chemical shift type 1 artifct

A

increase the rBW (broader bandwidth decreases the artifact)

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7
Q

what will make chemical shift type 1 artifact worse?

A

narrow receiver bandwidth or HIGH field strength (worse on 3T compared to 1.5T)

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8
Q

with what sequences do you see chemical shift type 1 artifact?

A

spin echo and gradient echo sequences

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9
Q

type 2 artifact only occurs on what type of sequence?

A

Gradient only

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10
Q

drop of signal in the liver on in phase imaging`

A

iron in the liver

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11
Q

what phase do you have to scan first in regards to in phase and out of phase imaging

A

HAVE to do OOP first, otherwise you can’t tell a an iron laden liver from a fatty liver

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12
Q

primary hemochromatosis involves the liver and _____

A

pancreas primary = “p” for pancreas

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13
Q

secondary hemochromatosis involves the liver and __________

A

spleen secondary = “s” for spleen

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14
Q

epidemiology of hemachromatosis

A

white dudes

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15
Q

hooked osteophytes on metacarpal heads

A

hemochromatosis

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16
Q

super dense liver

A

AMIODARONE should be your first thought others include: Wilsons/hemochromatosis, Thorotrast, Glycogen storage disease

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17
Q

Caroli’s disease associations

A

ADPKD, Medullary sponge kidney, cholangiocarcinoma

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18
Q

what biliary disease is associated with ADPKD and medullary sponge kidney

A

Caroli disease

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19
Q

PSC gender predisposition

A

PSC is way more common in males

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20
Q

male with biliary strictures

A

PSC

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21
Q

associated with PSC

A

UC

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22
Q

UC is associated with what biliary pathology?

A

PSC

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23
Q

when is cholangiocarcinoma unresectable?

A

second order radicles, main portal vein is encased or occluded contralateral hepatic artery and portal vein are involved

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24
Q

hepatic capsular retraction in a male patient with PSC

A

cholangiocarcinoma

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25
Q

big bloody hepatic mass in a young person (female or young dude on roids)

A

hepatic adenoma

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26
Q

blood hepatic mass in a cirrhotic

A

HCC

27
Q

can hepatic adenomas undergo malignant degeneration?

A

yes the “beta catenin mutated” ones

28
Q

why do they resect hepatic adenomas>?

A

two things: 1) risk of bleeding, and 2) risk of malignant transformation (beta catenin)

29
Q

transporter that allows Eovist into hepatocytes

A

OATP (not present in cancer cells –> HCC and other cancers are big black holes on Eovist scans)

30
Q

most common location for a hepatic adenoma

A

right lobe, subcapsular (85%)

31
Q

multiple hepatic adenomas, systemic disease association

A

glycogen storage disease (Von Gierke’s)

32
Q

glycogen storage disease (Von Gierke’s) is associated with what hepatic finding

A

multiple hepatic adenomas

33
Q

what signal characteristic is your main differentiator between dysplatic nodule and HCC?

A

T2! HCC will be T2 bright

34
Q
A
35
Q

your tech tells this pt that they have cancer and you are called to the scanner to console the patient. What is your next step?

A

get delayed imaging. See if it fills in and is a cavernous hemangioma

36
Q
A
37
Q
A

FNH

nuc med study = sulfur colloid (Kupffer cells in the FNH are hungry for colloid)

38
Q
A

biliary hamartomas (Von meyenberg complexes)

key on MRCP –> the bright cysts don’t connect to the biliary system

39
Q

young dude with UC

A

PSC

40
Q
A
41
Q
A
42
Q

is PET good for HCC

A

No, PET is absolute shit to evaluate for HCC

43
Q
A

secondary hemochromatosis

Secondary = Spleen

44
Q
A

primary hemochromatosis

Primary = Pancreas

45
Q
A

sickle cell, auto infarcted spleen

46
Q
A

Iron overload

“drop” of signal on In phase

47
Q
A

Fatty liver

Obese, drop of signal on Out of phase

48
Q
A
49
Q
A

heaptic veins are out

50
Q
A

if show hepatic veins out…Budd Chiari

if showed reflux of contrast into IVC..Right heart failure

51
Q
A

“rocky liver” = calcificaiton in the liver

mucinous adenocarcinoma met (CRC)

52
Q

key imaging feature of fibrolamellar HCC?

A

it calcifies!

53
Q

18 yo F

A

fibrolamellar HCC

calcifies, central scar, young people\

rare AF

54
Q

has HIV and likes IV drugs

A

infection, infection, infection

55
Q

elevated LFTs

A

hepatitis

GB wall thickness >1cm is NOT acute cholecystitis

56
Q
A

hepatitis

periportal edema without anyother imaging clues for other etiologies

57
Q

is there any situation when an adult can get HCC in a non cirrhotic liver

A

HBV

58
Q
A

hot quadrate lobe + chest wall collaterals

= SVC syndrome

59
Q

what is the most likely diagnosis?

A

biliary necrosis secondary to hepatic artery stenosis

60
Q

describe the following waveforms and what they are indicative of

A
61
Q

what waveform is sensitive for severe hepatic artery stenosis

A

elevated diastolic flow

62
Q

sensitive AND specific signs for severe proximal stenosis

A

Elevated diastolic flow AND tardus parvus

63
Q

What other imaging should you get?

A

Get a brain MRI to look for brain abscesses.

This is HHT –> right to left shunt –> bugs to brain

64
Q

pregnant lady

A

HELLP