Lecture 9: Integumentary System Flashcards

1
Q

Skin

A
  • largest organ in body
  • accounts for 15-20% of body weight
  • consists of 3 layers: epidermis, dermis, subcutaneous tissue
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2
Q

Function of Skin

A
  • protect underlying structures from external injury and harmful substances
  • primarily an insulator
  • holds organs together
  • sensory perception
  • fluid balance
  • temperature control
  • absorption of UV radiation
  • metabolism of vitamin D
  • synthesis of epidermal lipids
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3
Q

Signs and Symptoms of Skin Disease

A
  • pruritus
  • urticaria (hives): vascular action of skin resulting in elevated patches of skin called wheals
  • rash: general term for eruption on skin usually accompanied by itching, can be anything from erythema to macular lesions to raised papules
  • blisters (vesicle or bulla): fluid-containing elevated skin lesions with clear watery or bloody contents
  • ichthyosis: genetic skin disorder characterized by dry, rough, discolored skin with formation of scaly desquamation
  • xeroderma pigmentosum: autosomal recessive genetic disorder of DNA repair in which ability to repair damage caused by UV light is deficient
  • unusual spots, moles, cysts, fibromas, nodules, swelling, changes in nail beds
  • any unusual spot that has appeared recently or changed since initial appearance should be documented and brought to Dr.’s attention
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4
Q

SIFTT About Skin Lesions

A
  • blisters of unknown cause may be first sign of underlying malignancy requiring immediate medical evaluation
  • when examining and documenting presence of skin disorder note location, size, and any irregularities in skin color, temperature, moisture, ulceration, texture, thickness, mobility, edema, turgor, odor, and tenderness
  • note whether lesions are unilateral or bilateral, note whether they are symmetric or asymmetric, and note arrangement of lesions
  • blisters should be opened and debrided (with few exceptions); blister fluid impairs normal function of neutrophils and lymphocytes
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5
Q

Aging and Integumentary System

A
  • wound healing impaired in intrinsically aged skin as compared to young skin that the rate of healing is appreciably slower
  • decreased elasticity and blood flow
  • both structural and functional changes occur in skin: diminished pain perception, increased vulnerability to injury, decreased vascularity, weakened inflammatory response
  • stratum corneum layer (outermost layer of epidermis) becomes thinner and becomes more translucent
  • lesions must be examined by a physician
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6
Q

Common Skin Disorders: Acne

A
  • chronic inflammatory disease of sebaceous glands
  • usually associated with high rate of sebum secretion
  • sebum carries dead skin cells through follicles to skin surface
  • small hair grows through follicle out of the skin
  • pimples grow when these follicles get blocked, resulting in an accumulation of oil under the skin
  • pathogenesis: androgens stimulate sebum production which is secreted into dilated hair follicles that contain bacteria; bacteria secrete lipase which interacts with sebum to produce free fatty acids which provoke inflammation; hair follicles produce more keratin, which forms a plug with sebun in dilated follicle
  • clinical manifestations: a closed comedo (clogged hair follicle) or white head-covered by epidermis, an open comedo or blackhead-not covered by epidermis, inflammation, acne pustules
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7
Q

Common Skin Disorders: Atopic Dermatitis

A
  • chronic inflammatory skin disease, most common type of eczema, frequently present during first year of life
  • etiology, risk factors, and pathogenesis: rubbing and scratching of itchy skin are responsible for clinical changes seen; hands in and out of water makes it worse
  • CM: begins as red, oozing, crusting rash
  • chronic form results in dry, thickened, and brownish gray skin
  • xerosis (dryness) and pruritus are major symptoms
  • S. aureus is most common bacterial infection
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8
Q

SIFTT for Atopic Dermatitis

A
  • patient education to avoid factors that precipitate or exacerbate inflammation
  • daily care (hydration and lubrication) of skin including applications (two or three times daily) of emollients that occlude skin to prevent evaporation and retain moisture should be recommended
  • older clients should bathe with tepid water using nondrying, non-fragranced soap
  • emollients must be applied to body within 5 minutes after showering or bathing especially in dry weather to prevent further drying of skin
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9
Q

Common Skin Disorders: Contact Dermatitis

A
  • acute or chronic skin inflammation caused by exposure to a chemical, mechanical , physical or biological agent
  • commonly appears as sharply demarcated inflammation of skin
  • CM: intense pruritus, erythema, blistering, and edema
  • may progress to vesiculation, oozing, crusting, and scaling
  • clearly defined lesions
  • delayed hypersensitivity
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10
Q

SIFTT for Contact Dermatitis

A
  • always consider clients reactions to external substances
  • skin must always be examined before and after intervention
  • client should be instructed to report any discomfort or unusual findings during or after treatment to therapist
  • use of alcohol-based lubricants or soaps, antifungal or antibacterial soaps without a rinsing agent, lanolin should be avoided
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11
Q

Common Skin Disorders: Stasis Dermatitis

A
  • development of areas of very dry, thin skin
  • usually due to venous insufficiency (usually begins with edema of leg due to slow venous return)
  • itching, a feeling of heaviness, and brown stained skin
  • lesions are slow to heal due to lack of oxygenated blood
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12
Q

Common Skin Disorders: Rosacea

A
  • chronic facial disorder of middle aged and older people (form of acne)
  • benign: reveals rosy appearance marked by reddened skin on cheeks, nose, and chin
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13
Q

Bacterial Infectiosn

A
  • normally skin harbors variety of bacterial flora, including major pathogenic varieties of staph and strep-usually enter skin via abrasions or puncture wounds
  • follicular lesions should not be squeezed because this will not hasten resolution of infection and may increase risk of making lesion worse or spreading infection
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14
Q

Impetigo

A
  • superficial skin infection commonly caused by staph or strep
  • usually found in infants, kids 2-5 and older people
  • close contact in living quarters, poor hygiene, malnutrition, etc
  • can be spread by direct contact or arthropod vector
  • presents as small macules rapidly developing into vesicles that become pustular
  • scratching spreads infection, a process called auto-inoculation
  • lesions leave depigmented areas
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15
Q

Cellulitis

A
  • infection of dermis or subcutaneous tissues that spreads widely through tissue spaces
  • not contagious
  • RF: diabetes, immunodeficiency, impaired circulation, neuropathy
  • patho: streptococcus pyogenes (GAS) or staphylococci are usual cause although others may be responsible; bacteria overwhelm defensive cells
  • usually occurs in loose tissue beneath skin, but may also occur in tissues beneath mucous membranes or around muscle bundles
  • CM: makes skin look erythematous and edematous, pain and warmth at site, fever
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16
Q

Warts

A
  • aka verrucae
  • common benign viral infection of skin and adjacent mucous membranes caused by HPVs
  • transmission is probably through direct contact
  • some respond to simple Rx and some disappear spontaneously
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17
Q

Fungal Infections (Dermatophytosis)

A
  • superficial infections by fungi that live on, not in, skin

- will spread without Rx

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18
Q

Ringworm (tinea corporis)

A
  • fungal infections of hair, skin, or nails-designated by latin word tinea
  • no association with worms but rather is marked by formation of ring-shaped pigmented patches covered with vesicles or scales that often become itchy
  • transmission can occur directly through contact with infected lesions or indirectly through fomites
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19
Q

Athletes Foot (tinea pedis)

A
  • causes erythema, skin peeling, and pruritus between toes that may spread from interdigital spaces to sole
  • may have strong odor as well
  • causes severe itching and pain on walking
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20
Q

SIFTT of Athletes Foot

A
  • client may consider nuisance or minimal problem that doesn’t require medical attention, it can be entry point for bacterial infections, especially in older adults
  • keeping it under control is important way to prevent cellulitis and especially in diabetics
21
Q

Scabies

A
  • parasitic infection
  • highly contagious skin eruption caused by a mite
  • female mite burrows into skin and deposits eggs that hatch into larvae in a few days
  • easily transmitted by skin-skin contact or fomites
  • sx: intense pruritus (from excreta deposits of mites), usually excoriated skin, the burrow-linear ridge with vesicle at one end (mite found in burrow)
22
Q

SIFTT for Scabies

A
  • wear gloves when touching affected person and a gown
  • gas autoclave blood pressure cuffs or other equipment used with affected person before using them on other people
  • treatment area must be thoroughly disinfected after each session
23
Q

Pediculosis (Lousiness)

A
  • parasitic infection
  • infestation of scalp, trunk, or pubic area by lice
  • transmission from person to person usually via shared personal items or furniture
  • parasite attaches to hair shaft with claws and feeds on blood
  • intense itching, dermatitis, inflammation
  • not carried or transmitted by pets
  • anyone can get it regardless of age or status
24
Q

Skin Cancer

A
  • nearly all white people older than 65 will get some form
  • exposure to solar radiation causes most skin cancers (UVB)
  • protection from sun during first 2 decades of life significantly reduces risk of skin cancer
25
Q

Benign Tumors of Skin Cancer

A
  • seborrheic keratosis: proliferation of basal cells presenting as multiple lesions
  • nevi (moles): pigmented or non-pigmented lesions that form from melanocytes; seldom transition to malignant melanoma
26
Q

Precancerous Conditions for Skin Cancer

A
  • actinic keratosis (solar keratosis): skin disease resulting from years of exposure to sun’s UV rays; well defined crusty or sandpaper-like patch or bump, affects nearly 100% of older caucasian population
  • bowen’s disease: presents as persistent brownish, scaly plaque with well-defined margins
27
Q

Malignant Neoplasms

A
  • basal cell carcinoma
  • squamous cell carcinoma
  • malignant melanoma
  • kaposi’s sarcoma (KS)
28
Q

Basal Cell Carcinoma

A
  • slow-growing surface epithelia skin tumor originating from undifferentiated basal cells contained in epidermis
  • rarely metastasizes beyond skin
  • does not invade blood or lymph vessels, but can cause significant local destruction
  • most common malignant tumor affecting caucasians
  • mohs micrographic sx
  • may present as crusty, open sore that will not heal-can appear as a shiny, pearly, or translucent pink, red, or white bump
  • if left untreated it may slowly destroy surrounding tissue
29
Q

Squamous Cell Carcinoma

A
  • second most common skin cancer in white people
  • tumor of epidermal keratinocytes and rarely occurs in dark-skinned people
  • persistent scaly, red patch that sometimes crusts or bleeds or an open sore that does not heal
  • may also present as raised or wart-like growth that may bleed
30
Q

Malignant Melanoma

A
  • neoplasm of skin originating from melanocytes or cells that synthesize melanin
  • majority appear to be associated with intensity rather than duration of sunlight exposure
  • can appear anywhere on body, not just exposed areas
  • more serious problem than other skin cancers, because it can spread quickly and insidiously, becoming life-threatening at earlier stage of development
  • 100% curable if detected early
31
Q

SIFTT for Malignant Melanoma

A
  • during observation and inspection of any client, therapist should be alert to potential signs of skin cancer
  • look for abnormal spots, especially in sun exposed areas, that are rough in textuer, persistently present, and bleed on minimal contact or with minimal friction
  • any change in wart or mole should be inspected by physician
  • ABCDE method
  • A: asymmetry-uneven edges, lopsided, half unlike other
  • B: border-irregularity, edges scalloped or poorly defined edges
  • C: color-black, shades of brown, red, white, pin, occasionally blue
  • D: diameter-larger than a pencil eraser
  • E: elevated or enlarging lesion
32
Q

Risk Factors for Malignant Melanoma

A
  • excessive exposure to sunlight
  • increased nevi
  • tendency to freckle from sun
  • family hx
  • red hair, fair skin, blue eyes
33
Q

Kaposi’s Sarcoma (KS)

A
  • malignancy of vascular tissue that presents as a skin disorder caused by herpes virus infection
  • classic KS occurs commonly on LEs and affected areas are red, purple, or dark blue macules
  • lesions may spread by metastasis to face and oral mucosa
34
Q

Psoriasis

A
  • associated with immune dysfunction
  • chronic, inherited, recurrent inflammatory, but non infectious dermatosis characterized by well-defined erythematous plaques covered with a silvery scale
  • turnover time for skin is 3-4 days vs. 26-28 days for normal skin
  • corticosteroids usually prescribed
35
Q

SIFTT for Psoriasis

A
  • teaching relaxation techniques and stress management should be encouraged daily, especially during periods of exacerbation
  • can result in psychological problems because skin lesions may cause person to feel contagious and untouchable
  • instruction and direct intervention should emphasize…
  • using gloves, steroid cream application in thin film, rubbed gently with downward motion until it disappears
  • medication should be applied only to affected lesions avoiding contact with normal surrounding skin
36
Q

Thermal Injuries: Frostbite

A
  • overexposure to cold are or water
  • results from prolonged exposure to dry temperatures far below freezing
  • patho: many biologic reactions and pathways become slowed
  • low body shell temperature can interfere with athletic ability: weakening and slowing muscle contractions, delaying nerve conduction time
  • when tissue temp drops to 2* C ice crystals form in tissues and expand extracellular spaces resulting in localized cold injuries
  • with compression of cells, membranes rupture, interrupting enzymatic and metabolic activities
  • with thawing you get release of histamine increasing capillary permeability, aggregation of RBCs and microvascular occlusion
  • may be deep: extends beyond subcutaneous tissue; usually affects hands or feet; skin becomes white until thawed then turns purplish blue, pain, blisters, tissue necrosis, gangrene
  • superficial: affects skin and subcutaneous tissue; will cause burning, tingling, numbness, swelling, mottled blue-gray skin color
37
Q

Medical Management for Localized Cold Injury

A
  • rewarming injured part without rubbing or massaging area to avoid further tissue damage
  • avoid weight bearing
  • if deep or severe, do not thaw till at hospital
  • clear blisters should be aspirated
38
Q

Burns

A
  • injury resulting from direct contact or exposure to any thermal, chemical, electrical, or radiation source
  • rule of 9’s used to estimate body percentage affected by burn
  • arms 9% each
  • legs 18% each
  • thorax 18% (front or back)
  • head 9%
  • inguinal area 1%
39
Q

Classification of Burns-Degree

A
  • superficial partial-thickness (first degree): localized injury or destruction to epidermis (sunburn)
  • deep partial-thickness (second degree): destruction to epidermis and some dermis with blisters developing along with edema and pain; white, waxy appearance
  • full thickness (third degree): extends through epidermis and dermis and into subcutaneous tissue; fluids and proteins shift from capillaries and to interstitial spacesC
40
Q

Classification of Burns-Mechanism of Injury

A
  • thermal: exposure or contact with sources such as flames, hot liquids, steam, semisolids (tar), or hot objects
  • chemical: caused by tissue contact, ingestion, inhalation, or injection with strong acids, alkalis, or organic compounds; can result from contact with certain household cleaning agents and various chemicals used in industry, agriculture, and military
  • electrical: caused by heat generated by electrical energy as it passesthrough body; can result from contact with exposed or faulty electrical wiring, high-voltage power lines, or lightning
  • radiation: least common, caused by exposure to radioactive source; associated with use of ionizing radiation in industry or from therapeutic radiation sources in medicine; sunburn considered a type of radiation burn
41
Q

Pathogenesis of Burns: Cardiovascular Changes

A
  • occur immediately following as vasoactive substances are released from injured tissue causing increase in capillary permeability
  • extensive burns result in: edema in both burned and non-burned tissues; decrease in circulating intravascular blood volume, increased HR and decreased CO
42
Q

Pathogenesis of Burns: Renal, Gastrointestinal, Immune, Respiratory Systems

A
  • blood shunted from kidneys and intestines leading to oliguria (decreased urine output) and intestinal dysfunction if TBSA is >25%
  • immune system function is depressed
  • respiratory system: pulmonary artery HTN, decreased lung compliance
43
Q

Medical Management for Burns

A
  • wound care: cleansing, debridement of loose nonviable tissue, application of topical antimicrobial creams or ointment and dressings
  • home care: observation for infection, AROM ex to maintain normal jt. function, decreased edema, and decreased scar formation
44
Q

SIFTT for Burns

A
  • non ER PTs: begin Rx when burn client is physiologically stable or at bedside to reduce morbidity and preserve function
  • burns and contracture: close assessment of ROM and muscle strength needed; encourage AROM every 2 hours while patient awake unless contraindicated by skin graft
45
Q

Pressure Ulcers

A
  • localized areas of cellular necrosis caused by unrelieved pressure: usually occur over bony prominences
  • graded or staged to classify degree of tissue damage
  • national pressure ulcer advisory panel added new stage-deep tissue injury
  • incidence: high volume, high-risk problems in most health care settings; LTC-regulatory agencies have designated development of pressure ulcers as indicator of quality of care provided
  • etiology and RF: interface pressure (externally), friction, shearing forces, maceration (softening and whitening of skin that’s kept constantly wet), decreased skin resilience
  • patho: continuous pressure on soft tissues between bony prominences and hard surfaces
  • most develop over 5 bony locations: sacral area, lateral malleolus, ischial tuberosity, greater trochanter, heel
  • compressed capillaries occlude blood flow
  • normal BP at arterial end of vascular bed averages 32 mmHg
  • CM: usually occur in circular pattern shaped like inverted volcano, may assume shape of objects causing pressure, irregular patterns indicate additional shearing forces or other contributing factors
46
Q

Stages of Pressure Ulcers

A
  • stage 1: skin remains intact, observable local changes in temp (warmth or coolness), texture (firm or boggy), color (red in light skin, red, blue, or purple in darker) or sensation (pain or itching)
  • stage 2: partial-thickness skin loss, ulcer involves epidermis, dermis, or both, superficial and may look like an abrasion, blister or shallow crater
  • stage 3: full-thickness skin loos, ulcer forms deep crater, adjacent tissue may be involved, damage to or necrosis of subcutaneous tissue, which may extend down to underlying fascia but fascia is not affected
  • stage 4: full-thickness skin loss accompanied by tissue necrosis or damage to muscle, bone or supporting structures such as tendon or joint capsule,extensive tissue destruction; sinus tracts may be present
  • suspected DTI: maroon or purple intact skin or blood-filled blister; may be painful; mushy, firm, or boggy; and warmer or cooler than other tissue before discoloration occurs
  • unstageable: involves full-thickness tissue loss, with base of ulcer covered by slough and yellow, tan, gray, green, or brown eschar; can’t be staged until slough and eschar are removed to expose wound base
47
Q

SIFTT for Pressure Ulcers

A
  • PT is pivotal in prevention and management
  • positioning, management of tissue load and good mobility are essential factors
  • high-risk client: frequent position changes (at least every 2 hours), utilize all turning surfaces, position at 35* angle when side-lying, elevate HOB no greater than 30* when supine
48
Q

Pigmentary Disorders

A
  • skin color determined by deposition of melanin; melanin formed by melanocytes in basal layer of epidermis
  • hyperpigmentation: abnormally increased pigmentation resulting from increased melanin production
  • hyperpigmentation disorders: pigmented nevi, cafe au lait spots (pigmented birth marks), mongolian spots, juvenile freckles, lentigines (liver spots) from sun exposure, hypermelanosis
  • hypopigmentation: abnormally decreased pigmentation due to decreased melanin production; ex-vitiligo in which melanocytes are destroyed