Lecture 9-Obesity Flashcards

1
Q

define obesity- WHO

A

excess fat accumulation in adipose tissue- extent that health may be impaired

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2
Q

BMI

A

weight in kg/height in metres squared

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3
Q

BMI for overweight and obesity

A

overweight- 25-30
obesity- >30 (1)
class II- >35
class III- >40

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4
Q

limitations of BMI

A

doesn’t take into account muscle mass at individual level, not at population level

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5
Q

why is waist circumference measured?

A

metabolically active fat

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6
Q

double burden

A

low and middle income countries- overnutrition alongside undernutrition

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7
Q

nutrition transition

A

obesity common from women–>men
higher socioeconomic status –>low socio economic status
more prevalence in children

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8
Q

global prevalence of obesity

A

increase started in rich world but now everywhere except sub saharan africa

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9
Q

australian adults obesity prevalence peaks?

A

at 55-64 then decreases

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10
Q

healthy survivor effect

A

people who are obese throughout their life not making it into older years

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11
Q

percentage of overweight/obese adults in australia

A

62.8%- normal to be overweight

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12
Q

prevalence of obesity gradual or sudden?

A

steady gradual increase not sudden

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13
Q

3.5 year old victorian children

A

prevalence of overweight/obesity decreased

- positive effects, first generation to come out

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14
Q

socio-economic differences in obesity

A

higher prevalence for most disadvantaged (especially women), decreases as you get to least disadvantaged

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15
Q

obesity and self-reported chronic diseases prevalence

A
  1. diseases of circulatory system
  2. mental/behavioural problems
  3. T2D
  4. IHD
  5. cerebrovascular disease
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16
Q

hypertension and obesity

A

triple for obese people compared to underweight

17
Q

dyslipidemia

A

almost double for obese compared to underweight

18
Q

type 2 diabetes and obesity

A

quite uncommon to have T2D in healthy weight,
quite common if you are obese
-higher prevalence in obese men compared to women

19
Q

biomarkers to diagnose T2D

A

fasting plasma glucose

HbA1C level

20
Q

obesity and economic consequences

A

productivity losses
health care costs
decreased quality of life
- costs borne both by individual and society

21
Q

indirect costs

A

absenteeism, government subsidies

22
Q

direct costs

A

GPs, health services, weight loss interventions, pharmaceuticals

23
Q

4 explanations for obesity epidemic

A
  1. energy imbalance
  2. genes and environment
  3. potential influences on population prevalence
  4. foresight causal map
24
Q

energy imbalance

A

level of individual

  • overnutrition and underactivity
  • NOT driven us to be obese society (need to see how its changed in last 30 years)
25
Q

genes interacting with environment

A

certain genes make more susceptible to obesity in certain environment
-NOT driven obese society

26
Q

potential influences on population prevalence

A

international factors–>national/regional–>community locality–>work/school/home–>individual

e. g. public transport, manufactured/imported food- community
e. g. globalisation of markets, media–>international

27
Q

foresight causal map

A

centre is energy balance

  • around drivers of obesity
    e. g. food production/consumption, physical activity, social/ individual psychology
28
Q

3 levels of obesity prevention

A
  1. universal/public health/primary intervention
  2. selective prevention
  3. targeted prevention
29
Q

public health/ primary intervention

A

prevent weight gain across population- all members of community

30
Q

selective prevention

A

identify groups/individuals at risk, prevent further weight gain, promote weight loss

31
Q

targeted prevention

A

identify those with existing weight problems, prevent downstream consequences e.g. diseases

32
Q

select committee into obesity- children in most points

2 reasons

A
  • primary prevention, childrens rates not yet high in australia
  • politically palatable- children are innocent, help them
33
Q

approaches to addressing obesity

agent–>structural

A

agent- individual makes choice

structural- individual choice removed e.g. school canteen

34
Q

approaches to addressing obesity

micro–>macro

A

micro- schools, worksites, homes

macro- national, state, community e.g. sugar tax

35
Q

community based interventions

A
  • healthy choices easy choices, healthier community environment, tailored to local context
    time frames- need at least 3 years to see effect
  • prevention measured in generations
36
Q

systems based community interventions

A

importance of linkages, relationships, feedback loops, interactions amongst systems parts

  • identify elements of community system that promote or prevent obesity
  • points of intervention
  • use existing resources to combat
  • feedback, shared understanding
37
Q

policy interventions

A

most policies happen outside health care system e.g. transport, taxation, education
-focus on government policy at all 3 levels plus international agreements

38
Q

sugar tax

A

reformulation to drive food companies to change what’s in their products

39
Q

8 critical actions

A
  1. toughen TV restriction
  2. food reformulation targets
  3. health star ratings
  4. active transport
  5. public health education campaigns
  6. 20% health levy on sugar drinks
  7. national obesity taskforce
  8. monitor diet, physical activity, weight