Lecture 3: Neuro Intervention: Body Structure and Function Flashcards

1
Q

Knowledge check: Which of the following key points of control is true for the starting points of NDT

A

Start proximal move distal

steat the pelvis and correct the posture before you move onto something

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

contractures due to immobility - think being in cast and not being able to move = leads to contracture

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

How do we asses for tone (not spasticity)

A

By passively moving someone
* not modified ashworth

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Prognosis: Prediction of motor recovery poststroke with a primary symptom of hypotonia has been reported to be associated w/ a poor outcome if there is a lack of voluntary motor control of the leg within the first week and notiviable arm movements at 4 weeks
* dont memorize the #’s
* So time really does mattter

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Slings can be good and bad for shoulder subluxations
* supports the arm, but tightens the streuctures in and around the arm (contractures)

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

contractures shouldnt happen and can be prevented

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Hypotonia: treatment techniques:

Weight bearing: - getting pts up in moving quickly is important because they get that weight bearing. Obviously if theres low tone and weakness you’re going to have to be safe about your movements
* enhance, promote, and stimulate more normal postural alignment, motor control, and functional extremeity use
* joint approximation - facilitation technique as a result of weight bearing
* Goal - muscle activitation: position of UE is latearl to the body in shoulder abduction, external rotation, elbow extension, wrist extension, and finger extension.
* Goal: osteoporosis prevention and circulatory stimulation.- EX: LE weight bearing post chronic SCI
* Goal postural alignment.
* Add facilitation techniques if needed. EX: think muscle tapping - this might help them get up and weight bear

Make these dynamic as you go, to progress exercise:
* Weight-shifting, unilatearl reaching, manipulation of objects with a fixed component, limb mobement from a target, and holding and sustaining graded movements

A

This is an example of wt bearing. shes approximating his shoulder

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Facilitation Tehcniques (for muscl activation)

Neuromuscular + Sensory stimulation techniques are combined for this
* remember its harder to target sensory but a lot of these techniques are both

Gaol: To increase muscle tone and muscular activity

Techniques include:
* Quick tapping
* Quick stretch
* Vibration
* Manual contacts
* Resistance
* Approximation
* Fast brushing/icing

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Quick Stretch/Tapping - to help treat hypotonicity

Goal: Facilitate muscle contraction

HOW: Manually apply a brief quick stretch to the muscle, quickly tap, or apply vibration to the muscle belly or tendon
* So its best to tap when its already lengthened out

Most effective when the muscle is first placed in a lengthened position, placing the muscle spindle on stretch

Utilizes the stretch reflex and results in a muscle contraction

Contraction is short lived –> follow with active motion or light tracking resistance
* Think if they’re trying to flex the elbow but can only get halfway, well I’m going to grab their arm and lightly push it back up into flexion = light tracking

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Manual Contacts, Apporximation and use of Resistance - for hypotonicity

Cocontraction - increases joint stability due to activation of muscles arounf the joint

Tracking resistance - don’t want to overpower the muscle

What is irradiation/overflow?

A

Irradiation/overflow - applying maximal resistance to a stronger muscle or muscle group may result in facilitation of a muscle contraction in weaker muscles within the same synergistic movement or in the contralatearl extremity
* EX: Resistance to the hip flexors may facilitate ankle DF’s in the same limb or resistance to ankle dorsiflexion on the right side may overflow to the left

Essentially power flows elsewhere when you’re contarcting a very strong
* resistance at hip flexors can make the Df’s go off
* YOu can use this to your advantage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Fast Brushing / Icing for muscle hypotonicity
* Activating tactile and thermoreceptors through brief quick strokes of the skin over a muscle with ice or with the fingertips may promote a muscle contraction
* Contraction is short lived –> follow with active motion or light tracking resistance
* Use with caution if patient presents with autonomic instability or who are already in a heightened state of arousal (could could be too much for someone) due to potential autonomic sympathetic response

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Strengthening - treatment for hypotonicity

Goal: To improve motor unit recruitment if able to facilitate muscle activity
* obvisouly if they dont have anything this is a different story

Perform facilitation techniques then follow up with resistance to improve muscle contraction

Light tracking resistance –> maximal allowable resistance while maintaing smooth coordinated movement against gravity
* so you’re essentially helping them when they get stuck
* Don’t want movement to be wonky

Tools: manual resistance, use of gravity and body weight, and tools such as weights, theraband, etc.

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Shoulder Strapping/Taping - for hypotonicity

Mechanism to reduce shoulder subluxation or prevent shoulder pain in individuals poststroke, either as a stand-alone intervention or in combination with electrical stimulation

Not much research to back this up

When taping, place GH in optimal starting position

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Neuromuscular electrical stimulation (NMES) - tx for hypotonicity

Delivers electrical current to specific muscles or peripheral nerves to (in the case of hypotonia or flaccidity) facilitate active muscle contraction and prevent disuse atrophy

Can decrease shoulder subluxation

Key muscles to reduce inferior displacement of humeral head: supraspinatus and posterior deltoid

Research shows potential in initiating early poststroke to prevent subluxation (inconclusive in chronic shoulder subluxation)

Parameters: stimulation should eleicit a motor contraction in the supraspinatus and posterior deltoid at more than 30 hz gradually progressing from 1 to 6 hours per day
* do not memorize parameters

note: subluxation isnt just joint, it can even lengthen muscles

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What two muscles are targets w/ NMES to reduce displacement of humeral head?

A

Posterior deltoid
Supraspinatus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Applying NMES to functional activities:

Functional electrical stimulation (FES) - subgroup of NMES - think using for footdrop after stroke to help that anterior tibialis work
* Applies NMES in an organnized manner to promote goal-oriented movement, which enhances functional motor learning

Functional activities
* Supine
* Sidelying
* Sitting
* Standing
* Quadruped
* Walking

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Examples of interventions for hypotonicity - split in body parts

So think strenghtening the joints is the common thread / wt bearing / activating those muscles

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

knowledge check: which of the following is a faciliattion technique as a result of wt bearing?

A

Approximation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Hypertonicity: Spastic / rigid

this is UMN’s

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

pt has rigidity, what part of the brain is likely imapcted?

A

Basal ganglia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

pt has hypotonia, what part of the brain is likely impacted?

A

Cerebellum

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

know that lesion ins the brainstem, subcortical white matter, and primary motor cortex lead to spastic tone - not just hypertonicity

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Form of hypertonicity that is a movement disorder in which involuntary sustained or intermittent muscle contractions cause twisting and repetitive movements, abnormal postures, or both

A

hypertonia (dystonia)

think cervical dystonia where the neck twists

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

When should we treat hypertonicity?

A

When it interferes w/ activity
* note, if they had a SCI and got hyptonciity in legs that helps to stabilize them, well we might not treat it because its actually helping their function

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

What are our 4 oral medications for hypertonicity?

A

Orals: baclofen, diazepam, dantrolene, tianidine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

What injection do we use for hypertonicity?

A

Botox

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

What does an implanted baclofen pump do?
* why is it better than oral medications for this?

A

Implante under skin @ lower abd, cath to SC out through CSF. Less general sedation effect than orals.
* helps treat hypertonicity
* great for SCI’s because it can be a targeted approach
* invasive risky surgery

NOTE: cannabis can be used to treat hyperetonicity (relaxes the body)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Interventions for hypertonicity:

Surgical interventions:
* Tendon lengthening (tenotomies) - common at Achille’s tendon - think high tone and walking on ankles - can fix this
* Selective dorsal rhizotomy - severs sensory nerve fibers, best candidate is peds with high tone - abnormal rootlets identified via EMG and cit

More drastic: osteotomies, arthrodesis

Deep brainstimulation
* invasive surgery
* helps w/ tone / tremor
* Helps at level of brain rather than level of extremeities

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

What does deep brain stimulation help w/

A

hypertonicity

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Interventions for hypertonicity:

Sustained positioning and PROM
* Serial casting

Handling and physical inhibition

Equipment
* Think AFO
* dynamic splint

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

Serial casting:

Common adverse events caused by the cast include skin irritation and breakdown, pain, swelling, numbness, and nerve impingement

General containdications to the use of casting include medical instability, open wounds, skin infection, and cellulitits

Precaustion should be used in individuals w/ impaired sensation, cognitition, or communication

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

interventions for hypertonicity: all designed to inhibit tone
* Deep pressure
* Joint traction
* Rhythmic rotation
* Sustained stretch
* Warm/cold (gentle heat, prolonged ice)
* Raping
* Biofeedback
* Vibration and sonic pulses
* Estim - apply to intervated antagonist muscle group
* Acupuncture

Rhythmic rotation

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

how is estem applied when treating hypertonicity

A

Apply to innervated antagonist muscle group (I think it will essentailly stretch out the agonist by having the antagonist contract)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

intventions for rigidity (not hypertonicity) - remember this is velocity independent (velocity doesnt matter)

  • Massive stretch - helpful to some degree, you don’t want to push to their max where they’re fighting you though
  • Physical activity and exercise
  • Aquatic exercise in warm water
  • Stretch exercise
  • Trager therapy (gentle rocking motion)
  • Whole body vibration
  • Botox - very tageted because its a focal injection
  • E-stem and magnetic pulse stimulation
A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

Aquatic therapy for hypertonicity:
* good because buoyancy - that upward force expressed on an object in water that creates a floating roce, creates greater degree of safety and stability for ambulatory activities, eliminates effects of gravity
* so its safer due to the decreased risk of falling

A
36
Q

Functional activity application for hypertonicity

Quadrupated position (food for breaking up extensor tone)

Rolling - rhythmic initation

Weight bearing activities
* encourages optimal limb use and attention to limb in natural ways

Tone reducing orthotics

Patient and caregiver education
* contacture prevention
* Emphasize wieght bearing as much as possible

A
37
Q

knowledge check: Which CNS damage area causes specifically rigidity

A

Basal ganglia

38
Q

What is an abnormal involuntary movement?
* What part of the brain is typically responsible for them?

A

Dyskinesia

Typically the basal ganglia causes them

think anything like tremora, dystonia, athetosis, chorea, choreoasthetosis, ballismus, tics, myoclonus

39
Q

Most common involuntary rhythmic oscillations of reciprocally innervated muscles - resting, action postural, essential

A

Tremor

think pronation/supination of forearm (oscillations)

40
Q

Persistent contraction

A

Dystonia

think head getting warped in a certain position

41
Q

Slow, writhing movement (not controllable, involuntary)

A

Athetosis

42
Q

Abrupt, involuntary, variable, jerky movement

A

Chorea

Think huntingtons

43
Q

Uncontrollable, violent, flailing

A

Ballismus

intense and severe - think someone trying to walk and throwing their limb

44
Q

Stereotypical movement or vocalization, habit spasms, sudden, nonrhythmic

A

tics

45
Q

Brief, sudden, rapid, asymmetrical movement

A

myoclonus

46
Q

Interventions for uncontrollable contractions and movements

Pharmalogical implications
* botox

Surgery
* deep brain stimulation

Therapeutic techniques
* Whole body viberation
* Orthoses
* resistance training w/ wts
* Stretched and seated karate
* Functional e-stem
* Peripheral cooling of limbs
* Active assited cycling
* Bright light therapy
* Neuromuscular massage
* Functional desterity training
* Transcranial stimulation

A
47
Q

Dystonia Intervention - she likes having people contract muscles in an isolated fashion. Working on motor control
* Sensorimotor training
* Mirror therapy or mirror box
* CIMT
* TENS
* TRanscranial magnetic stimulation (TMS)
* Biofeedback
* Orthoses
* ARM cervical spine/whole body relaxation
* KinesioTape application

A
48
Q

Is rehab effective for chorea movements?

A

No.

usually anything that relaxes the pt is helpful
* medications
* general exercise, customized
* Relaxation techniques
* Hydrotherapy
* Rhythmic auditory stimulation

49
Q

KNOW: adding wt to extremity with tremor - ADL impact - practice
* causes co contraction of agonist and antagonistic muscles which temporally removes that osiliation - think like a seesaw - trying to balance it in the middle

A
50
Q

Knowledge check: which of the following dyskinesias would the pt present with uncontorllable violent flailing movements?

A

Ballismus

51
Q

Coordination requires precise cooperation between opposing muscle groups, releated interjoint muscle groups, and interlimb muscle groups for functional activities, along with normal postural control during a volitional movement

A
52
Q
A
53
Q

Is ataxia muscle weakness?

A

No, its incoordination
* so its not a strength issue, these pts can be very strong

Result of either the lack/absence of sensory (proprioceptive) input from the periphery to the cerebellum, or higher sensory centers, or a lesion/disruption in the interaction of upper and lower motor neurons
* so basically saying the cerebellum is largely responsible for it

Patients are unable to perform smooth, skilled, adequately ranged movements
* Lack of cooperation between agonist/antagonist
* don’t perform those smooth movements

Deficits seen: dysmetria (inability to reach the target), dysdiadochokinesia (difficult with rapid alternating movement), tremors, hypermetria (over shooting)
* releated: dysarthria (speaking), titubation (rhythmic shaking, type of essential tremor), nystagmus - because of the effect of the vestibular on the cerebellum

Assessment - within outcome measures
* cerebellar ataxia - imbalance regarddless if eyes are open or closed
* Sensory ataxia

54
Q

Interventions for ataxia

Pharmaological interventions - some medications exist, recent in research, medication commonly prescribed for primary pathology which may influence ataxia
* mostly targeted at whatever the underlying cause of ataxia is, not one underlying cause for ataxia so not one set medication to treat it

Primary focus for rehab: functional training to obtain central, proximal and distal postural control

Visuomotor training

Equipment - weighted vest (promote proprioceptive awareness), weighted ADL tools, cycle ergometry
* if someone is feeling understable that weighted vest will calm them down

PNF Pt exercises focused on coordination

Frenkle exercises - vision as principle source of feedback

Cawthorne-Cooksey exercises - vestibular component

stable closed chain exercises are good for these pts

A
55
Q

When treating ataxia what is the order of obtaining new control that we strive for?

A

Central –> proximal –> distal

56
Q

Functional intervention for ataxic / uncoordinated pts

Mat activities - good because it promotes that closed chain safe method of training

Balance training (in functional positions)

Establishing central (trunk stability) before promoting proximal (shoulder and pelvic girdle) or distal contorl (limb)
* remember you want to start w/ the trunk first because we move central –> Proximal –> distal

Considerations
* Limit resistance/tasks the patient performs against gravity requiring execessive effort
* Mental fatigue from visual and cognitive control of movement - mental fatigue is real w/ these pts (when they’re trying to get the correct movement patterns down)
* remember, at first we want to focus on trunk/proximal areas, so limit that resistance in the distal extrememities (at least at first)

Remember, you can get ataxia from stroke / MS etc… (its motor incoordination)
* because someone w/ MS could absoutely have damage to that cerebellum (which is what causes most of that ataxia)

A
57
Q

Knowledge check: Which part of the CNS does movement planning

A

Motor cortex (precentral gyrus)

58
Q

Weakness in neuromotor disorders
* need to figure out if its PNS or CNS
* So weakness can originate in the CNS or PNS depending on the underlying pathology

CNS - Weakness example - Stroke

Weakness is due to an underlying cause due to primary central activation deficits, transynaptic degeneration, leads to secondary changes in muscle (think atrophy)
* CNS releated
* because something hapened in the brain and signals have a hard time getting outward
* Not due to that muscle distally - its the brain having a hard time connecting to it
* however, secondary changes due happen in the muscle because that UMN is having trouble connecting to that LMN, which means the muscle isnt activating corectly
* With weakness strength deficits are greater in distal than proximal muscles - makes sense, if its already a weak signal and has to travel further its not going to work well the further out it is.

Weakness in the PNS caused by GBS

Weakness is the primary clinical manifestation that affects function - primary changes
* this is a LMN disorder, so those muscles are affected directly, not indirectly
* there are now primary changes in the muscle unlike w/ UMN weakness where the muscle changes are secondary. The muscle is directly imapcted because those LMN’s are directly impacted.

Ascending loss, distal to proximal
* meaning that theres something wrong w/ those motor tracts going back up. And most distally is once again impacted here

MS is different. However, it is a CNS issue, but impacts the PNS as well.

A
59
Q

What happens to motor units following a stroke?
* how long does it take disuse atrophy to come following a stroke
* what happens to motor unit innervation ratios
* What muscle fiber type atrophies the most following a stroke

A

They’re decreased

Takes 4-6 weeks
* these would be the secondary muscle changes following a storke (because stroke is a CNS disorder / weakness)
* we jump in to decrease the impact of m atrophy

Increased motor unit innervation ratios (meaning that the existing motor units have to innervate my myofibirils because there are less of them)

Impaired firing rate regulation –> leads to decreased type 2 fibers, meaning theres a higher percentage of type 1 fibers
* so its changing the relationship of the MU’s now
* know that the literal composition of the muscle is impacted / changed

60
Q

Strength training in the neuro population

How long does the program have to be to promote neural adaptation?

What intensity do you need to train at to gain neuroplasticity

A

Program should be 6-8 weeks to see neural adaptation

Moderate- to high intentiy training may also facilitate neural plasticity at the cortical, spinal and neuromuscular levels

Benefical to both the acute and chronic stages

Specific training varies in population
* ALS vs Stroke (remembr training w/ ALS is great) but w/ stroke it is

61
Q

for neuro population

A
62
Q

Interventions for incoordiantion / ataxia
* Facilitation techniques
* NMES
* EMG biofeedback / NMES
* PNF
* Aqutics
* Exercise machines
* Traditional - OKC (PRes - progressive resistive exercise) (isokinetic exercise)
* Task specific - work in functional synergies, not isolation

A
63
Q
A
64
Q

Pre cautions for aquatic therapy

A

Precautions
* Lack of a gag reflex
* Diabetes
* Homeostatic abnormalities
* Controlled seizures
* Breathing difficulty
* Autonomic dysreflexia
* Nasogastric tube
* Colostomy/G-tube
* Serial casting
* Hypertension
* Superficial wounds
* Fear of water
* Limited water safety skills
* Chemical hypersensitivity

65
Q

Contraindications for aquatic therspy
* Open wounds
* Infections
* Fever
* Bladder incontinence
* Bowel incontinence
* Tracheostomy
* Uncontrolled BP
* Casts due to fractures
* Uncontrolled agitation
* Isolation precautions
* Continuous nasal oxygen
* IV or indwelling catheter
* Incompatibility with facility infection control proceudes
* Uncontrolled seizures
* Unstable fractures

A
66
Q

What temperature does the water have the be below for someone w/ MS’s heat sensitivity?

A

Below 85

67
Q

Functional activities for weakness

Task oriented functional strength training

Individualized

A
68
Q

Knowledge check: If someone is very weak you can’t just throw them into a sit to stand, you need to start w/ those faciliation techniques and then move forward.

A
69
Q

Knowledge check: Percaution for aquatic therapy = superficial wounds

A
70
Q

Limited PROM - Effects of immobilization

Patients with CNS pathology may not actively move their limbs as often or as far as people w/o pathology due to
* Impaired functional mobility
* Impaired motor contorl
* Impaired strength
* Frequent use of splints/orthoses

However, ecause of this they are at risk for:
* Contracture formation
* Heterotopic ossification = excessive bone somewhere

A
71
Q

what two things can immobilization cause

A

Contracture formation

Heterotopic ossification

72
Q

Interventions to effects of immoblization (think those contractures / hetertropic ossification)

Surgical release - surgical lengthening (tenotomies) or “release”

Therapeutic techniques
* Stretch to shortened tissue –> needs to be slow and sustained 4x15 seconds isnt gonna cut it here
* Splinting
* Serial casting
* Joint mobiizations
* heat modalities
* Referral for surgical release

A
73
Q

Things to do for that immoblization (functional activities)

A
74
Q

Stability: The aspect of motor control that allows a joint or body segment to be stable at a time when movement is not supposed to occur there

Other synonymous terms: neuromotor stability, stability motor control

Problem is releated to the way the brain controls movement

PT assessment - observation and within outcome measures + patient/caregiver report

These are what happens in theres instability at this area of the body

A
75
Q

A brain lesion where causes trouble initiating movement

A

Basal ganglia

76
Q

A lesion where causes issues w/ motor planning?
* think any voluntary motor control

A

Cerebral cortex (makes sense, think the motor contorl center in the prefrontal gyrus)

77
Q

Damage where in the brain causes ataxia

A

cerebellum

78
Q

Where is cognition in the brain?

A

Frontal lobe

79
Q

Interventions for lack of stability
* NDT
* PNF
* Stance weight bearing exercise
* Postural EX
* Biofeedback
* E-stim
* Body-weight support treadmill training
* Adjunct therapies (taping/strapping, splinting/bracing, casting, orthotics, pressure garments)

A
80
Q

Functional Activities Applications - For pts w/ instability

Remember joint approximation doesnt have to be in the close packed position. Its just anything that loads the joint and pushes it togther

Transitioning between supine and sitting
* Consider quadruped position

Transitioning between sit and stand

Transitioning between stand and squat

Complex functional activities

Adjusting to the environment

A
81
Q

Knowledge check: In which diagnosis is sensory impairment perminant?

SCI
* have to use those compensatory strategies because they arent going to regain the function they’ve lost

A
82
Q

If cardiorespiratory health and fitness is decreased, than quality of life is decreased

Can also have pulmonary impairments –> muscular weakness (lack of respiration)

A
83
Q

Aerobic EX primes neuroplasticity
* it gets us ready - strenghtens the chance of neurplasticity
* so do something hard w/ your pts following aerobic activity - maybe that neurplasticity will encode better

A
84
Q

Interventions for cardiovascular/pulmonary pathologies
* Physical activity vs aerobic exercise - pop an RPE scale on these
* Promote long term enfafement in aerobic EX
* Meaningful and enjoyable to the individual pt

A
85
Q

Knowledge check: When is the optimal time in a pt session to prime neurplasticity
* Before you do the hard activity / before motor training

A