Lecture 15: Cardiac Failure, Heart Sounds, and Circulatory Shock Flashcards

1
Q

What is cardiac failure?

A

failure of the heart to pump enough blood to satisfy the needs of the body

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2
Q

How does the ANS compensate for acute cardiac failure?

A

baroreceptor reflex
chemoreceptor reflex
CNS ischemic response

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3
Q

In acute cardiac failure, if all the ventricular musculature is diffusely damaged but still functional, ________ strengthens this damaged musculature

A

sympathetic strengthens

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4
Q

If part of the musculature is nonfunctional and part is still normal, the normal muscle is ______

A

strongly stimulated

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5
Q

Strong sympathetic stimulation increases tone of most of the blood vessels in circulation and therefore increase ______

A

venous return

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6
Q

What are the acute effects of a heart attack?

A

reduced cardiac output

damming of blood in the veins—increased venous pressure

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7
Q

In an acute heart attack, what does the compensation?

A

sympathetic nervous system

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8
Q

What are the chronic compensations after an acute heart attack?

A
  • results for partial heart recovery and rental mention of fluid
  • max pump ability is depressed to less than one half normal
  • any heavy exercise causes immediate return to symptoms of acute failure
  • increase in right atrial pressure can maintain cardiac output near normal level
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9
Q

In left heart failure, blood pumps into the lungs but ______

A

not adequately out of them

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10
Q

If blood cannot be pumped out of the lungs, what happens?

A

mean pulmonary filing pressure rises because of shift of large volumes of blood from the systemic circulation into the pulmonary circulation
pulmonary capillary pressure increases

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11
Q

What are the two major problems with left heart failure?

A

pulmonary vascular congestion

pulmonary edema

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12
Q

In severe acute left heart failure, pulmonary edema may happen so rapidly that it can cause death by ______ in _____ minutes

A

suffocation, 20-30

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13
Q

What does an arteriovenous fistula do?

A

overloads heart because of excessive venous return

venous return curve rotates upward

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14
Q

What is beriberi?

A

thymine deficiency that leads to weakening of the heart
decreased blood flow to kidney — fluid retention
increased mean filling pressure
venous curve shifts to right

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15
Q

_______ results in major decrease in peripheral vascular resistance

A

AV fistula

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16
Q

What is the first heart sound?

A

AV valves close on at the onset of ventricular systole

17
Q

What is the second heart sound?

A

semilunar values close at end of systole

18
Q

What is a left to right congenital defect?

A

blood flows backward and fails to flow through systemic circulation

19
Q

What is right to left congenital defect?

A

blood flows from right to left side of heart, bypassing lungs

20
Q

What type of defect is ductus arteriosus?

A

left to right

21
Q

What type of defect is tetralogy of fallot?

A

right to left

22
Q

What is the definition of circulatory schock?

A

generalized inadequate blood flow through the body, to the extent that the body tissues are damaged, especially because of too little oxygen and other nutrients delivered to the tissue cells

23
Q

What are some cardiac abnormalities that decrease the ability of the heart to pump blood?

A
myocardial infarction
toxic state of the heart
severe valve dysfunction
heart arrhythmias 
circulatory shock
24
Q

What are factors the decrease venous return?

A

diminished blood volume
decreased vascular tone
obstruction of blood flow

25
Q

What are powerful sympathetic reflexes initiated by?

A

arterial baroreceptors and other vascular stretch receptors

26
Q

What does powerful sympathetic reflexes result from?

A

decrease in arterial pressure after hemorrhage

decreases in pressures in the pulmonary arteries and veins in the thorax

27
Q

What effects result from powerful sympathetic reflexes

A

arterioles constriction in most parts of the systemic circulation
veins and venous reservoirs constrict
heart activity increases markedly

28
Q

Compensatory mechanisms that return blood volume back to normal after shock

A

absorption of large quantities of fluid from intestinal tract
absorption of fluids into blood capillaries from the interstitial spaces of the body
conservation of water and salt by kidneys
increased this and appetite fro salt

29
Q

What is non progressive shock?

A

sympathetic reflexes and other factors compensate enough to prevent further deterioration of the circulation
negative feedback mechanisms

30
Q

What is progressive shock?

A

positive feedbacks

when arterial pressure falls low enough, coronary blood flow decreases below requirement for nutrition of the myocardium

31
Q

What is one of the important features of progressive shock?

A

whether or not it is hemorrhagic in origin

32
Q

What are the factors in final lethal progression of schock?

A
vasomotor failure
blockage of small vessels 
increased vascular permeability 
release of toxins by necrotic tissue
cardiac depression caused by endotoxin
generalized cellular deterioation
33
Q

What is neurogenic shock?

A

shock occurring from any loss of blood volume

34
Q

What are the causes of neurogenic shock?

A

deep general anesthesia
spinal anesthesia
brain damage