Lesson 8- Biological Approach- Influence of biological structures & neurochemistry on behaviour πβββ Flashcards
What are biological structures?
Biological structures- biological structures are organs (e.g. π§ ) & systems (e.g. nervous system) that influence human πβββ
What are the 2 main biological structures?
1) Brain
2) Nervous System & Neurons
What is the Nervous system comprised of?
Nervous System & Neurons:
- CNS (central nervous system)- consists of π§ & spinal cord- controls breathing & π rate
- PNS (peripheral nervous system)- sends & receives messages from CNS to other parts of body
- Neurons- transmit nerve impulses in the form of electricalβ‘οΈ signals
What is the brain comprised of?
Made up of 4 lobes- see image β‘οΈ:
Parietal- processes info about temp, taste π, touchβοΈ & movement
Frontal- important for cognitive π§ functions & control of voluntary movement/activity
Temporal- processes memories, integrating them with sensations of taste π , sound π, sight π & touchβοΈ
Occipital- primarily responsible for vision π
Cerebrum- makes up 85% of π§
Cerebral cortex- covers cerebrum & is responsible for high β¬οΈ order thinking π€ & language
What are the evaluation points for biological structures as an explanation of behaviour?
π- biological model criticised- applicable at explaining causes of some πβββ BUT βοΈ others- infection of π§ can-> schizophrenia BUT phobias usually learnt rather than due to π§ malfunction
π- study of π§ relies on use of scans- e.g. PET (Positron Emission Tomography) & MRI (Magnetic Resonance Imaging) & post mortem studies add to scientific π§ͺ evidence- helps support biological approach- 1 of the most scientific π§ͺ models/approaches when compared to π§ & πβββ approach
π- psychologists still βοΈ know 100% about π§ & its functions- β¦ potential problems of trying to identify which part of π§ responsible for certain πβββ
π- scientifically & objectively investigated in order to test how π§ structures might affect πβββ- e.g. use of FMRI & PET scanning gives psychologists understanding of how π§ functions
Why might schizophrenia develop?
Schizophrenia- might develop due to structural & functional π§ abnormalities
What did they do in the past to investigate patients with schizophrenia?
PAST- used post mortemβs to investigate structure of π§ in patients who had schizophrenia
What do they do now to investigate patients with schizophrenia?
NOW- use fMRI (Functional Magnetic Resonance Imaging)- shows images of π§ in action using magnetic 𧲠& radio π» waves
NOW- patients with schizophrenia given cognitive π§ & memory tasks to do- compared with normal healthy patients whilst also conducting fMRI
Which psychologist did a study about brain structure?
Brain π§ structure- Neural correlates- study by Swayze
What method was used for Swayzeβs neural correlates brain study?
Swayze reviewed 50 studies of schizophrenic patients & examined their π§ imaging using MRI (Magnetic Resonance Imaging)
What were the findings for Swayzeβs neural correlates brain study?
From these brain images- structure of π§ examined & found that schizophrenic patientsβ have structural abnormalities in their π§ including:
- Decreased β¬οΈ brain weight
- Larger β¬οΈ ventricles (that are filled with water)
- Smaller β¬οΈ hypothalamus
- Less β¬οΈ grey matter (intelligence held here & β¦ seems to have deteriorated)
- Structural abnormalities in pre-frontal cortex (where personality held)
What were the evaluation points for Swayzeβs neural correlates brain study?
π- Andreason (1982)- criticised neural correlates explanation for schizophrenia- found that extent to which ventricles in π§ enlarged by in schizophrenic patients βοΈ significant & β¦ very small difference between neural correlates of schizophrenic patientsβ & normal people β¦ neural correlates might βοΈ be a major factor in causing schizophrenia
π- supporting psychological evidence- states that schizophrenia caused by neural correlates changing in π§ - occur during pre-natal development in womb & why schizophrenia occurs in early adulthood explained by Weinberger (1987)- pre-frontal cortex (area effected by schizophrenia) develops during adolescence & therefore damage to this part of π§ only noticed during adolescence & adulthood (when symptoms of schizophrenia become apparent)
π- cause & effect βοΈ established- abnormal neural correlates cause schizophrenia or schizophrenia occur 1st (due to other factors) & then cause π§ structure/neural correlates to alter?
What are neurotransmitters and what are its roles in the body?
Neurotransmitters- π§ chemicals that communicate info throughout π§ & body- relay signals between nerve cells called neurons
- Nerve impulse reaches end of a neuron & neurotransmitter released- travels from 1 neuron to the next across synapse
What is dopamine and what is its role in the body?
Dopamine- neurotransmitter released by nerve cells to send signals to other nerve cells
- plays major role in motivational component of reward-motivated πβββ
- expectation of rewards β¬οΈ dopamine in π§
- involved in motor control & in controlling release of various hormones
What are excitatory neurotransmitters?
Excitatory neurotransmitters- trigger nerve impulses in receiving neuron & stimulate π§ activity e.g. β¬οΈ levels of dopamine β¬οΈ drive & motivation β¦ dopamine excitatory neuron
What are inhibitory neurotransmitters?
Inhibitory neurotransmitters- hinder nerve impulses & calm the person, these are called, e.g. serotonin βοΈ stimulate π§
What is the endocrine system?
Endocrine system- responsible for producing hormones in body
- Consists of ductless glands that release hormones into body that affect πβββ
What are the evaluation points for neurochemistry/neurotransmitters and their affect of behaviour?
π- criticised by π§ approach- states that πβββ caused by thought patterns & π€ rather than neurochemistry/chemicals (neurotransmitters) e.g. depression caused by β π€ according to π§ approach rather than by β¬οΈ serotonin
π- Neurotransmitters- measured objectively & scientifically which is a strength- e.g. dopamine measured by inserting needle into spine & extracting spinal plasma fluid- scientific π§ͺ measurements of neurotransmitters-> accurate findings
π- cause & effect βοΈ established- β¬οΈ/β¬οΈ levels of neurotransmitters cause a change in πβββ or does πβββ change-> β¬οΈ/β¬οΈ in neurotransmitters
π- better treatment- if we know lack/excess of particular neurotransmitter-> change in πβββ the we can give people suitable to treatment to fix neurotransmitter imbalances e.g. β¬οΈ levels of serotonin-> depression β¦ give depressed ppl SSRI drugs which β¬οΈ serotonin
Who studied neurochemistry & schizophrenia further to come up with the dopamine hypothesis?
Davis & Neale
What are the levels of dopamine like in schizophrenic patients?
Schizophrenic patients have β¬οΈ levels of neurotransmitter dopamine in π§ -> β¬οΈ firing of neurons
What drugs can be given to schizophrenic patients to reduce dopamine levels? ALSO how are dopamine levels reduced & what are the effects?
When drugs e.g. phenothiazines given to patientsβ that block dopamine at synapses in π§ - symptoms of schizophrenia β¬οΈ (hallucinations & delusions)
What drug can increase the levels of dopamine and what are the effects of it in schizophrenic patients?
Drug called L-Dopa β¬οΈ dopamine levels in π§ -> symptoms of schizophrenia
How do the amount of dopamine receptors & dopamine in schizophrenic patients compare to normal people & where is brain activity greatest?
Post mortem studies found that schizophrenic patientsβ have β¬οΈ amount of dopamine receptors & dopamine in left amygdala of π§
What is dopamine metabolism like in schizophrenic patients & how is this monitored?
Dopamine metabolism in schizophrenic patients ALSO abnormal- monitored by PET scans (Positron Emission Tomography)
What are the evaluation points of the dopamine hypothesis & its effect on behaviour?
π- Cause & effect βοΈ clear- does β¬οΈ in dopamine-> schizophrenia OR does schizophrenia develop (due to another cause) -> β¬οΈ in dopamine β¦ difficult to identify whether β¬οΈ dopamine causes schizophrenia
π- research support- Davidson found that when schizophrenic patients given drug L-Dopa (β¬οΈ dopamine)- their schizophrenic symptoms got worse & intensified- β¦ seems that dopamine has key role to play when examining causes symptoms of schizophrenia
π- reductionist- reduces complicated causes of schizophrenia down to component of dopamine alone- potential other factors/causes of schizophrenia ignored
π- scientific π§ͺ evidence & support- research used evidence from π§ scans (PET & fMRI)-> β¬οΈ valid & reliable info- π§ͺ & objective data helps psychologists look at root cause of schizophrenia