Lecture 27: Exotic Animal Ax (Exam 4) Flashcards

1
Q

Describe ax in ferrets

A
  • Ax & analgesic tech/protocols similar to dogs & cats
  • Fasting is not necessary b/c they are very prone to hypoglycemia & have a really high GI transient time
  • Be careful w/ sharp teeth
  • S/N, insulinoma, adrenal tumors, FB, & trauma
  • Monitor hypoglycemia & hypothermia
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2
Q

Describe ax in rodents

A
  • Small px
  • Obtain accurate BW
  • Small rodents & rabbits don’t vomit so NPO not req
  • Intraperitoneal injection should be considered
  • Don’t use ketamine alone but can combine w/ ace or a2 agonist
  • Buprenorphine can cause pica but is considered analgesic of choice in lab animals
  • Morphine sedates rats but excites mice
  • Increased SA to BW ratio leads to rapid cooling under as
  • Use a box to give gas ax
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3
Q

Describe ax in rabbits

A
  • Increased ax risk compared to dogs & cats
  • Min fasting to no more than 1 H
  • Impt to restrain by supporting the rump & tuck head under your arm or make a bunny burrito
  • High MBR & rapid elim of drugs
  • Always premed
  • Don’t use ketamine alone but can combine w/ benzo, ace, or a2 agonist
  • NSAIDs, opioids, & LAs are useful
  • Give IM injection in quads
  • Iso & sevo are typically used for maintenance
  • Monitor closely for hypothermia & hypoglycemia
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4
Q

Describe intubation of rabbits

A
  • Preoxygenation is a must
  • Intubation is the standard of car
  • Keep the head 7 neck extended in extension to prevent obstruction
  • Do not persist for more than a few mins or if evidence of laryngeal edema/hemorrhage
  • Can use supraglottic airway devices
  • They are obligate nasal breathers so check patency of the nares & nasopharynx after extubation
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5
Q

What are some differences in the anatomy & physiology of birds

A
  • There respiratory system has separate ventilator & gas exchange compartments
  • Complete tracheal rings that can collapse during restraint
  • No diaphragm
  • Inspiration & expiration are active
  • Pneumatized bones
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6
Q

Describe birds ax

A
  • Apnea can occur from breathing in room air (must be treated immediately)
  • HR varies by species
  • Catecholamines are released during stress & pain & can cause arrhythmias
  • Have a renal portal system
  • Correct hypoglycemia before ax
  • Use a towel to restrain them
  • Give warm fluids
  • Pre oxygenate for 5 mins before induction
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7
Q

Describe birds & ax drugs

A
  • Avoid midazolam in macaws & cockatoos
  • a2 agonists have many adverse effect so use w/ great caution
  • Ketamine should not be given alone (combine w/ benzo)
  • Propofol has narrow margin of safety in pigeons & chickens
  • Iso or sevo can be used (more likely to cause apnea)
  • NSAIDs & opioids are commonly used
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8
Q

Describe monitory & recovery in birds

A
  • Monitor trends of vital sx
  • Palpate pulse/doppler placement (brachial, medial, metatarsal, carotid, or palatine a)
  • Place ECG close to body
  • Can do indirect BP around thigh or distal humerus
  • ETCO2 is useful but SpO2 is not validated in birds
  • Absence of palpebral & corneal reflex = excessive depth
  • Extubate when jaw tone returns
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9
Q

Describe ax in ungulates

A
  • Treated like ruminants
  • Ruminal tympany
  • Hypothermia
  • Hypoxemia
  • Bloating
  • Regurgitation
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10
Q

Describe exertional myopath

A
  • Life threatening syndrome caused by a combo of sympathetic exhaustion from sustained stress & intense muscular exertion
  • Prevent w/ limited capture time to < 2 min
  • Captured wild animals should not be handled or stressed for @ least 6 W
  • Mortality rate > 2% indicates ax protocol should be re evaled
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11
Q

What is acute death syndrome

A
  • AKA capture shock syndrome
  • Weak & depressed & remains recumbent after reversal
  • Shock & death w/in 3 to 4 H
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12
Q

What is delayed peracute death syndrome

A
  • Appears to be in good condition after capture
  • When stressed again they will get ventricular fibrillation & cardiac arrest
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13
Q

What is ataxic myoglobinuric syndrome

A
  • Ataxia, brownish urine, & usually die w/in hours or days after capture due to kidney failure following myoglobin mediated tubular destruction
  • Increased CK, LDH, SGOT, & BUN
  • Most common syndrome
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14
Q

What is rupture muscle syndrome

A

W/ 1 to 2 days post capture the animal is unable to support weight on the hind legs. The hocks are hyperflexed & there is increase CK, LDH, & SGOT (animal usually dies in 3 to 4 W)

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15
Q

What are the highlights given about zebras

A

Tx like equine but a more close watched drug protocol

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16
Q

Describe the highlights on giraffe

A

Highest increased ax risk

17
Q

Describe the rhino highlights

A

Treat them like a horse and have the same issues as horses

18
Q

Describe the elephant highlights

A

Biggest issues are the respiratory system (place them in a lateral recumbency) & Pink foam syndrome

19
Q

What highlights of carnivores

A

Safety is the number one consideration

20
Q

Describe the highlights of tigers

A
  • Tigers have more ax complications
  • Can get hyperkalemia
  • Extubate them a lot sooner (pull tube when see an ear flick)
21
Q

Describe the highlights of bears

A
  • Black bear approaches are diff than polar bear diff
  • Safety is the number concerns
  • Think bout the time of year & how much fat they have
  • Ketamine can lead to sudden recovery in brown & polar bears
22
Q

Describe the highlights of fish ax

A
  • Have no lungs but still have to have oxygenated water over the gills
  • Dissolve ax in the water
  • Need to be fasted
  • Don’t let them dry out
  • Can be removed from the water for 1 - 4 min
  • SpO2 is not effective
  • Use MS-222 for ax (approved by FDA)
23
Q

What is one thing to remember about reptiles

A
  • Recovery levels take a really long times
  • Low O2 levels are needed in recovery