Lecture 4 - Continuous vs Interval Training Flashcards

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1
Q

List the 12 muscular adaptations of aerobic training?

  • Cellular
  • Anatomical
A

Cellular

  • Increased mitochondrial content and size.
  • Increased glycogen concentration
  • Increased oxidative enzyme activity
  • Increased myoglobin concentrations
  • Increased glycotic enzyme activity
  • Increased uptake of blood fats
  • Increased fat utilisation
  • Decreased glycogen utilisation
  • Decreased lactate production.

Anatomical

  • Increased capillaries per fibre
  • Decreased diffusion distance of 02 substrates and metabolites between blood and muscle
  • Fast twitch fibres become slower
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2
Q

Why is iron important for athletes and how much 02 binds to it.

A

Iron is important because it has a high affinity for 02 binding and transport, so depleted can hinder performance.

Each haemoglobin molecule has 4 iron groups, meaning 1 haemoglobin can carry 4 O2 molecule.

1g of haemoglobin can carry 1.34ml O2

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3
Q

What are the effects of endurance training on muscle metabolic homeostasis during exercises.

A

Homeostasis in maintained more effectively because their is less PCr and glycogen utilisation and a reduced building of of acidosis because of the better buffering capacity.

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4
Q

What is the best training method for well trained athletes ?

A

SIT as it can improve performance even in well trained athletes. However you cannot entirely remove the MICT as it is essential to baseline conditioning and central adaptions.

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5
Q

What are the central cardiovascular adaptations to exercises training ?

A

Reduced HR
Increased QMax
Increased blood volume (plasma, RBC, Heamaglobin)

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6
Q

How does endurance training improve endurance performance. An overview.

A

When running at the same velocity as before training an individual is:
– More economical so uses less energy
– Depletes the energy reserves less (improved economy and fat oxidation spares muscle PCr and glycogen)
– Produces less metabolites linked to muscle fatigue (ADP, Pi, H+)
– Able to perform better in the sprint finish
• Capable of running faster for the same depletion of energy reserves and metabolite accumulation as before training
– We can run the ‘kick’ out of our competitors.

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7
Q

What are the implication of having an increased heamatocrit (ratio of RBC to toal volume).

A

Increased blood volume and 02 carrying capacity.

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8
Q

How much energy in ATP do you gain from glycogen vs fats ?

A
Glycogen = 39ATP
Fat = 129ATP
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9
Q

What is the problem of work matching performance?

A

It can skew the conclusion as you may perform a few sprints, however, if you only completed 30 mins moderate activity it is not enough to stimulate a response.

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10
Q

What are some standard adaptations to endurance endurance training ?

A
  • VO2max
  • Lactate threshold
  • Economy
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11
Q

Describe the 4 signalling cascades for endurance adaptation from training?

A
  • Low glycogen leads to the activation of MAPKp38 + AMPK.

High levels of ADP,AMP AND low PCr lead to activation of AMPK

When calcium is released this stimulates caMKII.

Adrenalin stimulates activation of PKA.

These four action then lead to the activation of PGC1a, which leads to mitochondrial biogenisis, fibre type conversion and blood vessel creation.

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12
Q

In 4 weeks time is it possible to increase mitochondria ?

A

No

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13
Q

What is the most effective method of training to improve VO2max?

HIIT, MICT or SIT ?

A

HIIT is best as it has the biggest response for developing SV, Qmax and VO2.

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14
Q

What is the best method to improved mitochondrial respiration

HIIT, MICT or SIT ?

A

SIT is best because it caused the largest disturbance, therefore allowing greatest remodelling adaptation, however, this only increased the effectiveness of the mitochondria not the overall content.

Showed highest levels of PGC1a and P53 increases.

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15
Q

What is a typical sprint interval training session?

A

30 seconds interval with 4 min recovery
4-7 repetitions
90-95% HRmax
170% Peak aerobic power

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16
Q

How does blood volume increase?

A

Initial because of an increase in plasma volume

However, with an extended period it is via increases in red blood cell count.

17
Q

For sprint interval training answer the following:

  • What systems does it improve?
  • What structural effects does it have ?
  • Does this translate into performance improvements in races?
  • Should it be the training of choice ?
  • What athletes should use this
  • What is another effect of SIT that needs to be considered.
A
  • SIT can improve aerobic and anaerobic metabolism
  • SIT can increase skeletal muscle hypertrophy, % of IIa fibres and capillarisation
  • SIT can improve sprint, repeated sprint and endurance performance (at least up to around 10 km)
  • SIT should be the ‘endurance training’ of choice for athletes wishing to also improve strength performance
  • SIT should be used by team sports athletes due to its numerous metabolic and performance improvements
  • Continuous training more important for longer distance athletes.
  • SIT can increase skeletal muscle hypertrophy, Meaning that if weight is an important factor this may need to be limited of applied intelligently.
18
Q

What does HIIT do to onset of fatigue ?

A

Central fatigue is reduced allowing greater peripheral fatigue as muscles are not limited so can contract for longer and harder.

19
Q

What is a typical high intensity training session?

A

1-5 mins exercise with similar recovery.
6-20 repetitions
85-90% HRmax
75-100% VO2max

20
Q

Give an overview of the effects of endurance training on the following:

  • Central and peripheral cardiovascular adaptations.
  • Energy utilisation
  • Substrate usage within cells
  • Metabolite accumulation (Which types change
  • Feedback responses.
A

Post endurance training
– ↑ Qmax, SV, muscle blood flow, capillarity density, mitochondrial content AND efficiency
– ↑ Fat oxidation, ↓ CHO oxidation
– ↓ Muscle glycogen and PCr decline
– ↓ Muscle ADP, Pi, H+, K+ and lactate accumulation
– ↓ Muscle afferent feedback
– ↓ Central fatigue
– ↑ Pain tolerance
– Allows the development of greater peripheral fatigue
– Improved performance

21
Q

How are muscle ion transporters effected by exercise training ?

A

The monocarboxylate (MCT) and exchanger isoform 1 (NHE) which are responsible for removing lactate and H+ from the muscle ino the blood were enhanced.

Moreover the Na, K pump was enhanced also, meaning that any potassium accumulating outside of the muscle is pumped back in, ensuring that there is no interference in muscle excitability, which potassium can cause fatigue.

22
Q

What is needed to increase mitochondrial content best ?

A

Mitochondrial content responds best to a increase in volume of training at a moderate/ low intensities, as apposed to increases in intensity.

23
Q

How does a 30 second wingate correlate to the work done during normal cycling ?

A

Equates to 5 mins moderate cycling.

24
Q

What causes the increase in red blood cell count ?

A

When the blood become oxygenated during exercise performing this repeatedly cause the kidneys to secrete the erythrocyte stimulating hormone erythropoietin (EPO).

EPO stimulates RBC production in bone marrow of long bones which increases the count of RBC and haemoglobin in blood.

25
Q

Why does HR reduce with endurance training ?

A

Improvement of Stoke volume via left ventricular hypertrophy and the increased blood volume.

This allows the chamber to fill to greater degree improving the ejection fraction, and subsequent stoke volume per beat, thus improving overall Qmax.

Therefore, the heart does not have to beat as often to supply the same amount of blood.

26
Q

How does endurance training change fibre recruitment ?

A

Less recruitment of type II muscle fibres at the same exercise intensity after training

  • Beneficial since type II muscle fibres are less economical (require more energy) and become fatigued quicker.
27
Q

What are the benefits of greater fat oxidation ?

A
  • More ATP produced than carbohydrate
  • Huge fat reserves compared to carbohydrate
  • Increasing fat oxidation spared the limited muscle glycogen reserves.
28
Q

Summarise the central adaptions to endurance training ?

A
  • Can pump more blood per beat.
  • Muscle blood flow is increased.
  • Blood contains more O2
  • Muscle 02 delivery is increased.
29
Q

What is Ficks equation for oxidative metabolism and how is is broken down?

A

VO2 = CO x a-vO2 difference.

The CO and the (a) represent the Central O2 delivery (Hence the CO)

Whereas the vO2 difference represents the O2 uptake by the muscle..

30
Q

What are the factors that determine mean race pace ?

A

Lactate threshold and critical velocity = sustainable %VO2max

VO2max and sustainable %VO2max = performance VO2

This represents the highest sustainable rate of ATP resynthesis

running economy fit in here to determine how much energy is demanded at a certain speed and this gives mean race pace.

31
Q

Name the SPORT AND FITT acronyms.

A
Specificity
Progression
Overload
Reversibility
Tedium

Frequency
Intensity
Type
Time

32
Q

What happens to energy utilisation after endurance training?

A

Fat oxidation increases and carbohydrate oxidation decreases.

33
Q

What is a Typical example of a moderate intensity continuous training session ?

A

60 minutes
70% HRmax
50-80% VO2max / Peak aerobic power.

34
Q

What is sprint interval training good for ?

A

SIT is very effective at getting peripheral improvements because of its large changes in the energy charge of the cells. It is also very time efficient.

35
Q

What is the other advantage of having a greater fat oxidation for energy systems ?

A

Means you will not rely on anaerobic energy as heavily, meaning less depletion of crucial PCr and glycogen stores, and lower accumulation of Pi, ADP and H+ in the muscle

36
Q

How do the HITT and MICT training methods effect pain sensation ?

A

HIIT improved pain sensation.