Likely Stations Flashcards

1
Q

Paediatric Head Injury

2023.2 History Taking Station

take a focused history from a parent of a paediatric patient with a head injury

EM rapid bombs on concussion ep 9

Rule out child abuse
PECARN
Using CT responsibly
- Radiation exposure
ALARA “as low as reasonably achievable”
sedation for CT - IN midazolam
Concussion

Medical Expertise: Assessment (40%)
 Elicit a focused relevant history de novo.
 Clarify aspects and inconsistencies in a pre-obtained history to refine diagnosis generation.
 Identify important historical details (red flags) diagnostic of an important condition.
 Generate a differential diagnosis, with an inherent focus on conditions requiring time critical
management.

Medical Expertise: Management– 30%
 Create an appropriate ongoing assessment and management plan.
 Manage multiple problems simultaneously by prioritising treatment options while remaining
vigilant for other potential problems.
 Provide a rationale to explain decisions about ongoing assessment.

Communication – 30%
 Use language appropriate to the patient’s level of understanding e.g., avoid jargon, explain
medical terms.
 Demonstrate a professional and respectful approach.
 Summarise the encounter and confirm patient understanding.

Candidates were required to meet with the parent of the child (role player) and to:
 Obtain a detailed history from the parent.
 Explain your management plan to the parent.
 Answer any further questions from the parent

A

PECARN:

age >2
*LOC
*Vomiting
*severe headache
*Severe mechanism
–> observation

*GCS < 15
*Altered mental status (Agitation, somnolence, repetitive questioning, or slow response to verbal communication)
*Signs of base of skull fracture
–> CT brain

HISTORY:

Exact mechanism of injury:
- when
- how

Severe Mechanism of injury includes:
- MVA with ejection
- MVA with roll over
- Death of passenger in same vehicle
- Pedestrian or cyclist w/o helmet struck by motorised vehicle
- fall >1m
- head struck by high impact object

Red flag symptoms:
- infant younger than 3 months
- abnormal behaviour
- drowsy/lethargic
- persistent irritability
- headache
- non-frontal scalp haematoma
- blood coming from ears (haemotympanum)
- clear liquid coming from ears or nose (CSR rhinorrhoea)
- bruising behind ears (battle sign)
- memory impairment
- loss of consciousness
- seizures (not impact seizures)
- difficulty walking
- intractable vomiting
- other injuries sustained

RED FLAGS FOR CHILD ABUSE:
- previous similar presentations
- changing story
- mechanism/pattern of injury inconsistent with developmental age

PMHx:
- bleeding disorders (haemophilia)
- VP shunts
- Neurodevelopmental disorders (autism) - difficult to assess –> lower threshold to CT

Meds:
anticoagulation

IMMUNISATIONS:

ALLERGIES

SOCIAL:
- where do you live
- who lives at home
- access to medical services, car, phone

EXAMINATION:
GCS:

Eye opening (4)
- spontaneous (4)
- to speech (3)
- to pain (2)
- none (1)

Verbal: (5)
- babbles/coos (5)
- irritable/cries (4)
- cries to pain (3)
- moans to pain (2)
- none (1)

Motor: (6)
- normal movements (6)
- withdraws to touch (5)
- withdraws to pain (4)
- abnormal flexion (3)
- abnormal extension (2)
- none (1)

mental status/behaviour - irritable, agitated, somnolent, repetitive questioning, slow response to questioning

Non-frontal boggy scalp haematomas >2cm in size
- high risk if occipital, parietal and temporal as opposed to frontal
- boggy suggestive of skull fracture

Signs of base of skull fracture:
- battle sign
- raccoon eyes
- CSF otorrhoea/rhinorrhoea
- haemotympanum

Assess for signs of raised ICP and brain herniation:
- reduced LOC
- blown pupil
- hemiparesis
- abnormal posturing (decorticate or decerebrate)
- cushings reflex (bradycardia, hypertension)

ASSESSMENT OF CERVICAL SPINE

ASSESS FOR NON-ACCIDENTAL INJURY:
- head to toe exam

MANAGEMENT:

  • Child with head injury is a very common presentation to the emergency department. It is something that we see and treat a lot.
  • The next step in my assessment is to decide whether or not we need to do a CT scan of the head.
  • We perform CT scans to looks for serious injuries to the brain that may need urgent treatments. For example a bleed on the brain.
  • As you may be aware, CT scans are not completely benign tests. They do expose the growing brain to radiation which does increase the lifetime risk of developing brain cancer. This risk is higher in younger children.

1: 1500 in 1yr old
1: 10,000 10yr old

We use a clinical decision tool to help us identify which patients need to have CT.

This tool helps us to balance the risk of missing a serious brain injury, and to minimise radiation induced cancer in the paediatric population.

Based on the information that I have gathered today. Your risk of having a serious injury inside your head is very low. Almost negligible. <0.05%.

Therefore we will not proceed to a CT scan. We will observe you for 6hrs from the time of injury, reassess you and check your progress.

CRITERIA FOR DISCHARGE HOME
- normal mental status with improving symptoms
-

CONCUSSION:

Concussion symptoms:
- poor balance, unsteady
- headache
- dizzy
- poor concentration “in a fog”
- sensitive to light and noisy
- fatigue

Cognitive rest - 1-2 day break from school with graded return to cognitive activities

Limit screen time

Physical rest - 1-2 days of physical rest with graded return to normal physical activity

sleep hygeine and adequate hydration are important aspects in the recovery

referral to multidisciplinary head injury team for children with prolonged concussion symptoms

return to contact sport and game play - need to be cleared by primary care physician

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2
Q

Paediatric Head Injury

2022.1 Case Based Discussion

discuss with an examiner, the assessment and
management of an 8 year old male who has presented to the ED following a fall at a playground.

Prioritisation and Decision Making: Assessment – 30%
* Provides a rationale to explain decisions about ongoing assessment.
Prioritisation and Decision Making: Management – 30%
* Provides an appropriate list of advantages and disadvantages (pros and cons) to explain
decisions made regarding management
* Provides a rationale to explain and justify decisions about ongoing treatment.
Medical Expertise – 40%
* Correctly interprets the results of an investigation within the scenario
* Correctly chooses time critical interventions based on assessment
* Initiates treatments specific to identified neurological pathologies.
Candidates were required to interact with the examiner and to:
* describe how they would decide if the child needed a CT brain
* answer further questions regarding the evolving case
* outline their reasoning or justify their rationale where appropriate.

A

PECARN identify patients at very low risk of intracranial injury who do not need a CT scan (all predictor variables negative, no CT required)

PECARN the only prospectively validated decision making rule

PECARN has highest sensitivity in ruling out serious pathology

It helps us to balance the risk of missing a significant injury, and to minimize the risk of radiation-induced cancer in the paediatric population

AGE <2yrs
GCS <14
Altered mental status
Loss of consciousness
Parietal, temporal, occipital haematoma
Not acting normal as per parent
Severe mechanism:
- MVC with patient ejection,
- death of another passenger
- rollover
- pedestrian or bicyclist w/o helmet struck by motorized vehicle;
- fall from >0.9m

AGE >2yrs
GCS<14
Signs of base of skull fracture (racoon eyes, battle sign)
Altered mental status
Intractable vomiting
Severe headache
Dangerous mechanism
- fall >1.5m

MODIFIED PAEDIATRIC GCS:
alteration in verbal response
coos or babbles = 5
irritable cry = 4
cries to pain = 3
moans to pain = 2
no response = 1

If PECARN negative:
CT is not required as per best practice evidence

The purpose of non-con CT brain is to detect critical intracranial injuries that require urgent neurosurgical intervention

Risk associated with procedural sedation required to obtain the CT scan - aspiration, respiratory depression

Radiation exposure increases lifetime risk of developing brain cancer

1: 1500 in 1yr
1: 10,000 10yr old

Concussion will reveal a normal CT scan and is managed conservatively

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3
Q

Paediatric Head Injury

2021.1 History taking station

take a history from the parent of an infant with a head injury.

Medical Expertise: Assessment – 40%
* Elicits a focused, relevant history de novo
* Clarifies aspects and inconsistencies in a pre-obtained history to refine diagnosis generation
* Identifies important historical details (red flags) diagnostic of an important condition
* Generates a differential diagnosis, with an inherent focus on conditions requiring time
critical management

Medical Expertise: Management – 30%
* Prioritises patient assessment and management
* Assesses clinical risk
* Justifies decision making

Communication – 30%
* Attempts to establish rapport and attempts to establishes the concerns/issues/needs of the
patient or carer
* Establishes the concerns/issues/needs of the patient or carer
* Uses appropriate verbal and non-verbal skills
* Actively listens and encourages questions

Candidates were required to meet with the child’s parent (role player) and to:
* obtain a detailed history
* explain their management plan, and
* answer any further questions from the parent (role player)

A

.

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4
Q

Head Injury Intubation

A

Prevent raised ICP - raise head of bed 30 degrees

Anticipate difficult intubation due to potential c-spine injury/in line immobilisation

Blunt sympathetic response from laryngeal manipulation - pre medication with fentanyl 1mcg/kg IV (need to give this 3-5min before induction)

Ketamine 1-2mg/kg for induction - haemodynamically stable

Rocuronium 1.2mg/kg IV

Avoid hypotension:
have vasopressors on stand by
adrenaline 1mcg/kg IV Q5min
adequately fluid resuscitate before induction

Avoid hypoxia - pre-oxygenate with high flow oxygen 15L NRBM

Treat raised ICP:
- 3% NS 3-5ml/kg IV following by infusion 0.1ml/kg/hr to maintain Na+ 155-165
OR
- Mannitol 0.5–1 gram/kg

Maintain SaO2 >90%, PCO2 35-40

Maintain normothermia 36-37

Maintain normoglycemia - check BSL Q1h - especially important in paediatric patients

Maintain SBP >70 + (age x2)
Invasive BP monitoring with arterial line
adrenaline infusion 0.05-1mcg/kg/min

Seizure prophylaxis:
levetiracetam 15-40mg/kg IV

Immediate Neurosurgical attendance to facilitate decompressive craniectomy

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5
Q

Brain Herniation

A

BRAIN HERNIATION:
uncal herniation:
- compression of the occulomotor nerve –> ispilateral fixed dilated pupil
- contralateral hemiparesis

Central transtentorial herniation:
- bilateral pin-point fixed pupils
- bilateral babiski
- increased tone
- progress to hyperventilation and decorticate posturing

Cerebellotonsillar herniation:
- flaccid paralysis
- bradycardia
- respiratory arrest
- sudden death

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6
Q

Obese Intubation & Ventilation

2022.1 Case based discussion

The intubation of an obese patient with impending respiratory failure

Medical Expertise: Preparation for intubation (40%)
* Recognises a difficult intubation scenario
* Makes appropriate preparation for a difficult intubation including adjusting patient, staff,
equipment and drug factors
* Optimises patient factors to minimise complications of the intubation.

Medical Expertise: Ventilator settings (20%)
* Adjusts ventilator settings to cater for initial patient clinical status
* Understands importance of ventilator parameters for an obese patient.

Prioritisation and Decision Making (40%)
* Prioritises the essential components of ongoing treatments
* Provides a rationale to explain and justify ongoing treatment
* Manages multiple problems simultaneously whilst remaining vigilant for patient deterioration.

Candidates were required to interact with the examiner and to:
* outline their preparation for the intubation of this patient
* establish ventilator settings and explain their rationale
* answer any questions the examiner may have.

A

Recognize DIFFICULT INTUBATION situation:

RAPID DESATURATION “safe apnoea time” is reduced
- Reduced FRC
- more fatty tissue = higher metabolic requirements

HIGHER AIRWAY PRESSURES TO VENTILATE
- reduced chest wall and pulmonary compliance

INCREASED RISK OF ASPIRATION:
- high intra-abdominal pressure

DRUG METABOLISM & PHARMAKOKINETICS ARE ALTERED
- Lipophilic drugs (eg. Propofol & suxamethonium) have a larger Vd since Vd is dependent on the amount of adipose tissue. Lipophilic drugs need to be dosed based on TOTAL BODY WEIGHT. Therefore these doses will be much higher than in average adults.

  • Hydrophilic drugs (eg. Ketamine) are dosed based on IDEAL BODY WEIGHT

AIRWAY MANAGEMENT:

PREPARATION & OPTIMIZATION

Place arterial line for accurate BP readings
- BP cuffs often don’t fit properly and overestimate BP which is dangerous on induction)
- fluid resuscitate prior to intubation
- 2 large bore IV lines
- Vasopressors on stand by

PREOXYGENATE
- patient sitting upright
- NIV positive pressure ventilation with Bipap (FiO2 100%, IPAP 20, EPAP 10 target TV 7-8ml/kg IBW) - alveolar recruitment
- at least 5min

Obese patients are at an increased risk for ASPIRATION
- have decanto or yankauer suction catheters ready

POSITION:
- RAMPED position
- tragus in line with sternal notch
- face plane parallel with the ceiling
- Reverse trendelenburg
(Improves respiratory mechanics and glottic view)

PLAN FOR SURGICAL AIRWAY - identify landmarks and mark front of neck incase of CICO

SEDATION with ketamine 1-2mg/kg ideal body weight

PARALYSIS with succinylcholine 2-3mg/kg IV (increases tone in lower oesophageal sphinchter to help prevent aspiration), quick onset and offset in case of failed intubation

Rocuronium with reversal agent (sugamadex) on stand by

Apnoeic oxygenation with HFNP 60L 100%

INTUBATION PLAN:

Plan A - most experienced operator, video laryngoscope, bougie

Plan B - 2 person BVM ventilation
oxygenation with bilateral NPA and OPA

Plan C - attempt oxygenation with LMA

Plan D is officially declaring “cannot intubate, cannot oxygenate” and proceeding with a surgical airway

CRICOTHYROTOMY:

Immobilise the larynx with the non-dominant hand

Identify the cricothyroid membrane (cricoid cartilage below, thyroid cartilage above). It’s 1cm in height and 2-3cm wide)

Prep the skin with chlorhexidine

Infiltrate the skin with 2ml xylocaine with adrenaline

Make a 1.5 cm transverse incision (avoid injury to anterior jugular veins) through skin, subcutaneous tissue and the cricothyroid membrane

A rush of air will be heard and bubbling seen

pass bougie through trachea

rail road size 6 ETT over bougie

BVM ventilate

Confirm placement
- auscultate bilateral lungs
- symmetrical chest wall rise and fall
- ETCO2 trace

VENTILATOR SETTINGS:

TV should be adjusted to IBW not TBW - 8ml/kg

RR - increased to accommodate higher metabolic demands (from 12-14 to 18-20).

PEEP should be increased (can be up to 20)
- increase FRC
- alveolar recruitment
(reverse trendelenburg can help decrease the pressures required)

FiO2 100% and titrate down to achieve SaO2 >90%

Peak pressure limit - try to keep the Pplat under 35 cmH2O

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7
Q

Penetrating Chest Trauma

EM rapid bombs
- ep 61 Ketamine facts
- ep 57 Thoracotomy
- ep 63 Tension pneumothorax

2022.1 Case based discussion

Discuss the assessment and
management of a male patient who has presented with a single stab wound to the right axilla.

Patient has sucking chest wound - manage this

Patient becomes agitated - manage this

Trauma call, involve surgery

Medical Expertise: Assessment and diagnosis (20%)
* Correctly interprets the CXR in the context of the patient presentation
* Formulates a provisional diagnosis to match the immediate issues.

Medical Expertise: Management (40%)
* Initiates treatments specific to identified abnormalities of airway and ventilation
* Initiates treatments specific to identifies abnormalities of circulation
* Outlines an overall plan for the ongoing treatment of the patient
* Adapts and initiates standard therapies to that patient, including drugs, fluids, gases, and
monitoring.

Prioritisation and Decision Making (40%)
* Summarises and prioritises key issues that must be addressed during and following the
emergency encounter
* Provides a rationale to explain and justify decisions about ongoing treatment
* Provides an appropriate list of advantages and disadvantages (pros and cons) to explain
decisions made.

Candidates were required to interact with the examiner and to:
* interpret the CXR in the context of the patient’s presentation
* answer further questions from the examiner.

A

Check list:

Analgesia -
Treat agitation -
Supine CXR - hard signs of trauma, check interventions (ETT, ICC, NGT)
CT chest with contrast is gold standard
Massive haemorrhage protocol
Trauma call
Cardiothoracics - surgical repair
Transfer to trauma centre by plane

Pathology:
- pneumothorax
- tension pneumothorax
- open sucking chest wound
- haemothorax
- diaphragm laceration/disruption with visceral herniation
- oesophageal disruption
- tracheobronchial disruption
- myocardial laceration with pericardial tamponade

HISTORY:

EXAMINATION:

eFAST - pericardial fluid

INVESTIGATIONS:

CT chest with contrast is the gold standard

MANAGEMENT:

Analgesia - ketamine

Agitation - ketamine

INTUBATION prior to transfer to CT

Optimize prior to intubation
- Resuscitate with blood products with rapid infuser
- Pre-oxygenate with nasal prongs and NRBM

Half dose ketamine in shocked state
- 0.5mg/kg IV
Higher dose paralytic in shocked state so that they reach the neuromuscular junction
- rocuronium 1.2-1.6mg/kg

DISCUSS THE EFFECTS OF POSITIVE PRESSURE VENTILATION IN PNEUMOTHORAX

PPV causes the patients intrathoracic pressures to exceed atmospheric pressure, worsening the pneumothorax.

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8
Q

Penetrating Chest Injuries

EM rapid bombs:
- ep 70 traumatic aortic injury

Open pneumothorax:
- A “sucking” chest wound exists when air enters the pleural cavity preferentially via an open chest wound, rather than the lungs via the trachea. Placement of an occlusive dressing, taped on three sides, over a sucking chest wound can seal off air entry into the pleural cavity and prevent the expansion of a pneumothorax.

Aorta laceration/disruption:
-

Diaphragm Laceration
- bowel can herniate into chest cavity
- diagnosed on CT or MRI OR laparoscopy/laparotomy
- requires surgical repair

Tension pneumopericardium:
- rare
- hypotension, Jugular vein distension
- requires urgent pericardiocentesis

A

MASSIVE HAEMTHORAX:
Large volume blood loss – >1500mls on initial chest tube placement

Ongoing blood loss from chest tube >200mls/hr for >2hrs

The need for ongoing blood transfusion due to haemodynamic instability

  • second to lung laceration or intercostal artery laceration
  • large 32F chest tube

Tracheobronchial injury:
- pneumomediastinum
- cervical surgical emphysema
- investigate with bronchoscopy
- needs surgical repair

Myocardial laceration and cardiac tamponade:
- eFAST
- hypotension, Jugular vein distension, diminished heart sounds
- resuscitative thoracotomy

Resuscitative thoracotomy
- penetrating chest injury
- signs of life in the field or on arrival
- cardiac arrest within 10min
- cardiothoracic surgeon within 30min

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9
Q

Chest Drain Insertion

2023.2

EM rapid bombs ep 132 - re-expansion pulmonary oedema

A

“Tension pneumothorax is a clinical diagnosis—ideally before a radiograph—and is immediately treated by needle decompression, finger thoracostomy and/or a tube thoracostomy”

“A site that is being more commonly used is the fourth to fifth intercostal space at the anterior axillary line, which is the shortest distance from the skin to the pleura”

Needle decompression
- 2nd intercostal space in the midclavicular line OR
- 4th intercostal space at the anterior axillary line

Safe triangle bordered by:
- 5th intercostal space
- pectoralis major
- latissimus dorsi.

SIZE

erect PA film measure apex - cupula <3cm = small

The British Thoracic Society definition:

Interpleural distance at the level of the hilum
2cm = 50% pneumothorax
<2cm = small
>2cm = large

Pneumothorax and POCUS

Absence of lung sliding.
Absence of Comet tails
Presence of the “lung point” or “transition point”
Absence of seashore sign in M mode (presence of
barcode sign)

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10
Q

Pregnant Trauma

2023.1 CBD station

the management of a pregnant patient who has been involved in an accident

interpret the chest xray

  • Do not defer radiology for concerns of fetal radiation exposure
  • Risk to fetus is highest during first 15 weeks of pregnancy
    *Discuss with radiologist can request low radiation dose protocol

Domestic violence:
- Incidence is increased in pregnancy
* Most commonly struck area is abdomen
* Consider domestic violence
as a cause of trauma

OBSTETRIC RELATED INJURIES:

Placental abruption - USS cannot rule out placental abruption

Uterine rupture

Preterm labour

Fetal-maternal haemorrhage
- Kleihauer test used to detect and quantify extent of FMH
- administer Rh D immunoglobulin if
Rh D negative

Amniotic fluid embolism

DIC

Premature rupture of membranes

Foetal demise

PREGNANCY RELATED INJURIES:
- liver and splenic injury (enlarged in pregnancy)
- bladder rupture
- ureteric rupture
- ovarian vein laceration –> retroperitoneal haemorrhage
Pelvic fractures –> high rate of foetal demise

Pneumothorax:
If chest tube indicated → insert 1–2 intercostal spaces higher than usual

Use cryoprecipitate early and aim to
maintain fibrinogen levels above 2.5 g/L

A

Cardiac monitoring and pulse oximetry

AIRWAY + BREATHING:
- HFNP 60L/min + 15L NRBM

Lateral displacement of uterus to left or left lateral tilt with a wedge

2x large bore IV lines

BSL, VBG, FBC, UEC, LFT, Coags including fibrinogen, Calcium, Group and Screen

Tranexamic Acid 1g IV, followed by 1g over 8hrs
Early cryoprecipitate
Early activation of Massive haemorrhage protocol

Perform eFAST
- free fluid is just as easy to see as non-pregnant patients
- foetal assessment - FHR, placenta, presentation, estimate gestational age

FOETAL MONITORING
- CTG in >23 weeks

CXR
Pelvic XR

Will need CT scans for chest trauma:
- rib fractures can injure the liver and spleen
- ovarian veins can rupture and cause retroperitoneal bleeding which cannot be detected with USS

INTUBATION PLAN:

POSITION:
- RAMPED
- Reverse trendelenburg
- Tragus in line with sternal notch
- Face parallel with ceiling

Increased risk of aspiration
- decanto or yankauer suction catheters

Increased risk of CICO
- prepare for surgical airway
- assess landmarks and mark front of neck

most experienced operator, video laryngoscope, hypercurved D blade with ETT preloaded onto a fixed stylet

Two person BVM
Rescue LMA

FONA

DRUGS:
Ketamine 1mg/kg IV
Suxamethonium 2mg/kg IV

Early involvement of obstetrics and neonatal teams

Steroids for premature labor

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11
Q

Rib Fractures

A

Rib fractures

” it takes great force to fracture the first and second ribs, Such fractures prompt investigation for myocardial injury, bronchial tears and major vascular injuries”

Complications:

  • Pneumothorax
  • Hemothorax
  • Pulmonary contusion

Imaging

CXR

  • 30% sensitive, 90% specific
  • “for each fracture that is visualised, there is another that is not”
  • Harder to see fractures that are not displaced

US

  • Time consuming to image all ribs
  • Uncomfortable for patient

CT
- CTA is the gold standard
- Indicated in any patient with 3 or more rib fractures seen on plain CXR
- Detects complications and may identify source of bleeding
o Intercostal and internal mammary arterial injuries may be suitable for interventional radiology

Management

Intercostal nerve blocks
Epidural if multiple lower rib fractures

Criteria for admission

  • 3 or more rib fractures
  • COPD, asthma, smokers
  • Have complications (pneumothorax, haemothorax, pulmonary contusion)
  • Pain not controlled with oral analgesia (must be able to deep breath and cough)
  • Inability to cope at home with no social supports

HDU/ICU admission for high risk groups

  • > 3 fractures
  • Flail chest
  • Elderly
  • Respiratory compromise

Erector spinae block

  • Preferred for 3 or more rib fractures
  • Ropivacaine through a catheter placed deep to the erector spinae muscle
  • Performed under US guidance
  • 15ml 0.2% ropivacaine every 3hrs
  • Simpler to perform than epidural
  • Avoids potential to cause pneumothorax from intercostal blocks

Intercostal nerve blocks
- Bupivacaine 0.5% with adrenaline
- Inject 2ml each segment (max 20ml)
- Anaesthetise level above and below the fracture, posterior to mid-axillary line,
- Suitable for 1-2 rib fractures
- Limitations:
o Only temporary relief, bupivacaine lasts 8-12hrs, bd injections required
o Risk of pneumothorax performing block 1.5% incidence for each rib blocked
o Difficult to block 1st-7th ribs and posterior rib fractures
o Bupivacaine cardiotoxic if accidental intravascular injection

Epidural
-	HDU/ICU setting
-	Reduces mortality and pulmonary complications
-	Complications
o	Total spinal anaesthesia
o	Hypotension
o	Masking of abdominal injuries
Chest wall stabilisation surgery
-	Not commonly performed
-	Possible indications
o	Flail chest with >3 rib fractures in >2 places
o	Especially if fractured and displaced
o	Chest wall deformity
o	Open rib fractures

FLAIL CHEST

  • 3 or more ribs fractured with at least 2 fractures of the same rib
  • Paradoxical chest wall motion
  • Pulmonary contusion common
  • Ventilatory support
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12
Q

Massive Transfusion Protocol

2023.2 RMO discussion

Teach a junior doctor about blood
transfusion protocols.

“7T’s of MTP”

Trigger
Team
TXA
Testing
Target
Temperature Control
Termination

know your local MTP protocol

HEPARIN

  • 1mg protamine per 100 units heparin
  • give maximum of 50mg protamine slow IVI as initial dose

CLEXANE (ENOXAPARIN):

  • 1mg protamine per 1mg clexane
  • give maximum of 50mg protamine slow IVI as initial dose

Warfarin reversal:
- prothrombinex 50IU/kg
- vitamin k 10mg IV

Dabigatran
- prothrombinex 50IU/kg IV
- tranexamic acid 1g IV
- idarucizumab (praxbind) 5g IV

Apixaban & Rivaroxaban
- prothrombinex 50IU/kg
- tranexamic acid 1g IV
- adexanet alpha (factor 10a) - not available in australia yet

Prothrombinex contains factors 2, 9, 10
- European countries give PCC’s and Cryoprecipitate instead of giving FFP
- can consider if you are in a rural hospital that does not have FFP
- PCC kept at room temperature (can be kept in resus)
- don’t need blood group
- no pathogens
- don’t cause TRALI

Studies currently looking at the use of whole blood instead of blood products - evidence is not out yet

BLOOD PRODUCT VOLUMES:

PRBCs 260mls +/- 15mls
Platelets 367mls +/- 16mls
FFP 278mls +/- 13 mls
Cryoprecipitate 36mls +/- 2mls

cryoprecipitate contains:

0.2g fibrinogen in each unit
von willebrand factor
factor 8
factor 13
give it if fibrinogen <1

tranexamic acid 1g IV bolus, then 1g IV in 100ml NS over 8hrs
**most beneficial if given in the <1h post trauma (needs to be given within 3hrs of trauma)
CRASH2 trial

A

TRIGGER: (when to activate the MTP)

Clinical Judgement:
- bleeding in shocked state
(positive shock index)

The Revised Assessment of Bleeding and Transfusion (RABT)
Score – 1 point each
1. Shock index > 1.0
2. Pelvic Fracture
3. Positive FAST
4. Penetrating Injury

A 2018 study showed that RABT score ≥ 2 performed better than
ABC score in predicting need for MHP.

Lower threshold to activate:
- elderly
- anticoagulation medication
- medications that blunt the sympathetic response (beta blockers)

TEAM:

Lab and blood bank notification

Haemorrhage control/Source control
- early notification of surgeon, gastroenterologist, interventional radiologist

TXA:
1g IV, followed by 1g over 8hrs within the first 3hrs in trauma (CRASH2 reduce mortality)

TESTING:
Baseline bloods:
- Fibrinogen
- Coags (INR & PT)
- VBG - pH
- FBC - Hb, platelets
- Calcium
- K+
- Bhcg (o negative blood)

Q1 hour bloods
- fibrinogen
- INR
- FBC (Hb & platelets)
- VBG (pH, lactate)
- Calcium
- K+

TARGETS: (haematologic and metabolic targets)
*Hb > 80
*Platelets >100
*Fibrinogen >1.5
*INR <1.5
*Ionised calcium >1
*pH >7.2

Fibrinogen needs to be >2g/L in obstetric patients

The citrate preservative in blood products binds to serum calcium making it inactive. Calcium is important in regulating coagulation and haemostasis.

TEMPERATURE:
36.5-37.5

TERMINATION:
- normalising haematological and metabolic parameters
- haemodynamic stability

Prevent blood wastage:
- return products as soon as you know you don’t need them

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13
Q

Pelvic Fractures

2016 station

PR exam to assess for open pelvic fractures - will need antibiotics and operative management of bowel transection

Retrograde urethrogram - to look for urethral injury that may warrant suprapubic catheter placement

A

RADIOLOGY

Lateral compression fracture
AP compression (open book)
Vertical shear
Acetabular fractures

XRAY FINDINGS:
- Pelvic binder in-situ
- Disruption pelvic ring
- Superior and inferior pubic rami
- Pubic symphysis diastasis (open book pelvic fracture)
- SIJ widening
- Disruption of arcuate lines –> sacral alar fracture
- Disruption of superior acetabular margin
- Disruption of shentons line - right
- Disruption of illeopectineal lines
- Disruption of ischiopectineal lines
- Femoral head dislocations

b)

Activate massive transfusion protocol - 1:1:1 PRBC: Platelet: FFP

Rapid infusion catheter or 2x large bore IVC at least 18G

1g tranexamic acid IV stat

Temperature control 36.5-37.5

Targets:
ionised calcium >1
INR < 1.5
Fibrinogen >1.5 (>2 in obstetric patients)
Hb >80
Plt >50
pH >7.2
lactate <4

Pelvic angiography +/- embolization internal illiac

REBOA (Resuscitative endovascular balloon occlusion of the aorta)

Pelvic packing in OT

ORIF in OT

Preparation for transfer:
- blood products
- analgesia - ketamine
- arterial line and monitoring
- IDC
- update family of plan

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14
Q

Teaching: Pelvic binder management

2022.1 - Teaching Station

*EM Board Bombs ep 11, 33, 106
*EM quick hits 30
*EM cases ep 119 trauma
*Emergency medicine procedures

Medical Expertise: Use and application of pelvic binders (45%)
* Selects an appropriate procedure after considering indications, contraindications, and
potential complications
* Describes the important features of common procedural equipment.

Medical Expertise: Management after binder application (30%)
* Anticipates and manages common complications during and after a procedure
* Adapts the performance of a procedure in response to unforeseen complications when
performing a procedure.

Scholarship and Teaching (25%)
* Effectively delivers a teaching session which teaches procedural skills and use of equipment
* Integrates basic principles of adult learning to proficiently deliver a teaching session to a
small audience.

Candidates were required to meet with the junior registrar (role player) and to:
* teach them about the use of pelvic binder systems
* describe the fitting of a pelvic binder system
* explain post pelvic binder application management
* answer any questions they may have.

A

TEACHING:
- introduce
- establish rapport
- enquire about level of experience
- prior learning
- check understanding
- opportunity to ask questions
- summarise
- suggested reading/learning resources

INDICATIONS:
- blunt trauma
- hypotension/cardiac arrest
- suspected pelvic fractures

HOW IT WORKS:
- decrease pelvic diameter,
- decreasing the anatomic bleed space in open book and vertical shear fractures

CONTRAINDICATIONS:
- lateral compression fracture

COMPLICATIONS:
- painful to apply if fractures are present
- incorrect application is common
- may cause further harm in lateral compression fractures
- pressure sore with prolonged use

Clinical assessment of pelvic injuries is unreliable

Early pelvic binding if shocked or suspect pelvic fracture

Place pelvic binder on bed prior to patient arrival

Given analgesia - ketamine 10mg IV
- this is painful for the patient

need to bind the greater trochanters

binding the legs together to further reduce the pelvic diameter

check correct position - greater trochanters
check tension
document time of application

Pitfalls:
- helps tamponade venous bleeding and cancellous bone bleeding, won’t tamponade arterial haemorrhage
- incorrect placement is common - need to bind the greater trochanters

WHEN TO REMOVE:
- if haemodynamically stable with no fractures on imaging

don’t log roll the patient

PR and PV exam for blood = open fracture requiring antibiotics

IDC - haematuria = bladder injury
(need for CT cystourethrogram)

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15
Q

Bronchiolitis

  • Viral respiratory tract infection
  • Affects infants <12 months of age
  • Peak severity is usually at around day 2-3 from the onset of increased work of breathing
  • self limiting illness with resolution over 7-10 days
  • cough may persist for weeks
  • treatment is supportive
  • ventilatory support with oxygen/NIV, mechanical ventilation
  • nutritional support support with feeding (NGT of IV fluids)

Cause:
*Respiratory syncitial virus (RSV) - most common
- Influenza
- parainfluenza
- covid
- human metapneumovirus
- rhinovirus

A

HISTORY:

Prodrome of coryzal symptoms
Followed by:
- cough
- fever
- increased work of breathing
- reduced feeding
- irritable/unsettled

Feeding
Urine output
Lethargy

ASSESS PARENTAL COPING

RISK FACTORS FOR SEVERE BRONCHIOLITIS:
- Chronic lung disease (cystic fibrosis
- Congenital heart disease (tetralogy of fallot)
- Chronic neurological conditions
- Premature
- Age < 6 weeks
- Failure to thrive/growth restriction
- Indigenous
- Immunodeficiency
- Trisomy 21
- Exposure to cigarette smoke

SOCIAL HISTORY:
- Exposure to cigarette smoke

REASONS FOR ADMISSION:
- oxygen requirement

  • marked increased work of breathing with potential need for positive pressure ventilation / NIV
  • poor feeding requiring NGT placement (<50% normal intake with evidence of dehydration)
  • early in illness i.e. day 1 with potential to deteriorate

INVESTIGATIONS:

not for chest xray
- CXR only indicated if superimposed bacterial infection, alternative diagnosis considered

not for blood tests

not for respiratory viral swabs
- viral swabs only indicated if patient is going to be admitted –> isolation to prevent cross infection between children on the ward

MANAGEMENT:

supportive:
monitoring of apnoea.

DISCHARGE ADVICE:
- reassurance
- smaller volume feeds more frequently (ensure adequate hydration)
- monitor feeding and urine output
- SMOKING

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16
Q

Paediatric Elbow Fracture

EM Rapid Bombs ep 225

2022.1 Teaching station

Medical Expertise: X-ray analysis and interpretation (40%)
* Analyses the results of an investigation to identify relevant findings or patterns
* Correctly interprets the results of an investigation within the scenario.

Medical Expertise: Examination and management – 40%
* Describes a focused structured and relevant physical examination for a paediatric patient
* Initiates appropriate initial supportive treatment for any presenting problem
* Anticipates and manages common complications during and after a procedure
* Provides clear instructions for the patient/carer on discharge and ensure comprehension.

Scholarship and Teaching – 20%
* Checks for level of prior knowledge/understanding, establishes rapport, encourages learner participation and allows questions
* Demonstrates well-paced communication, covers the topic in appropriate detail,
demonstrates a structured approach, gives clear explanations, explains rationale
* Ensures the learner has received and understood the information, checks understanding
* Invites questions and clarifies any areas of uncertainty.

Candidates were required to meet with a junior doctor (role player) and to:
* explain an approach for paediatric elbow X-ray interpretation
* interpret the X-rays
* explain an approach to examining a 5 year old with an elbow injury
* describe a management plan for the patient.

Interpret and teach approach to paediatric elbow xray to junior doctor

  • Explore what they know about xray interpretation and paediatric elbow fractures
  • Suggest radiopedia lectures for further learning

Explain your approach to elbow examination in the distressed child

  • importance of establishing rapport with the parent and child
  • consideration of the developmental age
  • analgesia
  • distration - ipad
  • getting the parents on board
  • minimal handling examination
A

Key Points:

*Approach to elbow xray
*Examination approach in distressed child - analgesia, distraction technique and minimal handling examination
*Gartland’s criteria - determines management
*Complications
*Discharge advice
*Fracture clinic follow up

APPROACH TO ELBOW XRAY:

Mechanism
Demographic
Likely pathology

AP and Lateral films

Soft tissue swelling
Joint effusion
Cortex disruptions
Trabeculae pattern disruptions
Alignment
Ossification centres

CRITOE - ossification centres appear at different stages

Capitellum 1yr
Radial head 3yr
Internal condyle 5yr
Trochlea 7yr
Olecranon 9yr
External condyle 11yr

MANAGEMENT is based on the GARTLAND’S CLASSIFICATION.

Type 1 fractures:
Nondisplaced
long arm back slab with elbow at 90deg, collar and cuff
fracture clinic follow up in 10days with repeat xray

Type 2 fractures:

  • Displaced with posterior cortex intact
  • needs orthopaedic review as type 2b fractures (>20 degrees angulation) require operative management
  • Closed reduction and immobilisation in ED + orthopaedic consultation

Type 3 fractures:

  • Complete displacement
  • Operative management - k wires or open reduction

COMPLICATIONS:

Incidence of nerve injuries associated with supracondylar fractures is 10-20%. The median nerve and the radial nerve are most often injured.

Anterior interosseous nerve - inability to make “OK” sign
(loss of strength of the thumb interphalangeal joint in flexion as well as the index DIP joint in flexion. This injury renders the patient unable to perform the “OK” sign)

Radial nerve - inability to extend at the wrist

Brachial artery injury (spasm/kinking) can lead to Volkmann’s contracture - compartment syndrome of the forearm.

EXAMINATION:
Analgesia:
- paracetamol 15mg/kg,
- ibuprofen 10mg/kg,
- IN fentanyl 1.5mcg/kg
- Nitrous oxide - relatively weak dissociative anaesthetic gas with anxiolytic, analgesic and amnesic properties. rapid onset, rapid offset. don’t require fasting.

Distraction techniques:
- music on parents smart phone of tablet device
- guided visual imagery

Engage and involve the parents

Neuropraxias – usually resolve spontaneously
- Anterior interosseous nerve (branch of median nerve) – most common injury
o Unable to flex interphalangeal joint of the thumb or flex DIP of index finger. Can’t make OK sign.
- Median nerve
o Loss of sensation over volar index finger
- Radial nerve – second most common
o Inability to extend wrist, MCP’s, thumb IP joint
- Brachial artery
o Palpate radial and ulnar artery
o Can use biphasic doppler
o Hand well perfused - warm, pink, OR poorly perfused – cold, pale, CRT >2sec

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17
Q

Sexually Transmitted Infections

EM Rapid Bombs ep 395

2022.1 History Taking

Medical Expertise: Assessment and management – 50%
* Elicits a focused history and identifies important historical details diagnostic of an important
condition
* Generates differential diagnoses
* Creates a focused investigation plan to confirm or exclude time critical diagnoses
* Outlines a treatment plan for the patient
* Outlines a clear discharge plan for the patient including safety net return advice.

Communication – 25%
* Demonstrates a professional and respectful approach
* Uses language appropriate to the patient’s level of health literacy to explain the treatment
and management plan
* Summarises the encounter and confirms the patient’s understanding.

Health Advocacy – 25%
* Demonstrates the ability to use medical expertise to protect and advance the health and
well-being of the patient
* Demonstrates the ability to integrate the broad range of factors that affect this patient
beyond the ED encounter
* Proactively engages in health promotion using available resources to intervene in order to
improve the health outcomes of this patient and those within his social circle.

Candidates were required to:
* take a focused history from the patient (role player)
* provide their differential diagnoses
* advise the patient of the investigation and management plan.

A

HISTORY:

*Reassure privacy and confidentiality
*Non-judegemental
*Screen for sexual assault

Symptoms:
- discharge (penile, rectal)
- ulcers
- lumps or sores
- genital rashes
- itching/burning
- abnormal vaginal or rectal bleeding
- irregular periods
- dysuria, difficulty passing urine
- dyspareunia (painful sex)
- pelvic pain
- joint pain
- mouth ulcers, throat pain
- occular symptoms
- vaccination history (Hep B, Hep A, HPV)

Partners:
- male or female or both
- stable partner
- last sexual encounter
- how many partners in the last year

Practices:
- type of intercourse - vaginal, anal, oral (insertive or receptive or both)
- **sex overseas in high risk countries (africa, asia)
- IVDU - sharing needles

Prevention:
- barrier protection
- Prep
- regular STI testing

Pregnancy:
- Contraception

Contact tracing

PMHx:
Medications:
Allergies:
Social:
- smoking
- drugs
- alcohol
- job (sex worker?)
- friends/family
- access to follow up?

DIFFERENTIAL DIAGNOSES:
Genital ulcers:
- Herpes Simplex Virus
- Syphilis
- Lymphogranuloma venerium - chlamydia
- Chancroid - Haemophilus ducreyi
- Granuloma inguinale - Donavanosis

INVESTIGATIONS:
HIV
Hepatitis C
Hepatitis B
Syphillis

NAAT/PCR
*Vaginal/endocervical swabs
*Urethral swabs
*First pass urine
*Pharyngeal swabs
*Rectal swabs
- Trichomonas
- Gonorrhoea
- Chlamydia
- Mycoplasma genitalium

Additional gonorrhoea culture and sensitivity due to increasing antibiotic resistance

MANAGEMENT:

  • Offer emergency contraception
  • Counsel on safe sex practices (abstinence, barrier protection)
  • Abstain from sexual intercourse for 7 days after treatment
  • Follow up with community sexual health - review results, discuss contraception, Prep
  • Address contact tracing - directly or through contact tracing website “Let them know” “the drama down under”
  • Immunisation against HPV, Hep A, Hep B (if not immune already)
  • PrEP for men who have sex with men
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18
Q

Sexually Transmitted Infections

PID:

Complications:
- infertility
- ectopic pregnancy
- chronic pelvic pain

History:
- sexually active
- pelvic pain
- abnormal vaginal discharge
- irregular periods
- dyspareunia

Exam:
- pelvic tenderness
- cervical motion tenderness
- adnexal tenderness

USS:
- thickened fallopian tubes >5mmg
- tubo-ovarian abscess
- cogwheel sign on cross section tubal view

Admission criteria:
- systemically unwell with fever or sepsis
- pregnancy
- surgical cause not excluded
- severe pain requiring IV opiates
- inability to tolerated outpatient regimen
- failed outpatient treatment

IUD removal with PID:
- can safely leave IUD in place
- PID second to IUD insertion occurs in the first 3 weeks of insertion
- Remove IUD if no clinical improvement within 72hrs of starting treatment

Fitz Hugh Curtis Syndrome
- PID with RUQ pain
- perihepatitis
- inflammation of liver capsule leading to adhesions
- LFT’s normal or mildly elevated transaminases
- seen on laparoscopy or laparotomy

A

SYPHILIS:
- on the rise among men who have sex with men
- associated with HIV infection

Primary Syphilis:
- painless ulcer to genitals, anus, mouth
- appears about 3 weeks after contact
- painless and usually solitary so often missed by patient
- ulcer heals over in 6 weeks

Secondary Syphilis:
- 4-10weeks after ulcer heals
- painless lymphadenopathy
- non-puritic maculopapular rash that also affects the palms and soles of feet
- condylomata lata
- aseptic meningitis
- nephrotic syndrome
- moth eaten allopecia
- ocular manifestations (uveitis, iritis, optic neuritis)

Latent phase (asymptomatic)
- lasts years

Tertiary phase
- gumma (skin and bone)
- syphilitic aortitis (aortic aneurysm, aortic regurgitation)
- neurosyphilis
*endarteritis leading to ischemic stroke
*tabes dorsalis (spinal cord wasting)
*dementia

Ix:
Swab ulcer - treponema pallidum NAAT or PCR
non-treponemal test (VDRL), if positive –> perform treponema pallidum particle agglutination assay or the IgM/IgG enzyme immunoassay

If both tests are positive = the patient is syphilis positive

LYMPHOGRANULOMA VENEREUM (LGV):
- chlamydia
- may have proctitis or rectal bleeding
- Untreated can lead to tenesmus, fistulas and strictures
- Rx doxycycline 100mg bd 21 days

CHANCROID:
- haemophilus ducreyi
- diagnosis of exclusion of syphilis and HSV
- treat with ceftriaxone 500mg IM

Granuloma inguinale - Donavanosis
- caused by klebsiella granulomatis
- requires punch biopsy
- Rx doxycycline 100mg bd 21 days

Herpes Simplex Virus:
- swab HSV NAAT
- rx valaciclovir 1g bd 7 days

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19
Q

Organophosphate / Carbamate Toxicity

2022.1 discussion with registrar station

Medical Expertise: Assessment – 40%
* Elicits a focused, relevant history of organophosphate exposure
* Performs a focused, structured and relevant physical examination of the patient with
organophosphate poisoning
* Identifies risks of deterioration/need for intervention in the patient with organophosphate
toxicity.
Medical Expertise: Management – 40%
* Outlines an appropriate plan regarding decontamination of a patient with organophosphate
poisoning
* Outlines an overall plan for the management of the patient with established organophosphate
toxicity.
Prioritisation and Decision Making – 20%
* Lists specific triggers for intubation in a patient with organophosphate toxicity.
Candidates were required to interact with a junior registrar (role player) and to:
* answer the registrar’s questions regarding this patient’s presentation.

A

Pesticides & insecticides used in agriculture
- carbamates are less toxic than organophosphates and have shorter duration of action
- any ingestion in children can be lethal
- deliberate ingestion in adults will cause life threatening toxicity
- accidental dermal or inhalational exposure causes toxicity but is rarely life threatening

***Large amounts of atropine, hundreds of milligrams, may be necessary in massive ingestions. Proactive contact with the hospital pharmacy (or even other centres) may be necessary to ensure access to adequate amounts of atropine.

MECHANISM:
- inhibits acetylcholinesterase
- accumulation of acetylcholine at muscurinic and nicotinic receptors

HISTORY:
Which agent ingested?
When?
How much?
Co-ingestion?
Self decontamination with emesis?
Symptoms since ingestion?

“Cholinergic crisis”
Muscurinic effects ‘DUMBBELS’
- diarrhoea & diaphoresis
- urinarion
- miosis
- bronchorrhoea & bronchospasm
- bradycardia & hypotension
- emesis
- lacrimation
- salivation

Nicotinic effects:
- fasciculations
- tremor
- weakness
- respiratory muscle paralysis
- tachycardia/hypertension

CNS effects- agitation, coma, seizures

Respiratory effects:
- progressive neuromuscular junction dysfunction and respiratory failure over 1 to 4 days
- Chemical pneumonitis (if contains hydrocarbons)

Intentional - Will need psychiatric assessment

EXAMINATION:
Bradycardia or tachycardia
hypotension or hypertension
Respiratory distress - dyspnoeic, hypoxic, wheeze

GCS - coma, confused, delirius
seizures

diaphoresis
miosis
lacrimation
salivation
vomiting

incontinent - urine, diarrhoea

INVESTIGATIONS:
ECG - arrythmia
CXR - chemical pneumonitis

*plasma cholinesterase activity - if the diagnosis is unclear

*red cell cholinesterase activity - better correlation with severity of poisoning

MANAGEMENT:
PPE - impervious gown, gloves, mask, eye protection for resus team

DECONTAMINATION:
- no role for activated charcoal with oral ingestion as rapidly absorbed and distributed
- remove and dispose of clothing, wash skin with water and detergent for dermal exposures

ENHANCED ELIMINATION:
- none

ANTIDOTE:
atropine 1.2mg (0.05mg/kg) IV
double dose every 5min until adequate atropinisation - chest clear, no wheeze
- HR 80
- SBP >80
then use 10-20% of the loading dose per hour infusion

reduce infusion if over atropinisation
- confusion
- pyrexia
- mydriasis
- absent bowel sounds

*Atropine has no effects on the neuromuscular junction

PRALIDOXIME:
- controversial
- causes reactivation of acetylcholinesterase with SOME organophosphate poisonings (not carbamate poisonings)
- studies have shown that although it does increase red cell cholinesterase activations, it does not improve survival or intubation rates
- if poisoning is refractory to atropine, discuss use of pralidoxime with toxicologist

CHEMICAL PNEUMONITIS
- oxygen
- ventilatory support
- steroids

SEIZURES:
- midazolam 5mg IV

AGITATION:
- midazolam

RESPIRATORY FAILURE:
- Intubation and mechanical ventilation

EARLY INTUBATION:
*atropine improves wheeze and clears secretions but DOES NOT reverse respiratory muscle paralysis
*also many have an aspiration pneumonitis

OPTIMISE PRE-INTUBATION:
- Preoxygenate
- Fluid resuscitation
- Noreadrenaline infusion

  • using ketamine 1mg/kg and rocuronium 1.2mg/kg

***A non-depolarizing agent should be used when neuromuscular blockade is needed.
Succinylcholine is metabolized by plasma butyrylcholinesterase; therefore, prolonged paralysis may result.

  • using video laryngoscope and experienced operator to increase likelihood of first pass success.

DISPOSITION:
ICU

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20
Q

Abdominal pain + Hypotension

EM Rapid bombs ep 164 - AAA

2022.1 Case based discusssion

Rural hospital, elderly patient with abdo pain and hypotension

Medical Expertise: Assessment and diagnosis – 30%
* Generates a differential diagnosis, with an inherent focus on conditions requiring time
critical management
* Identifies important historical details (red flags) diagnostic of an important condition
* Differentiates expected physical signs for different conditions.

Medical Expertise: Management – 30%
* Outlines an overall plan for resuscitating a patient
* Correctly chooses time critical interventions based on assessment
* Outlines an overall plan for ongoing treatment of a patient
* Creates a safe and clear discharge plan for a patient.

Health Advocacy – 40%
* Incorporates the patient’s chronic clinical state and the patient’s wishes when making
decisions
* Explains to patients and/or their guardians the rationale for management decisions
* Incorporates the patient’s belief system to create a personalised management plan
* Integrates specific communication skills relevant to the patient’s culture, to enhance delivery
of health advice to patients.

Candidates were required to interact with the examiner to:
* outline their differential diagnosis
* describe how to assess the patient to differentiate between possible diagnoses
* manage the patient within the limitations of the available resources.

A

DIFFERENTIAL DIAGNOSES:

Vascular:
- ruptured AAA
- aortic dissection
- mesenteric ischemia

Infection:
- urosepsis

Surgical:
- perforated viscous with peritonitis
- cholecystitis
- appendicitis
- diverticulitis
- pancreatitis
- acending cholangitis

Trauma:
- solid organ injury with haemorrhage

ASSESSMENT

HISTORY:

SOCRATES
- fever
- nausea, vomiting
- diarrhoea
- GI bleeding

surgical history
known AAA
- previous endovascular aneurysm repair (EVAR) have 25% chance of leaking at the graft site

gallstones
renal stones
UTI’s

Alcohol
Abdominal trauma
Anticoagulation
AF or coagulopathy predisposing to ischemic gut

EXAMINATION:

pulsatile abdominal mass at umbilicus

signs of lower limb ischemia
- thromboembolism to lower limbs

Neurological finding due to spinal cord ischemia (T10-T12)

signs of rupture - shock, pallor, peritonism with ruptures into intraperitoneal cavity

POCUS:
ED AAA POCUS - studies shown 99% sensitive 98% specific for ED physicians

curvilinear probe
xiphoid to bifurcation
IVC on patients right side
aorta on patients left side
vertebral body deep
measure largest transverse diameter (outer wall to outer wall)
normal < 2cm
can’t exclude AAA rupture (can’t assess for retroperitoneal haemorrhage)
rupture usually occurs >5.5cm

limitations of POCUS:
- obesity
- overlying bowel gas
- need to see aorta in entirety
- can’t assess for retroperitoneal haemorrhage

INVESTIGATIONS:

Urianlysis - WCC, nitrites, blood
VGB - lactic acidosis with end-organ hypoperfusion and ischemic gut
Lipase - pancreatitis
LFT’s - obstructive hepatitis in cholangitis
Inflammatory markers - CRP, WCC
UEC - renal failure with hypoperfusion
FBC - Hb in haemorrhage
Group and Screen - for transfusion

CT abdomen with contrast:
- only if haemodynamically stable
- can diagnose rupture or impending rupture
- useful in surgical planning

MANAGEMENT:

Resuscitation room
continuous cardiac monitoring
pulse oximetry and Q2min BP
high flow oxygen 15L NRBM
2x large bore IV cannulae or rapid infusion catheter

urgent vascular or general surgery attendance

activation of massive haemorrhage protocol - 4 unit o negative blood

place arterial line - permissive hypotension MAP 50-60 to maintain organ perfusion
- assess mentation, UO,

IDC - monitor UO/end-organ perfusion (target 30ml/hr)

Analgesia - morphine 5mg IV - reassess and titrate to patient comfort

PROGNOSIS:
Poor prognostic factors for open repair
- age >80
- hypotension SBP <90
- acute renal failure
- cardiac arrest
- LOC/syncope
- IHD

multiple risk factors for poor outcome should prompt consideration for comfort care

DECISION TO TRANSFER FOR SURGERY:
For patients who present to a facility where local surgical expertise is not available, transfer is appropriate.

If transfer is chosen, the patient and their family should be informed of the risk of deterioration during transfer, and the transfer should be accomplished as soon as possible.

ADVANCED CARE DIRECTIVES/PATIENT WISHES

It is unclear whether endovascular repair decreases mortality in patients with ruptured AAA.

A decision must be made according to the wishes of the patient (if known) and family whether to proceed with repair or provide comfort measures.

ASSESS CAPACITY:

  • Cognitive ability to understand and retain information in regards to their medical situation
  • Compare the treatment options and understand the consequences of each
  • Engage in rational deliberation about the proposed treatment and communicate a choice

RESPECT FOR PATIENT AUTONOMY
- every competent patient has the moral right to choose what happens to their body
- therefore patients have the right to freely accept or reject a physicians recommendations

DUTY OF CARE:
Common Law has established that doctors have a ‘duty of care’ to act in the best interest of their patients.

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21
Q

Bowel Obstruction

2023.2 Case based discussion

Medical Expertise: Investigation Interpretation – 30%
 Analyse the results of an investigation to identify relevant findings or patterns.
 Correctly interpret the results of an investigation within the scenario.
 Act on time critical investigation results as they arise.

Medical Expertise: Treatment Including Resuscitation – 30%
 Outline an overall plan for resuscitating a patient.
 Recognise and expedite any specific intervention to resuscitation.
 Initiate treatments specific to identified abnormalities in airway, ventilation or circulation.
 Adapt and initiate standard therapies to that patient.
Prioritisation and Decision Making – 40%

 Manage multiple problems simultaneously by prioritising treatment options whilst remaining
vigilant for other potential problems.
 Summarise and prioritise the key issues that must be addressed during and following the
emergency encounter.
 Provide a rationale to explain and justify decisions about ongoing treatment.

Candidates were required to interact with the examiner and to:
 Describe and interpret the patient’s abdominal X-rays.
 Outline their management of this patient.
 Answer further questions asked by the examiner.

A

Mechanical bowel obstruction vs. paralytic illeus

Small bowel vs. large bowel obstruction

Complete vs. partial obstruction

Complicated vs. uncomplicated

SMALL BOWEL OBSTRUCTION

Causes:
- adhesions
- intussusception (lymphoma as lead point)
- malignancy
- incarcerated hernia
- strictures (inflammatory bowel disease)
- gallstone illeus (obstructs illeo-caecal valve +pneumobilia)
- radiation enteritis
- bezoars
- blunt abdominal trauma (duodenal haematoma)

LARGE BOWEL OBSTRUCTION:
- malignancy
- strictures
- fecal impaction
- volvulus (caecal, sigmoid)

COMPLICATIONS:
Intestinal ischemia
Perforation & peritonitis
Sepsis & multiorgan failure
Malabsorption & Dehydration
Electrolyte disturbance

ASSESSMENT:

History:
Symptoms:
- abdo pain, distension, vomiting, passing flatus, opening bowels, pr bleeding, weight loss, fevers, urine output

Risk factors:
- Previous abdominal surgeries
- Previous bowel obstructions
- Malignancy, lymphoma
- Inflammatory bowel disease
- Hernias
- Volvulus common in institutionalised elderly
- Preveious radiation therapy

Consider other differential diagnoses:
- pancreatits
- AAA

EXAMINATION:
GCS
Airway patent
Breathing spontaneously + Oxygenation
Circulation - maintaining HR & BP
Temp
BSL
Hydration - mucous membranes, skin tugor, POCUS assessment of IVC

Abdominal distension
Tenderness
Peritonism
Resonant to percussion
High pitched tinkling bowel sounds

LABORATORY:
VBG - lactate, BSL
Electrolytes -

IMAGAING:

Xrays:
Erect CXR and erect/supine AXR films (or lateral decubitus film if the patient cannot sit upright) - these have a sensitivity of 70-83% and specificity of 67-83% for small bowel obstruction.

Small bowel obstruction:
- Dilated loops of small bowel > 3 cm
- Central dilated loops
- Valvulae conniventes or plicae circulares are present
- Gas-fluid levels

Large bowel obstruction:
- Distended colon >6cm or 9cm at caecum
- contains gas and faecal matter (no fluid because water is absorbed)
- Located peripherally
- Haustral folds

Pneumoperitoneum
Pneumotosis intestinalis
Riglers sign
Lead pipe colon - chronic inflammation
Thumbprint sign - bowel wall oedema

CT abdomen:

More sensitive and specific

Can identifying transition point

Can determine the cause (hernias, adhesion, tumours, inflammation etc)

Can identify complications - necrosis, perforation

Can distinguish between true obstruction and pseudo-obstruction

MANAGEMENT:

NBM

Fluid resuscitation guided by HR, BP and POCUS assessment of IVC

IDC placed to monitor urine output aiming for 0.5ml/kg/hr

Analgesia
- Morphine 2.5-5mg IV - reassess and titrate to patient pain level
- IV paracetamol 1g

Consider NG tube for gastric decompression (vomiting or severe symptoms of gastric distension)

IV antibiotics

Surgical consultation

DEFINITIVE MANAGEMENT:

Small bowel obstruction often managed conservatively initially for 48-72hrs then surgery if no resolution. Surgery for perforation or strangulated hernia.

Gastrografin may be diagnostic and therapeutic in SBO due to adhesions

Large bowel obstruction often requires surgical management

Sigmoid volvulus –> decompression with flexible or rigid sigmoidoscopy + insertion of rectal tube

Closed loop obstruction, Caecal volvulus and bowel necrosis, perforation and peritonitis require emergency surgery

TOXIC MEGACOLON
- inflammatory bowel disease and C. diff colitis
- colon dilatation and systemic toxicity

Diagnosis:
Radiology:
- dilated colon >6cm
- multiple air fluid levels
- loss of haustral folds
PLUS at least 3 of the following:
- fever
- tachycardia
- neutrophilia
- anaemia
PLUS at least 1 of the following:
- hypotension
- dehydration
- altered mentation
- electrolyte disturbance

The main goal of treatment is to reduce the severity of colitis in order to restore normal colonic motility and decrease the likelihood of perforation.

PSEUDO-OBSTRUCTION/PARALYTIC ILLEUS/Ogilvies syndrome

  • functional obstruction (uncoordinated muscle contraction)

Causes:
- post abdominal surgery
- peritonitis
- drugs (anticholinergics, opiates)
- trauma
- ischemic bowel
exacerbated by electrolyte disturbance (hypokalemia)
- disturbance of enteric nervous system or intestinal smooth muscle

MANAGEMENT:
Aim is to restore motility and prevent ischemia and perforation

Neostigmine and erythromycin.

Endoscopic Decompression in patients with Ogilvie’s syndrome

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22
Q

Bowel Obstruction

2021.2 Case based discussion

assessment and management of previously well adult who presented with a 24 hour
history of severe generalised abdominal pain and profuse vomiting.

Medical Expertise: Investigation interpretation – 30%
* Analyses the results of an investigation to identify relevant findings or patterns
* Correctly interprets the results of an investigation within the scenario
* Acts on time critical investigation results as they arise

Medical Expertise: Treatment including resuscitation – 30%
* Outlines an overall plan for resuscitating a patient
* Recognises and expedites any specific intervention to resuscitation
* Initiates treatments specific to identified abnormalities in airway, ventilation or circulation
* Adapts and initiates standard therapies to that patient

Prioritisation and Decision Making – 40%
* Manages multiple problems simultaneously by prioritising treatment options whilst remaining
vigilant for other potential problems
* Summarises and prioritises the key issues that must be addressed during and following the
emergency encounter
* Provides a rationale to explain and justify decisions about ongoing treatment

Candidates were required to:
* describe and interpret the patient’s abdominal X-rays for the examiner
* outline their management of the patient
* answer further questions asked by the examiner.

A

.

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23
Q

Discharge Against Medical Advice

2022.1 Registrar discussion

discuss the assessment of high risk patients in the context of discharge against medical advice.

Health Advocacy – 50%
* Promotes family centred or patient centred care when describing to junior doctors their
approach to patients
* Implements strategies to prevent a patient ceasing their emergency care prematurely
* Identifies factors that increase the likelihood of a patient being vulnerable
* Integrates extra services to increase the likelihood of successful treatment in vulnerable
patients.
Professionalism – 30%
* Describes the following medico-legal principles: duty of care
* Describes how medico-legal frameworks and principles are applied in the assessment of
capacity to make decisions
* Balances respect for patient autonomy with best clinical practice in patient encounters.
Leadership and Management – 20%
* Provides time critical counselling for an underperforming junior staff member
* Demonstrates a capacity to understand and manage emotions in a professional and effective
manner
* Demonstrates an awareness of patient safety principles in the management of an Emergency
Department.
Candidates were required to meet with a registrar (role player) to:
* provide feedback regarding their management of a patient
* discuss the issues to be considered when deciding if this or any other patient can discharge
against medical advice
* discuss strategies to prevent patients discharging against medical advice
* discuss ways to identify high risk patients who should be prioritised during a busy shift

A

ASSESS CAPACITY:

  • Cognitive ability to understand and retain information in regards to their medical situation
  • Compare the treatment options and understand the consequences of each
  • Engage in rational deliberation about the proposed treatment and communicate a choice

Identifying barriers to decisional capacity:
- acute intoxication with alcohol or drugs
- acute psychiatric illness
- cognitive (dementia)

Identify the reason for wanting to leave:
- this maybe easily rectified

Involve trusted others

Educate the patient:
- about the risks associated with refusing to complete evaluation

Maintain rapport and invite the patient to represent if they have any concerns

Meticulous Documentation:
- document behaviour and features of intact capacity with examples
- investigations and treatments that were offered
- risks were explained
- advised patient that they are welcome to represent at anytime

24
Q

End of Life Decision Discussions

2016 - 86y Nursing home resident with severe dementia with sepsis and aspiration pneumoniae - the patient is dying and has no capacity. Meet with the daughter (NOK) to discuss palliation.

A

Palliative care
“treatment that is not aimed at a cure but at caring for the patient by keeping him/her as physically comfortable and pain-free as possible, while also attending to his/her emotional, mental, social and spiritual needs”

25
Q

End of Life Discussions

2021.1 Communication station

interact with the relative of a patient (role player) about
end of life care in the setting of dementia

Communication: Assessment – 30%
* Introduces self and purpose, establishes rapport
* Actively listens to and validates the concerns of the relative
* Establishes the issues/needs of the patient early in the encounter
* Explains decisions regarding treatment for the dying patient

Communication: Management – 30%
* Explains the rationale for investigation, management and disposition decisions.
* Uses appropriate verbal skills for the situation: appropriate language, avoids jargon, explains
medical terms
* Discusses options for focus of care and location

Health Advocacy: End of life care – 40%
* Summarises the key issues that must be met in the patient’s care
* Explains the decisions around medical management and the goals of end of life care
* Arranges appropriate supports to aid in treating the imminently dying patient and their
relatives

Candidates were required to:
* meet with the relative of the patient (role player)
* discuss the patient’s prognosis
* develop an approach to the management of the patient.

A

SETTING THE SCENE:

Private room
Allow enough time
No interruptions
Allow family supports (or offer ED social worker)

Introduce self and role
Thank you for coming into hospital for meeting
Apologies to meet under these circumstances
Reason - to discuss current medical situation and the plan going forward
“Do you have a support person or other family members who you wish to be here?”

GAUGE PERCEPTION:

Assess the families understanding about the current clinical situation

“what is your understanding of your loved ones illness”

Quality of life:
GATHER INFORMATION:

find out what the patient and/or family know about their condition

Live limiting co-morbid conditions
- COPD, CHF, dementia, renal failure

Premorbid function and quality of life

“what is her day to day life like”

Assess activity of daily living:
- living independently or nursing home
- bed ridden
- cognitive function (dementia)?
- Do they need assistance for toileting, feeding, bathing, dressing?

Is there an advanced care directive in place?
Who is the surrogate decision maker?

Ask family to rate the following three categories in order of importance to the patient:
1) life prolongation,
2) quality of life,
3) comfort.

Values and religious beliefs?

INVITATION TO RECEIVE INFORMATION

“would you like me to tell you all the details of your mother’s condition?

GIVE INFORMATION

warning shot: “I’m afraid, based on what we know now, that his/her health is deteriorating”

Explain the current medical situation clearly and communicate that patient is seriously ill and likely to die

“Based on what I see, I think that he/she is dying.”

RESPOND TO EMOTIONS

“This must be incredibly hard to hear.”
“I can not imagine how scary this must be.”

ESTABLISH GOALS OF CARE

“If they were able to partake in this conversation - would would they say, what would they want”

Explain why most appropriate plan to return to nursing home to die peacefully in familliar environment surrounded by staff and family known to her

Explain that further medical interventions would be futile, prolong suffering, not lead to recovery

COMFORT CARE VS CURATIVE CARE

“Based on what you have told me, I recommend that we focus more on aggressive symptom management”

“We will refocus our efforts on his/her priorities and make this as dignified and comfortable an experience as possible.”

“We will make sure we use all our resources to support him/her and all of you in these final moments of his/her life.”

EXPLAIN CLINICAL COURSE AND PLAN:

explain to the family what to expect - clinical course of the dying patient

Guarantee:
Not withdrawal of treatment and care - infact, more aggressive treatment and care.
Focus is comfort rather than curative.
- pain relief with morphine
- antinausea
- agitation & delirium
- secretions
- dry mouth - oral cares
- palliative care reviews

DISPOSITION:
- nursing home with palliative care
- inpatient palliative care

give family opportunity to express concerns/issues

26
Q

End of Life Discussions

2023.2 - Communication station

Communication: Assessment – 20%
 Introduce self and purpose, establish rapport.
 Actively listen to and validate the concerns of the relative.
 Establish the issues/needs of the patient early in the encounter.
 Explain decisions regarding treatment for the dying patient.

Communication: Management – 40%
 Explain the rationale for investigation, management and disposition decisions.
 Use appropriate verbal skills for the situation: appropriate language, avoids jargon, explains
medical terms.
 Discuss options for focus of care and location.

Health Advocacy – 40%
 Summarise the key issues that must be met in the patient’s care.
 Explain the decisions around medical management and the goals of end of life care.
 Arrange appropriate supports to aid in treating the imminently dying patient and their
relatives.

Candidates were required to meet with the parent (role player) and to:
 Discuss the patient’s prognosis.
 Develop an approach to management of the patient.

A

arrange private room for patient and family
explain to the family what to expect - clinical course of they dying patient
offer religious supports i.e. chaplain/priest
consistent nursing presence
remove all monitoring, remove catheters and cannulas
stop all life sustaining treatments including IV fluids
analgesia through a subcutaneous butterfly needle

PRN MEDICATIONS:
pain & dyspnoea - morphine 5mg sc Q1h PRN
agitation & anxiety - midazolam 5mg sc Q1h PRN
respiratory secretions - glycopyrolate 200mcg sc Q2h
nausea & vomiting - ondansetron 4mg sublingual wafers Q8h

27
Q

End of Life Care

2023.1 Case based discussion

Medical Expertise – 40%
* Outline an overall plan for ongoing treatment of a patient.
* Adapt and initiate standard therapies to that patient, e.g., drugs, fluids, gases, and monitoring.
* Consult / refer to inpatient team (palliative care).
* Specify the resources that will be required to address ongoing post disposition needs, with
consideration of social and cultural factors.

Health Advocacy – 40%
* Incorporate the patient’s chronic clinical state and the patient’s wishes when making
decisions.
* Initiate an end of life discussion with the patient and / or relative.
* Justify what supportive treatment is appropriate for the dying patient.
* Arrange extra supports to aid in treating the imminently dying patient and the patient’s
relatives.

Prioritisation and Decision Making – 20%
* Explain the decision to limit assessment and treatment when they do not alter patient
prognosis.
* Incorporate patient and family needs as part of shared decision-making.

Candidates were required to interact with the examiner and to:
* Outline an overall plan for ongoing management of this patient.
* Answer the further questions asked by the examiner.

A

Need to ask about advanced care directives

28
Q

End of Life Discussions

2022.1 Case based discussion

Patient has end stage COPD

Prioritisation and Decision Making – 40%
* Explains the risk stratification process used by incorporating patient safety principles when
making key treatment decisions and disposition decisions
* Explains the decision to limit assessment and treatment when these do not alter patient
prognosis
* Provides a rationale to explain and justify decisions about ongoing treatment.

Health Advocacy – 30%
* Incorporates the patient’s chronic clinical state and the patient’s wishes when making
decisions
* Defines and identifies medically futile treatment
* Initiates an end of life discussion with the patient and/or relative
* Explains the decision of limiting the provision of invasive treatment for the dying patient
* Justifies what supportive treatment is appropriate for the dying patient
* Arranges extra supports to aid in treating the imminently dying patient and supporting the
patient’s relatives.

Leadership and Management – 30%
* Uses advanced communication techniques to defuse the anger/anxiety in the patient or
relative
* Outlines major principles of conflict resolution in the initial or bedside response.

Candidates were required to interact with the examiner and to:
* outline how to determine limits of care in this patient
* describe how you would communicate this to the family
* answer any further questions asked by the examiner.

A

arrange private room for patient and family
explain to the family what to expect - clinical course of they dying patient
offer religious supports i.e. chaplain/priest
consistent nursing presence
remove all monitoring, remove catheters and cannulas
stop all life sustaining treatments including IV fluids
analgesia through a subcutaneous butterfly needle

PRN MEDICATIONS:
pain & dyspnoea - morphine 5mg sc Q1h PRN
agitation & anxiety - midazolam 5mg sc Q1h PRN
respiratory secretions - glycopyrolate 200mcg sc Q2h
nausea & vomiting - ondansetron 4mg sublingual wafers Q8h

29
Q

Otitis Media

2022.1 Communication

EM rapid bombs ep 269, 270 otitis media

interact with the parent (role player) of a child with
earache, who initially presented to the ED five days previously.

Communication – 30%
* Introduces self and purpose and attempts to establish rapport
* Establishes (asks) the concerns/issues/needs of the parent early in the encounter
* Demonstrates empathy and allows the parent to react emotionally to the situation
* Demonstrates a professional and respectful approach.

Medical Expertise: Management – 30%
* Adapts and initiates standard therapies to that patient
* Describes treatments specific to the condition
* Outlines an overall plan for ongoing treatment of the patient
* Identifies risks of deterioration in the patient – complications of the condition.

Leadership and Management – 40%
* Listens respectfully
* Acknowledges the parent’s concerns
* Uses advanced communication techniques to defuse the anger/anxiety in the parent
* Outlines the immediate management plan to resolve the issue.

Candidates were required to meet with the parent (role player) and to:
* explain the current situation and the plan for the patient
* address their concerns about the presentations.

A

HISTORY:
- fever, malaise, crying, irritability
- ear pain, otorrhoea
- nausea and vomiting
- upper respiratory tract infection

EXAMINATION:
- bulging, motionless tympanic membrane
- red tympanic membrane can occur with high fever or after prolonged crying and is not a sign of otitis media
- light reflex
- retracted
- perforation with otorrhoea

RED FLAGS/RISK FACTORS:
- indigenous/aboriginal
- immune compromised
- craniofacial abnormalities (cleft palate)
- cochlear implant
- age < 6months
- systemically unwell/toxic appearing

SHARED DECISION MAKING WITH THE PARENT:

EDUCATE:
- Acute otitis media predominantly caused by a viral infection
- Can be caused by bacteria
- Can be both viral and bacterial

*Strep. pneumoniae
*Haemophilus influenza
*Moraxella catarrhalis

REASSURANCE:
- Regardless of the cause, it is usually self-limiting.
- Resolves spontaneously in 80% of children within 2 to 3 days with or without antibiotics
- severe complications are rare

DISCUSS LIMITED BENEFITS
- even with a bacterial cause, antibiotics have very little benefit
- studies have shown that for every 100 children treated with antibiotics, only 5 children will feel better at 3 days
- antibiotics do not improve pain at 24hrs
- antibiotics only shorten the duration of illness by about 12hrs

DISCUSS THE POTENTIAL HARMS OF ANTIBIOTIC USE:
- antibiotics have unwanted side effects:
* vomiting and diarrhoea
* rash
* allergic reactions
- for every 100 children treated with antibiotics, 7 children will experience an antibiotic adverse effect.

Opportunistic infections:
- antibiotics disrupt the balance of bacteria in the body (the microbiome) which can lead to other infections such as thrush

Multidrug resistance
- antibiotics can cause bacteria in the body to become more resistant so that future infections are harder to treat.

Multidrug resistant bacteria or “superbugs” which is problematic for the entire community.

Drug interactions:
between antibiotics and regular medications

COMPLICATIONS:
- ear drum perforations
- mastoiditis
- meningitis
- brain abscess
- lateral sinus thrombosis
- cholesteatoma
- facial nerve palsy

SIGNS OF ACUTE MASTOIDITIS:
- very rare but serious complication
- with ongoing otitis media, infection spreads to the the mastoid bone
- fever, red, hot, tender swelling behind the ear
- need to come to ED
- IV cefotaxime 50mg/kg bd (pseudomonas cover for immunecompromised, grommets, aboriginal)
- admit under ENT

MANAGEMENT:

Regular analgesia:
- paracetamol 15mg/kg
- ibuprofen 10mg/kg
- 2% topical lignocaine drops (intact TM)
- antihistamines, decongestants and steroids are not effective in OM

Expectant management

Follow up in 2-3 days (if no improvement can start antibiotics)

Return to ED if symptoms worsen or no improvement in 2-3 days

A delayed prescription for antibiotics if the parent is unable to return for review

Prevent exposure to cigarette smoke

Written information about otitis media

Amoxycillin 30mg/kg PO BD 5 days

30
Q

Paediatric fever - Otitis Media, Kawasaki’s

EM rapid bombs ep 55 Kawasakis

2022.2 History taking

take a focused history from a parent (role player) whose
child has presented with fever.

Medical Expertise – 40%
 Elicits a focused, relevant history de novo
 Identifies important historical details (red flags) diagnostic of an important condition
 Generates a relevant list of differential diagnoses after synthesising clinical information found
on initial assessment.

Prioritisation and Decision Making – 30%
 Links clinical information and evidence-based practice to explain decisions
 Provides a rationale to explain decisions about ongoing assessment
 Provides a rationale to explain and justify decisions about ongoing treatment.

Communication – 30%
 Establishes (asks) the concerns/issues/needs of the patient/relative/person early in the
encounter
 Uses language appropriate to the patient’s level of understanding / avoids jargon/ explains
medical terms
 Demonstrates empathy
 Demonstrates a professional and respectful approach.

Candidates were required to:
 take a focused history from the parent (role player)
 answer any questions the parent may have.

A

DIFFERENTIAL DIAGNOSIS:
Group A streptococcal infections: *tonsillitis,
*scarlet fever,
*acute rheumatic fever

Viral infections including:
*EBV,
*CMV,
*Adenovirus,
*Covid

Systemic juvenile idiopathic arthritis (JIA)

Sepsis
*uti
*pneumoniae
*cellulitis

Toxic shock syndrome (staphylococcal or streptococcal)

Stevens-Johnson syndrome

Drug reaction

Malignancy

KAWASAKI’s
Vasculitis
Ages 6 months to 5yrs
boys > girls

Diagnosis:
- fever > 5days PLUS 4 of the following
(can have less than 4 if coronary artery abnormalities on echo)

POLYMORPHOUS RASH

CERVICAL LYMPHADENOPATHY
- unilateral
- affect that anterior chain

BILATERAL CONJUNCTIVITIS:
- no exudate
- painless
- limic sparing ‘halo around cornea’

STRAWBERRY TONGUE:
- dry, cracked lips
- oropharyngeal erythema

PALMAR & SOLE OEDEMA + ERYTHEMA
- progress to periungal desquamation in 2-4wks as fever subsides

INVESTIGATIONS:
ECG - arrythmias, MI
FBC - anaemia, *high WCC, *high platelets
CRP/ESR - *critically high
LFT’s - raised GGT, raised ALT, low albumin

Urinalysis - sterile pyuria

Echo at presentation, 2wks, 6wks, 12 months
- identify coronary artery aneurysms
- or signs of coronary vasculitis (ectasia/dilatation, perivascular brightness)

COMPLICATIONS:
Coronary artery aneurysms –> thrombosis –> MI

Myocarditis can lead to conduction defects, arrhythmias and heart failure

Hepatitis
Arthritis
Urethritis/dysuria

MANAGEMENT:
To prevent cardiovascular complications

Echo at presentation, 2wks, 6-8wks

Aspirin 5mg/kg orally daily until normal echo

IV immunoglobulin 2g/kg IV over 12hrs - if started within 10 days can reduce the risk of coronary artery aneurysm from 20% to 3%

Give second dose of IVIG 36hrs later if no response to first dose

Haemolytic anaemia can occur 1 week after IVIG

Limited evidence for corticosteroids
Consider corticosteroids in high-risk groups:
- Demographics:
*age <12 months of age,
*Asian ethnicity

  • Investigation abnormalities:
    *ALT >100 IU/L,
    *albumin ≤30 g/L,
    *sodium ≤133 mmol/L,
    *platelets ≤30 x 109/L,
    *CRP >100 mg/L,
    *anaemia for age
  • Cardiac or coronary artery involvement on echo at presentation
31
Q

Massive Upper GI Bleeding

EM rapid bombs ep 5, 168 - oesophageal varices

IBCC 3 - Approach to GI bleeding

2023.2 Registrar Discussion

Medical Expertise: Management – 50%
- Outline an overall plan for resuscitating a patient.
- Recognise and expedite any specific intervention essential to resuscitation.
- Initiate treatments specific to identified severe abnormalities in circulation.

Medical Expertise: Assessment – 30%
- Identify important historical details (red flags) to ascertain risk.
- Differentiate expected physical signs.
- Recognise signs on physical examination that indicate the patient is at risk of imminent
deterioration.
- Explain the reasons for selecting tests in the investigation plan.

Prioritisation and Decision Making – 20%
- Manage multiple problems simultaneously by prioritising treatment options.
- Highlight high risk features identified during initial patient assessment.
- Prioritise chosen treatment options to create an appropriate escalating treatment plan.
- Summarise and prioritise the key issues that must be addressed.

Candidates were required to meet with a junior doctor (role player) and to:
- Outline their approach to the assessment of the patient.
- Outline their approach to the management of the patient.

A

Upper GI bleed likely if:
- malena
- urea/creatinine ratio >30
- age <50

Imitators of malena - iron tablets, black foods - liquorice
malena is black, tarry and putrid odour

CAUSES:
Peptic ulcer disease - most common
Oesophageal varices
aorto-enteric fistulae
delafoys lesion

ASSESSMENT:
known PUD, liver disease/varices, previous EVAR

Antiplatelets and anticoagulation? need for reversal?

MANAGEMENT:

Resus

Call resus team for help

Attach patient to Cardiac monitoring, Pulse oximetry and cycle BP q2min

Oxygen - HFNP 60L/min target SaO2 100% (no mask as patient will be vomiting)

2x large bore IV access 14-16g
Rapid infusion catheter

–> send bloods VBG, FBC, UEC, LFT’s, full coagulation panel including fibrinogen, group and screen and cross match 4units of blood

CBC - Hb, platelet count
PTT - identifies those on NOACs
Fibrinogen - need for cryoprecipitate
INR <1.8 in liver disease has preserved coagulation

Fluid resuscitate with blood through fluid warmer

Temperature - keep patient warm - reduce exposure, warm blankets, fluid warmers - target temp 37

If there is a delay with blood can give a small crystalloid bolus 250-500ml to maintain MAP >60

Do not over resuscitate - disrupt clot formation and cause further bleeding

Permissive hypotension - aim for SBP low enough to maintain end organ perfusion SBP 90-100

Place invasive BP monitoring - arterial line

Activate massive haemorrhage protocol if:
- need over 4units RBC
- positive shock index
- massive haemorrhage
- on anticoagulation
- elderly

Liase blood bank and haematologist
Early discussion with gastroenterologist for definative haemorrhage control with endoscopy
Liase with ICU

give blood products in a 1:1:1 ratio

Monitor our metabolic and haematologic targets Q1h:
Hb >80
Platelents >50
Ionised calcium >1.1
INR <1.5
Fibrinogen >1.5
Lactate <4

Tranexamic acid 1g IV
Contraindications:
- coronary stents
- haematuria (clot formation and urinary retention)
- history of VTE

MEDICATIONS:

Octreotide/somatostatin 50mcg IV followed by 50mcg/hr infusion for 5 days (splanchnic constrictor, reduce portal venous pressure)

Terlipressin 1.7g IV Q4h for 5 days

Propranolol 20mg TDS

Tranexamic acid 1g IV

Prophylactic antibiotics 1g ceftriaxone daily or po norfloxacin bd has been shown to reduce early re-bleeding rate and improve survival, reduce mortality

Oral sucralfate (local anti-fibrinolytic effect)

Pantoprazole 80mg IV followed by infusion 8mg/hr

AIRWAY:

RSI + Intubation

Sit up right + Preoxygenate with HFNP 60/min

Prokinetics to empty the stomach - metaclopramide 10mg IV or erythromycin

Most experienced operator - difficult intubation

PPE - eye protection, impervius

Direct laryngoscopy - blood will obscure video laryngoscopy

SALAD technique
(suction assisted laryngoscopy airway decontamination)

Use a lower dose induction agent to avoid hypotension - ketamine 0.5-1mg/kg IV

Use standard paralytic dose as do not want to cause vomiting and aspiration on laryngoscopy
rocuronium 1.2mg/kg IV

Decontaminate the airway if they vomit - place in trendelenburg and have double suction set up

push dose vasopressors on standby encase of sudden deterioration
adrenanline 10mcg/ml in 10ml syringe

DEFINITIVE MANAGEMENT:

Endoscopy

Balloon tamponade (Blakemore or Minnesota tube)
- Balloon placed in stomach
o Balloon inflated with 50ml
- CXR to confirm below diaphragm (prevent oesophageal perforation)
- fully inflate gastric balloon
- apply traction which compresses oesophageal varices
- inflate oesophageal balloon if still bleeding
- transfer to interventional radiology for TIPS

  • Trans-jugular intrahepatic porto-systemic shunt (TIPS)
    o stent between hepatic vein and portal vein – reduces portal pressure
    o Needs to be done by interventional radiologist
    o Increased risk of hepatic encephalopathy

CT angiography to identify bleeding site +/- embolization

Surgery

DISPOSITION:
ICU

32
Q

Rectal Bleeding

2022.2 Case based discussion

PR bleeding in Jehova’s witness

2016 - Registrar discussion

74y M on dabigatran with heavy rectal bleeding

Medical Expertise: Initial management – 30%
 Outlines an overall plan for resuscitating a patient
 Recognises and expedites any specific intervention/s essential to resuscitation
 Initiates appropriate time critical interventions
 Outlines an overall plan for ongoing treatment of a patient.

Professionalism: Informed consent – 30%
 Applies appropriate medico-legal and ethical frameworks and principles in assessment of
capacity to make decisions
 Balances respect for patient autonomy/religious beliefs with best clinical practice in patient
encounters
 Justifies setting limits in providing clinical care in challenging situations using ethical
principles.

Prioritisation and Decision Making – 40%
 Highlights high-risk features identified during initial patient assessment
 Explains the specific benefits and risks of a treatment modality
 Provides a rationale to explain and justify decisions about ongoing treatment
 Provides an appropriate list of advantages and disadvantages to explain decisions made
 Incorporates patient and family/whānau needs as part of shared decision-making.

Candidates were required to interact with the examiner and to discuss:
 their approach to management of this patient
 the ethical considerations in this case
 ongoing management decisions.

A

DIFFERENTIAL DIAGNOSIS:

Diverticulitis
Angiodysplasia
Haemorrhoids
Infective colitis
- salmonella
- campylobacter
- shingela
- entamoeba
- E. coli 0157:H7
Ischemic colitis
Radiation colitis
Anticoagulation
Inflammatory bowel disease
Post polypectomy
Malignancy
Anal fissures/fistulas
Rectal ulcers
Rectal trauma - foreign body insertion, sexual practices
Aortoenteric fistulas - after EVAR

HISTORY:

first episode or recurrent bleeding?
bright red blood?
volume of blood loss?
mixed in with stool?
spontaneous bleeding independent of bowel motion?

Potential causes:
Medications - anticoagulation? antiplatelets?

Bleeding disorders (haemophilia?)

Diverticular disease?

Inflammatory bowel disease?

Recent colonoscopy + polypectomy?

Previous endovascular abdominal aortic aneurysm repair? (aortoenteric fistula)

Haemorrhoids?

Colorectal cancer?

Anal fistulas/fissures/ulcers

Rectal trauma - foreign body insertion

Liver cirrhosis/oesophageal varices?

EXAMINATION:

Signs of haemorrhagic shock:
- tachycardia
- hypotension
- tachypnoea
- pallor
- cool peripheries
- anuria
- altered mentation

abdominal tenderness
acute abdomen ?perforated diverticulitis ?colitis

signs of coagulopathy - bruising, blood coming from canulae

signs of anaemia - pallor, pale conjunctiva

PR - anal fissures/ulcers, trauma, masses, blood (fresh, malena)

INVESTIGATIONS:

Group and screen - xmatch blood for transfusion

FBC - Hb count, platelet count, WCC - leukocytosis in IBD, colitis, diverticulitis

UEC - AKI in renal hypoperfusion

LFTs - ischemic hepatitis with hypoperfusion

full coagulation profile - extended clotting times in coagulopathy

CMP - calcium important with massive transfusion

VBG - lactate with end-organ hypoperfusion

Sigmoidoscopy/colonoscopy:
- for stable patients who are bleeding slowly
- need adequate bowel prep
- can identify source of bleeding and take biopsies
- can stop bleeding by adrenaline injection, diathermy, or haemoclipping

CT Mesenteric Angiography:
- blush amenable to embolization
- note risk of post contrast nephropathy given AKI and hypotension (but benefits outweigh risk in this instance)

SPECIALIST CONSULTATION:
*interventional radiologist
*general surgeon:
- encase IR not available or it fails
- need for laparotomy and bowel resection
*haematologist - advice for anticoagulation reversal
*intensivist - admission to ICU

DISPOSITION:
- ICU

33
Q

Massive Haemorrhage Protocol

2023.2 Registrar discussion

Medical Expertise: Management – 40%
 Create an appropriate ongoing reassessment and management plan.
 Initiate appropriate time critical interventions.

Medical Expertise: Assessment – 40%
 Identify risks of deterioration during a massive transfusion protocol.
 Perform a structured initial assessment on a critically ill patient.

Scholarship and Teaching – 20%
 Demonstrate a structured approach to the topic.
 Establish rapport.
 Make the learner feel safe and supported.
 Demonstrate well-paced communication.

Candidates were required to meet with a junior doctor (role player) and to:
 Discuss the use of massive blood transfusion protocols.
 Answer any questions the junior registrar may have.

Teach a junior doctor about blood
transfusion protocols.

“7T’s of MTP”

Trigger
Team
TXA
Testing
Target
Temperature Control
Termination

know your local MTP protocol

HEPARIN

  • 1mg protamine per 100 units heparin
  • give maximum of 50mg protamine slow IVI as initial dose

CLEXANE (ENOXAPARIN):

  • 1mg protamine per 1mg clexane
  • give maximum of 50mg protamine slow IVI as initial dose

Warfarin reversal:
- FFP
- prothrombinex 50IU/kg
- vitamin k 10mg IV

Dabigatran
- prothrombinex 50IU/kg IV
- tranexamic acid 1g IV
- idarucizumab (praxbind) 5g IV

Apixaban & Rivaroxaban (factor 10a inhibitors)
- prothrombinex 50IU/kg
- tranexamic acid 1g IV
- adexanet alpha (factor 10a) - not available in australia yet

Prothrombinex contains factors 9, 10, 7, 2
- European countries give PCC’s and Cryoprecipitate instead of giving FFP
- can consider if you are in a rural hospital that does not have FFP
- PCC kept at room temperature (can be kept in resus)
- don’t need blood group
- no pathogens
- don’t cause TRALI

Studies currently looking at the use of whole blood instead of blood products - evidence is not out yet

BLOOD PRODUCT VOLUMES:

PRBCs 260mls +/- 15mls
Platelets 367mls +/- 16mls
FFP 278mls +/- 13 mls
Cryoprecipitate 36mls +/- 2mls

cryoprecipitate contains:

0.2g fibrinogen in each unit
von willebrand factor
factor 8
factor 13
give it if fibrinogen <1

tranexamic acid 1g IV bolus, then 1g IV in 100ml NS over 8hrs
**most beneficial if given in the <1h post trauma (needs to be given within 3hrs of trauma)
CRASH2 trial

A

TRIGGER: (when to activate the MTP)

Clinical Judgement:
- bleeding in shocked state
(positive shock index)

Lower threshold to activate:
- elderly
- anticoagulation medication
- medications that blunt the sympathetic response (beta blockers)

MHP PACK 1:
- RBC 4 units
- FFP 2 units
- fibrinogen concentrate or cryoprecipitate

MHP PACK 2:
- RBC 4 unit
- fibrinogen concentrate or cryoprecipitate
- platelets

TEAM:

Lab and blood bank notification

Haemorrhage control/Source control
- early notification of surgeon, gastroenterologist, interventional radiologist

Haematologist - reversal of anticoagulation

TXA:
1g IV, followed by 1g over 8hrs within the first 3hrs in trauma (CRASH2 reduce mortality)

Relative TXA contraindications:
- cardiac stents
- haematuria (may precipitate clot formation and lead to urinary retention)
- history of VTE

TESTING:
Baseline bloods:

  • Fibrinogen
  • Calcium
  • Coags (INR & PT)
    PT is a one time screening test to check for congenital bleeding disorders or anticoagulants such as DOACs dabigatran
  • VBG - pH, base excess, lactate, ionised calcium, K+
  • FBC - Hb, platelets
  • Bhcg (o negative blood)

Q1 hour bloods
- fibrinogen
- INR
- FBC (Hb & platelets)
- VBG (pH, base excess, lactate, ionised calcium, K+)

TARGETS: (haematologic and metabolic targets)
*Temperature >35
*pH >7.2
*lactate <4
*BE -6 to 6
*Hb > 80
*Platelets >50
*Fibrinogen >2
*INR <1.5
*Ionised calcium >1.1

Fibrinogen needs to be >2.5g/L in obstetric patients
- give cryoprecipitate 10 units IV
- OR fibrinogen 70mg/kg IV

The citrate preservative in blood products binds to serum calcium making it inactive. Calcium is important in regulating coagulation and haemostasis.

TEMPERATURE:
36.5-37.5

TERMINATION:
- haemorrhage control
- hemodynamically stable
- normalising haematologic and metabolic targets

STEWARDSHIP: Prevent blood wastage

  • return products as soon as you know you don’t need them
34
Q

Toxic Alcohol Poisoning
- methanol
- ethylene glycol

EM rapid bombs ep 177, 179, 181

2021.1 CBD station

Assessment and management of an adult female who has been brought to the ED in a highly agitated state.

Interpret VBG

Differential diagnosis for HAGMA
“MUD PILES”

Metformin, Methanol
Uremia (renal failure)
DKA
Paracetamol toxicity
Iron toxicity, Isoniazid
Lactic acidosis (sepsis)
Ethanol, ethylene glycol
Salicylates

Anion gap = Na+ - Cl - HCO3-
normal anion gap =12

Osmolar gap = measured osmolarity - calculated osmolarity

calculated osmolarity = (2x Na+) + glucose + urea

normal osmolar gap = <10

Outline your care for the obtunded patient

Signs of hypocalcemia “CATs go numb”
Convulsions
Arrythmias (prolonged QTc)
Tetany
numbness (perioral, hands, feet parasthesias)

Trousseau sign
Chvostek sign

A

Ethylene glycol + Methanol

Methanol - poorly distilled alcohol

Ethylene glycol - antifreeze, break fluid and some solvents

Metabolised by alcohol dehydrogenase and aldehyde dehydrogenase to form toxic metabolites

ethylene glycol –> oxalic acid –> calcium oxalate precipitation in kidneys and brain –> ATN –> oliguric renal failure –> ethylene glycol elimination half life increases as it has to be eliminated exclusively by the kidneys –> dialysis

calcium oxylate crystals also deposit in the brain causing basal ganglia haemorrhages

methanol –> formic acid –> affects retina + basal ganglia
- optic neuritis

Fomepizole and ethanol inhibits alcohol dehydrogenase to prevent formation of toxic metabolites

haemodialysis removes toxic metabolites
- formic acid
- glycolic acid
- oxalic acid

PRESENTATION:
- GI upset (nausea and vomiting)
- drunk/altered mentation/slurred speech
- hyperventilation (due to metabolic acidosis)
- reduced visual acuity with methanol - “snow storm vision”
- become more obtunded instead of sobering up
- seizure (due to hypocalcemia)

EXAMINATION:
Altered mentation
Slurred speech
Nystagmus

Tachypnoea

Visual changes - reduced VA
RAPD in methanol poisoning

INVESTIGATIONS:

ECG - prolonged QTc with hypocalcemia

VBG:
*HAGMA
*raised osmolar gap
*lactic acidosis

*hypocalcemia
*hypoglycemia
*hyperkalemia

Calcium level
Serum osmolality
UEC - acute renal failure

*Urine microscopy - calcium oxylate crystals

*raised serum methanol or ethylene glycol levels
- gas chromatography is not widely available
*methanol level >15mmol/L
*ethylene glycol level >8mmol/L
are associated with severe toxicity in adults and children

*serum ethanol levels
- serum ethanol levels 11-22mmol/L competitively inhibit alcohol dehydrogenase

*paracetamol level

GOALS OF MANAGEMENT:
- Block formation of toxic metabolites
- Correct pH with bicarbonate
- Eliminate toxic metabolites with dialysis

DECONTAMINATION:
No role for gastrointestinal decontamination
- toxic alcohols are rapidly absorbed
- charcoal doesn’t bind alcohols

ANTIDOTE:
- fomepizole (inhibit alcohol dehydrogenase)
- 15mg/kg Q12hr
- very expensive and not readily available
- toxic alcohols have long half lives so prolonged treatment required

Second line:
- Ethanol 10% 6 mL/kg IV or NGT loading dose, followed by 50-100ml/hr infusion

If ethanol 10% is not available, then white spirits (eg vodka), can be administered orally or via a nasogastric tube. Use serial serum ethanol concentrations to guide the rate of administration.

Need to double the dose of ethanol if having dialysis. Aim for serum ethanol 22-44mmol/L

ENHANCED ELIMINATION:
- haemodialysis removes toxic metabolites
- shorten hospital length of stay

Indications for haemodialysis:
- severe metabolic acidosis pH <7.2
- osmolar gap >10
- acute renal failure

continue fomepizole during dialysis

MODIFIED AIRWAY MANAGEMENT FOR SEVERE ACIDOSIS:

Patient is obtunded and has a seizure. Outline your management.

support airway - jaw thrust/chin lift
100% oxygen 15L NRBM
suction airway if required

Terminate seizures with midazolam 5mg IV
Correct calcium in light of seizure
- give calcium gluconate 10 ml IV

Prepare for intubation
- Sodium bicarbonate 50-100ml IV bolus for acidemia

  • hyperventilate to counteract acidosis

ELECTROLYTES:
don’t treat hypocalcium unless tetany, seizures, QT prolongation - giving calcium will precipitate calcium oxylate in the tissues

ANTIDOTES:
Ethanol 10% 6 mL/kg IV or NGT loading dose, followed by 50-100ml/hr infusion

Fomepizole 15 mg/kg IV loading, then 10mg/kg IV BD, dosing increases to Q4h during dialysis

CO-FACTOR THERAPY:
*Folic acid 50mg IV QID - for methanol poisoning. metabolises formic acid to non-toxic metabolite.

*Thiamine 300mg IV daily OR *Pyridoxime 50mg IV QID for ethylene glycol poisoning. metabolises oxalic acid to non-toxic metabolite.

Dialysis

SPECIALIST CONSULTATION:
- Toxicologist
- ICU (dialysis)

DISPOSITION:
- ICU

35
Q

PV Bleeding in Early Pregnancy

EM rapid bombs
ep 213 - alloimunization
ep 51, 85 - ectopic pregnancy

2022.2 Communication

discuss with a pregnant patient, her assessment and management plan.

Do Bhcg on all females of childbearing age.
Even if tubal ligation - tubal ligation is not 100% effective and this increases risk of ectopic pregnancy

Screening for domestic violence as this increases in pregnancy

Speculum exam is an opportunity for STI screening and to check for non-obstetric causes of bleeding

Molar pregnancy:
“tumors from abnormal fertilization of an ovum, with over proliferation of trophoblastic tissue”
- uterus large for dates
- abnormally high bhcg - hyperemesis
- USS ‘snow storm’ appearance
treatment is surgical with a work up for metastatic disease

ED POCUS for ectopic pregnancy:
- empty uterus with serum bhcg > 6500
- complex adnexal mass
- FAST positive
- tubal ring or blob sign

Medical Management of Ectopic Pregnancy with Methotrexate:
- folic acid antagonist
Suitable for:
- BhCG <5000
- no fetal cardiac activity
- ectopic mass <4cm,
- hemodynamically stable (no sign of rupture)
- reliable for follow up
- normal baseline liver and renal function tests

Counselling:
- need to discontinue folic acid supplements and avoid alcohol
- need to avoid strenuous exercise and sexual intercourse due to risk of tubal rupture
- 5% failure rate, need to represent if symptoms of rupture
- follow up is crucial

Contraindications to Methotrexate: Tintinalli’s table

  • embryonic cardiac activity detected by TV US
  • ectopic pregnancy >4cm on TV US
  • Bhcg >5000
  • unreliable for follow up
  • immunocompromised
  • allergy to methotrexate
  • liver or renal disease
  • active peptic ulcer disease
  • haemodynamically unstable
  • breast feeding

MANAGEMENT STABLE MISCARRIAGE:
- expectant
- medical (misoprostol)
- surgical

A

DIFFERENTIAL DIAGNOSIS:
Pregnancy related:
*ectopic pregnancy
*miscarriage
viable - threatened
non-viable - missed, incomplete, complete
*Anembryonic pregnancy (blighted ovum) - “gestational sac with no yolk sac, no foetal pole”
*Gestational trophoblastic disease (molar pregnancy)

Non-pregnancy related
- infections - cervicitis, vaginitis
- trauma
- retained foreign body (tampon)
- cervical malignancy
- cervical ectropion

HISTRORY:
How pregnant?
- urinary bhcg
- bloods bhcg
- USS
- LMP, regular cycles

IVF

Blood group if known

previous pregnancies, terminations, miscarriages, live births

how much bleeding - spotting, clots, tissue,

abdominal pain (central vs. lateral)

history of trauma (sexual intercourse)

genital infections - pv discharge, malodorous

bleeding disorders - anticoagulation

Ectopic pregnancy red flags:
- previous ectopic pregnancy
- previous STI/PID
- previous tubal surgery, previous ruptured appendix (peritonitis)
- IVF pregnancy (risk of heterotopic pregnancy)
- conception with IUD in place
- Cigarette smoking (may alter embryo tubal transport)

Symptoms of ectopic rupture:
- syncope
- orthostatic hypotension
- right shoulder tip pain

Screening for domestic violence

EXAMINATION:
Haemodynamic status

Abdominal tenderness, peritonism

bimanual examination - adnexal tenderness, cervical motion tenderness, adnexal masses, assess uterus size ?consistent with dates

cervical os - open, closed, cervical masses

speculum - visualise vaginal walls, cervix to localise source of bleeding, obtain swabs to screen for infections

FAST scan - free fluid, IUP, adnexal mass, tubal ring sign, subchorionic haematoma

INVESTIGATIONS:
Group and screen - confirm Rhesus status (need for anti-D)

FBC - Hb, platelet count

Bhcg - confirm pregnancy and determine discriminatory zone

Pelvic USS
- determine location and viability

DISCRIMINATORY ZONE:
>1500 - IUP visible transvaginal USS
>6500 - IUP visible trans-abdominal USS

RUPTURED ECTOPIC CLINICAL FEATURES:
- syncope
- right shoulder tip pain (intra-peritoneal blood irritating the diaphragmatic parietal peritoneum)
- orthostatic hypotension
- abdominal tenderness with perotonism
- Adnexal mass

MASSIVE BLEEDING IN MISCARRIAGE:
- speculum exam to remove contents from the os
- oxytocin 40IU in 500ml NS over 4hrs
- ergometrine 250mcg IM
- tranexamic acid 1g IV
- OT for D&C

ANTI-D FOR Rh (D) NEGATIVE

Prevent alloimmunisation - prevent development of antibodies to foetal red cells that are Rh (D) positive

needs to be given in the first 72hrs

first trimester - 250IU for singleton pregnancy, 625IU for multiple pregnancy

Second & 3rd trimester - 625IU

successive dosing should be guided by the kleihauer test and degree of fetomaternal haemorrhage

is a blood product

small risk

if they have had one dose in early pregnancy and present with multiple episodes of bleeding. the initial dose will cover them up until 20 weeks so no need to repeat the dose.

36
Q

Hyperemesis Gravidarum

EM rapid bombs ep 233

2023.2 Junior doctor discussion

meet with a junior doctor to discuss the assessment and management of a patient with nausea and vomiting in pregnancy.

Medical Expertise: Assessment and Diagnosis: Investigations – 40%
- Analyse the results of an investigation to identify relevant findings or patterns.
- Correctly interpret the results of an investigation within the scenario.
- Create a focused investigation plan that confirms or excludes time critical diagnoses.
- Explain the reasons for selecting these tests in that investigation plan.
- Generate a relevant list of differential diagnoses after synthesising clinical information found on initial assessment.
- Formulate a provisional diagnosis to match the immediate issues.

Medical Expertise: Management – 30%
- Outline an overall plan for ongoing treatment of a patient.
- Adapt and initiate standard therapies to that patient e.g., drugs, fluids, gases and monitoring.
- Recognise patients who do not respond to therapy as expected and adjust the approach
accordingly.
- Create an appropriate ongoing reassessment and management plan.

Prioritisation and Decision Making – 30%
- Explain the specific benefits and risks of a treatment modality.
- Provide a rationale to explain and justify decisions about ongoing treatment.
- Incorporate patient and family/whānau needs as part of shared decision making.

Candidates were required to meet with the junior doctor (role player) and to:
- Interpret the VBG.
- Outline further differential diagnoses, investigations and management.

A

DIFFERENTIAL DIAGNOSIS:
Pregnancy related:
- Hyperemesis gravidarum
- Molar pregnancy
- Twin or multiple pregnancy

Infection:
- UTI/pyelonephritis
- gastroenteritis
- covid

Gastritis
Pancreatitis

Surgical causes:
- cholecystitis
- Appendicitis
- bowel obstruction

Metabolic:
- diabetes/DKA

HISTORY:

extent of vomiting

therapies tried so far if any
- dietary or lifestyle modification

history of GORD

weight loss
dehydration (urine output)
light headedness/pre-syncope

impact on day to day life
- employment
- relationships
- child care

screen for domestic violence

EXAMINATION:
sign of infection - fever, tachycardia, hypotension

confusion - wernicke’s encephalopathy

Mini-mental exam - depression

hydration status:
- tachycardia
- tachypnoea
- postural hypotension
- dry mm’s
- poor skin tugor
-POCUS assessment of the IVC and LV

abdominal exam:
- tenderness
- renal angle tenderness

INVESTIGATIONS:

Urine - ketonuria, exclude UTI

Bloods:
- BSL (hypoglycemia or hyperglycemia in DKA)
- Serum ketones (DKA, starvation)
- VBG (metabolic alkalosis, loss of HCl with vomiting)
- Urinalysis (pyelonephritis)
- UEC & CMP (renal failure & electrolyte disturbance - hypokalemia)
- Lipase (pancreatitis)
- CRP (raised with infection and inflammation)
- TSH (transient hyperthyroidism)
- Bhcg >100,000 (molar or multiple pregnancy)
- USS (molar, multiple pregnancy)

HYPEREMESIS GRAVIDARUM:
diagnosis of exclusion
starts week 5, settles by week 14

COMPLICATIONS:
- dehydration
- weight loss
- electrolyte derrangements
- renal failure
- transient hyperthyroidism
- wernicke’s encephalopathy (thiamine deficiency)
- peripheral neuropathies (vitamin B12, B6 deficiency)
- mallory weiss tears
- pneumomediastinum
- splenic avulsion
- psychological (anxiety depression)

MANAGEMENT:

NON-PHARMACOLOGICAL:
- eating small, frequent, high-carbohydrate, low-fat meals
- changing to a multivitamin without iron
- maintaining adequate hydration with cold drinks, carbonated drinks, or ice chips as tolerated
- snacking on high-protein foods between meals (nuts)
- eating crackers or plain biscuits before getting out of bed in the morning
- avoiding spicy foods and strong odours
- adequate sleep is important - fatigue will exacerbate symptoms
- over the counter ginger tablets

pharmacologic:
1st line:
- pyridoxime B6 25mg PO TDS
- doxylamine 12.5mg PO mane, midi, 25mg nocte

2nd line:
Metaclopramide (class A)
OR
Ondansetron (classe B1)
OR
Prochlorperizine 12.5mg IM (class C)

IV fluids - Hartmann’s

Thiamine 100mg IV

Replace K+, Mg+, Ca+

Maintenance with 5% dextrose

IV pantoprazole if GORD symptoms

SPECIALTY CONSULTATION:
Obstetric and gynaecology

DISPOSITION:
Short stay unit or admission

Category A
Drugs which have been taken by a large number of pregnant women and women of childbearing age without any proven increase in the frequency of malformations or other direct or indirect harmful effects on the fetus having been observed.

Category B1
Drugs which have been taken by only a limited number of pregnant women and women of childbearing age, without an increase in the frequency of malformation or other direct or indirect harmful effects on the human fetus having been observed.

Studies in animals have not shown evidence of an increased occurrence of fetal damage.

37
Q

Pulmonary Embolism in Pregnancy

EM cases - EM quick hits - radiation exposure in pregnant patients

2021.1 Junior doctor discussion

advise a junior doctor (role player) on conducting a risk
assessment for pulmonary embolism in a pregnant patient.

Prioritisation and Decision Making – 40%
* Explains the risk stratification process used by incorporating patient safety principles when
making key treatment decisions and disposition decisions
* Links clinical information and evidence-based practice to explain decisions made
* Provides a rationale to explain decisions about ongoing assessment

Medical Expertise – 40%
* Creates a focused investigation plan that confirms or excludes time critical diagnoses
* Explains the reasons for selecting those tests in that investigation plan
* States the theoretical accuracy of an investigation for confirming a diagnosis

Leadership and Management – 20%
* Rectifies sub-optimal treatment plans:
* Establishes rapport and makes learner feel supported
* Provides appropriate level of detail to meet the needs of the patient:

Candidates were required to:
* advise the junior doctor (role player) how to assess a pregnant patient with suspected PE
* respond to the junior doctor’s subsequent questions regarding this patient.

A

Controversial
No good evidence to guide us yet
Only expert opinion

DIAGNOSTIC DILEMA:
- Shortness of breath and leg swelling are common symptoms of pregnancy

  • there are no pre-test probability scoring systems that are validated in pregnancy - determined by clinical gestalt
  • D-dimer starts to rise in the second trimester of pregnancy

DIPEP study concluded that negative D-dimers are insufficient to rule out PE in pregnancy

No D-dimer unless 1st trimester

PE DIAGNOSTIC PATHWAY:

1) What is your pre-test probability or Gestalt? If you have a pregnant patient and PE is the most likely diagnosis then place the patient as high risk and progress to imaging (see below).

2) If the signs and symptoms are suggestive of PE but there are other diagnosis equally likely and the patient is in first trimester perform a D-dimer. If this is negative you can stop.

3) If the patient is high risk or has a positive d-dimer you should now perform a chest x-ray. You will need this to aid in the choice between V/Q and CTPA. It also may show an area of consolidation or a pneumothorax.

4) Prior to consideration of V/Q or CTPA perform bilateral venous dopplers. If this is positive you can stop here and treat for PE.

5) V/Q or CTPA? The main concern is radiation risk for both mother and fetus. The risk of death from an undiagnosed PE is much higher than the risk of malignancy due to radiation. When considering V/Q as an option also be aware that you may still need to go on to do a CTPA if inconclusive.

CXR

  • readily available
  • extremely low dose of radiation to fetus and mother
  • useful in detecting alternative diagnoses i.e. pneumoniae, pneumothorax, pleural effusion, pulmonary oedema - need CXR to decide between VQ scan and CTPA
  • high probability of non-diagnostic VQ scan if pulmonary pathology on CXR
  • If chest x-ray is normal the patient should undergo a half-dose perfusion scan. If the chest x-ray is abnormal the patient should undergo a CTPA.
  • sometimes pulmonary infiltrate can represent pulmonary infarction caused by PE - review CXR findings with radiologist if possible

US lower limbs for DVT
- readily available, cheaper than CT and VQ scans
- no radiation or contrast exposure
- if DVT present, can stop investigation and commence anticoagulation

CTPA
- readily available
- is the gold standard for identifying PE
- less foetal radiation exposure compared to VQ scan
(CTPA = 0.1mGy, VQ scan = 0.5mGy)
- alternative pathologies can be identified (pneumoniae)
- theoretic risk of foetal hypothyroidism with iodine contrast
- radiation dose to maternal breast tissue from CTPA is 20-100 times that of V/Q scan (increasing the women’s lifetime risk of breast cancer)
- can use a bismuth shield to reduce radiation to breasts

VQ scan
- less radiation exposure to maternal breast tissue - this is useful in those who have strong family hx of breast cancer

  • not readily available
  • time consuming study
  • exposes foetus to 0.5 mGy radiation (more radiation than CTPA)
  • non diagnostic scans are high in those with lung disease
  • need IDC to reduce radiation to bladder

Bedside ECHO

  • insufficient to exclude subsegmental PE.
  • increased R heart strain may be normal physiology of pregnancy

Patient counselling:

PE in pregnancy is a leading cause of mortality in pregnancy so it must be excluded

Scans required to exclude PE –> radiation exposure to fetus and maternal breast tissue

breast tissue is rapidly proliferating, making it more radiosensitive and increases lifetime risk of breast cancer

amount of radiation to cause fetal harm/teratogen effect is 50mGy (=0.05Gy)

background radiation in one year = 2mGy

CXR - 5 days background radiation
CTPA = 0.1mGy (3 weeks)
VQ scan = 0.5mGy (3 months)

Discuss low dose radiation protocols with radiologist

Shared decision making with patient

Involve obstetrics and respiratory physicians.

38
Q

Pre-eclampsia

EM rapid bombs ep 69, 126,

2021.2 Station

Discuss with an examiner their assessment and management of a pregnant female having a seizure.

Medical Expertise: Initial assessment and management – 30%
* Seeks evidence of time critical diagnoses when performing assessment
* Creates a focused investigation plan that confirms or excludes time critical diagnoses
* Initiates treatments specific to identified abnormalities in airway and/or ventilation

Medical Expertise: Further management – 30%
* Outlines an overall plan for ongoing treatment of the patient
* Initiates treatments specific to identified abnormalities in circulation
* Identifies important aspects of supportive care in this patient
* Anticipates and manages common complications during and after a procedure

Prioritisation and Decision Making – 40%
* Prioritises a differential diagnosis list to determine the most likely diagnoses in a patient
* Prioritises essential components of ongoing care a of any patient in the emergency
department
* Initiates treatments specific to identified abnormalities in airway and/or ventilation
* Provides a rationale to explain and justify decisions about ongoing treatment

Candidates were required to:
* outline their initial approach to the assessment and management of the patient to the
examiner
* answer further questions from the examiner regarding the evolving case.

A

DEFINITION:
a)
>20weeks gestation
+
BP > 140/90 OR 20mmHg higher than booking BP
+
1 or more signs of end-organ dsyfunction
*proteinuria
*renal failure/hyperuricemia
*transaminitis/RUQ pain
*headache/visual impairment
*haemolytic anaemia
*thrombotic microangiopathy
*DIC
*pulmonary oedema
*peripheral oedema

INVESTIGATIONS:
FBC - thrombotic microangiopathy & haemolytic anaemia
*low Hb
*low haematocrit
*low platelets <100

UEC - raised creatinine

LFT - transaminitis (in HELLP sydrome)

LDH - raised in haemolysis (HELLP syndrome)

Uric acid - raised

Coagulation panel - DIC
*prolonged PT
*prolonged APTT
*low fibrinogen

Urinalysis - proteinuria, raised protein/creatinine ratio >0.3

24hr urine collection >0.3g protein

CTG - fetal distress, placental abruption, contractions in pre-term labour

CT brain - intracranial haemorrhage

COMPLICATIONS:
- placental abruption
- DIC
- Intracranial haemorrhage
- subcapsular liver haematoma
- splenic haematoma
- multi-organ failure

INITIAL ASSESSMENT & MANAGEMENT:

Left lateral position

High flow 100% Oxygen 15L NRBM
Guedel airway, jaw thrust and chin lift

Check GLUCOSE - 2ml/kg 10% dextrose IV - target BSL >5mmol/L

Check BLOOD PRESSURE

STOP SEIZURE:
- Magnesium 4g IV over 20min, Magnesium infusion 1g/hr
- Midazolam 5mg IV or 10mg IM for refractory seizures

Check RR and patellar reflexes hourly
Check serum magnesium if toxicity is suspected
Therapeutic serum magnesium level 1.7–3.5mmol/L

BLOOD PRESSURE CONTROL:

Labetalol 20mg iv, repeat in 10min (max 80mg) - followed by infusion 20mg/hr - fetal bradycardia

Hydralazine 5mg IV bolus every 10min (max 30mg) - may cause excessive hypotension and reflex tachycardia

GTN infusion starting at 10mcg/min increasing by 5mcg every 10-15min until desired effect

Aim for BP 130/80
Arterial line
IDC to monitor UO

SEIZURE PROPHYLAXIS:

Magnesium sulphate 4g IV over 20min
Continue magnesium sulphate infusion at 1g/hr
Contraindicated in myasthenia gravis
Reduce dose in renal insufficiency

Monitor for:
- respiratory depression
- heart block –> cardiac arrest
- loss of patellar reflexes (first sign of toxicity)
- symptomatic hypocalcemia

Antitode: calcium gluconate 1g IV over 5min
(10ml calcium gluconate 10%)

CTG Monitoring (fetal bradycardia with labetalol)

Consider betamethasone 11.4mg IM in <37/40 - promote fetal lung maturity

O&G for imminent delivery by C-section

39
Q

Subarachnoid Haemorrhage

2023.1 OSCE Station:
42y F with sudden onset severe headache
- Discuss investigations (CT, LP, CTA)
- Interpret CT scan
- GCS drops to 5. Outline your management

ASSESSMENT:
HISTORY:
- thunderclap
- loss of consciousness/syncope
- neck stiffness
- onset on exertion/sex
- vomiting
- eye pain

Sentinel bleed - headache in the preceding days/weeks

Risk factors:
- hypertension
- smoking
- heavy alcohol
- sympathomimetic use - cocaine
- personal history of cerebral
- aneurysm
- polycystic kidney disease
- connective tissue disease - ehlers danlos

EXAMINATION:
Temp - may have low grade temperature (meningeal irritation)
Hypertension - will need to be reduced

Neck stiffness/meningism

Eyes:
- optic fundi (subhyaloid haemorrhage)
- IOP (glaucoma)
- unequal pupils, diplopia, disconjugate gaze (cranial nerve 3,4,6 palsy)
POCUS - optic sheath diametre (raised ICP/hydrocephalus)

Gross neurological examination:
- GCS
- motor function
- speech
- vision

INVESTIGATIONS:
BSL - rule out hypoglycemia as mimic
Hb - anaemia more difficult to detect SAH on CT
ECG - deep ST depression and T wave inversion in praecordial leads

Non-contrast CT
- 99.7% sensitive within 6hrs of headache onset if modern CT scanner and interpreted by radiologist
- sensitivity declines with time

LUMBAR PUNCTURE:
- indicated if negative CT beyond 6hrs
- increase ED length of stay (wait 12hrs for xanthochromia)

Contraindications to LP
- anticoagulation
- spinal metal hardware in situ
- cellulitis overlying the lumbar spine

Complications
- post LP headache
- traumatic tap
- increase ED length of stay

Pro’s
- can diagnose meningitis
- cost effective
- may avoid need for CTA which comes with radiation dose and contrast load

CT angiography
- higher radiation dose
- can’t detect microaneurysms <3mm
- incidental finding of aneurysm 2.5% of population may lead to unnecessary invasive intervention

A

Differential diagnosis for THUNDERCLAP HEADACHE
“severe headache with maximal intensity within 1hr of onset”

  • subarachnoid haemorrhage
  • carotid/vertebral artery dissection
  • pituitary apoplexy
  • reversible cerebral vasoconstriction syndrome
  • PRES (posterior reversible encephalopathy syndrome)
  • intracerebral haemorrhage
  • acute closed angle glaucoma
  • cerebral venous sinus thrombosis

CAUSES SAH:
Traumatic - most common cause

Spontaneous atraumatic:
- aneurysm (85%)
- AV malformation
- CVST
- RCVS
- amyloid angiopathy

RISK FACTORS:
- hypertension
- age >50
- smoking
- heavy alcohol or sympathomimetic use
- known aneurysm >5mm or in posterior circulation
- family history aneurysms
- connective tissue disorder (ehlers danlos)
- polycystic kidney disease

Increase likelihood ratio:
- thunderclap headache plus
+ vomiting
+ neck stiffness
+ syncope/loc
+ similar recent headache indicative of sentinel bleed

SAH can be ruled out with 100% sensitivity with the OTTAWA SAH RULE F0R HEADACHE evaluation.
- < 40 years of age
- without neck pain or stiffness
- without a witnessed loss of consciousness
- without onset during exertion
- without a thunderclap headache
- and without limited neck flexion

INVESTIGATIONS:
“Less likely to see SAH on CT if patient is anaemic”

Modern Non-contrast CT within 6 hours
- 99.7% sensitive
- >99.5% specific
sensitivity decreases with time therefore a SAH cannot be excluded with a normal CT after 6hrs

“a normal head CT within 6 hours of headache onset is extremely sensitive in ruling out aneurysmal subarachnoid haemorrhage”

CT 6-12hrs
- 98% sensitive

91-93% at 24hrs
50% at 7days

Ref: Tintinalli’s

If CT is performed beyond 6hrs and is negative for SAH. Need to proceed to LP.

CSF for xanthochromia (bilirubin released from RBC)
Takes 12hrs for xanthochromia to develop.

CSF RBC count in the 3rd and 4th tube.

Comparison RBC counts between consecutive tubes or between tubes 1 and 4 can be used to differentiate SAH from a traumatic lumbar puncture

Unfortunately, no agreed threshold number of RBCs needed in the CSF to diagnose SAH.

CT angiography for those who refuse LP, LP is contraindicated or have failed LP
- can’t see micro aneurysms
- incidental finding of aneurysms leading on unnecessary intervention

Negative CT, no xanthochromia, and zero or <5 RBCs in tube 4 CSF exclude SAH.

Negative CT with xanthrochromia or elevated RBC count in tube 4 is diagnostic for SAH.

CAUSES FOR DROP IN GCS:
- seizure
- hydrocephalus
- re-bleed
- vasospasm with ischemia
- iatrogenic - excessive opioids/sedation
- hypercapnoeic respiratory failure

40
Q

Subarachnoid Haemorrhage Management

COMPLICATIONS:

  • rebleeding/haematoma expansion
  • vasospasm - nimodipine
  • infarction/ischemia (careful BP management to maintain CPP)
  • obstructive hydrocephalus - RBCs block arachnoid villi and CSF drainage - need for VP shunt
  • raised intracranial pressure and brain herniation

MANAGEMENT:

Analgesia - morphine 2.5-5mg IV titrate to patient comfort

Anti-emetic - ondansetron 4-8mg IV

BP control - target SBP 140-160
Treat hypertension with esmolol 500 micrograms/kg intravenously, over 1 minute, followed by 50 to 200 micrograms/kg/minute by intravenous infusion

Avoid hypotension (to maintain CPP) have noradrenaline infusion on stand by (0.02mcg/kg/min)

Prevent vasospasm - nimodipine 60mg NGT/PO Q4hrs

Prevent raised ICP
- raise head of bed 30 degree

Prevent hypoxia - high flow oxygen 15L NRBM (target SaO2 >95%)

Monitor for and manage raised ICP:
cushings reflex
POCUS eye - pappiloedema, optic sheath diammeter >5mm
- raise head of bed 30 deg
- 3% hypertonic saline

Seizure prophylaxis levetiracetam 45-60mg/kg IV

Neuroprotective intubation

PROGNOSIS:

Hunt & Hess grading system for severity - clinical scoring system that estimates mortality based on GCS and neurological deficits

DEFINITIVE MANAGEMENT:

Neurosurgery

endovascular coiling or neurosurgical clipping of aneurysm

manage hydrocephalus with external ventricular drain

DISPOSITION:

Neuro ICU

A

NEUROPROTECTIVE INTUBATION:

POSITION:
elevate head of bed 30 deg
tragus in line with sternal notch
face parallel with ceiling

PREOXYGENATION:
100% oxygen with 15L NRBM and NP

PREMEDICATION with fentanyl 100mcg iv (blunt sympathetic response with laryngeal manipulation)

SEDATION with ketamine 1-2mg/kg iv (haemodynamically stable)

PARALYSIS with rocuronium 1.2mg/kg iv

Apnoeic BVM - Prevent hypoxia and hypercapnoea
Post intubation sedation with propofol and fentanyl

Treat hypertension - esmolol aim SBP 140-160

BP support with noradrenaline 0.1-0.2mcg/kg/min titrate to SBP 120-160

Invasive blood pressure monitoring - arterial line

Prevent raised ICP:
- Elevate head of bed 30 degrees
- Loose neck ties

Aim for normal physiological parametres:
- normotensive SBP 110-140
- normal CO2 PCO2 35-40
- normal glucose 4-10mmol/L
- normal temperature 36-37

Seizure prophylaxis
- Levitiracetam 60mg/kg iv

Regularly evaluate for signs of raised ICP and brain herniation
- unilateral blown pupil
- focal neurological deficit (hemiparesis)
- cushings reflex (bradycardia, hypertension)
- increased tone, upgoing babinski, decorticate posturing

Repeat CT scan

Urgent neurosurgical consult

41
Q

Paediatric Fever

EM rapid bombs ep 33, 188 - pneumonia
EM rapid bombs ep 16, 60, 62 - meningitis

2022.2 History taking

Take a focused history from a parent whose child has presented with fever.

Medical Expertise – 40%
- Elicits a focused, relevant history de novo
- Identifies important historical details (red flags) diagnostic of an important condition
- Generates a relevant list of differential diagnoses after synthesising clinical information found
on initial assessment.

Prioritisation and Decision Making – 30%
- Links clinical information and evidence-based practice to explain decisions
- Provides a rationale to explain decisions about ongoing assessment
- Provides a rationale to explain and justify decisions about ongoing treatment.

Communication – 30%
- Establishes (asks) the concerns/issues/needs of the patient/relative/person early in the
encounter
- Uses language appropriate to the patient’s level of understanding / avoids jargon/ explains
medical terms
- Demonstrates empathy
- Demonstrates a professional and respectful approach.

Candidates were required to:
- take a focused history from the parent (role player)
- answer any questions the parent may have.

RED FLAGS:
*neonates (<28days)
*incomplete immunisations
*immunocompromise - oncology patients
*asplenic children
*sickle cell disease
*fever despite antibiotics
*fever with complex seizures
*children with medical co-morbidities (cystic fibrosis, congenital cardiac disease)
*children with in-dwelling devices (VP shunts, central lines, IDC’s)

A

Core temperature = temperature in pulmonary artery

Rectal temperature = gold standard

Tympanic thermometers - accurate but probe may be too large for infants ear canal

oral temp requires co-operation
axillary temp is inaccurate

*Antipyretics may prolong viral shedding, impair antibody response to infection
*Antipyretics can cause hepatic dysfunction, GI bleeding, Reye syndrome, metabolic acidosis
*Antipyretics are used to treat discomfort and pain not they height of the temperature
*Antipyretics do not prevent febrile seizures

The majority of children with fever will have self limiting viral infections
The challenge is to identify children at risk of serious bacterial infection
- UTI
- occult bacteremia (bacteria in the blood stream)
- meningitis

DIFFERENTIAL DIAGNOSES:
self limiting viral illness
serious bacterial infection
- meningitis
- occult bacteremia
- UTI/pyelonephritis
- pneumoniae
- cellulitis/septic arthritis

Malignancy
Kawasaki’s disease

HISTORY:
Feeding (<50% in 24hrs)
Urine output (<50% in 24hrs)

Overseas travel
Sick contacts - day care
Contact with animals

Immunisation status

PMHx:
- immune compromised
- cystic fibrosis
- congenital cardiac disease
- indwelling devices
- recent operations

Medications:
- immunosuppression
- antibiotics

Allergies:

Social History:
- parents, siblings
- home life
- occupation, car, phone
- alcohol, smoking drugs
- social supports

Developmental:
- gaining weight
- reaching milestones
- daycare, schooling

EXAMINATION:
Reduced level of alertness, subdude, less active

Increased work of breathing - tachypnoea

Poor perfusion, dehydration

Pallor, purpuric rash
bulging fontanelle

INVESTIGATIONS:

FBC - neutrophilia or neutropenia

blood cultures

Urinalysis, microscopy and culture

PLAN:

Review in 24hrs

42
Q

Pediatric UTI

EM rapid bombs ep 182, 183, 184 (UTI)

*Midstream urine (MSU): preferred method for toilet-trained children — contamination rate 25%

*Clean catch: appropriate for pre-continent children who cannot void on request, but are not seriously unwell (yield may be improved by gently rubbing child’s suprapubic area with gauze soaked in cold fluid, see urine tests) — contamination rate 25%

*Suprapubic aspirate (see SPA): gold standard — contamination rate 1%

*In/out catheter: useful if there is little urine in the bladder, such as after failed clean catch or SPA (discard first few drops of urine if possible to reduce contamination) — contamination rate 10%

*Bag urine: not recommended for culture due to high false positive rates — contamination rate 50%

Rx Cephalexin 33mg/kg TDS for UTI
Rx Cephalexin 45mg/kg TDS for pyelonephritis

A

Pediatric Pneumoniae

43
Q

Bradycardia & Transcutaneous Pacing

EM cases ep 155 bradycardia
- video on transcutaneous pacing

IBCC ep 5 - bradycardia

EM rapid bombs ep 61 ketamine facts

2022.2 Procedure/Equipment

teach a junior registrar the use of
transcutaneous cardiac pacing.

Medical Expertise: Application of transcutaneous pacing – 40%
- Describe the important features of common procedural equipment (transcutaneous pacing)
- Appropriately use procedural and monitoring equipment
- Proficiently perform the procedure.

Medical Expertise: Issues in transcutaneous pacing / further management – 40%
- Anticipate and manage common complications during and after a procedure
- Adapt the performance of a procedure in response to unforeseen complications when
performing a procedure.

Scholarship and Teaching – 20%
- Establish rapport, make the learner feel safe and supported
- Demonstrate well-paced communication, use language appropriate to the learner’s level
- Check understanding in learner (regularly and at conclusion), invite questions and clarify any
areas of uncertainty
- Summarise the session with appropriate emphasis of key elements.

Candidates were required to meet with the junior registrar (role player) and to:
- demonstrate / teach the use of transcutaneous pacing
- answer any questions they may have.

A

CAUSES:
- hyperkalemia
- MI (inferior and anterior MI)
- toxicological (calcium channel blockers, beta blockers, digoxin, clonidine)
- Intracranial pathology/raised ICP/imminent herniation (cushing’s reflex)
- hypothermia
- hypothyroid myxoedema
- post cardiac/valve surgery
- infection (myocarditis, endocarditis)
- neurogenic shock

ASSESSMENT:

Syncope/Presyncope
Chest pain
SOB
Heart failure
Altered mental state

Medication review - beta blockers, calcium channel blockers, digoxin, clonidine

ECG
- MI (anterior vs inferior MI)
- signs of hyperkalemia
- deep TWI in precordial leads in raised ICP
- identify location of problem (SA/AV node vs his purkinje system)

Electrolytes
- hyper K+
- hyper Mg+
- hypo Ca+

MEDICATIONS:
atropine 600mcg IV repeat in 3min max 3mg

adrenaline infusion 2-10mcg/min titrate to effect

isoprenaline 2-10mcg/min titrate to effect

Calcium gluconate 3g IV

treat the underlying cause of bradycardia

Myocardial infarction –> cath lab for revascularization

Hypothermia –> rewarming

Hypothyroid myxoedema –> hydrocortisone and thyroxine

beta blocker/calcium channel blocker overdose –> calcium + high dose insulin therapy

digoxin toxicity –> digibind

TRANSCUTANEOUS PACING
INDICATIONS:
- haemodynamically significant bradycardia refractory to medications
- fastest strategy to increase the heart rate
- Temporising measure until patient receives transvenous pacing or definitive management with pacemaker

COMPLICATIONS:
- failure to achieve mechanical capture
- external pacing impulses cause chest wall muscle contractions which are painful to the patient

SEDATION:
- low dose ketamine 10-20mg IV boluses then infusion 0.3mg/kg/hr
- sedation, dissociation, amnesic, analgesic
- maintain own airway
- haemodynamically stable

CORRECT PAD PLACEMENT
- Air is a poor conductor of electricity, so placing pads that overlie the lungs is a poor strategy
- AP position
- anterior pad covers the left parasternal window of the heart
- posterior pad just below the left scapula

Turn on
Pacer mode
Set rate to 60bpm

CURRENT
- If patient is in bradysystolic arrest, start at maximal current and work your way down after the patient has stabilized.

Otherwise start at 30mA and titrate up until

Electrical capture - QRS present after each electrical impulse

Mechanical capture - confirm by palpating distal pulse, visualize cardiac contractility on bedside echo or waveform capnography

Continue pacing at 10-20 mA above the minimum energy required for capture.
Usually ~40-80 mA required to achieve capture (possibly more in obesity or obstructive lung disease).
continue pacing 10-20mA above minimum energy required for capture
usually 40-80mA but may need higher current in obesity and COPD

Be aware of pseudopacing
check for mechanical capture:
- palpate femoral artery or dorsalis pedis
- pulse oximetry
- POCUS

44
Q

VBG Interpretation - HAGMA

HAGMA “KULT”

KETONES
- DKA
- alcoholic ketoacidosis
- starvation ketoacidosis

check blood ketones

UREMIC RENAL FAILURE

  • usually occur when GFR <20

check UEC (urea, creatinine, GFR)

LACTATE

Shock of any etiology:
- Septic shock.
- Cardiogenic shock.
- Obstructive shock (PE, tamponade).
- Hypovolemic shock.

Regional hypoperfusion:
- Ischemic limb.
- Mesenteric ischemia.
- Muscle hyperactivity:
- Generalized seizure.
- Extreme exertion.
- Extreme anemia

TOXINS

Massive paracetamol poisoning
Metformin
Toxic alcohols
- methanol
- ethanol
- ethylene glycol
Toluene
Carbon monoxide.
Cyanide.
Salicylate.
Sympathomimetics
- methamphetamines
Iron
Isoniazid.

A

Anion gap = Na+ - Cl- -HCO3-

expected CO2 = 1.5 x HCO3- +8

Corrected Na+ in hyperglycaemia
Corrected Na+ = Na+ + (Glucose – 5 ÷ 3)

Shifts K to intravascular space so increasing the serum K
Correction of acidosis will produce a decrease in serum K (may drop precipitously eg correction of DKA)

Delta ratio = (change in anion gap) / (change in bicarbonate)
* This can reveal any mixed acid-base disorders

  • Less than 0.4 = pure normal anion gap acidosis
  • 0.4-0.8 = mixed high and normal anion gap acidosis
  • 0.8-2.0 = pure high anion gap acidosis
  • More than 2.0= high anion gap acidosis and a pre-existing metabolic alkalosis

Calculated osmolarity = (2 x [Na+]) + [glucose] + [urea])
Osmolar gap = Osmolality (measured) – Osmolarity (calculated)
normal = < 10

45
Q

VBG interpretation - Metabolic alkalosis

Vomiting in pregnancy

A

complete respiratory compensation
expected CO2 in metabolic alkalosis
= (0.7 x HCO3) + 20

46
Q

Anaphylaxis

vasodilatory shock
bronchospasm
upper airway oedema

7 Maximum Medications to consider in Crashing Anaphylaxis: Epinephrine, Rocuronium, Ketamine, Bronchodilators, Magnesium Sulphate, Vasopressors, Steroids

1.Push dose epinephrine 1mcg/kg IV push then 1mL/kg/min and titrate

Adrenaline formulations:
Adrenaline 1:1000 (1 mg/mL) Adrenaline 1:10,000 (1 mg/10 mL)

0.5ml of 1:10,1000 = 50mcg

2.Rocuronium 1.2mg/kg IV push paralytic if patient is maintaining muscle tone

3.Ketamine 1-2mg/kg IV induction

ketamine infusion 1-10 mg/kg/hr for it’s bronchodilator properties

4.Continuous bronchodilators in circuit (salbutamol 15 mg/h) + ipatropium 3 x 500 mcg for a 1-hour continuous nebulization)

IV Salbutamol 10mcg/kg loading then 5mcg/min

5.IV Magnesium sulphate 2g (40 mg/kg) over 20

  1. Adrenaline infusion 0.05mcg/kg/min

7.IV steroids:
- Methylprednisolone 2mg/kg (max 80mg) or Hydrocortisone 5mg/kg (max 400 mg)]

A

For persistent hypotension/shock
* give normal saline (maximum 50 mL/kg in the first 30 min)

For upper airway obstruction
* nebulised adrenaline (5 mL, i.e. 5 ampoules of 1:1000)
* intubation if skills and equipment are available

Need intubation
only cuffed ETT can withstand the high airway pressures required to ventilate the severe asthmatic/anaphylaxis

47
Q

Threatened Airway - Paediatric
Stridor

EM rapid bombs - ep 138 ludwigs angina
EM quick hits 5 - ludwigs angina

2023.1 Case based discussion

discuss with an examiner, the airway management plan for a child with airway compromise.

Medical Expertise: Assessment – 20%
* Perform a structured initial assessment on a critically ill patient.
* Identify risks of deterioration in the patient.
* Identify important historical details (red flags) diagnostic of an important condition.
* Analyse the results of an investigation to identify relevant findings or patterns.

Medical Expertise: Treatment – 40%
* Initiate treatments specific to identified abnormalities in airway and/or ventilation.
* Outline an overall plan for resuscitating a patient.
* Anticipate and manage common complications during and after a procedure.
* Create an appropriate ongoing reassessment and management plan.

Prioritisation and Decision Making – 40%
* Prioritise the essential tasks in a high complexity patient.
* Prioritise chosen treatment options to create an appropriate escalating treatment plan.
* Provide a rationale to explain and justify decisions about ongoing treatment.
* Justify own decisions as they occur and make timely corrections.

Candidates were required to interact with the examiner and to:
* Respond to the examiner’s questions regarding the airway assessment and management of
this patient.

Paediatrics with acute epiglottitis are more likely to lose their airway because they have smaller diameter airway and larger epiglottis

Collateral history:
- age of child
- events leading to presentation
- preceeding URI or pharyngitis
- timing of stridor (after eating, acute, subacute, chronic)
- dental pain/infection (ludwigs angina)
- vaccination status (diptheria, haemophilus influenza in acute epiglottitis)
- asthma/anaphylaxis
- congenital airway abnormalities

  • sore throat
  • dysphagia
  • odynodysphagia
  • fever
  • drooling, inability to swallow secretions
  • dysphonia (change in voice)
  • inability to lie flat

EXAMINATION:

Vital signs:
- Fever (infectious aetiology)
- Tachypnoea
- Hypoxia
- Tachycardia

  • respiratory distress (increased work of breathing, retractions)
  • stridor
  • facial swelling
  • neck swelling
  • dysphonia “hot potato voice”
  • drooling, inability to swallow secretions
  • trisum
  • raised tongue
  • woody texture to floor of mouth
  • submandibular crepitus
  • tripod stance
  • increased work of breathing
  • intercostal/subcostal retractions
  • altered mental status and fatigue
  • tender anterior neck
  • cervical lymphadenopathy

MEDICAL MANAGEMENT:
Acute epiglottitis:
Ceftriaxone 50mg/kg IV or Cefotaxime 50mg/kg
Moxifloxacin 10mg/kg IV if penicillin allergy

Dexamethasone 0.6mg/kg IV (10mg IV) - controversial, no studies show benefit

Nebulised adrenaline 5mg

Ludwigs angina:
- benzylpenicillin 60mg/kg (2.4g)
- metronidazole 12.5mg/kg (500mg)

A

DIFFERENTIAL DIAGNOSES:

INFECTION:
- retropharyngeal abscess
- peritonsillar abscess
- bacterial tracheiitis
- Ludwig’s angina
- acute epiglottitis
- croup
- diptheria

ALLERGY/IMMUNE
- anaphylaxis
- hereditary angioedma

TRAUMA:
- blunt or penetrating
- burns
- caustic inhalation/ingestion
- thermal epiglotitis (hot drink)

FOREIGN BODY

CONGENITAL
- tracheomalacia
- laryngomalacia
- vocal cord paralysis

ACQUIRED
- vocal cord paralysis

ASSESSMENT of deterioration:
*Worsening stridor despite medical therapy
*GCS - altered mental status/agitation
*Fatigue & lethargy
*Worsening hypoxia despite oxygen therapy
*Bradycardia
*VBG - respiratory acidosis, hypercapnia

INVESTIGATIONS:

VBG - respiratory acidosis with hypercapnoea

lateral neck xrays - foreign body, thumbprint sign and vallecula sign in acute epiglottitis,

AP neck xray - steeple sign in croup

AIRWAY MANAGEMENT:

**PARENTS
Designate staff member to accompany parents - parents to be in the resuscitation room with the understanding that they will be escorted out in emergency situation

**DIFFICULT INTUBATION
Anatomically difficulty intubutation due to airway oedema - likely to faile

**SAFER ALTERNATIVE
Safer to perform awake fibreoptic intubation in theatre with anaesthetics and ENT ready to perform surgical airway “dual airway setup”

Call for help from anaesthetics and ENT

POSITION upright or position of comfort for patient - improves gas exchange

PREOXYGENATE
15L NP +
15L NRBM 15L/min for at least 3min targeting SaO2 >95%
15L BVM

If unable to target SaO2 >95% due to patient agitation
NIV with CPAP
Delayed sequence induction with ketamine 1mg/kg IV bolus

INDUCTION AGENTS:
Ketamine 1-2mg/kg IV - haemodynamically stable, maintain respirations, bronchodilator properties
Rocuronium 1.6mg/kg IV
Push dose adrenaline 10mcg/ml in 20ml syringe
give 1mcg/kg IV push
Adrenaline infusion 0.1-1mcg/kg/min targeting MAP 80

Dual airway setup
Second provider in PPE ready to perform front of neck access/ needle cricothyroidotomy

Most experienced operator to intubate

Video laryngoscope + bougie

ETT = age/4 + 3.5 (use smaller ETT due to airway oedema)

ETT depth age/2 + 12

If intubation fails,

Rescue LMA - will likely worsen obstruction

Declare CICO to team
Second provider to perform needle cricothyrotomy + jet ventilation

Can convert to seldinger cricothyrotomy if needle cricothyrotomy fails

POST PROCEDURE

confirm ETT placement
- continuous waveform capnograpghy ETCO2

Secure ETT to avoid inadvertent extubation

POST INTUBATION sedation
- propofol and fentanyl
- adrenaline infusion

DISPOSITION
- Paediatric ICU

Update family/parents

48
Q

Procedural Sedation/Threatened airway

obese patient undergoing procedural sedation ? reflux causing laryngospam

Best Case Ever 57 PREPARE mnemonic for Airway Management

2022.1 Case based discussion

Sedation in obese patients
Situational awareness
Airway assessment
- History
- Examination
Fasting status
Informed consent
Laryngospasm management

Medical Expertise: Assessment and Management – 50%
* Recognises and expedites any specific intervention essential to resuscitation
* Initiates treatments specific to identified abnormalities in airway and/or ventilation
* Adapts and initiates standard therapies to that patient, including drugs, fluids, gases, and
monitoring
* Adapts the performance of a procedure in response to unforeseen complications when
performing a procedure.

Prioritisation and Decision Making – 25%
* Highlights high-risk features identified during initial patient assessment
* Provides a rationale to explain and justify decisions about ongoing treatment.

Leadership and Management – 25%
* Provides strategies to improve patient safety in a given scenario
* Outlines major principles in initial or bedside response
* Outlines the further processes to optimise improvements in patient care.
Candidates were required to interact with the examiner and to:
* outline their initial approach to the assessment and management of the patient
* answer further questions from the examiner regarding the evolving case.

A

*Is the procedure necessary? - is the patient going to OT anyway?
*Is there a safer alternative? - nerve block/nitrous oxide
*is the ED the safest place to perform sedation
- patient factors (anticipated difficult airway, haemodynamic instability)
- departmental factors (busy department, adequate staffing)
*resources
- resuscitation room
- monitoring
- staffing and expertise

DIFFICULT AIRWAY ASSESSMENT:

HISTORY:
“AMPLE”

Allergies
Medications
Past medical history/Family history - malignant hyperthermia
Last ate/drank
Events - leading to presentation

EXAMINATION:

General:

Obesity, Pregnancy, OSA

Facial hair (unable to get a good seal with BVM ventilation)

Others:
- facial trauma
- airway burns
- angioedema
- masses
- ludwigs angina

Mouth:
- small mouth, large tongue, high arched palate
- large protruding teeth, large overbite
- receding mandible and chin
- mallampati 3 (only see base of uvula)
- mallampati 4 (only see hard palate)

Jaw opening <6cm
Thyromental distance <6cm

Neck:
- short thick neck circumference >40cm
- inability to extend (trauma, c-spine collar, arthritis, spinal fusion)

*Ensure FASTING for at least 4hrs if non-emergent procedure
*Always mentally prepare for SURGICAL AIRWAY
- assess FON and mark landmarks
*Obese patients are at risk of ASPIRATION
- have Ducanto or Yankauer suction catheters ready to suction posterior pharynx

INFORMED CONSENT

OPTIMIZE:
Prevent hypotension
- fluid resuscitate (optimise fluid status before sedation)
- push dose vasopressors on stand by

Prevent hypoxia:
Pre-oxygenate
- sit up right
- HFNP 100% oxygen 60L/min + NRBM 15L/min for at least 5min

POSITION:
- RAMPED
- Reverse trendelenburg
improves respiratory mechanics
moves weight of off chest to allow expansion
- ear to sternal notch
- face plane parallel to the ceiling

MONITORING:
- level of consciousness
- continuous cardiac monitoring
- continuous pulse oximetry
- BP monitoring q5min
- ETCO2 capnography - continuous

SEDATION:
- ketamine 1-2mg/kg
- administer slowly

LARYNGOSPASM MANAGEMENT:

stop the procedure
call for more help

suction posterior pharynx if vomiting

Positive pressure ventilation with 100% oxygen:
2 person BVM ventilation, tight seal, high PEEP 20cm H2O

LARSONS MANOUVER + jaw thrust:
- firm pressure on posterior rami of mandible

DEEPEN SEDATION:
- propofol 1-2mg/kg IV

PARALYSE:
- suxemethonium 2mg/kg IV

INTUBATE:
- CHEST THRUST before passing the ETT through the cords

49
Q

Paediatric Blunt Trauma

EM rapid bombs - ep 321
Radiopedia - paediatric trauma

2021.2 Case based discussion

Paediatric patient injured by a car

Medical Expertise: Assessment – 30%
* Correctly identifies and interprets the results of an investigation within the scenario
* Creates a focused investigation plan that confirms or excludes time critical diagnoses
* Recognises signs on physical examination that indicate the patient is or at risk of imminent
deterioration.

Medical Expertise: Management – 30%
* Outlines an overall plan for resuscitating a patient
* Initiates treatments specific to identified severe abnormalities in circulation
* Creates an appropriate ongoing reassessment and management plan
* Adapts and initiates standard therapies to that patient, including drugs, fluids, gases, and
monitoring.

Prioritisation and Decision Making – 40%
* Highlights high-risk features identified during initial patient assessment
* Provides a rationale to explain and justify decisions about ongoing treatment
* Explains the specific benefits and risks of a treatment modality
* Provides an appropriate list of advantages and disadvantages (pros and cons) to explain
decisions made.

Candidates were required to:
* interpret the X-ray in the context of the patient’s presentation
* answer questions from the examiner regarding progression of the case.

A

Pulmonary contusion is the most common pulmonary injury

deflating the stomach to improve breathing

Use POCUS for pneumothorax and tamponade

Small pneumothorax are usually managed conservatively

deflate the stomach before chest tube placement

use pigtail drain - useful in pneumo and haemothorax,

CT

50
Q

Paediatric Blunt Trauma

EM rapid bombs ep 131 blunt abdominal trauma

2022.2 Case based discussion

Rural hospital. Young child who has been injured in a fall from a tree.

Medical Expertise: Investigation – 30%
- Recognises signs on physical examination that indicate the patient is or at risk of imminent
deterioration
- Creates a focused investigation plan that confirms or excludes time critical diagnoses
- Explains the reasons for selecting those tests in that investigation plan
- Analyses the results of an investigation to identify relevant findings or patterns
- Correctly interprets the results of an investigation within the scenario.

Medical Expertise: Management – 30%
- Correctly chooses time critical interventions based on assessment
- Initiates treatments specific to identified severe abnormalities in circulation
- Outlines an overall plan for ongoing treatment of a patient
- Adapts and initiates standard therapies to that patient, including drugs, fluids, gases, and
monitoring
- Creates an appropriate ongoing reassessment and management plan
- Appropriately uses procedural and monitoring equipment when describing procedural
sedation of a child
- Consults/refers to inpatient team urgently.

Prioritisation and Decision Making – 40%
- Highlights high-risk features identified during initial patient assessment
- Prioritises a differential diagnosis list to determine the most likely diagnoses in a patient
- Summarises and prioritises the key issues that must be addressed during and following the
emergency encounter
- Creates a safe and clear admission plan for a patient
- Justifies investigation selection by linking the pre-test probability, risk-benefit ratio and
availability within a hospital of that investigation to the patient’s need for that investigation
- Links clinical information and evidence-based practice to explain decisions made
- Provides a rationale to explain and justify decisions about ongoing assessment, treatment
and disposition decisions
- Provides an appropriate list of advantages and disadvantages (pros and cons) to explain
decisions made.

Candidates were required to interact with the examiner to:
- discuss their initial approach to the investigation and management of the patient
- answer questions from the examiner regarding the progress of the case.

A

PAEDIATRIC TRAUMA:

Be mindful of non-accidental trauma

deflating stomach with NG tube can optimise ventilation and should be done before chest tube placement

PREPARATION:

trauma call - surgeon

weight = age + 4 x2
ETT = age/4 + 3.5 (cuffed ETT)

ketamine 2mg/kg
rocuronium 1.2mg/kg
fentanyl 3mcg/kg
atropine 20mcg/kg
tranexamic acid 15mg/kg

venous access - have IO’s ready

pelvic binder or sheet laid down

warmed crystalloids

warm the room, have warm blankets

ASSESSMENT:
Immobilise cervical spine - manual in line stabilisation

GCS or AVPU

Haemodynamic instability:
- hypotension is a late sign because children compensate well. hypotension indicates >30% of blood volume loss
Signs of compensated shock
- tachycardia
- slow capillary refill
- cool peripheries
- mottled skin
- altered mental status

  • respiratory distress
  • tachypnoea
  • hypoxia
  • Temp
  • BSL

eFAST
- highly sensitive for pneumothorax
- poorly sensitive for abdominal injuries in children

INVESTIGATIONS:

VBG - metabolic acidosis, lactic acidosis in shocked state
FBC - Hb, platelets
Group and screen - blood transfusion
UEC - end organ perfusion
LFT - end organ perfusion (signs of solid organ injury)
lipase - trauma induced pancreatitis
full coagulation panel - DIC, coagulopathy
fibrinogen
bhcg in females of childbearing age

CXR

CT abdomen:
- concerning history (handle bar)
- abdominal tenderness, peritonism, seat belt or handle bar sign, abdominal wall bruising
- transaminitis
- gross haematuria
- positive fast scan

MANAGEMENT:
Immobilise cervical spine - manual in line stabilisation (poorly fitting soft collars can cause more harm)

Airway & Breathing:
- OPA to get tongue of the way
- jaw thrust
- towel under the shoulder to prevent hyperflexion
- oxygen 15L NRBM + 15L NP target SaO2 100%

IV access or early IO
2x IO into tibia
will need definitive IV access once resuscitated
large bore IV or central venous access

fluid resuscitate - warmed crystalloid 10ml/kg IV target BP 90-100
NOT for permissive hypotension
(early blood in decompensated shock)

Tranexamic acid 15mg/kg IV

Analgesia - fentanyl 0.5 - 1mcg/kg IV

Check BSL - treat hypoglycemia with 2ml/kg 10% dextrose target BSL 4-7mmol/L

Keep warm:
- reduce exposure
- warm blankets
- check temp Q1h target Temp 37

early NG tube placement - deflating stomach

Family presence:
- reduces stress and anxiety
- they can help with history and child co-operation
- designate staff member to accompany them

MASSIVE HAEMORRHAGE PROTOCOL:

Trigger - haemodynamic instability despite fluid resuscitation, decompensated shock

Team:
- blood bank
- haematologist
- surgeon

Tranexamic acid 15mg/kg IV

Testing
Q1h
FBC - Hb, platelets
VBG - lactate, ionised calcium
full coagulation panel
fibrinogen

Targets - haematological + metabolic:
- Hb >80
- platelets >50
- ionised calcium >1.1 (calcium gluconate 0.5ml/kg max 20ml)
- fibrinogen >1.5 (cryoprecipitate 10ml/kg IV)
- INR <1.5
- lactate < 4
- pH >7.2

MTP RBC: FFP: platelets 1:1:1 ratio

Normoglycemia target 4-10mmol/L

Temperature 36-37

INTUBATION:

Haemodynamically challenging:
- shocked state
- metabolic acidosis
- increased vagal response to laryngoscopy

Anatomically challenging:
- cervical spine immobilisation
- large tongue and epiglottis

Preoxygenation
- 15L NP + 15L NRBM for at least 3min
- target SaO2 >95%
- allow adequate preoxygenation time (3-5min). children have low FRC and shorter safe apnoea times

Optimise:
- sufficient fluid resuscitation
- NG tube placement for stomach decompression for full diaphragmatic excursion

Position:
- towel under shoulders
- tragus in line with sternal notch
- face parallel with ceiling

straight blade laryngoscope - move epiglottis out of the way

video laryngoscope + bougie

ETT size = age/4 + 3.5

most experienced operator

Sedation:
- ketamine 1mg/kg IV

Paralytic:
- rocuronium 1.2mg/kg IV

Atropine 20mcg/kg IV encase of bradycardia

POST INTUBATION:

sedation - propofol + fentanyl

vasopressors - adrenaline infusion 0.01-1mcg/kg/min

CXR - confirm ETT in carina and not Right mainstem bronchus, check NGT below diaphragm

TRANSFER TO TRAUMA CENTRE
- identified and addressed life threats
- Intubated, ETT secure, sedation and paralysis
- EtCO2 monitor
- invasive BP monitoring
- IDC
- analgesia
- 2x vascular access
- blood products

51
Q

Paediatric Blunt Abdominal Trauma

EM rapid bombs ep 131

2023.2 Case based discussion

discuss with an examiner, the assessment and management of a child with a serious abdominal injury, as well as discuss issues around consent for therapy.

Medical Expertise: Assessment and Diagnosis: Investigations – 20%
- Analyse imaging results to identify relevant findings or patterns.
- Correctly interpret the imaging results within the scenario.
Medical Expertise: Management:

Treatment including resuscitation – 40%
- Outline an overall plan for resuscitating a paediatric patient.
- Initiate treatments specific to identified abnormalities in circulation.

Professionalism – 40%
- Describe how ethical principles are applied in clinical practice with cultural differences
(including religious aspects).
- Balance respect for patient autonomy with best clinical practice in patient encounters.
- Describe how medico-legal frameworks and principles are applied in the care of children.

Candidates were required to interact with the examiner and to:
- Describe and interpret the CT for this patient.
- Answer Any further questions from the examiner.

A
  • always consider non-accidental injury
  • Relatively large organs to body size
  • Less abdominal wall musculature and subcutaneous fat means less protection of intra-abdominal solid organs
  • Compensate for hypovolemia / hemorrhage very well.
  • Can maintain blood pressure even up to 30% blood volume loss.
  • Hypotension is a late clinical indicator of hemorrhage.
  • Signs of compensated shock - tachycardia, cold peripheries, slow CRT, mottled skin, altered mental status

CT abdomen not always necessary
- have to weigh up the risk of missed injury vs. adverse effects
- serial abdominal exams
- monitoring of vital signs for decompensated shock
- serial FAST exam
- LFT’s transaminitis AST >200 ALT >125
- haematuria
- falling haematocrit

Majority of solid organ injury are managed conservatively

If positive FAST - discuss with surgeons need for CT or laparotomy

Interventions:
- laparotomy
- interventional radiology + embolization
- blood transfusion

IONIZING RADIATION
CT scans in childhood do increase lifetime incidence of cancer
- more radiosensitive tissue
- more time to develop cancer after insult

52
Q

Ataxia - Cerebellar Exam

2021.1 Examination station

Youtube - cerebellar exam stanford medicine 25

Adult with unsteady GAIT (cerebellar dysfunction)

1) list differential diagnoses
2) perform the cerebellar neurological exam
3) outline investigations with explanation

Dyssynergia (breakdown of movements into parts),

dysmetria (inaccurate fine movements),

dysdiadochokinesia (clumsy rapid movements) may indicate a lateral cerebellar lesion.

A

Watch Geeky medics cerebellar exam video

DIFFERENTIAL DIAGNOSES:

Toxins:
- Alcohol and sedatives
- Antiepileptics (lithium, phenytoin, carbarmazepines, sodium valproate)

Vascular:
- cerebellar stroke (ischemic or haemorrhagic)
- cerebral venous sinus thrombosis

Trauma/Non-accidental injury
- ICH

Metabolic:
- Wernicke’s encephalopathy
- Hyponatremia
- Vitamin B12 deficiency

Malignancy
- Posterior fossa tumours (astrocytoma, haemangioblastoma)
- Metastatic disease

Infection
- Cerebellar abscess
- Meningitis
- Post infectious ataxia

Environmental:
- Extreme hyperthermia

Autoimmune:
- Multiple sclerosis

Neurological:
- basilar artery migraine
- hydrocephalus

EXAMINATION:

Testing co-ordination

Level of alertness - GCS
Altered mentation
Essential tremors

Signs of uncal herniation:
- ispilateral blown pupil (occulomotor nerve compression)
- contralateral hemiparesis

Vital signs:
- fever (suggest infective cause)
- meningism

TRUNCAL ATAXIA:
- observe in sitting position
- do they sway or need help sitting upright

STANDING BALANCE:
- observe stance and balance
- feet together

GAIT ASSESSMENT:
- walk, turn around and walk back
- walk on heels and toes
? broad based GAIT
? high stepping GAIT
? staggering GAIT

TANDEM GAIT ASSESSMENT:

ROMBERG’S TEST: proprioception rather than cerebellar function

CO-ORDINATION:

FINGER NOSE TEST:
?dysmetria
?intention tremor
past pointing in cerebellar disease

DYSDIADOCHOKINESIA
- impaired rapid alternating movements

REBOUND PHENOMENA

HEEL SHIN TEST:
?lower limb dysmetria

SPEECH:
- Ataxic dysarthria or slurred staccato speech
“baby hippopotamus”

NYSTAGMUS
- gaze provoked nystagmus

TONE:
- hypotonia in cerebellar disease

REFLEXES:
- hyporeflexia in cerebellar disease

INVESTIGATIONS:

BSL

FBC - megaloblastic anaemia
B12 levels

Electrolytes - hyponatremia

Anti-epileptic levels

Ethanol/ethylene glycol

LP - after neuroimaging

Non - contrast CT brain
- hydrocephalus
- haemorrhage

CT angiography head and neck
- vertebral artery dissection
- basilar artery thrombosis

CT venogram
- cerebral venous sinus thrombosis

MRI
- tumours
- surgical planning

53
Q

History Taking - Back Pain

EM rapid bombs - epidural spinal abscess ep 229
EM rapid bombs - back pain ep 434

2023.1 - lower back pain after an injury
2022.1 - lower back pain

Take a focussed history
Interpret examination findings given
List differential diagnoses
Outline investigations with justification

DIFFERENTIAL DIAGNOSIS:

Intervertebral disc prolapse
Degenerative arthritis
Spinal stenosis

Inflammatory:
- Transverse myelitis
- Ankylosing spondylitis
- Rheumatoid arthritis

Infection:
- discitis
- osteomyelitis
- epidural abscess

Congenital:
- kyphosis
- scoliosis

Haematological:
- sickle cell crisis
- bleeding tendency causing epidural haematoma

Malignancy - spinal tumour

Vertebral fracture:
- pathological
- traumatic

Vascular:
- spinal cord infarction

Referred pain from abdominal pathology:
- AAA
- Renal colic
- UTI/pyelonephritis
- Prostatitis
- Pancreatitis
- Mesenteric ischemia
- PID
- Endometriosis

Referred pain from hips/pelvis
- arthritis
- occult fracture

RED FLAGS:
- pain worse at night lying supine
- fevers, weight loss, night sweats
- malignancy
- current infections (cellulitis, pneumoniae, UTI) homogenous spread
- immunesuppression (diabetes, HIV, transplant)
- immune suppression medication
- IVDU
- trauma
- chronic corticosteroids
- osteoporosis
- inflammatory arthritis
- saddle anaesthesia
- weakness, numbness or tingling in legs
- bowel or bladder dysfunction

YELLOW FLAGS:
- Belief that pain and activity are harmful
- Sickness behaviours
- Low or negative moods, mental illness
- Treatment that does not fit with best practice
- Problems with compensation system
- Previous history of back pain with time off work
- Problems at work, poor job satisfaction
- Overprotective family or lack of social support

A

History:

“can you tell me about the issues you’ve been experiencing”

Site -
Onset -
Character -
Radiation -
Alleviating & Aggrevating factors (worse pain at night and lying down)
Progression -
Response to analgesia -

Key Symptoms:

  • numbness, tingling or weakness
  • urinary incontinence or retention
  • bowel incontinence or constipation
  • fever
  • infections (cellulitis, UTI, LRTI’s)
  • constitutional symptoms (lethargy, weight loss, night sweats)
  • trauma or injury
  • malignancy
  • previous spinal surgery
  • IVDU
  • medications - prolonged steroid use, analgesia, anticoagulation
  • family history of arthritis (ankylosing spondylitis)
  • impact on employment and daily activities

Dysuria (pyelonephritis, UTI)
AAA

Pain relief and pain management strategies

PMHX:
*Malignancy
*Osteoporosis
*Inflammatory arthritis - ankylosing spondylitis
*Bleeding disorder - epidural haematoma
*Clotting disorder - spinal infarction
*AAA

MEDICATIONS:

*chronic steroids - osteoporosis & immune compromise

*anticoagulation - epidural haematoma

*Immune suppression - azathioprine

SOCIAL:
*work - problems at work, sick days, poor job satifaction
*activities of daily living
*mental health - depression, suicidal ideation
*social supports
*IVDU
*alcohol
*smoking

54
Q

Communication - Back pain

2021.1 - communication with patient - low back pain, no red flags
- discuss disposition and plan

Communication – 50%
* Develops rapport
* Uses appropriate communication techniques
* Appropriately responds to the patient
Prioritisation and Decision Making – 30%
Assessing clinical risk:
* Explains risk stratification
Justifying decision making:
* Explains benefits and risks of investigation modality
* Justifies investigation selection
* Links clinical information and evidence-based medicine to explain decisions
Medical Expertise – 20%
* Creates an appropriate management plan
* Creates an appropriate follow up plan with safety net
Candidates were required to:
* explore and respond to the patient’s (role player) questions
* explain to the patient the appropriate management for his condition.

A

COUNSELLING:

Back pain is very common

Only a very small proportion have a serious diagnosis

pain and activity is not harmful to your spine

*not for imaging in ED - cause harm
Radiation exposure:
- Xrays lumbar spine - equivalent to having 100 chest xrays

  • CT lumbar spine - equivalent to having 500 chest xrays
  • Overdiagnosis

*Trial analgesia and physiotherapy and if ongoing pain can discuss with GP about having an outpatient MRI

Non-pharmacological:
- get up and get moving
- physiotherapy referral - tailored exercise plan
- massage
Psychologist referral

Pharmacological:
- paracetamol 1g QID
- short course NSAID ibuprofen 400mg TDS (commence PPI)
- short course of oxycodone (advise prn, highly addictive, don’t mix with alcohol, cannot operate heavy machinery)

*Patients with spinal stenosis or disc prolapse with severe radicular pain may hand inpatient CT guided injections (lumber and sacral nerve root and facet joint injections)

55
Q

Q421 - Rapid Bomb Blitz Part 6 - Neurology focus

A