Lecture 11: Orthopaedic Management of Spasticity Flashcards

1
Q

Functions of the skeleton

A
  1. Body structure & shape
  2. Movement (lever systems)
  3. Vital organ protection
  4. Physiological contributions
     Blood cell production (via bone marrow)
     Mineral storage
     Endocrine regulation
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2
Q

What is the treatment for femur fracture
* what are the draw backs?

A

A metal rod forced into the medullary cavity of a bone (medullary rod). This will serve as a load sharing device allowing for early weight bearing and the facilitation of bone healing.

Removes mechanical barrier to movement anymore

  • people who have not alot of fat may feel the screws which are uncomfy. screws can be removed overtime
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3
Q

Two types of bone healing

A

primary: use metal plates to piece together misaligned bones. no callus formation

secondary: bone forms a callus to bridge the gap between broken ends. eventually replaced with bone in 6 weeks. occurs when cast, external fixation, splints, or nails are used

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4
Q

arthrodesis

A

surgical immobilization of a joint by fusion of the adjacent bones.

often performed on the bones of the ankle and foot

usually accompanied by contracture release surgery for fuller correction of the joint deformity.

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5
Q

high tibial osteomy
* what is it
* when is it used
* give some statistics on surgical outcomes

A
  • surgical procedure to correct varus malalignment and provide relief of unicompartmental osteoarthritis of the medial compartment of knee
  • change weightbearing axis to back to straight down middle.
  • used to prolong native joint lifespan. often performed to delay or prevent total knee replacement
  • 10 years after the surgery, 80% people still like their joint.

small wedge of bone is removed to allow the bone to be repositioned or reshaped

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6
Q

total knee replacement

A

all knee compartments wear away. no cartilage

Surgical procedure to resurface a knee damaged by arthritis

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7
Q

typical patient for high tibial osteomy

A

40-60
- bowlegged (varus)
- osteoarthritis is isolated to medial compartment of knee joint

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8
Q

Tendon Transfer Principles

A
  1. Joints must be supple prior to transfer (full rom, free of pathology; otherwise you will lose function of transferred tendon)
  2. Soft tissue must be at equilibrium (timing) (can’t have local inflammation)
  3. Donor muscle must have adequate excursion (amount of movement of muscle tendon unit; example: wrist flexor has about 30mm of excursion while finger flexors have greater excursion. transferinging a wrist flexor to a finger flexor is bad but you can do the other way around due to excursion)
  4. Donor muscle must have adequate strength (strength scale of 0-5. 3 is antigravity; 5 is full power; 1 is a flicker. donor should be at least 4 or 5 since itll lose 1 grade of power when you transfer it. muscles that have been paralyzed you shouldn’t use bc it wont have full power)
  5. Expendable donor muscle (never transfer a quadricep tendon since you’ll lose extensor)
  6. Straight line of pull (the muscle will pull in a direct line from its origin to the insertion of the tendon being substituted.)
  7. Synergy
    - muscle groups that work together. tendon transfer work more effectively when you use a synergist
  8. Replace a single function per transfer
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9
Q

Types of Cerebral Palsy

A

Spastic Diplegia: Affects legs > arms

Spastic Quadriplegia: Affects the entire body

Dyskinetic Cerebral Palsy (Athetoid): Often affects entire body The baby often presents with perinatal asphyxia and/or kernicterus (bilirubin-induced brain dysfunction) that affects the basal ganglia, cerebellum and sometimes thalamus

Spastic Hemiplegia– Affects the arm and leg of one side. Imaging shows unilateral UMN abnormalities. perhaps only one side of the brain was lacking oxygen during pregnancy

Ataxic CP Involves the entire body

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9
Q

etiology of cerebral palsy

A

Intrauterine growth restriction (34%)
Intrauterine Infections (28%)
Prematurity (78%)
Perinatal asphyxia (10%)

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9
Q

cerebral palsy definition

A

Heterogeneous group of non-progressive disorders
characterized by motor and postural dysfunction

Range in severity due to abnormalities in developing brain

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10
Q

9% of the population is missing a

A

palmaris longus
tendon

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11
Q

Describe the following descriptors:
* athetoid/dystonic
* ataxic

A
  • Athetoid/Dystonic – abnormal posture, involuntary movements
  • Ataxic – Lack coordination
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12
Q

Orthopaedic
Classification
GMFCS

A

Gross, motor, function, classification scale
* level 1: children can walk and stairclimb without limitaiton
* level 2: climb stairs with railing; experiences limitations on uneven surfaces, inclines, or crowded spaces
* level 3: need assistive mobility devices; may be able to climb stairs; can push self in wheelchair
* level 4: can walk short distances with a walker and relies on wheeled mobility device
* level 5: all areas of motor function are limited. physical impairment restricts voluntary movement control and antigravity head and trunk control

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13
Q

CP Orthopaedic Manifestations (presentations)

A
  • Upper extremity deformities
  • Hip subluxation and dislocation
  • Spine deformities
  • Foot deformities
  • Gait disorders
  • Contractures and fractures
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14
Q

What are the functional and technical objectives as well as management options for CP

A
  1. Functional Objectives
    * Improve ADL’s
    * Improve gait
    * Pain relief
    * Ease of care
  2. Technical objectives
    * Tone reduction
    * Improved ROM and joint position
  3. Management options
    * Eliminate noxious stimuli
    * PT/OT
    * Oral medications
    * Intrathecal baclofen
    * Chemodenervation
    * Surgery (last line)
15
Q

Goals of orthopaedic surgery for CP

A

Goals
Reduce spasticity
Increase ROM
Improve ADL’s
Reduce pain

16
Q

Types of orthopaedic surgery for CP

A
  1. Contracture release
  2. Tendon transfer
  3. Osteotomy
  4. Arthrodesis
17
Q

what are 4 types of foot deformities seen in those with CP

A
  1. Equinus – Plantarflexion contracture
  2. Hallux Valgus – “Bunion”
  3. Equinoplanovalgus
    * Plantarflexion contracture
    * Planus – flat foot
    * Hindfoot valgus – heel angled laterally away from midline
  4. Equinocavovarus
    * Cavus – High arch
    * Hindfoot varus – heel angled toward midline
18
Q

Pathophysiology and presentation for: equinus

A

Pathophysiology
* Spasticity/contracture of gastrocnemius-soleus complex
* Muscle imbalance: Plantarflexors > Dorsiflexors

Presentation
* Toe walking / heel strike is absent
* Forefoot calluses
* Hyperextended knee with heel contact

19
Q

Pathophysiology and presentation for: hallux valgus

A

Pathophysiology
* Adductor hallucis spasticity
* Secondary to equinovalgus deformity
* Forces toe into valgus – angled laterally away from midline

Presentation
* Pain and difficulty with shoe wear
* “Bunion” – callus over 1st metatarsal medial head

20
Q

Pathophysiology and presentation for: equinoplanovalgus

A

Pathophysiology
* Muscle imbalance: Plantar Flexors > Dorsi Flexors
* Eversion spasticity (Peroneus longus > brevis)
* Inversion weakness (tibialis posterior, tibialis anterior)

Presentation
* Weight Bearing on medial border
* Planus – flat foot
* Hindfoot valgus – angled laterally away from midline

often seen with hallux valgus

21
Q

Pathophysiology and presentation for: equinocavovarus

A

Pathophysiology
* Muscle imbalance: Plantar Flexors > Dorsi Flexors
* Spastic peroneus longus and tibialis posterior
* Weak tibialis anterior and peroneus brevis
Presentation
* Cavus – High arch
* Hindfoot varus – angled toward midline
* Forefoot pronation with compensatory hindfoot varus in attempt to
keep foot plantigrade

22
Q

What is a split tibial posterior tendon transfer

A

split tibialis posterior tendon.

insert it to lateral cuneiform

brings foot back to antegrade

point: obtain a plantegrade foot

correct spastic foot varus

23
Q

Pulvertaft Weave

A

attaching a tendons to eachother.

split one, pass the white tendon through the other one.

you can split the white tendon too and push it through the other one

suture it all together

24
Q

In the case study of the CP patient with left hemiplegia, GMFCS II, plantarflexion contractures, toe walking, foot falling into cavus and varus, what was done surgically to correct this?

A

goal: make foot plantigrade
* lengthen achililes tendon
* plantarfascia release
* adductor hallucis lengthening
* Split tibialis posterior tendon transfer (SPOTT)

may require ankle foot orthroses for stability

25
Q

Achilles Z Lengthening

A

split the tendon and lengthen

need a boot after?

26
Q

plantar fascia release
* why is it done?
* what is the post op recommendations following

A
  • relieve tension and inflammation
  • non weightbearing until wound heals
27
Q

Why do hip abnormalities occur?

A

lack of weightbearing during development results in the acetabulum not forming fully and spasticity in the hip abdductors and flexors

28
Q

Hip Subluxation/Dislocation
* occurs in what CP population
* what causes it

A
  • Progressive, occurs in 50% of kids with spastic quadriparesis
  • Spastic hip adductors and flexors + lack of weightbearing lead to scissoring gait –> subluxation –> dislocation
  • femoral heads are not under acetabulum.
29
Q

How are hip dislocations managed?

A
  • Soft tissue: adductor +/- psoas tenotomy, abduction brace
    put abduction brace for 6 weeks after so they heal in a lengthened position. makes the pelvis more level.
  • Bony: Joint preserving procedures
    1. Varus Derotation Osteotomy (femur)
    2. Pelvic osteotomy to increase femoral head coverage (involves taking a wedge out of the femur, derotating it, and placing wedge in pelvis
30
Q

what are some upper extremity deformities in CP kids

A
  • waiters tip
  • shoulder internal rotation, elbow flexion, forearm pronation, wrist flexion, thumb in palm, finger flexion
31
Q

Spine deformities seen with CP

A

Scoliosis
* Long, C-shaped, stiff
* Pelvic obliquity
* Sagittal plane deformity
(kyphotic/lordotic)
* Do not do well with non
operative management

32
Q

Spinal correction for CP

A
  • spinal fusion

cut a wedge out of the concave aspect of curve in order to bring curve straight again

33
Q

secondary issues with spinal correction

A

issues with gut function. changes their whole physiology

34
Q

contracture release
* what is it
* what is done during operation and post op
* where is it done most often?

A
  • partially or fully cut the tendon of a contractured muscle
  • reposition joint at a normal angle and apply a cast. serial casting overtime. tendon is allowed to regrow at new length over several weeks and joint is gradually stretched
  • requires physical therapy to strengthen muscle and improve ROM
  • often done at the Achilles tendon to correct equinus deformity