Lecture 14: HF/CAD/DM Cardiac Rehab pt. 2 Flashcards

1
Q

describe phase 2 cardiac rehab

A

outpatient setting

starts as soon as possible after hospital dc

36 sessions (3x/wk x 12 wks)- Medicare pays this much

led largely by exercise physiologists

Vitals taken before, during, and after activity with adjustments made based on exercise response

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2
Q

factors that help determine frequency of monitoring and degree of direct supervision in CR exercise program

A

prior clinical course (complicated vs uncomplicated)

GXT results

degree of LV impairment

initial assessment

risk stratification

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3
Q

exercise components in phase 2 CR should consist of

A

combo of aerobic, resistance, and flexibility

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4
Q

specificity for CR

A

training should mirror pts regular functional tasks

changes will occur in specific mm groups or aerobic tasks that are challenged

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5
Q

mode of exercise for CR phase 2

A

should engage mm used for regular functional tasks

variety of exercise equipment to accomplish task

take pt preferences into account if able

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6
Q

intensity for phase 2 cardiac rehab

A

too intense = hazard

not intense enough = ineffective

aerobic training effects occur at 70-85% HR max, but as little as 40-60% HR max in elderly or more compromised pts

use HR max and RPE to monitor

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7
Q

type of activities for CR phase 2

A

warm up (5-10 min) = allows mm to stretch to max length used in peak exercise

steady state exercise

cool down (5-15 min) = allows slowed return to rest since abrupt end can reduce blood return to heart

interval training can be used for pts who are not yet able to tolerate long duration aerobic activities

combination of BLE and BUE exercise will produce a higher VO2 max consumption

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8
Q

duration of phase 2 cardiac rehab per session

A

goal = 45 min continuous steady state

can start with shorter period and progress per individualized pt needs

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9
Q

frequency phase 2 CR

A

typical schedule = 3x/week

more impaired pts should start with low intensity, short duration, high frequency exercise program if possible

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10
Q

resistance training guidelines for phase 2 CR

A

initiated after aerobic

goal = 30-50% 1RM, 8-10 reps, 2-3x/wk to major mm groups

can increase weights by 5-10 lbs when 12-15 reps are comfortable

train large mm before small

exhale with exertion to avoid valsalva

take individual pt needs into consideration to determine focus on mm power vs endurance

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11
Q

describe phase 3 CR

A

maintenance stage

focus on lifelong exercise involvement

progressive independence and self monitoring of symptoms and health

larger exercise groups with less individualized supervision (i.e. YMCA, senior centers, private gyms)

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12
Q

benefits of CR

A

47% lower mortality risk

31% lower risk of repeat MI if 36 sessions attended

decreased hospital readmission

higher QOL

lower cost to healthcare system

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13
Q

barriers to cardiac rehab

A

pts who would benefit but are not being referred (greatest predictor of participation is strength of MD recommendation)

geographic location; 30% less likely to participate if living outside of large city

long wait times; every day a pt waits to enroll, they are 1% less likely to ever do so

finances; pts who make >$75k are 2x as likely to participate

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14
Q

safety precautions in CR

A

avoid exercise 1-2 hours post meal

avoid isometrics and breath holds

add warm up and cool down periods to lower risk of adverse event

take shorter showers and not extreme temps to maintain normal blood flow/distribution

staff should be trained on how to respond to emergency

CR programs in high altitude ares or environments of extreme temps should adjust properly

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15
Q

CVD prevention methods

A

healthy lifestyle reduces risk of MI by 81-94%; pharm treatment alone is only 20-30% reduction

most effective treatment = combo of pharm management and lifestyle choices

key changes = diet, physical activity, weight management, smoking cessation, and stress management

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16
Q

nutrition recommendations for prevention and management of CVD

A

adherence to mediterranean diet

increased intake fruits/vegetables

limit processed foods

balance macronutrient absorption

reduce sodium and sugar intake

regular monitoring and education

17
Q

components of the mediterranean diet

A

lots of fruits/vegetables (antioxidants to reduce systemic inflammation)

whole grains

healthy fats from EVOO, nuts, seeds

legumes (plant based protein and fiber)

fish/seafood (omega 3 to lower cholesterol)

moderate consumption of eggs, poultry, and dairy

infrequent consumption of red meat and sugar

herbs and spices in place of salt

red wine in moderation

18
Q

recommended sodium intake per day to prevent HTN

A

<2000mg

19
Q

benefits of mediterranean diet

A

reduced risk of CVD and CVA

lower indidence of type 2DM

better weight management/reduced obesity rate

enhanced cognitive function and reduced risk of neurodegenerative diseases

overall longevity and improved QOL

20
Q

pharm management of type 1 DM

A

needs insulin replaced

made with human and animal products

meds have various characteristics and can be a combo of short acting and long acting properties

21
Q

pharm management for type 2 DM

A

effective only for type 2

different mechanism to lower blood sugar than type I
- slow rate pf glucose released by liver
- stimulate insulin secretion by beta cells of pancreas
- blocks glucose reabsorption in kidneys
- enhance insulin sensitivity in peripheral tissues
- artificial replacement of insulin

22
Q

how does metformin (glucophage) work and what are the side effects

A
  1. slows rate of glucose released by liver
  2. enhances insulin sensitivity in peripheral tissues

first drug of choice in type 2 DM with Hgb A1c >7%

very low risk of hypoglycemia when used alone (doesn’t affect insulin secretion in pancreas)

side effects = GI distress and increased lactic acidosis with exercise

23
Q

types of insulin stimulators

A

GLP-1 agonists

DPP-4 inhibitors

Sulfonylureas

24
Q

how do GLP-1 agonists work

A

mimic a GO hormone released with digestion that stimulates pancreas to produce/release insulin

successful in weight reduction

25
Q

how do DPP-4 inhibitors work

A

prevent breakdown of GLP-1 allowing it to act on the pancreas to produce/release insulin

26
Q

how do sulfonylureas work

A

directly stimulate pancreas to release insulin

higher risk of hypoglycemia compared to others

27
Q

what is thiazolidinediones (TZDs)

A

insulin sensitizer

enhances insulin sensitivity in adipose, muscle, and liver tissue

insulin is utilized at the cell membrane to make cells more receptive to blood glucose

higher risk of side effects; no prescribed as much anymore

28
Q

how is glucose lowered with the kidneys (what drugs)

A

SGLT-2 inhibitors

block glucose absorption in kidneys, allowing a higher excretion of glucose in urine

side effects = hypotension, UTI, yeast infections

29
Q

impact of exercise on DM

A

sustained PA reduces insulin secretion

decreased insulin secretion is compensated for by heightened sensitivity of peripheral tissues to insulin, causing more rapid glucose uptake by mm

help decrease BS and HgbA1c

resistance training = more effective at improving glycemic control when targeting major mm groups with high intensity strength training (3 sets of 8-10 reps at weight that incudes near fatigue 3x/wk)

30
Q

rehab managent of type 1 DM

A

avoid exercise induced hypoglycemia (can occur during, immediately after, or up to 24 hours post exercise)

avoid exercise during peak effect of insulin- reduces risk of hypoglycemia

aim for blood sugar of 120-160 prior to exercise

avoid exercise of limb used for insulin injection for 60-90 min or until after peak effect has been reached

ensure adequate hydration

31
Q

tips to avoid exercise induced hypoglycemia

A

eat a meal 60% carbs 1-3 hours prior to exercise

check BS 60 and 30 min prior to exercise to assess trend
- if BS <70 = eat more cards, no exercise until BS 100-120
- if BS >250 = may need insulin, no exercise until BS 100-120

> 250 mL fluid intake 20 min prior to exercise

monitor BS every 30 min during long duration exercise

insert short bouts of intense anaerobic exercise throughout aerobic exercise to blunt hypoglycemic effects

monitor BS for 24 hours and adjust insulin dosing if needed

eat slow acting carbs to protect against late onset hypoglycemia

32
Q

rehab management of type 2 DM

A

during low-mod intensity exercise = mm have increased glucose uptake for energy production and reduces hyperglycemia

combined aerobic and resistance training has more benefit on HgbA1c than either in isolation

weight loss benefits all risk factors of DM2

greatest CVD risk reduction achieved with >4 hours/wk of mod intensity exercise

> 7 hours/wk mod intensity exercise is more successful in achieving and maintaining weight loss

33
Q
A