Lecture 3+4 Flashcards

1
Q

pericarditis

A

inflammation of the pericardium

cause:
infection, renal failure, MI, post-surgical

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2
Q

pericardial friction rub

A

A resulting sound due to roughness of the parietal lining from inflammation

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3
Q

pericardial effusion

A

the accumulation of excess fluid within the pericardial space

cause:
pericarditis
CT disease
hypothyroidism

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4
Q

cardiac tamponade

A

rapid large fluid (blood) accumulation in the pericardial space that leads to compression which prevents adequate filling of the chambers

clinical signs: beck’s triad

  1. hypotension
  2. distant heart sounds
  3. distended neck veins

causes:
Stab wound, MI, or Rupture of heart muscle

treatment: pericardiocentesis

paraxiphoid approach: tip of xiphoid, or between xiphoid and left costal margin, angles towards shoulder

apical approach: left 5th or 6th intercostal space

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5
Q

where does the right atrium get blood from?

A

gets blood from the IVC, SVC, and coronary sinus

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6
Q

left atrium

A

gets blood from 4 pulmonary arteries

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7
Q

location of aortic valve and where to auscultate

A

location: posterior to left side of sternum at 3rd intercostal space

Auscultate: right sternal border at 2nd intercostal space

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8
Q

location of pulmonary valve and where to auscultate?

A

location: left 3rd costal cartilage
auscultate: left costal border at 2nd intercostal space

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9
Q

location of tricuspid valve and where to auscultate

A

location: behind sternum at 4/5 intercostal space
auscultate: left sternal border at 5th intercostal space

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10
Q

mitral valve location and where to auscultate

A

location: behind sternum at 4th costal cartilage
auscultate: 5th left intercostal space in midclavicular line

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11
Q

structures supplied by the right coronary A.

A
right atrium 
right ventricle 
SAN
AVN
interventricular septum
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12
Q

structures supplied by the left coronary A

A

left atrium and left ventricle

interventricular septum

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13
Q

What is S1 and S2?

A

S1 (Lub) is the closing of the mitral and tricuspid valve.. systole

S2 (Dub) is the closure of the aortic and pulmonary valve… diastole

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14
Q

What is diastole and systole?

A

diastole is when the heart is filling with blood and is not contracting (S2 to S1)

systole is the period of contraction (S1 to S2)

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15
Q

The 7 steps of the cardiac cycle

A
  1. Arterial systole (diastole)
    (S1)
  2. isovolumetric contraction (systole: mitral valve closure)
  3. rapid ventricular ejection (systole: Aortic valve open)
  4. reduced ventricular ejection (systole: aortic valve open)
    (S2)
  5. isovolumetric ventricular relaxation (diastole: aortic valve closure)
  6. rapid ventricular filling (diastole: mitral valve open)
  7. reduced ventricular filling (diastole: mitral valve open)
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16
Q

Electrocardiogram (electrical activity)

A

P = arterial depolarization

QRS = ventricular depolarization

T = ventricular repolarization

17
Q

Normal cardiac pressures for LA, LV, and aorta (mmHg)

A

LA: 12
LV: 130/10
Aorta: 130/90

18
Q

Normal cardiac pressures for RA, RV, pulmonary A. (mmHg)

A

RA: 5
RV: 25/5
pulmonary A: 25/10

have lower pressures compared to the left side because the pulmonary system is a low resistance circulation

19
Q

S1, S2, S3, and S4

origin and location? Normal/Abnormal?

A

S1: mitral and tricuspid valve closure
loudest at the mitral valve region
normally present

S2: aortic and pulmonary valve closure
loudest at left upper external border
normally present

S3: In early diastole during rapid ventricular filling phase
Can be found in children, young adults, and pregnant
women (can be normal; but not normally present)

Abnormal in adults: Dilated ventricles (more common), increased filling pressure: mitral regurgitation. May lead to heart failure (left ventricular failure)

S4: during late diastole (atrial kick)
best heard at the apex with patient in left lateral decubitus position

abnormal regardless of age: high atrial pressure, ventricular hypertrophy, stiff left ventricular wall

20
Q

splitting patterns of S2

A
  1. wide-splitting: This is due to a delay of RV emptying
    pulmonic stenosis
  2. fixed-splitting: produces a great delay in the pulmonic valve
    arterial septal defect
  3. paradoxical splitting: delay aortic valve closure
    aortic stenosis
21
Q

systolic murmurs

A
  1. aortic stenosis
    crescendo and decrescendo
    usually due to age-related calcification
  2. Mitral regurgitation
    high-pitch “blowing murmur” (loudest in apex)
    due to rheumatic fever and/or ischemic heart disease
  3. mitral valve prolapse (most frequent)
    late systolic crescendo (loudest just before S2 at apex)
    due to rheumatic fever and/or Marfan syndrome and/or ehlers-danlos syndrome
  4. Tricuspid regurgitation
    high-pitched “blowing murmur” in tricuspid area
    due to right ventricle dilation and rheumatic fever
  5. ventricular septal defect
    harsh sound, loudest in tricuspid area
22
Q

Diastolic murmur

A
  1. Aortic regurgitation
    high-pitched blowing early diastolic
    causes: aortic root dilation, bicuspid aortic valve, endocarditis, rheumatic fever
  2. mitral stenosis
    delayed rumbling
    caused by sequela of rheumatic fever
23
Q

continuous mumur

A

patent ductus arteriosus:

machine-like murmur
best heard in the left infraclavicular area (loudest at S2)

causes: congenital rubella / prematurity