Lecture 4.2 Flashcards

1
Q

What are the spoken language characteristics of Wernicke’s aphasia?

A

fluent except for periods of anomia

normal to excessive verbal output
- logorrhea

semantically empty, primarily functors

morphosyntax and grammar near normal

articulation and prosody normal

all types of paraphasias

  • unintended syllables/words during effort to speak
  • paragrammatism
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2
Q

What is paragrammatism?

A

morphological substitution errors (ex. she for he)

well-constructed syntax of sentences with errors in grammatical morphemes, substitutions of and blending among lexical items

may occur in writing as well as speech

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3
Q

What is theorized to account for the difference in error types in aphasias?

A

neurological deficits affecting different stages of sentence processing

paragrammatic errors occurring later in sentence formulation process

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4
Q

What is literal/phonemic paraphasia?

A

phonemes or syllables of a word are produced early but in incorrect order OR word is distorted by production of unintended sounds

usually some phonemic features of the word are preserved

cat for mat, light for live

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5
Q

What is a neologism?

A

extreme literal paraphasic error such that the word is grossly distorted to such a degree that it is unintelligible

slammazer, carporta

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6
Q

What is a verbal paraphasia?

A

intelligible word used in place of intended/target word

tall for sail

run for sing

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7
Q

What is a semantic paraphasia?

A

substituted word has some lexical relationship to the intended word

can be random/irrelevant or perseverative

bowl for dish, girl for boy

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8
Q

What is extended jargon?

A

running/connected utterances (phrases or sentences) which include senseless words or neologisms

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9
Q

What are some other characteristics of Wernicke’s aphasia?

A

paraphasias pervasive

word finding difficulties severe

doesn’t respond to phonemic cues

resists interruption

unaware of communication difficulties

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10
Q

How is repetition affected in Wernicke’s aphasia?

A

disturbed

may be related to severe auditory comprehension difficulties

contains paraphasias

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11
Q

How is auditory comprehension affected in Wernicke’s aphasia?

A

severely impaired

some understand very little while others can comprehend simple words/phrases

system overloads easily

may have phoneme discrimination problems

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12
Q

How is reading comprehension affected in Wernicke’s aphasia?

A

often reading deficit parallels auditory comprehension deficit

comprehension and reading aloud

reading learned through auditory system so if it is impaired then reading is too

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13
Q

How is writing affected in Wernicke’s aphasia?

A

mechanical writing ability is good

well-formed letters

but content reflects paraphasic output at verbal levels, lacks meaning and semantically empty

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14
Q

Where is the site of lesion in Wernicke’s aphasia?

A

Wernicke’s area

posterior 1/3 of superior temporal gyrus plus inferior parietal lobe

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15
Q

What are some emotional and physical effects of Wernicke’s aphasia?

A

usually no hard neurological signs

may exhibit initial paresis but brief
- visual and ambulatory losses rare

tactile losses may be present

may become paranoid as others focus on deficits that clients can’t see

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16
Q

What are the spoken language effects of conduction aphasia?

A

generally fluent aphasia but verbal output limited to brief bursts of utterances
- words/min approaches normal

conversational output impaired but fluency maintained

  • literal/phonemic errors
  • impaired phoneme selection

disjointed spoken output
- facilitated by singing/rhythmic pattern

normal intonation, syntax

aware of errors

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17
Q

How is repetition affected in conduction aphasia?

A

serious problems

understand what they hear but can’t transfer to Broca’s to be repeated

classic sign of conduction aphasia

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18
Q

How is auditory and reading comprehension in conduction aphasia?

A

good!

contrasts with Wernicke’s

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19
Q

How is reading aloud impacted in conduction aphasia?

A

impaired because information cannot be transmitted to Broca’s area for verbal output

20
Q

How is writing impacted in conduction aphasia?

A

impaired on linguistic level but not motoric

spelling errors because of storage disturbances to angular gyrus

words omitted and interchanged in sentences

21
Q

Where is the lesion site in conduction aphasia?

A

along arcuate fasciculus

- white matter pathways under supramarginal gyrus

22
Q

What are the physical and emotional effects of conduction aphasia?

A

often no neurological deficits

motor hemiparesis varies based on site and size of lesion
- same with sensory disturbances (but usually only fingers or right side of face)

visual field impairments usually apparent, important implications for treatment to move items into field

23
Q

What are the spoken language effects on anomic aphasia?

A

anomia - inability to retreive known words

semantically empty speech

circumlocutions that are vague and bizarre

24
Q

How is repetition impacted in anomic aphasia?

A

evoking names spontaneously poor but repetition is good

25
Q

How is auditory comprehension impacted in anomic aphasia?

A

inconsistent

can be relatively good for connected language but poorer for isolated words

26
Q

How is writing and reading impacted in anomic aphasia?

A

reading and writing varies along a broad spectrum

spelling skills vary considerably

27
Q

Where is the lesion site in anomic aphasia?

A

least reliably localized

usually in L temperoparietal area

may extend into angular gyrus resulting in severe alexia and agraphia

28
Q

What are the physical and emotional effects of anomic aphasia?

A

do not usually see hard or soft neurological signs

29
Q

What is transcortical sensory aphasia?

A

damage to temporal lobe that cuts off Wernicke’s and Broca’s area from the rest of the brain

30
Q

What are the spoken language effects of transcortical sensory aphasia?

A

fluent output devoid of meaning

  • irrelevant, semantically empty
  • literal paraphagias and neologisms

naming severely impaired because of angular gyrus involvement

does not initiate utterance but those that are evoked are fluent yet empty

31
Q

How is repetition affected in TCSA?

A

excellent

may be considered echolalic - involuntary repetition of everything heard

32
Q

How is auditory and reading comprehension affected in TCSA?

A

impaired/poor

moderate to severe levels

33
Q

How is reading aloud and writing impacted in TCSA?

A

wide range of performance from preserved to defective

writing skills similar to Wernicke’s aphasia (poor)

34
Q

What is the site of lesion in TCS aphasia?

A

watershed lesion

posterior parieto-temporal

  • sparing Wernicke’s area
  • parietal and temporal border zones

disruption in blood supply from PCA affecting inferior temporal lobe and anterior occipital lobe

35
Q

What are the physcial and emotional effects of TCSA?

A

sensory impairments

Gerstman’s syndrome

  • R/L disorientation
  • finger agnosia (can’t tell what finger is touched when eyes closed)
  • agraphia
  • acalculia
36
Q

How can subcortical lesions cause aphasia?

A

thalamus and basal ganglia play direct regulatory or indirect gating roles in cortical language functions

subcortical vascular lesions may have widespread effects, creating hypofusion in L hemisphere Perisylvian regions

combination of 1 and 2

37
Q

What are the types of subcortical aphasias?

A

anterior capsule/putamen
posterior capsule/putamen
thalamic

38
Q

Describe anterior/putamen subcortical aphasias.

A

combination of TCM and Broca’s characteristics

short phrase length, impaired articulation, poor nonverbal skills

relatively good repetition and auditory comprehension

39
Q

Describe posterior/putamen subcortical aphasias.

A

combination of Broca’s and Wernicke’s symptoms

good articulation and verbal skills, poor nonverbal skills

poor repetition and auditory comprehension

40
Q

Describe thalamic subcortical aphasias.

A

TCM and TCS like linguistic characteristics

fluent aphasia with semantic paraphasias and neologisms

spared repetition, variable comprehension

41
Q

Describe pure alexia/pure word blindness.

A

alexia without agraphia

most common single language modality syndrome

can’t read or recognize written whole words
- can perceive letter by letter

can name objects presented visually, recognize numbers, and comprehend oral spelling

42
Q

What is the site of lesion in pure alexia?

A

complex lesion or series of lesions

L visual cortex and connections between R visual cortex and L hemisphere destroyed

absence of visual input to intact language areas of L hemisphere

43
Q

What is pure word deafness/subcortical sensory aphasia?

A

rare

can’t repeat or understand spoken utterances

  • but hearing still present
  • can interpret environmental sounds

can still speak, read, and write normally
- paraphasias usually present acutely

vowel contrasts appreciated but less so for consonant contrasts

44
Q

What is the site of lesion in subcortical sensory aphasia?

A

pathology in 2 locations

lesions isolate Wernicke’s area from primary auditory cortex in BOTH temporal lobes
- fibres that bring info from auditory association areas in R hemisphere destroyed

subcortical lesions in language-dominant superior temporal gyrus with destruction of primary auditory cortex (Heschl’s gyrus)

45
Q

What is pure agraphia/subcortical aphasia?

A

writing problems in absence of other significant language disturbances

may be due to focal lesion or acute confusional state

motor and spelling errors present but spelling errors dependent on lesion location

5 types

46
Q

What is anterior disconnection syndrome?

A

unilaterail tactile agnosia, agraphia, apraxia

unilateral L hand apraxia, can’t follow verbal directions with left hand

can’t name or talk about objects in L hand though can usually draw item or demonstrate its function

lesion in anterior corpus callosum
- interruption of connection between verbal areas in L language dominant hemisphere and sensory areas of L hand in right hemisphere

47
Q

What is posterior disconnection syndrome?

A

objects seen in L visual field can not be named

non-specific visual disturbances

lesion in posterior portion corpus callosum